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1 comes in animal models of retinal injury and retinal degenerative disease.
2 ay be an effective therapy for some cases of retinal degenerative disease.
3 ently, there was no approved treatment for a retinal degenerative disease.
4 dead photoreceptor cells in a mouse model of retinal degenerative disease.
5 f immunomodulatory therapeutic approaches to retinal degenerative disease.
6 generation of the mammalian retina following retinal degenerative disease.
7 dCVF gene therapy has potential for treating retinal degenerative disease.
8 inked to retinitis pigmentosa, a progressive retinal degenerative disease.
9 th use in the treatment of genetic causes of retinal degenerative disease.
10 tation-based approaches for the treatment of retinal degenerative disease.
11 pigment epithelium (RPE) are associated with retinal degenerative disease.
12 transplantation is a potential treatment for retinal degenerative disease.
13 identified in patients with several forms of retinal degenerative disease.
14 st report of a transgenic X. laevis model of retinal degenerative disease.
15 considerable potential for the treatment of retinal degenerative disease.
16 s that BCDO may also be a candidate gene for retinal degenerative disease.
17 in vivo quantitation of retinal structure in retinal degenerative disease.
18 These CD-1 mice do not develop the retinal degenerative disease.
19 dominant retinitis pigmentosa, a progressive retinal degenerative disease.
20 ould have significant prophylactic value for retinal degenerative diseases.
21 receptors are affected at the early stage of retinal degenerative diseases.
22 etinal metabolic ecosystem and RPE-initiated retinal degenerative diseases.
23 nistic investigation of alphaSyn lesions and retinal degenerative diseases.
24 es in retinal cells and their involvement in retinal degenerative diseases.
25 unctive treatment in iatrogenic RD and other retinal degenerative diseases.
26 inography, and genetic testing for inherited retinal degenerative diseases.
27 while presenting new therapeutic promise for retinal degenerative diseases.
28 RPE dysfunction plays a significant role in retinal degenerative diseases.
29 the understanding of the early pathology of retinal degenerative diseases.
30 eceptors occurs in inherited and age-related retinal degenerative diseases.
31 restoration of sight in patients blinded by retinal degenerative diseases.
32 potential new therapeutic agent for treating retinal degenerative diseases.
33 ntial treatment options for certain blinding retinal degenerative diseases.
34 nal prosthetics offer hope for patients with retinal degenerative diseases.
35 nd for further drug development for treating retinal degenerative diseases.
36 tions for halting the progression of several retinal degenerative diseases.
37 , which may be a novel therapeutic target in retinal degenerative diseases.
38 hR2-based gene therapy for treating blinding retinal degenerative diseases.
39 el approach for treating blindness caused by retinal degenerative diseases.
40 rtunity for the development of therapies for retinal degenerative diseases.
41 e have been identified in a variety of human retinal degenerative diseases.
42 avonoids could have a prophylactic value for retinal degenerative diseases.
43 a, but they also suggest novel ways to treat retinal degenerative diseases.
44 n in a wide variety of genetically disparate retinal degenerative diseases.
45 velopment of therapeutic approaches to treat retinal degenerative diseases.
46 ses (AAVs) show promise for the treatment of retinal degenerative diseases.
47 volved in the pathology of certain inherited retinal degenerative diseases.
48 ess and detachment in small animal models of retinal degenerative diseases.
49 AG-1 may serve as a potential means to treat retinal degenerative diseases.
50 ons in CRX have been associated with several retinal degenerative diseases.
51 (rAAV) is a promising vector for therapy of retinal degenerative diseases.
52 e significantly to the pathobiology of major retinal degenerative diseases.
53 utility for the treatment and prevention of retinal degenerative diseases.
54 enable characterization of animal models of retinal degenerative diseases and evaluation of therapeu
55 ion playing a central if not primary role in retinal degenerative diseases, and this association shou
56 me editing approaches for treating inherited retinal degenerative diseases, and we discuss important
61 gnaling may be especially useful in treating retinal degenerative diseases arising from rhodopsin mis
62 ause Stargardt disease (STGD1), an inherited retinal degenerative disease associated with the accumul
63 g RBP1 as a promising therapeutic target for retinal degenerative diseases associated with impaired a
64 838) were first identified by linkage to the retinal degenerative disease, autosomal dominant cone ro
65 dysfunction plays a central role in various retinal degenerative diseases, but knowledge is limited
66 raftment yield in regenerative approaches to retinal degenerative disease by stabilising the fate of
75 udy was an autosomal recessive, early-onset, retinal degenerative disease in Persian cats that is lik
79 tion and preventing vision loss secondary to retinal degenerative diseases, including age-related mac
80 tinal barrier (oBRB) is a central feature of retinal degenerative diseases, including age-related mac
81 the molecular mechanisms underlying certain retinal degenerative diseases, including retinitis pigme
82 to image the macular region in patients with retinal degenerative diseases, including two patients wi
85 Photoreceptor cell replacement therapy for retinal degenerative diseases is a promising approach.
87 of all-trans-retinal adducts, resulting in a retinal degenerative disease known as Stargardt-1 diseas
88 re associated with a wide range of inherited retinal degenerative diseases, leading to blindness.
89 as a potential therapeutic strategy to treat retinal degenerative diseases like retinitis pigmentosa
90 potential to restore vision to patients with retinal degenerative diseases like retinitis pigmentosa.
92 The death of photoreceptor cells caused by retinal degenerative diseases often results in a complet
93 e-causing variants in relatives of inherited retinal degenerative disease patients with multiple ABCA
95 d retinal vascular disease; gene therapy for retinal degenerative disease; regenerative medicine, inc
99 sion of Bcl-2 in photoreceptors of mice with retinal degenerative disease slows progression of the di
100 ing photoreceptors in the retina, leading to retinal degenerative diseases such as age-related macula
101 tages of RP is very similar to that of other retinal degenerative diseases such as age-related macula
102 bind sigmaR1 ligands may prove beneficial in retinal degenerative diseases such as diabetic retinopat
103 useful vision to people who are affected by retinal degenerative diseases such as retinitis pigmento
104 cells holds great promise for patients with retinal degenerative diseases, such as age-related macul
108 ed with Leber congenital amaurosis, a severe retinal degenerative disease that causes blindness durin
109 Age-related macular degeneration (AMD) is a retinal degenerative disease that is the leading cause o
112 Leber's congenital amaurosis, a progressive retinal degenerative disease that severely impairs sight
113 restoring functional vision in patients with retinal degenerative diseases, the eyes of blind human s
114 function or morphogenesis leads to blinding retinal degenerative diseases, the majority of which hav
115 restoring functional vision in patients with retinal degenerative diseases, USC/Second Sight Medical
117 mitigate the development and progression of retinal degenerative diseases via an array of distinct m
119 a (RP) is the most common group of inherited retinal degenerative diseases, whose most debilitating p
120 d retinitis pigmentosa is a severe inherited retinal degenerative disease with a frequency of 1 in 10
121 Retinitis pigmentosa (RP) is an inherited retinal degenerative disease with severe vision impairme
122 ighlight the severe nature of this inherited retinal degenerative disease with significant damage to
124 kinetic profile for the treatment of chronic retinal degenerative diseases without systemic exposure.