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1 uding causes such as axonal degeneration and retrograde degeneration.
2 ing axon protection in these acute models of retrograde degeneration.
3 d 90% of beta retinal ganglion cells die via retrograde degeneration.
4 reas undergoing secondary anterograde and/or retrograde degeneration.
5 he axon terminal that mediate proNGF-induced retrograde degeneration.
6 nt with a shared mechanism for Wallerian and retrograde degeneration.
7 of eyes with optic neuritis, suggesting that retrograde degeneration after optic neuritis may not ext
8 s, a surviving intact branch suppresses both retrograde degeneration and regeneration of the injured
9 e (LPZ, the sector of the LGN that underwent retrograde degeneration following a V1 lesion).
10              Serotonin axons undergo massive retrograde degeneration following amphetamine treatment
11 eing clarified in rodent models, focusing on retrograde degeneration following axonal damage, excitot
12  in this subtype than may be expected due to retrograde degeneration from either typical clinical or
13  nucleus; and (b) the lesions did not induce retrograde degeneration nor diminution of Fos label in t
14 ing of axons demonstrated that Wallerian and retrograde degeneration occurred synchronously.
15 lammatory process results in anterograde and retrograde degeneration of axons, leading to the tempora
16   Transection of the fimbria fornix leads to retrograde degeneration of axotomised septal cholinergic
17 r (proNGF) in the cerebral cortex results in retrograde degeneration of BFCNs through activation of p
18                Removal of the AOB results in retrograde degeneration of both Gi alpha 2-ir and G(o al
19                          This results in the retrograde degeneration of ganglion cells in the retina,
20                                Transneuronal retrograde degeneration of retinal ganglion cells after
21 e is experimental evidence of trans-synaptic retrograde degeneration of retinal ganglion cells follow
22 ization of visual pathways, including severe retrograde degeneration of retinal ganglion cells of the
23        For many years it has been known that retrograde degeneration of thalamic neurons occurs follo
24 tinal anatomy suggests that MME is caused by retrograde degeneration of the inner retinal layers, res
25         Postmortem histology revealed severe retrograde degeneration of the ipsilesional lateral geni
26 er bulbectomy, a manipulation known to cause retrograde degeneration of the mature ORNs.
27 d in diabetic neuropathy mice, likely due to retrograde degeneration of the Meissner-corpuscle innerv
28 mary retinal neuronopathy, in the absence of retrograde degeneration of the retinal nerve fibre layer
29 r to assess the time course of Wallerian and retrograde degeneration of unmyelinated retinal ganglion
30 t require a separate intervention to prevent retrograde degeneration of upper motoneurons in the cort
31               The existence of transsynaptic retrograde degeneration (TRD) in the human visual system
32  in actively demyelinating cortical lesions, retrograde degeneration was mainly related to demyelinat