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3 tion of the primate SCN overlaps that of the retinohypothalamic input and the VIP neuronal population
5 photic integration and suggest a dichotomous retinohypothalamic network leading to circadian phase sh
7 ypothalamic area supports the view that this retinohypothalamic projection is anatomically and functi
8 had no effect on glutamate release from the retinohypothalamic projection, suggesting a direct actio
10 ic manipulation in mice to show that another retinohypothalamic target, the anterior lateral hypothal
11 A are manifested downstream from presynaptic retinohypothalamic terminals and therefore are intrinsic
12 ves input from the ventrolateral SCN and the retinohypothalamic tract (Leak and Moore [] J Comp Neuro
13 receives photic information directly via the retinohypothalamic tract (RHT) and indirectly from retin
16 Photic information is transmitted via the retinohypothalamic tract (RHT) to the suprachiasmatic nu
19 (SCN) receives direct retinal input via the retinohypothalamic tract (RHT), and the retinal ganglion
20 which entraining stimuli reach the SCN: the retinohypothalamic tract (RHT), which transmits light in
25 rojection overlaps the terminal field of the retinohypothalamic tract in the SCN core, the central pa
26 matic nucleus (SCN) are at the origin of the retinohypothalamic tract that transmits the light signal
27 sting a direct action of cannabinoids on the retinohypothalamic tract was unlikely to explain the inh
29 ssed in retinal ganglion cells that form the retinohypothalamic tract, a neuronal connection between
30 ing, including electrical stimulation of the retinohypothalamic tract, produce releasate mass spectra
31 nucleus (SCN), the SCN's primary relay, the retinohypothalamic tract, secondary pathways originating
33 The primary entertainment pathway is the retinohypothalamic tract, which terminates in the circad
34 surrogate biomarker for the integrity of the retinohypothalamic tract, with potential utility for inv
35 these peptides, little SAAS, exhibits robust retinohypothalamic tract-stimulated release from the SCN