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1 c variants, and visualization of the central retinal vein.
2 nal hemorrhage than occlusion of the central retinal vein.
3 etinal hemorrhages, and dilated and tortuous retinal veins.
4 anchnic, upper-extremity, renal, ovarian, or retinal veins.
5 chemic by laser occlusion of the main branch retinal veins.
7 arterial occlusion (5/16), combined central retinal vein and cilioretinal artery occlusion (4/16), a
9 th, elevation of the pressure in the central retinal vein, and impaired perfusion of the neurons as t
10 was used to measure P(O2) separately in the retinal veins, arteries, and capillaries and in the chor
12 and hemodynamic changes in a major temporal retinal vein by laser Doppler, before and after plasmaph
13 SPCA), central retinal artery (CRA), central retinal vein (CRV), peripapillary choroid and sclera, an
14 e vision loss due to blockage of the central retinal vein (CRVO) or a branch retinal vein (BRVO).
17 al retinal artery equivalent (CRAE), central retinal vein equivalent (CRVE), and arteriole-to-venule
18 al retinal artery equivalent (CRAE), central retinal vein equivalent (CRVE), and arteriole-to-venule
19 etinal artery equivalent (CRAE), and central retinal vein equivalent (CRVE), in pediatric subjects wi
20 t veins in zone B, summarized by the central retinal vein equivalent (CRVE), the arteriole to venule
21 entral retinal artery equivalent and central retinal vein equivalent from baseline to 1 week (from 17
26 were measured in the major superior temporal retinal vein in each subject by using a laser Doppler in
27 d by a robot micromanipulator, while a major retinal vein near the optic disc was occluded by argon l
28 hile 7 eyes demonstrated obvious findings of retinal vein obstruction (5 with central and 2 with hemi
30 ondary to outflow obstruction from a central retinal vein obstruction appears to be the most common c
31 bstruction (typically central or hemicentral retinal vein obstruction) using en face optical coherenc
33 pathy; (3) laser photocoagulation for branch retinal vein obstruction; (4) diabetic vitrectomy; (5) t
37 d by diabetic retinopathy (11 eyes), central retinal vein occlusion (6 eyes), ocular ischemic syndrom
38 urtscher's retinopathy (4 eyes, 3 patients), retinal vein occlusion (7 eyes, 7 patients), central ret
39 was associated with a 40% increased rate of retinal vein occlusion (AIRR 1.4, 95% Confidence Interva
41 ety outcomes in eyes with branch and central retinal vein occlusion (BRVO and CRVO) treated with dexa
44 tral retinal vein occlusion (CRVO) or branch retinal vein occlusion (BRVO) causes hypoperfusion, high
45 ral retinal vein occlusion (CRVO) and branch retinal vein occlusion (BRVO) complicated by macular ede
47 olume distensibility in patients with branch retinal vein occlusion (BRVO) in comparison with normal
48 natural history of visual outcome in branch retinal vein occlusion (BRVO) is fundamental to its mana
51 optic nerve head (ONH) parameters and branch retinal vein occlusion (BRVO) using spectral domain opti
52 venous crossings are risk factors for branch retinal vein occlusion (BRVO), an eye disease in which c
59 ng data exists on whether central and branch retinal vein occlusion (CRVO and BRVO) are linked to sys
60 aphy angiography (OCTA) in eyes with central retinal vein occlusion (CRVO) and branch retinal vein oc
61 o review the definition of ischaemic central retinal vein occlusion (CRVO) and stratify the risk of n
62 retinal artery occlusion (CRAO) and central retinal vein occlusion (CRVO) as a complication of persi
64 ed retinal vein occlusion (BRVO) and central retinal vein occlusion (CRVO) have better functional res
67 rticipants with macular edema due to central retinal vein occlusion (CRVO) or hemiretinal vein occlus
68 ents with macular edema secondary to central retinal vein occlusion (CRVO) or hemiretinal vein occlus
69 ble to macular edema associated with central retinal vein occlusion (CRVO) or hemiretinal vein occlus
72 fty-five patients with a nonischemic central retinal vein occlusion (CRVO) who were randomized to rec
73 ibe a patient who developed combined central retinal vein occlusion (CRVO), cilioretinal artery occlu
74 To review the clinical picture of central retinal vein occlusion (CRVO), with an emphasis on recen
83 etic macular edema (DME), central and branch retinal vein occlusion (CRVO/BRVO), central serous chori
84 eneration (HR = 1.29; 95% CI: 1.08-1.54) and retinal vein occlusion (HR = 3.94; 95% CI: 3.11-4.99).
