ライフサイエンスコーパス: rhinovirus

1 primary leucocytes following treatment with rhinovirus.

2 duced in epithelial cells by IL-13 and human rhinovirus.

3 tible to acute wheezing episodes provoked by rhinovirus.

4 ally prolonged, especially for bocavirus and rhinovirus.

5 in allergic asthmatic subjects infected with rhinovirus.

6 asynchronous circulation of influenza A and rhinovirus.

7 ild asthma were experimentally infected with rhinovirus.

8 up of 45- to 64-year-olds were attributed to rhinovirus.

9 tory syndrome (SARS), coxsackie viruses, and rhinoviruses.

10 ion of human respiratory syncytial virus and rhinoviruses.

11 ement among ICAM-1-binding enteroviruses and rhinoviruses.

12 eroviruses, it seems to occur more rarely in rhinoviruses.

13 enteroviruses but appears to be sporadic in rhinoviruses.

14 .7%) tested for other respiratory pathogens, rhinovirus (107 [12.8%]), adenovirus (64 [6.0%]), respir

15 diates the replication of enterovirus 68 and rhinovirus 16.

16 enterovirus 68 but disadvantageous to human rhinovirus 16.

17 the RH-dependent survival of airborne human rhinovirus-16 (HRV-16).

18 (mepolizumab), followed by a challenge with rhinovirus-16 (RV16).

19 response to an experimental inoculation with rhinovirus-16 among asthmatics with high levels of total

20 response to an experimental inoculation with rhinovirus-16 in allergic asthmatics with the response i

21 ) beta was administered with fluticasone and rhinovirus 1B in both groups of mice.

22 IFN beta (10(4) units) with fluticasone and rhinovirus 1B led to upregulation of interferon-inducibl

23 lfoxide, control), 1 h before infection with rhinovirus 1B.

24 ytic degradation of eIF4G alone by the human rhinovirus 2A protease abrogated this translation strate

25 Respiratory syncytial virus (83%) and human rhinovirus (34%) were most commonly detected.

26 or adenovirus (100%), metapneumovirus (44%), rhinovirus (34%), and parainfluenza virus type 3 (28%);

27 ad life-threatening disease, 575 (24.8%) had rhinovirus, 347 (14.9%) had adenovirus (ADV), and 30 (1.

28 < .01), Coxsackieviruses (aOR 5.1, P = .09), rhinovirus A (aOR 3.5, P = .34), and rhinovirus C (aOR 2

29 on of rhinovirus C (OR, 2.85; P < 0.001) and rhinovirus A (OR, 2.92; P < 0.001), as well as, to a les

30 Closely related variants of rhinovirus A or B were identified in 2 to 4 children fro

31 We have shown that rhinovirus, a cause of asthma exacerbation, colocalizes

32 Infants with rhinovirus-A were more likely to have Haemophilus-domina

33 The severity and rates of rhinovirus acute respiratory illnesses (ARIs) in adults

34 We identified the seasonality of rhinovirus, adenovirus, influenza A and B viruses, human

35 justed OR, 4.8; P = .014), and wheezing with rhinovirus (adjusted OR, 5.0; P = .035).

36 ma, early-life exposure to tobacco smoke and rhinovirus aetiology were early-life risk factors for la

37 ive samples; this risk was also present with rhinovirus alone (aHR for mortality, 2.6; 95% CI, 1.2-5.

38 th control mice treated with fluticasone and rhinovirus alone and improved viral clearance without ha

39 tection when feasible, even for detection of rhinovirus alone.

40 uch as respiratory syncytial virus (RSV) and rhinovirus, also seemed to have an impact on IPD: RSV co

41 ng immune mechanisms and interaction between rhinovirus and allergy remain enigmatic, and current par

42 e asthma AMs and was associated with reduced rhinovirus and imiquimod-induced IFN responses by these

43 zumab treatment were stimulated ex vivo with rhinovirus and influenza in the presence or absence of I

44 l/throat swabs were collected and tested for rhinovirus and other viruses by using RT-PCR.

