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1 is and directing treatment in cases of acute retinal necrosis.
2 ral eye becomes infected and undergoes acute retinal necrosis.
3 e that T cells play a role in development of retinal necrosis.
4 al retina compatible with diagnosis of acute retinal necrosis.
5 e retina, despite initial lack of observable retinal necrosis.
6               Six of 7 specimens had foci of retinal necrosis.
7 gmentation and thinning, iris recession with retinal necrosis and hypotony, a filtering conjunctival
8                                              Retinal necrosis and loss of retinal architecture throug
9 arrive in the sensory retina at the onset of retinal necrosis and not during acute retinitis and the
10 al injury, with central sclopetaria retinae, retinal necrosis, and surrounding commotio retinae with
11 etinal whitening without clinically apparent retinal necrosis] and necrotizing retinochoroiditis) at
12 antiviral therapy for the treatment of acute retinal necrosis (ARN) and risk factors impacting visual
13                                        Acute retinal necrosis (ARN) is an infectious retinitis primar
14 fy determinants of adverse outcomes in acute retinal necrosis (ARN), presenting characteristics and i
15 simplex virus-type 2 (HSV-2) can cause acute retinal necrosis (ARN), which can lead to exudative and
16 s about the diagnosis and treatment of acute retinal necrosis (ARN).
17 le antiviral therapy for patients with acute retinal necrosis (ARN).
18  poor visual outcomes in patients with acute retinal necrosis (ARN).
19 c necrotizing MCMV retinitis (full-thickness retinal necrosis associated with virus inclusions and cy
20  in Sight) Registry Study showing that acute retinal necrosis cases treated with systemic antivirals
21            Herein, we report a case of acute retinal necrosis caused by co-infection with herpes simp
22                                        Acute retinal necrosis caused by co-infection with multiple vi
23                              We report acute retinal necrosis caused by the vaccine Oka strain follow
24  sensory retina coincident with the onset of retinal necrosis (day 11 p.i.), and CD4+ and CD8+ T cell
25 e considered in the physiopathology of acute retinal necrosis, especially in immunosuppressed patient
26 /6 mice and exhibited absolute resistance to retinal necrosis following subretinal MCMV inoculation,
27                                        Acute retinal necrosis has been described as a clinical entity
28                  The pure enzyme also caused retinal necrosis in vivo.
29                                        Acute retinal necrosis is a potentially visually devastating n
30                                        Acute retinal necrosis is caused by the herpes group of viruse
31                                        Acute retinal necrosis is considered a rare infectious uveitis
32 emorrhagic retinitis (n = 13) and peripheral retinal necrosis (n = 7).
33 d other rarer ocular disorders such as acute retinal necrosis syndrome or neuroretinitis.
34   We also propose a new hypothesis for acute retinal necrosis that occurs post-immunization.
35 d recurrent lymphocytic meningitis and acute retinal necrosis, we identified two patients heterozygou