85 occlusion (HR: 1.13, 95% CI: 1.02-1.26) and retinal vein occlusion (HR: 1.26, 95% CI: 1.20-1.33).
86 86), glaucoma (HR: 1.40, 95% CI: 1.05-1.88), retinal vein occlusion (HR: 1.58, 95% CI: 1.23-2.03), co
88 h retinal arterial occlusion (n = 3), branch retinal vein occlusion (n = 1), vitamin A deficiency (n
89 E (RR 19.5), diabetic retinopathy (RR 13.1), retinal vein occlusion (RR 12.9), macular hole (RR 7.7),
90 e (ERM) (RR, 4.1, CI, 2.63-6.19), history of retinal vein occlusion (RR, 2.94, CI, 1.75-4.93), diabet
91 .07), uveitis (RR, 2.88; 95% CI, 1.50-5.51), retinal vein occlusion (RR, 4.47; 95% CI, 2.56-5.92), or
94 R), retinopathie of prematurity (ROP) or the retinal vein occlusion (RVO) are caused through a hypoxi
95 he clinical unmet needs in the management of Retinal Vein Occlusion (RVO) associated Macular Edema (M
97 Results of ocular biometric measurements in retinal vein occlusion (RVO) eyes are still inconclusive
98 is study evaluated the treatment outcomes of retinal vein occlusion (RVO) in a routine clinical pract
99 the prevalence, pattern and risk factors of retinal vein occlusion (RVO) in an elderly population of
102 the treatment of macular edema secondary to retinal vein occlusion (RVO) in treatment-naive patients
108 ic, and gender composition of the cohorts of retinal vein occlusion (RVO) macular edema (ME) clinical
112 ion (AMD), diabetic macular edema (DME), and retinal vein occlusion (RVO) were evaluated by Pearson c
114 sented with no RVD, 20 patients (25.6%) with retinal vein occlusion (RVO), 16 patients (20.5%) with r
115 ses, including diabetic retinopathy (DR) and retinal vein occlusion (RVO), and neovascular macular di
116 lopathy including diabetic retinopathy (DR), retinal vein occlusion (RVO), and neovascular-age relate
117 ents with diabetic retinopathy (DR), AMD and retinal vein occlusion (RVO), and to identify potential
118 Participants were diagnosed with ME from retinal vein occlusion (RVO), diabetic retinopathy (DR;
119 pants (n = 1063) were diagnosed with ME from retinal vein occlusion (RVO), diabetic retinopathy (DR;
120 horoidal neovascularization (CNV) and 1 with retinal vein occlusion (RVO), experienced disease quiesc
121 ve regimen for diabetic macular edema (DME), retinal vein occlusion (RVO), noninfectious uveitis macu
122 ation (AMD), diabetic macular edema (DME) or retinal vein occlusion (RVO), receiving intravitreal tre
135 ), diabetic macular edema (DME, n = 400), or retinal vein occlusion (RVO, n = 400) acquired with Zeis
136 The Standard of Care vs Corticosteroid for Retinal Vein Occlusion (SCORE) Study showed that intravi
137 the Standard Care versus COrticosteroid for REtinal Vein Occlusion (SCORE)-CRVO trial, CRUISE Study,
138 ercept in Subjects with Macular Edema Due to Retinal Vein Occlusion (TANZANITE) study who received ei
139 nsive retinopathy 0.88%, macular scar 0.37%, retinal vein occlusion 0.50%, macular hole 0.20%, retini
140 s in the Study of COmparative Treatments for REtinal Vein Occlusion 2 (SCORE2) and to compare with co
141 d in the Study of Comparative Treatments for Retinal Vein Occlusion 2 (SCORE2) to be noninferior to a
143 ) in the Study of COmparative Treatments for REtinal Vein Occlusion 2 (SCORE2); evaluate the baseline
144 from the Study of Comparative Treatments for Retinal Vein Occlusion 2 randomized clinical trial inclu
145 d in the Study of Comparative Treatments for Retinal Vein Occlusion 2, and 88 participants randomized
147 at 6 and 12 months was 15.0 to 16.5 (central retinal vein occlusion [CRVO]) and 17.4 to 19.1 (branch
148 hundred eyes of 100 patients (79 with branch retinal vein occlusion and 21 with central retinal vein