45 y chemokine responses of airway epithelia to rhinovirus and viral mimics and decreased nuclear transl

46 xperimentally the recombination potential of rhinoviruses and analyze recombination sites.

47 Interspecies recombination is even rarer for rhinoviruses and mostly is related to ancient events whi

48 infection by a broad panel of EVs, including rhinoviruses and non-polio EVs increasingly linked to se

49 During wheezing illnesses, detection of rhinoviruses and predominance of Moraxella were associat

50 ole of specific respiratory viruses, such as rhinoviruses and respiratory syncytial virus, in asthma

51 5 (1 of the 7 naturally pleconaril-resistant rhinoviruses) and OBR-5-340, a bioavailable pyrazolopyri

52 detections of parainfluenza, coronaviruses, rhinovirus, and adenovirus, especially in children, requ

53 iral effect against hepatitis C virus, human rhinovirus, and coxsackievirus B3.

54 l virus, parainfluenza viruses, coronavirus, rhinovirus, and human metapneumovirus, represent a consi

55 on with adenovirus, coronavirus, enterovirus/rhinovirus, and influenza virus (P = .062-.093), while m

56 for early inflammatory responses induced by rhinovirus, and TLR2(+) macrophages are sufficient to co

57 Omalizumab improved IFN-alpha responses to rhinovirus, and within the omalizumab group, greater IFN

58 Omalizumab treatment increased rhinovirus- and influenza-induced PBMC and rhinovirus-in

59 Minor-group rhinoviruses are more immunogenic in laboratory studies,

60 on in respiratory viral infection.IMPORTANCE Rhinoviruses are one of the causes of the common cold.

61 Rhinoviruses are the most common causes of the common co

62 ease, viral infections, including those with rhinovirus, are the commonest cause of exacerbations.

63 umans, including poliovirus, coxsackievirus, rhinovirus, as well as newly emerging global health thre

64 eas additional effort is needed in regard to rhinovirus, as well as other respiratory tract viruses.

65 We sought to determine rhinovirus-associated ARI rates in adults presenting for

66 can reduce the likelihood of presenting with rhinovirus-associated ARIs.

67 There were 7 rhinovirus-associated ED visits and 3 hospitalizations p

68 mouse airway epithelial cells infected with rhinovirus at 33 degrees C vs. 37 degrees C.

69 P < 0.001), as well as, to a lesser extent, rhinovirus B (OR, 1.98; P = 0.019).

70 za viruses, respiratory syncytial virus, and rhinoviruses being the most frequent and with the highes

71 One vaccinee had grade 2 wheezing with rhinovirus but without concurrent LID/DeltaM2-2/1030s sh

72 ignaling drove the impaired IFN responses to rhinovirus by asthmatic alveolar macrophages (AMs); the

73 = .09), rhinovirus A (aOR 3.5, P = .34), and rhinovirus C (aOR 2.9, P = .57).

74 ingle-stranded RNA poliovirus (PV) and human rhinovirus C (HRV-C).

75 e significantly associated with detection of rhinovirus C (OR, 2.85; P < 0.001) and rhinovirus A (OR,

76 Isolates of rhinovirus C (RV-C), a recently identified Enterovirus (

77 Infection by Rhinovirus-C (RV-C), a species of Picornaviridae Enterov

78 r which underlies variable susceptibility to rhinovirus-C infection and is associated with severe chi

79 ominant microbiota profile, while those with rhinovirus-C were more likely to have Moraxella-dominant

80 infection of the respiratory epithelium with rhinovirus can antagonize tolerance to inhaled antigen t

81 Rhinoviruses cause the common cold and exacerbations of

82 se, IL-33 levels were persistently high, and rhinovirus challenge exacerbated the type 2 inflammatory

83 ls were precisely monitored before and after rhinovirus challenge in allergic asthmatic subjects (tot

