戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ture microdissection was used to isolate the retinal blood vessels.
2 nt epithelium, whereas DGLbeta is limited to retinal blood vessels.
3 iber bundle patterns occurs just above major retinal blood vessels.
4 nner plexiform layer, occasionally alongside retinal blood vessels.
5 anglion cell layers, nerve fiber layers, and retinal blood vessels.
6 ntegrity of the retina, and condition of the retinal blood vessels.
7 netrin-4 was found to be localised to mature retinal blood vessels.
8 e located in the nerve fiber layer and along retinal blood vessels and have been hypothesized to part
9 VIII) immunoreactivity was observed in large retinal blood vessels and in capillaries in some individ
10 wed quantitative P(O2) measurements in large retinal blood vessels and in retinal capillaries.
11  in the retina, resulting in the dilation of retinal blood vessels and increased blood flow.
12                 Some branches ended on large retinal blood vessels and others in dense clusters in th
13 , as did endothelial cells of some posterior retinal blood vessels and some retinal pigment epithelia
14 phosphatase (ADPase) activity (labels viable retinal blood vessels) and flat-embedded in JB-4.
15                               Pathologies of retinal blood vessels are among the major causes of blin
16                              Shadows cast by retinal blood vessels are represented in striate cortex
17 tes highly realistic digital models of human retinal blood vessels, based on established biophysical
18  Occasional cell bodies were associated with retinal blood vessels, but their identity as pericytes,
19  insight into the possible role of tbdn-1 in retinal blood vessels by characterizing its expression p
20            However, accurate segmentation of retinal blood vessels can be challenging due to imbalanc
21                Pathological proliferation of retinal blood vessels commonly causes vision impairment
22 e layer (RNFL) thickness, macular volume and retinal blood vessel diameter were measured and paramete
23 nsient but significant defect in pruning and retinal blood vessel extension.
24       Our work also suggested that the major retinal blood vessels form a "skeleton"-the configuratio
25 ontributes significantly in triggering inner retinal blood vessel formation.
26                     Slc38a5 is suppressed in retinal blood vessels from Lrp5(-/-) and Ndp(y/-) mice,
27 tion network was able to efficiently segment retinal blood vessels from OCT scans.
28  beta2 and gamma3 chains regulate subsequent retinal blood vessel growth and maintain vascular integr
29                        Aberrant, neovascular retinal blood vessel growth is a vision-threatening comp
30 ce in immunoreactivity to both antibodies in retinal blood vessels in aged control eyes.
31 n vivo, GSK101 increased the permeability of retinal blood vessels in wild type but not in TRPV4 knoc
32 ession of FcRn receptor was confirmed in the retinal blood vessels, iris blood vessels, optic nerve v
33 sistent with the concept that the absence of retinal blood vessels is essential for foveal pit develo
34        Angiogenesis, or the formation of new retinal blood vessels, is a key feature of many prolifer
35 ity of stimulation of cones in the shadow of retinal blood vessels (penumbral cones).
36                                              Retinal blood vessel positional shifts occurred in eyes
37                                              Retinal blood vessels provide information on the risk of
38 rity and diabetic retinopathy, overgrowth of retinal blood vessels results in the formation of pathol
39                       The representations of retinal blood vessels separated by only 0.65 degrees wer
40                      We found that activated retinal blood vessels share many of the endothelial and
41                                              Retinal blood vessel shift was present in 12.1% of minim
42                                              Retinal blood vessel shifts were noted in 33 of 125 (26.
43 ndothelial precursors stabilizes and rescues retinal blood vessels that would ordinarily completely d
44                          The distribution of retinal blood vessels was used to map the location of se
45                        Preretinal nuclei and retinal blood vessels were quantified at peak disease (P
46                                   Leakage of retinal blood vessels, which is an essential element of
47                 Endostatin staining outlined retinal blood vessels, which suggested endostatin bindin