149 derived anatomic measurements between branch retinal vein occlusion and central retinal vein occlusio
152 thickness in patients suffering from central retinal vein occlusion and low visual acuity (<0.1) in c
154 retinopathy, retinopathy of prematurity and retinal vein occlusion are potentially blinding conditio
155 acentral acute middle maculopathy (PAMM) and retinal vein occlusion are uncommon concurrent findings
156 sive patient, who developed bilateral branch retinal vein occlusion at 6T, the therapy was discontinu
157 d points, AF was not associated with central retinal vein occlusion but was associated with urinary t
160 en were associated with an increased rate of retinal vein occlusion compared to non-use, although the
161 uently developed retinal artery occlusion or retinal vein occlusion compared with patients undergoing
163 e tomography angiography (OCTA) among branch retinal vein occlusion disease (BRVO) cases with macular
165 and the absence of such a difference in the retinal vein occlusion group could be explained by edema
166 = 0.024); however, the same analysis in the retinal vein occlusion group revealed no significant dif
167 en branch retinal vein occlusion and central retinal vein occlusion groups (all P >/= 0.058); therefo
169 to 28 % of patients presenting with central retinal vein occlusion have a baseline BCVA of less than
171 ged 18 years with macular edema secondary to retinal vein occlusion in the branch vein occlusion (BRA
174 Retinal ischemic damage associated with retinal vein occlusion is exacerbated by fluid extravasa
177 pressure in the cat after acute experimental retinal vein occlusion may define the role of intravascu
179 retinal vein occlusions (both <0.1 per 1000 retinal vein occlusion patients in 2011, 5.6 and 140.2 i
180 retrospective chart review was performed on retinal vein occlusion patients treated by three anti-va
182 pathy, age-related macular degeneration, and retinal vein occlusion receiving intravitreal injections
186 2 patients, one each with Eales' disease and retinal vein occlusion the procedure was unsuccessful, n
189 rom 13 patients with diabetic retinopathy or retinal vein occlusion underwent both UWF-FA (Optos Silv
194 tinal artery occlusion and 463 patients with retinal vein occlusion were enrolled and matched for age
198 Treatment of macular edema secondary to retinal vein occlusion with ranibizumab has been shown t
199 rysm with subretinal hemorrhage (1), central retinal vein occlusion with vitreous hemorrhage (1), and
200 h retinal vein occlusion and 21 with central retinal vein occlusion) were recruited in the study.
201 ction (5 with central and 2 with hemicentral retinal vein occlusion), 4 eyes were unremarkable at pre
203 tinal artery occlusion and 463 patients with retinal vein occlusion, 66 (58.4%) and 245 (52.9%) were
204 omplete data; concomitant diseases including retinal vein occlusion, age-related macular degeneration
205 contributor to macular edema resulting from retinal vein occlusion, also confirmed in multicenter tr
206 17 trials (23%) included 3757 patients with retinal vein occlusion, and 1 trial (1%) included 122 pa
208 , in models of retinal degeneration, central retinal vein occlusion, and oxygen-induced retinopathy.
209 nal vascular diseases, diabetic retinopathy, retinal vein occlusion, and retinal artery occlusion, ma
210 such as proliferative diabetic retinopathy, retinal vein occlusion, and retinal detachment, than in
211 omplication of retinal ischemia in diabetes, retinal vein occlusion, and retinopathy of prematurity.
212 nal diseases including diabetic retinopathy, retinal vein occlusion, and retinopathy of prematurity.