84 ere we devised a strategy based on the polio:rhinovirus chimera PVSRIPO, devoid of viral neuropathoge

85 raspecies recombination gives rise to viable rhinovirus chimeras in the polyprotein coding region and

86 Rhinovirus, common human coronaviruses, and respiratory

87 Patients with rhinovirus, compared with virus-negative ARI, were more

88 The 25 children with greater than 7000 rhinovirus copies/mL (most sensitive cutoff) benefitted

89 human metapneumovirus, adenovirus, and human rhinoviruses, coronaviruses, and bocavirus.

90 Multiple infections with nearly identical rhinoviruses could be detected within each village, like

91 f infection by picornaviruses, such as human rhinovirus, coxsackievirus, poliovirus, foot-and-mouth d

92 ettings and identify factors associated with rhinovirus detection.

93 ions were usually symptomatic; bocavirus and rhinovirus detections were often asymptomatic.

94 compound that had been developed as an anti-rhinovirus drug.

95 Vitamin D supplementation may have an anti-rhinovirus effect.

96 cies rhinovirus/rhinovirus or rhinovirus/non-rhinovirus enterovirus recombinants are fully viable.

97 with increasing age and infection with RSV, rhinovirus, enterovirus, adenovirus, and bocavirus.

98 all detected respiratory viruses followed by rhinovirus/enterovirus (13%), influenza virus (12%), cor

99 he most common viruses detected by BFPP were rhinovirus/enterovirus (4.5%), influenza A virus (3%), a

100 ew York, from 2014 to 2018 were examined for rhinovirus/enterovirus (RhV/EV) by the FilmArray respira

101 re [ARF]), 21.3% had a virus detected (56.4% rhinovirus/enterovirus and 30.7% influenza/parainfluenza

102 Positive rhinovirus/enterovirus FA targets revealed patterns loos

103 ay (FA) respiratory panel was used to detect rhinovirus/enterovirus in respiratory specimens; suspect

104 arainfluenza virus 3, parainfluenza virus 4, rhinovirus/enterovirus, respiratory syncytial virus A an

105 ytes: coronaviruses 229E, NL63, and OC43 and rhinovirus/enterovirus.

106 ombination is described frequently among non-rhinovirus enteroviruses but appears to be sporadic in r

107 ombination is described frequently among non-rhinovirus enteroviruses, it seems to occur more rarely

108 Rhinovirus frequently causes asthma exacerbations among

109 After confirming rhinovirus from nasopharyngeal aspirate by using PCR, 79

110 At study entry, viral etiology, rhinovirus genome load, atopic and clinical characterist

111 Here, we show that human rhinovirus (HRV) 3A also recruited PI4KIIIbeta to replic

112 Respiratory syncytial virus (RSV) and human rhinovirus (HRV) are the most common viruses associated

113 The pathogenesis of human rhinovirus (HRV) during severe respiratory disease remai

114 First, infection of HeLa cells with human rhinovirus (HRV) induced the phosphorylation of PKD.

115 d whole blood samples during natural RSV and rhinovirus (hRV) infection (acute versus early recovery

116 Human rhinovirus (HRV) infection is a common trigger for child

117 Airway Mycoplasma pneumoniae and human rhinovirus (HRV) infections are linked to neutrophilic i

118 Rationale: Human rhinovirus (HRV) is a common cause of chronic obstructiv

119 Human Rhinovirus (HRV) is a major cause of common cold, bronch

120 Human rhinovirus (hRV) is frequently detected in the upper res

121 aetiological agent of the common cold, human rhinovirus (HRV) is the leading cause of human infectiou

122 nfections, caused by viruses including human rhinovirus (HRV), influenza virus, and respiratory syncy

123 DHR3) protein serves as a receptor for human rhinovirus (HRV)-C.

124 disease severity and the frequency of human rhinovirus (HRV)-initiated exacerbations.

