ライフサイエンスコーパス: retrograde amnesia

1 loss of explicit, recently formed memories (retrograde amnesia).

2 hat was acquired before the damage occurred (retrograde amnesia).

3 nts that occurred shortly before the injury (retrograde amnesia).

4 e also may have contributed to his extensive retrograde amnesia.

5 as directly addressed the area of malingered retrograde amnesia.

6 which was prevented under anisomycin-induced retrograde amnesia.

7 ant to extending the malingering paradigm to retrograde amnesia.

8 though the same lesions produced devastating retrograde amnesia.

9 grade amnesia and a 1-30 d temporally graded retrograde amnesia.

10 arning to determine the nature and extent of retrograde amnesia.

11 to surgery, indicating that they had a dense retrograde amnesia.

12 ade amnesia with no temporal gradient in the retrograde amnesia.

13 campal formation can cause temporally graded retrograde amnesia.

14 se, and recovery from functional and organic retrograde amnesia.

15 which gradually progresses into more severe retrograde amnesia.

16 Retrograde amnesia after an associative learning task ca

17 Observations of temporally graded retrograde amnesia after hippocampal damage suggest that

18 evious explanations of memory recovery after retrograde amnesia and critically challenges the traditi

19 that addresses the assessment of malingered retrograde amnesia and evidences that a critical moment

20 molecular and cellular mechanisms underlying retrograde amnesia and memory.

21 function, which is revealed behaviorally as retrograde amnesia and physiologically as a blockade of

22 gs emphasize the difference in the extent of retrograde amnesia associated with hippocampal lesions a

23 Y-83583 produced significant anterograde and retrograde amnesia at doses that did not impair performa

24 retrieval of previously stored information (retrograde amnesia), but did not produce anterograde amn

25 the 2-month follow-up, patients had reduced retrograde amnesia, but continued to show deficits in re

26 their crimes has emphasized that malingered retrograde amnesia can be identified with relevant asses

27 emporal lobe (MTL) lesions typically produce retrograde amnesia characterized by the disproportionate

28 ch hub identified after learning resulted in retrograde amnesia, confirming the behavioral significan

29 (3) Extensive, temporally graded retrograde amnesia covering 15 years or more can occur a

30 st, the H group exhibited temporally limited retrograde amnesia covering approximately 5 years.

31 re anterograde amnesia and temporally graded retrograde amnesia covering as much as 25 years.

32 e test, the MTL group exhibited an extensive retrograde amnesia covering decades.

33 pacity for learning facts and events and had retrograde amnesia covering several decades.

34 de amnesia), together with temporally graded retrograde amnesia covering ~5 y prior to the cardiac ar

35 0 sec or 15 sec after trial 1 induced clear retrograde amnesia: escape latencies on trial 2 were no

36 ied 45 sec or 30 sec after trial 1 caused no retrograde amnesia: escape latencies on trial 2 were the

37 (v) Can retrograde amnesia ever be "isolated"?

38 retrograde amnesia; (iii) psychogenic focal retrograde amnesia following a minor neurological episod

39 Results from recent studies of retrograde amnesia following damage to the hippocampal c

40 These studies may help explain retrograde amnesia following seizures.

41 her, following selective hippocampal damage: retrograde amnesia for episodic memories is temporally l

42 ately severe anterograde amnesia and limited retrograde amnesia for facts and events that affected, a

43 The patients also exhibit temporally limited retrograde amnesia for factual information from the seve

44 ippocampal damage produces temporally graded retrograde amnesia for the social transmission of a food

45 ross all time periods, whereas the two focal retrograde amnesia groups showed a 'reversed' temporal g

46 By contrast, the two focal retrograde amnesia groups showed less improvement and co

47 en exhaustively studied, whereas research on retrograde amnesia has tended to focus upon functional a

48 zed as: (i) fugue state; (ii) fugue-to-focal retrograde amnesia; (iii) psychogenic focal retrograde a

49 tion results in a profound temporally graded retrograde amnesia, implying that it is necessary for me

50 ircuits in the neocortex mediate progressive retrograde amnesia in AD.

51 oadly, triggering windows of anterograde and retrograde amnesia in healthy people.

52 activation of the engram network, leading to retrograde amnesia in HFHI mice.

53 t prominent in recent discussion: studies of retrograde amnesia in memory-impaired patients who have

54 urrent data on the nature of anterograde and retrograde amnesia in the degenerative diseases, and als

55 viding evidence for the occurrence of 'focal retrograde amnesia' in transient epileptic amnesia.

56 fect of thalamocortical synaptic coupling on retrograde amnesia induced by hippocampal lesions.

57 ed by 5-HTT polymorphism, an emotion-induced retrograde amnesia is expressed solely in the presence o

58 human and non-human subjects have shown that retrograde amnesia is extensive and can encompass much o

59 The results suggest that whether retrograde amnesia is temporally limited or very extensi

60 Retrograde amnesia is the most persistent cognitive adve

61 a brief recall results in a non-recoverable retrograde amnesia, known as reconsolidation blockade.

62 Furthermore, the results demonstrate that retrograde amnesia occurs as a result of subcortical dam

63 Here, we examine retrograde amnesia of contextual fear produced by DH les

64 Retrograde amnesia of learned associative memories is el

65 In particular, we examined the pattern of retrograde amnesia on an assessment of autobiographical

66 on, (b) showed that the effect is not due to retrograde amnesia produced by LiCl, and (c) confirmed t

67 The relationship between anterograde and retrograde amnesia remains unclear.

68 The Morris water maze (MWM) retrograde amnesia test was conducted on day 12 post-CCI

69 The PC lesions produced a significant retrograde amnesia that was not temporally graded.

70 inhibition with tatCN19o did not cause such retrograde amnesia that would pose a contraindication fo

71 ade amnesia and the temporal gradient of the retrograde amnesia were noted, 4 had a more severe retro

72 Furthermore, 7-NI produced retrograde amnesia when given immediately following trai

73 elatively greater degree of anterograde than retrograde amnesia, whereas damage to discrete regions o

74 Furthermore, MK-801 did not produce retrograde amnesia, whereas L-NAME did, suggesting that

75 isease (AD) initially show temporally graded retrograde amnesia, which gradually progresses into more

76 no temporal gradient and 2 had a less severe retrograde amnesia with a temporal gradient.

77 rade amnesia were noted, 4 had a more severe retrograde amnesia with no temporal gradient and 2 had a

78 We assessed retrograde amnesia with the Autobiographical Memory Inte