213 n were the number of drug injections, having retinal vein occlusion, and under 60 years of age, while
215 ab and steroid implants can be considered in retinal vein occlusion, but trials are awaited to determ
216 ration (nAMD), diabetic macular edema (DME), retinal vein occlusion, choroidal neovascularization (CN
217 ular diseases (e.g., diabetic macular edema, retinal vein occlusion, choroidal neovascularization) ha
218 ataract, uveitis, dry eye disease, glaucoma, retinal vein occlusion, conjunctivitis, meibomian gland
219 ataract, uveitis, dry eye disease, glaucoma, retinal vein occlusion, conjunctivitis, meibomian gland
220 the amount of neovascularization induced by retinal vein occlusion, corneal growth factor pellets, a
221 cation of diabetes mellitus, prematurity, or retinal vein occlusion, is a major cause of blindness wo
223 g, contralateral PCME, diabetic retinopathy, retinal vein occlusion, macular hole, epiretinal membran
224 macular edema, macular edema associated with retinal vein occlusion, myopic choroidal neovascularizat
225 , diarrhoea, myocardial infarction, pyrexia, retinal vein occlusion, n=1 each; placebo: vomiting, whi
226 , leakage such as macular edema secondary to retinal vein occlusion, or a combination of proliferatio
229 This suggests that prompt treatment for retinal vein occlusion, particularly BRVO, may be associ
230 agent ranibizumab in diabetic macular edema, retinal vein occlusion, pseudophakic macular edema, and
231 diverse conditions as diabetic retinopathy, retinal vein occlusion, retinopathy of prematurity, exud
232 condary to AMD or Macular Edema Secondary to Retinal Vein Occlusion, Safety Assessment of Intravitrea
233 conditions, including diabetic retinopathy, retinal vein occlusion, sickle cell retinopathy, uveitis
234 te to progression of retinal nonperfusion in retinal vein occlusion, the same is true in patients wit
235 , and 4 negative control end points (central retinal vein occlusion, urinary tract infection, humeral
236 mic diseases including diabetic retinopathy, retinal vein occlusion, uveitis, and HIV-related retinit
238 e patients, 3 with branch and 9 with central retinal vein occlusion, were imaged in 27 sessions with
239 tic neuropathy in contrast to what occurs in retinal vein occlusion, where edema affects all retinal
240 eyes with macular edema secondary to central retinal vein occlusion, which were treated with a dexame
241 (nAMD), diabetic macular edema (DME), branch retinal vein occlusion-related macular edema (BRVO-ME),
242 related macular edema (BRVO-ME), and central retinal vein occlusion-related macular edema (CRVO-ME).
269 patients with diabetic retinal diseases and retinal vein occlusions (both <0.1 per 1000 retinal vein
270 pth (VCD) and both central (CRVO) and branch retinal vein occlusions (BRVO) using optical low coheren
271 sting macular edema (ME) due to diabetes and retinal vein occlusions (RVO) make up a growing populati
272 rative diabetic retinopathy (PDR), 23.3% had retinal vein occlusions (RVO), and the remaining 15.0% s
273 story of retinal artery occlusions (RAOs) or retinal vein occlusions (RVOs) more than 6 months before
274 phase 3 Study of COmparative Treatments for REtinal Vein Occlusions 2 (SCORE2) clinical trial includ
275 sc were made for 13 patients with DE (7 with retinal vein occlusions and 6 with inflammatory optic ne
276 ckings (AVNs) in the retina are the cause of retinal vein occlusions and are also surrogates of cereb
277 ortant implications for the understanding of retinal vein occlusions and of cerebrovascular aging.
279 Although both retinal artery occlusions and retinal vein occlusions are associated with increased ag
280 with stroke expertise, whereas treatment of retinal vein occlusions is provided by ophthalmologists.
281 caused by inflammatory optic neuropathies or retinal vein occlusions on optical coherence tomography
282 jacent retinal arterioles than to blood from retinal vein occlusions or adjacent retinal venules, sug
283 CE PATTERN(R) GUIDELINES: New evidence-based Retinal Vein Occlusions Preferred Practice Pattern(R) (P
287 erative diabetic retinopathy), 8.3% to treat retinal vein occlusions, and 12.9% for all other uses.
288 eovascular age-related macular degeneration, retinal vein occlusions, or diabetic macular edema were
293 horoidal sinus communicates with the central retinal vein, the choriocapillaris, and with large veins
294 ersus 4.78 +/- 0.37 um, p = .041), and wider retinal vein trajectories (c' = 2.48 +/- 0.84 vs 3.39 +/
295 ue plasminogen activator, 3) cannulating the retinal vein transvitreally, or 4) transecting the poste
296 inogen activator (rt-PA), 3) cannulating the retinal vein transvitreally, or 4) transecting the poste
297 ere used to create a vascular overlay of the retinal veins vs the retinal arteries to map the distrib