125 ts) delineating the natural history of human rhinovirus (HRV).

126 tients were exposed to bacteria and/or human rhinovirus (HRV).

127 ith acute respiratory infection due to human rhinovirus (HRV; n = 52) or respiratory syncytial virus

128 Human rhinoviruses (HRV) comprise 3 species representing more

129 Human rhinoviruses (HRVs) are a major trigger of asthma exacer

130 Human rhinoviruses (HRVs) are one of the main causes of virus-

131 Human rhinoviruses (HRVs) commonly precipitate asthma exacerba

132 Human rhinoviruses (HRVs), human enteroviruses (HEVs) and huma

133 teristics and clinical significance of human rhinoviruses, human coronaviruses, human metapneumovirus

134 events with influenza virus, coronaviruses, rhinoviruses, human metapneumovirus, respiratory syncyti

135 at the 17q21 genetic locus who wheeze during rhinovirus illnesses have a greatly increased likelihood

136 tions, a mechanism through which IgE impairs rhinovirus immunity and underlies asthma exacerbations w

137 (respiratory syncytial virus in 51.4%, human rhinovirus in 11.7%).

138 ects of omalizumab on IFN-alpha responses to rhinovirus in PBMCs were examined.

139 l human coronaviruses, influenza viruses and rhinoviruses in exhaled breath and coughs of children an

140 The most common pathogens were human rhinovirus (in 9% of patients), influenza virus (in 6%),

141 In animal models of AAD and rhinovirus-induced AAD exacerbations, pDCs were recruite

142 he magnitude of type 2 inflammation during a rhinovirus-induced acute exacerbation; however, only ant

143 ncreasing viral loads, fluticasone inhibited rhinovirus-induced airway inflammation as evidenced by s

144 macrophages are required and sufficient for rhinovirus-induced airway inflammation in vivo.

145 wild-type, OVA-treated C57BL/6 mice blocked rhinovirus-induced airway responses, whereas transfer of

146 the requirement and sufficiency of TLR2 for rhinovirus-induced airway responses.

147 uggest that fluticasone treatment suppresses rhinovirus-induced airways inflammation in vivo but also

148 The role of type I interferon and PPRs in rhinovirus-induced asthma exacerbations in vivo are unce

149 and bronchoalveolar lavage before and after rhinovirus-induced asthma exacerbations.

150 y represent potential therapeutic targets of rhinovirus-induced asthma exacerbations.

151 ndings indicate that the regulation of human rhinovirus-induced B-cell responses could be a novel app

152 We have also shown that rhinovirus-induced cytokine expression is abolished in T

153 etermine the relationship between ORMDL3 and rhinovirus-induced ER stress and type I IFN in human leu

154 ociation between higher ORMDL3 and increased rhinovirus-induced HSPA5 and type I IFN gene expression.

155 es and transcription factors involved in the rhinovirus-induced interferon production and inflammator

156 rfactant protein C expression and suppressed rhinovirus-induced IP-10 secretion, which was associated

157 After allergen exposure, rhinovirus-induced neutrophilic and eosinophilic airway

158 Rhinovirus-induced ORMDL3 expression in primary leucocyt

159 The degree of rhinovirus-induced ORMDL3, HSPA5 and IFNB1 expression va

160 d rhinovirus- and influenza-induced PBMC and rhinovirus-induced pDC IFN-alpha responses in the presen

161 The interaction between allergen and rhinovirus-induced symptoms and inflammation over time i

162 sufficient to recapitulate many features of rhinovirus-induced type-2 immune responses and asthma pa

163 ment of the first acute, moderate-to-severe, rhinovirus-induced wheezing episode in young children.

164 Rhinovirus-induced wheezing is an important risk factor

165 ecific monoclonal antibody is available; (b) rhinovirus-induced wheezing, associated with atopic pred

166 Rhinovirus induces robust T(H)1 responses in allergic as

167 , and SOCS1-deficient mice, SOCS1 suppressed rhinovirus induction of interferons.

