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1 ate in cells after the 3' processing step of retroviral integration.
2  processes such as genetic recombination and retroviral integration.
3 ves viral end DNA during the initial step of retroviral integration.
4 y steps of transpositional recombination and retroviral integration.
5 roach to expand stem cells and to facilitate retroviral integration.
6 dentify the barriers and factors involved in retroviral integration.
7 that significantly decreases genotoxicity of retroviral integration.
8 able to catalyze the strand transfer step of retroviral integration.
9 hich ATR has been depleted are competent for retroviral integration.
10 ially informing future strategies to prevent retroviral integration.
11 effects on cellular growth, gene control and retroviral integration.
12 oove interactions within the LTR termini for retroviral integration.
13 eviously shown to have a role in mitosis and retroviral integration.
14 nd suggest that target site-selection limits retroviral integration.
15 act as host-supplied cofactors necessary for retroviral integration.
16 ng as host-supplied proteins that facilitate retroviral integration.
17 st cell protein that plays a crucial role in retroviral integration.
18 repair synthesis were required for efficient retroviral integration.
19 ion of segmental duplications, and bursts of retroviral integrations.
20                      INSTIs work by blocking retroviral integration; an essential step in the viral l
21 d fragment, a structure like that created by retroviral integration and all known transposition react
22 initial steps reflect a common theme seen in retroviral integration and prokaryotic transposition, an
23  several viral and host factors that promote retroviral integration and regulate integration targetin
24 1) preserves genomic integrity by preventing retroviral integration and retrotransposition during str
25 lly active DNA is not a preferred target for retroviral integration and that transcriptional activity
26  new questions about the role of histones in retroviral integration and transcription.
27 ransposition paved the way for understanding retroviral integration and V(D)J recombination as well a
28      Preintegration complexes (PICs) mediate retroviral integration, and recent results indicate an i
29 irally encoded integrase protein carries out retroviral integration, and to do so, it must make speci
30 e results suggest that the initial events in retroviral integration are detected as DNA damage by the
31 e DNA-breaking and -joining steps initiating retroviral integration are well understood, but the late
32     Here, we develop a biophysical model for retroviral integration as stochastic and quasi-equilibri
33           Herein, I review the mechanisms of retroviral integration as well as the promises and chall
34                        Our results show that retroviral integrations at intergenic sites can mark and
35 has been proposed to play a role late during retroviral integration, because infection by human immun
36 irus, apparently reflecting an occurrence of retroviral integration by homologous recombination.
37                                     However, retroviral integration can take place at a variety of ch
38 roviral integration sites to common sites of retroviral integration (CISs).
39 fundamental differences in the way different retroviral integration complexes interact with host-cell
40  intasome is the basic recombination unit of retroviral integration, comprising the integrase protein
41 ealization that proto-oncogene activation by retroviral integration could contribute to leukemia.
42  dependent on NHEJ, a 5-10-fold reduction in retroviral integration efficiency was observed in Brca1(
43  MTAP exons arose from early and independent retroviral-integration events in primate genomes at leas
44  advances in understanding the mechanisms of retroviral integration, focusing on LEDGF/p75--the first
45                                              Retroviral integration has been implicated in several bi
46                                              Retroviral integration has far-reaching consequences, fr
47 ibed in vitro assays to define inhibitors of retroviral integration has not been formerly demonstrate
48                      Factors contributing to retroviral integration have been intractable because pas
49    Here we show that Evi24, a common site of retroviral integration in AKXD B cell and BXH-2 myeloid
50  we showed that Evi3 is a frequent target of retroviral integration in AKXD27 B-cell lymphomas.
51 ferred hG6PD driven by these promoters after retroviral integration in bulk cultures and in individua
52 ied through its location at a common site of retroviral integration in BXH2 murine myeloid leukemias.
53 and either Hoxa7 or Hoxa9 are coactivated by retroviral integration in BXH2 murine myeloid leukemias.
54                    Evi27 is a common site of retroviral integration in BXH2 murine myeloid leukemias.
55  not strictly required for growth control or retroviral integration in DT40 cells and may well be red
56 entifies genomic regions that are targets of retroviral integration in more than one tumor (common in
57 dies identified 152 loci that are targets of retroviral integration in more than one tumor (common re
58      Hhex is one of the most common sites of retroviral integration in mouse models.
59  is an oncogene whose upregulation following retroviral integration in murine B cells leads to an arr
60 Meis1 and Hoxa9 expression is upregulated by retroviral integration in murine myeloid leukemias and i
61                     Evi5 is a common site of retroviral integration in T-cell lymphomas of AKXD mice.
62 lls, and that there is no apparent defect in retroviral integration in the absence of HMGA1 or HMGA2.
63  and HMGA2 have also been found to stimulate retroviral integration in vitro.
