コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 iary body hemorrhage, and 11% had peripheral retinal hemorrhage).
2 Large focal leak was observed as an evolving retinal hemorrhage.
3 classical posterior localization of neonatal retinal hemorrhage.
4 astrocyte-associated vasculature leading to retinal hemorrhage.
5 teristics of ROP, pre-plus/plus disease, and retinal hemorrhage.
6 on existed between vaccination injection and retinal hemorrhage.
7 7, 14, or 21 days preceding examination and retinal hemorrhage.
8 of beta-APP and ubiquitin immunostaining and retinal hemorrhage.
9 LFL was observed as an evolving retinal hemorrhage.
10 inal vascular permeability, leukostasis, and retinal hemorrhage.
11 ction of collagenase mimicked PK's effect on retinal hemorrhage.
12 yes, a potential pitfall in the diagnosis of retinal hemorrhages.
13 ndant microaneurysms, leaky capillaries, and retinal hemorrhages.
14 linically appreciated, especially related to retinal hemorrhages.
15 by the presence of cotton wool spots and/or retinal hemorrhages.
16 actures, subdural hematoma of the brain, and retinal hemorrhages.
17 a "double-ring sign" accompanied by diffuse retinal hemorrhages.
18 e of 0.18 (95% CI: 0.10-0.30, I(2) = 94.1%), retinal hemorrhage 0.08 (95% CI: 0.05-0.12, I(2) = 77.8%
19 he most frequent ocular AEs (study eye) were retinal hemorrhage (12.8%; 1 event related to study drug
20 bridging vein thrombosis, 74 of 99 (75%) had retinal hemorrhages, 23 of 72 (32%) had fractures, 26 of
21 The most common ocular manifestations were retinal hemorrhages (25.86%, n = 15), Purtscher-like ret
22 one spicules, retinal vascular sheathing, or retinal hemorrhages; (3) examination with >1+ anterior c
25 s with diabetic retinopathy, suggesting that retinal hemorrhage and erythrocyte lysis contribute to t
26 linical significance in a patient's risk for retinal hemorrhage and likely warrant periodic surveilla
27 rent literature regarding the association of retinal hemorrhage and subarachnoid bleeds in infants wh
28 n significantly inhibited the development of retinal hemorrhages and acellular capillaries over the 5
29 r Purtscher-like retinopathy is diagnosed by retinal hemorrhages and areas of retinal whitening on fu
31 n the left eye, it was associated with intra-retinal hemorrhages and diagnosed as macular branch reti
34 ays) revealed extensive vascular thrombosis, retinal hemorrhages and necrosis, and a dense inflammato
36 occur early in the disease process, whereas retinal hemorrhages and retinal lipid may occur later.
38 r agreement for overall retinal pathologies, retinal hemorrhage, and maculopathy were substantial bot
40 he presence of preplus disease, stage 2 ROP, retinal hemorrhage, and the need for ventilation at time
42 5%) had extraaxial hemorrhages, 52 (51%) had retinal hemorrhages, and 35 (35%) had evidence of acute
44 trauma score, worse prerepair visual acuity, retinal hemorrhage, anterior vitrectomy at primary repai
47 the retina, consistent with the finding that retinal hemorrhages are often found in multiple layers o
50 rrhages, optic nerve sheath hemorrhages, and retinal hemorrhages--are generally thought to be limited
52 ome was the association between COVID-19 and retinal hemorrhage assessed across 4 periods: retinal ex
53 hod provides an automated means of detecting retinal hemorrhages associated with malarial retinopathy
54 dus exhibited superior venous tortuosity and retinal hemorrhages compatible with incomplete branch re
55 posterior segment findings that include both retinal hemorrhages, cotton wool spots, and macular edem
56 liferative diabetic retinopathy (PDR), cause retinal hemorrhage, detachment, and eventually blindness
58 fundus examinations, 9 of 5177 children had retinal hemorrhage for a prevalence of 0.17% (95% CI, 0.
