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1 levels of IAP transcripts, Gag proteins, and reverse transcription products.
2  viral entry, before the generation of viral reverse transcription products.
3  in the ability of MLV particles to generate reverse transcription products.
4  approximate size expected for a full-length reverse transcription product and with plus-strand stron
5 G20K/R drastically decreased wild-type HIV-1 reverse transcription products and infectivity.
6 esult in decreased amounts of early and late reverse transcription products and integrated virus rela
7 ment three-way junction structure diminished reverse transcription products and led to reduced viral
8 at A3G exosomes reduce accumulation of HIV-1 reverse transcription products and steady-state levels o
9  sequencing strategy to characterize nascent reverse transcription products and their precise 3'-term
10 nfection, results in reduced accumulation of reverse transcription products, and is dominant in heter
11                 The standard is added to the reverse transcription product before PCR.
12 d PBMCs and MDDCs revealed similar levels of reverse transcription products, but increased nuclear im
13 troviruses by preventing the accumulation of reverse transcription products, but the underlying mecha
14  cells revealed that the generation of early reverse transcription products coincides with the timing
15 but not integration, indicating that a viral reverse transcription product comprises the HIV-1 pathog
16        All mutants tested produced levels of reverse transcription products either similar to or only
17                               Finally, HIV-1 reverse transcription products escaping rhesus TRIM5alph
18 d allows the normal generation of HIV-1 late reverse transcription products, even though HIV-1 infect
19 t early after virion entry and prevent viral reverse transcription products from accumulating.
20 ucin gene was determined by real-time PCR on reverse transcription products from RNA isolated from hu
21 esistant to other cytosolic sensors of viral reverse transcription products in newly infected cells.
22                                  Analysis of reverse transcription products in OMK cells revealed tha
23 ral core, prior to the accumulation of viral reverse transcription products in the target cell.
24 ome inhibition, prevents the accumulation of reverse transcription products in the target cell.
25 ar mass aggregates promote synthesis of long reverse transcription products in vitro by concentrating
26                             Other classes of reverse transcription products, including some that resu
27 Quantitative real-time PCR analysis of early reverse transcription products indicated that HIV-1 reve
28 64)K variant was impaired in generating late reverse transcription products, indicating that replicat
29 position, and mediate the integration of the reverse-transcription product into the genome of the hos
30 tion in multiple ways and that inhibition of reverse transcription products is not necessary for TRIM
31 s and that synthesis of long double-stranded reverse transcription products is required to trigger ef
32                  For this study, we examined reverse transcription products of human immunodeficiency
33 tion, quantitative real-time PCR analysis of reverse transcription products revealed that the mutant
34                     Detailed analyses of the reverse transcription products synthesized within nucleo
35 unodeficiency virus type 1 (HIV-1), allowing reverse transcription products to accumulate even though
36 HIV replication, including synthesis of late reverse transcription products, two-long terminal repeat
37 antitative methods that monitor formation of reverse transcription products, two-LTR circles and inte
38 photonic microring resonators to detect cDNA reverse transcription products via a subsequent enzymati
39                                     However, reverse transcription products were substantially increa
40  results indicate that faithful, full-length reverse transcription products were underrepresented in
41 24 production and quantitative PCR for HIV-1 reverse transcription products, whereas treatment of the
42 quantitative real-time PCR analysis of early reverse transcription products, with initiation at the H
43 mRNA synthesis.IMPORTANCE The fates of HIV-1 reverse transcription products within infected cells are
44 acellular viral DNA as well as intracellular reverse transcription products, without affecting HBV RN