64                                              Retroviral integration in vivo is mediated by preintegra
65 rsensitivity, and Southern blot analysis for retroviral integration indicated that the disease was ol
66                         In the first step of retroviral integration, integrase cleaves the linear vir
67  that H2AX is not required for repair of the retroviral integration intermediate as determined by sta
68 ap and 5' two-base flap structure present in retroviral integration intermediates and tested candidat
69  infection in resting CD4+ T cells is due to retroviral integration into chromosomal regions that are
70  studies of chromosomal lesions arising from retroviral integration into human compared with schistos
71                                        Thus, retroviral integration into nucleosomes involves the loo
72 ications play an outsized role in regulating retroviral integration into specific regions of the host
73                                        Avian retroviral integration into the c-myb locus is casually
74  T-cell activation is required for effective retroviral integration into the host cell genome and sub
75                                              Retroviral integration into the host genome is not entir
76                              The location of retroviral integration into the human genome is thought
77                                              Retroviral integration involves cleavage of the host DNA
78 n of a suitable chromatin environment during retroviral integration is a tightly regulated process.
79 at repair of these gaps flanking the site of retroviral integration is achieved by host DNA repair ma
80 en the PIC and host nucleosomes that promote retroviral integration is an understudied area.
81                                              Retroviral integration is catalysed by a tetramer of int
82                                     In vivo, retroviral integration is mediated by a large nucleoprot
83                                              Retroviral integration is mediated by a preintegration c
84                                              Retroviral integration is mediated by viral preintegrati
85                                    Increased retroviral integration is the first major phenotype desc
86                                              Retroviral integration, like all forms of DNA transposit
87 s into the organization and mechanism of the retroviral integration machinery and highlight open ques
88    A total of 26% (11 of 42) of the mice had retroviral integrations near Zeb2, a transcriptional cor
89  we present the analysis of a common site of retroviral integration on mouse chromosome 15, which inc
90                              Detecting novel retroviral integrations (ones not in the reference genom
91 step has never been investigated, either for retroviral integration or for any other transposition pr
92                                              Retroviral integration proceeds via two integrase activi
93 talytic strand transfer intermediates of the retroviral integration process.
94 st-replication/translesion DNA repair in the retroviral integration process.
95                                              Retroviral integration protein (IN) has been shown to be
96 ng T cells, generating the substrate for the retroviral integration reaction.
97 e-specific recombination, transposition, and retroviral integration reactions involve the collaborati
98                                              Retroviral integration results in the stable and coordin
99                  Here, we show that a common retroviral integration site (RIS) in AKXD B-cell lymphom
100                                              Retroviral integration site analysis in 4 animals reveal
101 line determines the genomic position of each retroviral integration site cloned from a mouse tumor, t
102                    The bic locus is a common retroviral integration site in avian leukosis virus (ALV
103   bic is a novel gene identified at a common retroviral integration site in avian leukosis virus-indu
104  activated by promoter insertion at a common retroviral integration site in B cell lymphomas induced
105     Molecular analysis demonstrated a common retroviral integration site in clonogenic hematopoietic
106      The chromosomal features that influence retroviral integration site selection are not well under
107 a that integrase is the major determinant of retroviral integration site selection.
108 usly unappreciated physical contributions to retroviral integration site selection.
109 ric (split) reads that span a putative novel retroviral integration site.
110                                          The retroviral integration sites (RISs) in these tumors thus
111 promotes transient formation of gammaH2AX at retroviral integration sites as detected by both immunoc
112 polymorphism (SNP)-based method, for mapping retroviral integration sites cloned from mouse tumors, a
113                           Clonal analysis of retroviral integration sites confirms a common stem cell
114 We used inverse PCR to clone and analyze 212 retroviral integration sites from 43 MZL at different st
115  these mutations occur in the same intron as retroviral integration sites in gene therapy-induced T-A
116                      Large-scale analysis of retroviral integration sites in these tumors revealed a
117 udies designed to determine whether specific retroviral integration sites might be associated with a
118 ) hybridization method, for localizing these retroviral integration sites to common sites of retrovir
119 l integration in more than one tumor (common retroviral integration sites, CISs) and therefore likely
120           The common interruption of p101 by retroviral integration suggests that the locus encodes a
121                               To investigate retroviral integration targeting on a nucleotide scale,
122              Some of the earliest studies of retroviral integration targeting reported that sites of
123 roglia-neuronal interactions in HIV and link retroviral integration to remodeling of the microglial 3
124                     BET proteins guide gamma-retroviral integration to transcription start sites and
125                     We cloned clone-specific retroviral integrations to identify candidate resistance
126     The selection of chromosomal targets for retroviral integration varies markedly, tracking with th
127                     No evidence of oncogenic retroviral integration was found with the use of multipl
128         To investigate the role of PARP-1 in retroviral integration, we infected wild-type and PARP-1
129 sposon, retrovirus-like retrotransposon, and retroviral integration, we propose a nonviral system tha
130   In an effort to examine the role of ATR in retroviral integration, we used RNA interference to sele
131 irally encoded integrase protein carries out retroviral integration, which requires specific interact
132     However, understanding of the biology of retroviral integration will require in vitro and in vivo
133               Molecular analysis infers that retroviral integration within Ikzf1 is an early event in
134 curred before the initial hydrolysis step of retroviral integration, work in the related Tn10 and V(D

 
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