60 children with positive COVID-19 testing and retinal hemorrhage had alternative plausible causes with
64 e found between COVID-19 and the presence of retinal hemorrhage in any of the periods: 7 days (odds r
65 hould not be considered a potential cause of retinal hemorrhage in children, and this unsupported the
66 ld not be considered as a potential cause of retinal hemorrhage in children, and this unsupported the
67 accinations have been proposed as a cause of retinal hemorrhage in children, primarily as part of a d
70 ssociation between vaccination injection and retinal hemorrhage in the prior 7 days (P > .99), 14 day
71 chamber and vitreous inflammation, sectoral retinal hemorrhages in areas of ischemia, and predilecti
73 important implications for the diagnosis of retinal hemorrhages in potential cases of nonaccidental
74 the biomechanical cause underlying neonatal retinal hemorrhage include elevated intracranial pressur
75 es, [kappa (k); 95 % CI = 0.59 (0.51-0.66)], retinal hemorrhage [k; 95 % CI = 0.60 (0.41-0.78)], and
76 0.52 (0.44-0.60)], substantial agreement for retinal hemorrhage [k; 95 % CI = 0.68 (0.52-0.84)], mode
77 dation (n = 6), epiretinal membrane (n = 3), retinal hemorrhage (n = 2), vitreous hemorrhage (n = 1),
78 r crack (n = 6, 1.8%), T-sign (n = 6, 1.8%), retinal hemorrhage (n = 3, 0.9%), active myopic choroida
79 , including conjunctival hemorrhage (n = 5), retinal hemorrhage (n = 4), and vitreous floaters (n = 4
80 seeds (n = 3), vitreous hemorrhage (n = 2), retinal hemorrhage (n = 4), subretinal fluid (n = 4), an
81 mplications included DES (n = 7 [14%]), (sub)retinal hemorrhage (n = 6 [12%]), optic disc edema (n =
82 ual loss associated with retinal vasculitis, retinal hemorrhage, non-confluent posterior retinal infi
88 ase in the mean number of microaneurysms and retinal hemorrhages on UWF CFP at M3 versus M0 (n = 40+/
89 otographs: 1) no retinopathy, 2) presence of retinal hemorrhages only, 3) presence of retinal microan
91 hage (OR, 2.43; 95% CI, 1.22-4.83; P = .01), retinal hemorrhage (OR, 2.17; 95% CI, 1.40-3.38; P < .00
92 5% CI, 2.13-8.00 vs no ROP), the presence of retinal hemorrhage (OR, 4.36; 95% CI, 1.57-12.1 vs absen
93 c nerve sheath, intrascleral hemorrhage, any retinal hemorrhage, ora-extended hemorrhage, cherry hemo
94 d children with head injury, the presence of retinal hemorrhages (positive LR, 11.0 [95% CI, 4.0-32.0
95 fundoscopic findings of optic disc edema and retinal hemorrhages, possibly consistent with a papillop
97 3 years, the presence of extensive bilateral retinal hemorrhages raises a very strong possibility of
98 als have shown that VEGF inhibition improves retinal hemorrhages, retinal vessel closure, and progres
99 n is often missed in medical encounters, and retinal hemorrhage (RH) is considered strong evidence fo
101 stress patterns consistent with the diffuse retinal hemorrhages (RH) typically found in the posterio
102 d retinopathy (ROR: 9.424 [1.081-3.406]) and retinal hemorrhage (ROR: 10.253 [0.319-4.336]) in non-T2
103 t surgery; retinal tear; retinal detachment; retinal hemorrhages; scotomas; and an increased number o
104 l deformation is a more likely mechanism for retinal hemorrhage than occlusion of the central retinal
110 he most common anticipated severe AE (severe retinal hemorrhage) was eliminated in the second cohort
116 al characteristics (subdural hemorrhages and retinal hemorrhages) were compared before and after the
117 p papillophlebitis with optic disc edema and retinal hemorrhages, which may be associated with a hype
118 ld intraocular inflammation in both eyes and retinal hemorrhages with an apparent choroidal neovascul