168 t-viral-challenge state resembled more other rhinovirus-infected asthmatics than their own pre-viral-

169 The latter phenotype was replicated in rhinovirus-infected human AECs, suggesting that anti-IL-

170 among asymptomatic controls with presence of rhinovirus infection (OR:4.23; 95%CI 1.16-14.22, p<0.05)

171 depletion after allergen challenge or during rhinovirus infection abrogated exacerbation of inflammat

172 , which was induced by allergen challenge or rhinovirus infection and conditioned pDCs for proinflamm

173 with exacerbation as the primary outcome and rhinovirus infection and respiratory illnesses as second

174 ent study collected individual data on human rhinovirus infection and sensitization to Alternaria and

175 f host double-stranded DNA (dsDNA) following rhinovirus infection and the exacerbation of type-2 alle

176 Rhinovirus infection at an early age has been associated

177 daptive capacity (AC) during an experimental rhinovirus infection in 24 healthy and asthmatic human v

178 ue of the lungs has been demonstrated during rhinovirus infection in allergic individuals.

179 ron and PRR expression at baseline and after rhinovirus infection in atopic asthmatic patients and co

180 asthma exacerbation-related cytokines and by rhinovirus infection in vitro.

181 effects of inhaled fluticasone propionate on rhinovirus infection in vivo, in a mouse model.

182 ociated with adverse clinical outcomes after rhinovirus infection in vivo.

183 nd polyinosinic-polycytidylic acid and human rhinovirus infection induced a potent antiviral protecti

184 Rhinovirus infection induced an amplified antiviral T(H)

185 Rhinovirus infection induced Il25, Il33, Il4, Il5, Il13,

186 sociated with viral load during experimental rhinovirus infection of asthmatic patients.

187 tory T (Treg) cells, and we examined whether rhinovirus infection of the respiratory tract can block

188 Rhinovirus infection triggers acute asthma exacerbations

189 lergic airway hypersensitivity, we show that rhinovirus infection triggers dsDNA release associated w

190 ma relate to the likelihood of exacerbation, rhinovirus infection, and respiratory illnesses during t

191 SV respiratory virus infection, asymptomatic rhinovirus infection, and virus-negative asymptomatic co

192 Following experimental rhinovirus infection, we observed increased respiratory

193 fficient for neutrophilic inflammation after rhinovirus infection, whereas macrophages treated with I

194 Rhinovirus infection-increased numbers of subepithelial

195 asthma hospitalizations independent of human rhinovirus infection.

196 y subjects and treated with anti-IL-33 after rhinovirus infection.

197 ness, and reductions in lung function during rhinovirus infection.

198 pithelial PRR expression were induced during rhinovirus infection.

199 -dominated assemblages increased the risk of rhinovirus infection.

200 s at baseline and on day 4 and 6 weeks after rhinovirus infection.

201 and non-respiratory syncytial virus (mostly rhinovirus) infection; profile B (49%), which has the la

202 Rhinovirus infections are the dominant cause of asthma e

203 ene-environment interactions (GEIs), such as rhinovirus infections, will improve detection of asthma

204 infection by RSV group B, and no prior human rhinovirus infections.

205 Asthma exacerbations are triggered by rhinovirus infections.

206 sociated with development of asthma, but how rhinovirus influences the immune response is not clear.

207 This technology successfully enriched rhinovirus, influenza virus, and parainfluenza viruses,

208 ted with inherent deficient IFN responses to rhinovirus, inhaled corticosteroids might interact syner

209 educed among allergic asthmatics early after rhinovirus inoculation but increased late in the infecti

210 To further assess the rhinovirus intra- and interspecies recombination potenti

211 Rhinovirus is associated with a substantial number of ED

212 Rhinovirus is linked to asthma exacerbations and chronic

213 The primary interaction analysis examined rhinovirus load.

214 how the interaction of increased ORMDL3 and rhinovirus may predispose to asthma.

