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1 ioretinal artery and not the ordinary branch retinal artery.
2 ttent retinal artery occlusions of different retinal arteries.
3 gions supplied by side branches of low-order retinal arteries.
4 tic NGRs, most leukocytes accumulated in the retinal arteries.
5 till multiple emboli in the conjunctival and retinal arteries.
8 on (PO(2)) was measured noninvasively in the retinal arteries and veins on optical section retinal im
9 erforming several microvascular maneuvers on retinal arteries and veins was demonstrated in porcine e
13 se the involved pathologically permeabilized retinal artery and normalize the vessel wall formation b
16 tinal vascular caliber measurements (Central Retinal Artery and Vein Equivalents; CRAE and CRVE) at E
17 peripheral vasculitis to occlusion of large retinal arteries around the optic nerve or macula with s
21 athing condition, mean P(O2) in the choroid, retinal arteries, capillaries, and veins were 58+/-2 mm
23 The shape of the blood velocity profile in retinal arteries changed systematically during the cardi
25 ose To identify and characterize the central retinal artery (CRA) using cone-beam CT (CBCT) images ob
26 posterior ciliary artery (PCA), and central retinal artery (CRA) were measured using Color Doppler U
27 ort posterior ciliary artery (SPCA), central retinal artery (CRA), central retinal vein (CRV), peripa
29 onsistently demonstrated the in vivo central retinal artery, demonstrating excellent potential for mu
30 erved a significant NO-dependent increase in retinal artery diameter in Cav-1 knockout mice, suggesti
32 al layer, and choroidal thicknesses, central retinal artery equivalent (CRAE), and central retinal ve
33 raphs (1 eye/participant) determined central retinal artery equivalent (CRAE), central retinal vein e
34 raphs (1 eye/participant) determined central retinal artery equivalent (CRAE), central retinal vein e
36 m the optic disc), summarized by the central retinal artery equivalent (CRAE), the widths of the 6 la
37 mean overall significant decrease of central retinal artery equivalent and central retinal vein equiv
41 te FA to be performed and if connection with retinal artery is proven, parents should be informed on
42 es as the blood pressure exiting the central retinal artery is varied between 28 and 40 mm Hg is used
45 riction, pupillary abnormalities, attenuated retinal arteries, loss of outer retinal signals on spect
46 ns included vitreous hemorrhage (2%), branch retinal artery obstruction (1%), ophthalmic artery spasm
47 ent ophthalmic artery spasm (n = 1), central retinal artery obstruction (n = 1), branch retinal arter
48 l retinal artery obstruction (n = 1), branch retinal artery obstruction (n = 2), and peripheral retin
51 vein occlusion (7 eyes, 7 patients), central retinal artery occlusion (1 eye, 1 patient), and idiopat
53 CRVO), cilioretinal artery occlusion, branch retinal artery occlusion (BRAO), and anterior ischaemic
54 set of paracentral scotoma, caused by branch retinal artery occlusion (BRAO), as a leading symptom of
57 estigate the visual outcome of acute central retinal artery occlusion (CRAO) after current standard t
58 eport, we present for the first time central retinal artery occlusion (CRAO) and central retinal vein
59 ygen therapy (HBOT) in patients with central retinal artery occlusion (CRAO) by analyzing changes in
61 nd compare macular vessel density in central retinal artery occlusion (CRAO) eyes with retinal arteri
64 aculopathy (PAMM) that progressed to central retinal artery occlusion (CRAO) on spectral domain-optic
65 been suggested for the treatment of central retinal artery occlusion (CRAO) such as ocular massage,
66 study was to evaluate a rat model of central retinal artery occlusion (CRAO) that simulates the clini
70 outcome analyses yielded similar results for retinal artery occlusion (HR: 1.13, 95% CI: 1.02-1.26) a
71 e of this study was to report case series of retinal artery occlusion (RAO) as one of the significant
72 overy in a 32-year-old woman with iatrogenic retinal artery occlusion (RAO) following glabella calciu
75 in occlusion (RVO), 16 patients (20.5%) with retinal artery occlusion (RAO), and 2 patients (2.6%) wi
76 ized as having retinal vein occlusion (RVO), retinal artery occlusion (RAO), and any retinal vascular
80 ements limitation, an infero-temporal branch retinal artery occlusion and multiple choroidal emboli.
81 Twenty-five patients (42%) had a confirmed retinal artery occlusion based on OCT and follow-up exam
86 sed the patient as having acute nasal branch retinal artery occlusion in the left eye and bilateral N
90 oing hemodialysis who subsequently developed retinal artery occlusion or retinal vein occlusion compa
91 nt with a history of left eye blindness from retinal artery occlusion presented with rapidly declinin
96 and Scopus using the following index terms: "retinal artery occlusion" OR "retinal ischemia" AND "thr
97 th increased risk of retinal stroke (central retinal artery occlusion), a subtype of ischemic stroke
98 ls without retinal occlusion, 6 had a branch retinal artery occlusion, and 9 had a central retinal ar
99 stimuli from a patient with a prior central retinal artery occlusion, and from two patients with sel
100 rized by the triad of encephalopathy, branch retinal artery occlusion, and sensorineural hearing loss
101 entricular assist device thrombosis, central retinal artery occlusion, cerebral venous sinus thrombos
102 each in the MicroShunt group having central retinal artery occlusion, choroidal hemorrhage (after pl
103 had significant visual complications (branch retinal artery occlusion, macular hole, and corneal deco
104 tic retinopathy, retinal vein occlusion, and retinal artery occlusion, may also be influenced by an a
105 ivided into central (CRAO) and branch (BRAO) retinal artery occlusion, ocular ischemic syndrome (OIS)
106 treating clinical conditions such as central retinal artery occlusion, retinoblastoma chemoembolizati
107 Under the impression of NAION with branch retinal artery occlusion, the patient was treated with i
108 gs of our similar study dealing with central retinal artery occlusion, where the young suffered much
120 ients were excluded if they had a history of retinal artery occlusions (RAOs) or retinal vein occlusi
123 temporary loss of vision due to intermittent retinal artery occlusions of different retinal arteries.
124 keratic precipitates, optic neuritis, branch retinal artery occlusions, and chorioretinal scarring in
126 sed on the intraluminal contrast patterns of retinal arteries on OCT, 3 independent graders categoriz
128 ized by variable occlusion of large or small retinal arteries, or both, and perivenular abnormalities
129 antly higher signal intensity changes in the retinal arteries (P=.001, compared with oxygen inhalatio
130 o was significantly increased in the central retinal artery (P < 0.01), temporal posterior ciliary ar
131 were decreased significantly in the central retinal artery (P < 0.02 and P < 0.01, respectively), te
132 ed with baseline measurements in the central retinal artery, posterior ciliary arteries, and superior
133 eathing and discontinuity of small and large retinal arteries, sclerotic arteries, regions of vascula
134 induce full vasodilation in isolated porcine retinal arteries suggested that the observed in vivo eff
135 included nerve fiber layer infarcts, central retinal artery thrombosis, hypertrophy and occlusion of
136 vascular overlay of the retinal veins vs the retinal arteries to map the distribution of PAMM with en
137 measurements of the blood flow in a temporal retinal artery using the bidirectional laser Doppler tec
139 The filler presumably enters the central retinal artery via the rich external-internal carotid an
140 dings included ophthalmic artery and central retinal artery wall dissection, fracturing of the intern
142 previously reported that blood speeds in the retinal arteries were significantly lower in patients wi
143 We measured blood flow in a major temporal retinal artery with subjects seated and then while recli