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1 ene), and M(retinol)+10 (from [(1)(3)C(1)(0)]retinyl acetate).
2 umber of cones that was ameliorated by 9-cis-retinyl acetate.
3 ne the affinity constants of fenretinide and retinyl acetate.
4 dependence and the differentiating power of retinyl acetate.
5 gredient, the 9-cis-retinal precursor, 9-cis-retinyl acetate.
6 cluding retinoic acid, retinol, retinal, and retinyl acetate.
8 g the response to the dose of [(1)(3)C(1)(0)]retinyl acetate (0.5 mg) as a reference, our results (wi
10 dioactive and nonradioactive form, 4-hydroxy-retinyl acetate (4-OH-RAc), and 5,6-epoxy-RA, all of whi
13 Long-term effects of treatment with 9-cis-retinyl acetate (9-cis-R-Ac), an artificial retinoid pro
14 congenital amaurosis with 9-cis-BC and 9-cis-retinyl-acetate, a well established 9-cis-retinal precur
16 orally with 30 micromol hexadeuterated (D6) retinyl acetate (all-trans-19,19,19,20,20,20-[2H6]retiny
17 yl acetate (all-trans-19,19,19,20,20,20-[2H6]retinyl acetate) and then with 37 micromol D6 beta-carot
18 retinoids, including retinoic acid, retinol, retinyl acetate, and other retinyl esters, using postsou
20 n (DRD) technique at a dose of 5 mg [(2)H(4)]retinyl acetate at baseline and 5 mg [(2)H(8)]retinyl ac
21 al of provitamin A carotenoids by using [2H4]retinyl acetate (d4-RA) as an extrinsic reference standa
25 ma retinol kinetics of an oral dose of [2H4] retinyl acetate in 4 healthy adults (2 men and 2 women)
26 drawn, and subjects consumed 1 mg [(13)C(10)]retinyl acetate in a 0.5-g corn oil capsule and 300 g wh
31 all-trans-retinoic acid (RA), 13-cis-RA, and retinyl acetate] markedly suppressed PMA-mediated increa
32 eriod, the women were given 30 micromol D(6) retinyl acetate orally, followed 1 wk later with 37 micr
33 h the artificial chromophore pro-drug, 9-cis-retinyl acetate, partially protected inferior retinal co
34 ough activation of c-Myc, and that all-trans-retinyl acetate (RAc) independently upregulates tumour n
38 employed for simultaneous quantification of retinyl acetate, retinyl palmitate, alpha-tocopherol and
40 cid (all-trans-RA), 9-cis-RA, 13-cis-RA, and retinyl acetate] suppressed both basal levels of Cox-2 a
41 n a defined culture medium supplemented with retinyl acetate, through HOXA-patterned mesoderm to hemo
42 94) were administered 1.0 mumol [14,15]-13C2-retinyl acetate to estimate total liver retinol reserves
43 e cleavage of the acetyl ester of vitamin A (retinyl acetate) to all-trans-retinal, also by isoform 1
48 ion for retinoic acid, retinal, retinol, and retinyl acetate were 23 pg, 1.0 ng, 0.5 ng, and 10 ng, r
49 amine), and an artificial chromophore (9-cis-retinyl acetate) were evaluated side by side in a recent