215 anation for how different types (strains) of rhinoviruses may elicit different cell responses that di

216 43/NL63/HKU1/SARS/MERS), human enteroviruses/rhinoviruses, measles virus, mumps virus, Hepatitis A-E

217 espiratory syncytial virus (RSV), influenza, rhinovirus, metapneumovirus, and adenovirus was highly a

218 3.4%); picornavirus (enteroviruses [n = 14], rhinovirus [n = 5], and parechovirus [n = 2]), hepatitis

219 were calculated and rhinovirus-positive and rhinovirus-negative patients were compared.

220 the first severe respiratory syncytial virus/rhinovirus-negative wheezing episode (adjusted OR, 8.0;

221 (UTR) interspecies rhinovirus/rhinovirus or rhinovirus/non-rhinovirus enterovirus recombinants are f

222 ected and examined for the presence of human rhinovirus or enterovirus using the FilmArray Respirator

223 ry virus types (respiratory syncytial virus, rhinoviruses, other picornaviruses, coronaviruses 229E a

224 ildren was 12 months (SD 6.0), 57 (75%) were rhinovirus positive, and 22 (30%) were sensitized.

225 its and hospitalizations were calculated and rhinovirus-positive and rhinovirus-negative patients wer

226 m severely ill patients nationwide, who were rhinovirus-positive or enterovirus-positive in hospital

227 ICAM-1 (intercellular adhesion molecule 1) (rhinovirus receptor as a comparator) in sputum cells fro

228 with high frequency, whereas no interspecies rhinovirus recombinants could be recovered.

229 s regarding the mechanism and limitations of rhinovirus recombination.

230 l approaches to improve current knowledge on rhinovirus recombination.

231 ings demonstrate that in mouse airway cells, rhinovirus replicates preferentially at nasal cavity tem

232 Anti-IL-33 decreased rhinovirus replication and increased IFN-lambda levels a

233 Rhinovirus replication was higher and IFN-lambda levels

234 ed 5' untranslated region (UTR) interspecies rhinovirus/rhinovirus or rhinovirus/non-rhinovirus enter

235 ks after which patients were challenged with rhinovirus (RV) 16.

236 and III production associated with enhanced rhinovirus (RV) and respiratory syncytial virus (RSV) re

237 OXO3a and play an important role in clearing rhinovirus (RV) by mounting antiviral type I and type II

238 The association of rhinovirus (RV) detection to illness is poorly understoo

239 would shorten the frequency and duration of rhinovirus (RV) illnesses in children with allergic asth

240 Mice were infected intranasally with rhinovirus (RV) immediately after O2 exposure.

241 ay epithelial cells are the major target for rhinovirus (RV) infection and express proinflammatory ch

242 Rhinovirus (RV) infection in asthma induces varying degr

243 Rhinovirus (RV) infection of 6-d-old mice, but not matur

244 Young children with rhinovirus (RV) infection-particularly bronchiolitis-are

245 Rhinovirus (RV) infections are closely linked to exacerb

246 Rhinovirus (RV) infections are common and have the poten

247 B cells play a crucial role during rhinovirus (RV) infections by production of virus-neutra

248 Data are scarce on the role of rhinovirus (RV) infections in this patient group.

249 Some children with rhinovirus (RV) infections wheeze, but it is unknown whe

250 ntiviral drugs to treat enterovirus (EV) and rhinovirus (RV) infections, which can be severe and occa

251 Producing rhinovirus (RV) inocula from nasal secretions has requir

252 Rhinovirus (RV) is a major cause of common cold and an i

253 ng-associated respiratory tract infection by rhinovirus (RV) is a risk factor for asthma development.

254 -associated respiratory infection with human rhinovirus (RV) is associated with asthma development.

255 in causative agent of bronchiolitis, whereas rhinovirus (RV) is most commonly detected in wheezing ch

256 y-life respiratory syncytial virus (RSV) and rhinovirus (RV) lower respiratory tract infections (LRTI

257 Rhinovirus (RV) recognition by pattern recognition recep

258 Early life rhinovirus (RV) wheezing illnesses and aeroallergen sens

259 an metapneumovirus (HMPV), adenovirus (AdV), rhinovirus (RV), bocavirus (BoV), and coronavirus (CoV).

260 ined as respiratory syncytial virus (RSV) or rhinovirus (RV), including coinfections with other virus

261 Most strains of rhinovirus (RV), the common cold virus, replicate better

262 FN-beta and IFN-lambda1) induction following rhinovirus (RV)-16 infection.

263 mucosal inflammation following experimental rhinovirus (RV)-16-induced COPD exacerbations and its re

264 TLR2 activation contributes to limitation of rhinovirus (RV)-induced IFN response in the airway epith

265 The RNA rhinoviruses (RV) encode 2A proteases (2A(pro)) that con

266 , target conserved regions in the capsids of rhinoviruses (RVs) and enteroviruses (EVs) by binding to

267 Rhinoviruses (RVs) are a major cause of symptomatic resp

268 Human rhinoviruses (RVs) are positive-strand RNA viruses that

269 Infections with human rhinoviruses (RVs) are responsible for millions of commo

270 Rhinoviruses (RVs) are the main cause of the common cold

271 Rhinoviruses (RVs) are ubiquitous respiratory pathogens

272 Human rhinoviruses (RVs) of the A, B, and C species are define

273 asthma have heightened antibody responses to rhinoviruses (RVs), although those specific for RV-C are

274 Rhinoviruses (RVs), respiratory enteroviruses (EVs), inf

275 irulence.IMPORTANCE Most colds are caused by rhinoviruses (RVs).

276 a preparation of a common cold virus (human rhinovirus serotype 2, HRV-A2) and (ii) the analysis of

277 may contribute to the very large numbers of rhinovirus serotypes that coexist while differing in vir

278 a subset known as the minor-group members of rhinovirus species A (RV-A) are correlated with the ince

279 Rhinovirus species and type were determined by partial g

280 such as enterovirus species groups A to H or rhinovirus species groups A to C.

281 Among 774 infants with severe bronchiolitis, rhinovirus species related to distinct nasopharyngeal mi

282 lyprotein region between members of the same rhinovirus species, but not between members of different

283 ay did not detect any other enteroviruses or rhinoviruses tested and did detect divergent strains of

284 Most isolates of human rhinovirus, the common cold virus, replicate more robust

285 arly and later life and then challenged with rhinovirus to model disease onset, progression, and chro

286 New evidence indicates that rhinovirus-triggered asthma exacerbations become more se

287 We show that serum nAb against many rhinovirus types can be induced by polyvalent, inactivat

288 pes.IMPORTANCE Genetic variation among human rhinovirus types includes unexpected diversity in the ge

289 Rhinovirus was associated with CAP among adults (AF, 0.9

290 ature-dependent IFN induction in response to rhinovirus was dependent on the MAVS protein, a key sign

291 Among 2351 enrollees, rhinovirus was detected in 247 (11%).

292 Rhinovirus was detected in 74% (n = 80/108) of the child

293 Rhinovirus was detected most often; multiple viruses wer

294 onal impact of other viruses such as RSV and rhinovirus was instead more important in the older popul

295 administering omalizumab on the response to rhinovirus was most pronounced during the early/innate p

296 Rhinovirus was the most frequently detected RV (42%).

297 In contrast to most other rhinoviruses, which bind intercellular adhesion molecule

298 factor that is required for cytotoxicity by rhinoviruses, which cause the common cold.

299 monocytes overexpressing ORMDL3 responded to rhinovirus with increased IFNB1 and HSPA5.

300 ailable assays that detect enteroviruses and rhinoviruses without distinguishing between the two, inc