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1 ading frames required for programmed -1 mRNA ribosomal frameshifting.
2 1) regulates the efficiency of programmed -1 ribosomal frameshifting.
3 eing a truncated version of tau arising from ribosomal frameshifting.
4 nce of a novel structure that can facilitate ribosomal frameshifting.
5 oacyl synthetase recognition, and programmed ribosomal frameshifting.
6 requirements and mechanism of programmed -1 ribosomal frameshifting.
7 promote significant levels of programmed -1 ribosomal frameshifting.
8 rocess may also have an impact on programmed ribosomal frameshifting.
9 shifted registers reminiscent of programmed ribosomal frameshifting.
10 tidyltransferase center affect programmed -1 ribosomal frameshifting.
11 l antiviral agents that target programmed -1 ribosomal frameshifting.
12 y alter the efficiency of -1, but not of +1, ribosomal frameshifting.
13 As, depends on a process known as programmed ribosomal frameshifting.
14 leus, and significantly impairing programmed ribosomal frameshifting.
15 ng the idea of two distinct mechanisms of +1 ribosomal frameshifting.
16 tau and a truncated gamma that is created by ribosomal frameshifting.
17 PKs to decipher the mechanism of programmed ribosomal frameshifting.
18 protein can function as a transactivator of ribosomal frameshifting.
19 s mainly due to PA-X, which was expressed by ribosomal frameshifting.
20 d open reading frame ("X-ORF"), accessed via ribosomal frameshifting.
21 through, ribosome biogenesis, and programmed ribosomal frameshifting.
22 All four pseudoknots cause -1 programmed ribosomal frameshifting.
23 active TK (TK-low phenotype), evidently via ribosomal frameshifting.
24 cane yellow leaf virus (ScYLV) stimulates -1 ribosomal frameshifting.
25 structure are required for the programmed -1 ribosomal frameshifting.
26 d signals that are involved in programmed -1 ribosomal frameshifting (-1 PRF) are typically two-stemm
27 determinants of stimulation of -1 programmed ribosomal frameshifting (-1 PRF) by RNA pseudoknots are
36 d related alphaviruses utilize programmed -1 ribosomal frameshifting (-1 PRF) to synthesize the viral
38 oit one such mechanism, termed -1 programmed ribosomal frameshifting (-1 PRF), to engineer ligand-res
40 vious studies have identified operational -1 ribosomal frameshifting (-1 RF) signals in eukaryotic ge
48 pe and increased efficiency of programmed -1 ribosomal frameshifting and conferred paromomycin sensit
49 the genomic mRNA was critical for sufficient ribosomal frameshifting and EIAV replication, while conc
50 rts a trans-dominant effect on programmed -1 ribosomal frameshifting and killer virus maintenance.
52 nsferase activity, stimulating programmed -1 ribosomal frameshifting and promoting virus propagation
53 the molecular determinants of WNV-programmed ribosomal frameshifting and provide a foundation for the
54 er refine the relationship between efficient ribosomal frameshifting and pseudoknot structure and sta
55 oted increased efficiencies of programmed -1 ribosomal frameshifting and rendered cells unable to mai
56 of the potential link between -1 programmed ribosomal frameshifting and response of a pseudoknot (PK
57 " model in which viruses use both programmed ribosomal frameshifting and translational attenuation to
58 CFTR transcript that stimulates efficient -1 ribosomal frameshifting and triggers the premature termi
59 not just unconventional initiation, but also ribosomal frameshifting and/or imperfect repeat DNA repl
61 domic analyses demonstrated the induction of ribosomal frameshifting, and the generation and presenta
62 domic analyses demonstrated the induction of ribosomal frameshifting, and the generation and presenta
64 molecular mechanisms governing programmed -1 ribosomal frameshifting are almost identical from yeast
65 , reinitiation, selenocysteine insertion, or ribosomal frameshifting, are then represented as branchi
66 ope whose expression results from incidental ribosomal frameshifting at a sequence element within the
67 f functional antizyme requires programmed +1 ribosomal frameshifting at the 3' end of the first of tw
68 mutation that increased the efficiency of -1 ribosomal frameshifting at the L-A virus frameshift site
71 ions in Euplotes ciliates ultimately specify ribosomal frameshifting by one or two nucleotides depend
72 he molecular mechanisms governing programmed ribosomal frameshifting by using two viruses of the yeas
74 in testing the hypothesis that programmed -1 ribosomal frameshifting can be used to control cellular
75 he basis of studies using cell-free systems, ribosomal frameshifting can explain this ability to expr
77 identification of novel frameshift proteins, ribosomal frameshifting, coding sequence detection and t
79 of translational recoding events (programmed ribosomal frameshifting, codon redefinition and translat
80 t killer virus phenotype, suggesting that -1 ribosomal frameshifting does not occur after the peptidy
81 there is an unusually high level, 15%, of +1 ribosomal frameshifting due to features of the nascent p
82 pe 1 (HIV-1) has an absolute requirement for ribosomal frameshifting during protein translation in or
84 It is generally believed that significant ribosomal frameshifting during translation does not occu
85 ved mechanism to influence the efficiency of ribosomal frameshifting during translation of viral RNA,
87 t signals, promoting increased programmed -1 ribosomal frameshifting efficiencies and subsequent loss
88 e inhibitors, anisomycin and sparsomycin, on ribosomal frameshifting efficiencies and the propagation
89 iral mRNA transcription, as well as impaired ribosomal frameshifting efficiency, are critical factors
90 0 significantly reduced the HIV-1 programmed ribosomal frameshifting efficiency, resulting in a shift
93 in of Rous sarcoma virus (RSV) requires a -1 ribosomal frameshifting event at the overlap region of t
94 lyses of alphavirus genomes suggested that a ribosomal frameshifting event occurs during translation
95 e RNA sequence that directs a programmed, +1 ribosomal frameshifting event required for Gag-Pol trans
96 t al. describe a novel, antibiotic-dependent ribosomal frameshifting event that activates translation
97 pected degree of mechanistic diversity among ribosomal frameshifting events and suggest that frameshi
100 West Nile virus (WNV) requires programmed -1 ribosomal frameshifting for translation of the viral gen
101 that a specific conformation is required for ribosomal frameshifting, further implying a specific int
102 li an autoregulatory mechanism of programmed ribosomal frameshifting governs the level of polypeptide
104 ghly accurate, a number of cases of directed ribosomal frameshifting have been reported in RNA viruse
105 nals are associated with sites of programmed ribosomal frameshifting, hopping, termination codon supp
108 ecific mRNA elements required for sufficient ribosomal frameshifting in equine anemia infectious viru
109 ation, specifically inhibits Ty1-directed +1 ribosomal frameshifting in intact yeast cells and in an
110 that provide one of the signals required for ribosomal frameshifting in mouse mammary tumor virus hav
111 h is a mutant of the pseudoknot required for ribosomal frameshifting in mouse mammary tumor virus, ha
113 inery, and ribosome may dynamically modulate ribosomal frameshifting in order to tune the processivit
114 fluenza virus virulence protein generated by ribosomal frameshifting in segment 3 of influenza virus
115 e cis-acting elements that promote efficient ribosomal frameshifting in the -1 (5') direction have be
116 research article describing the discovery of ribosomal frameshifting in the bacterial CopA gene also
118 -methylpseudouridine into mRNA results in +1 ribosomal frameshifting in vitro and that cellular immun
120 these drugs also change the efficiency of -1 ribosomal frameshifting in yeast and mammalian in vitro
126 totiviruses, the efficiency of programmed -1 ribosomal frameshifting is critical for ensuring the pro
129 y support the mechanistic hypothesis that -1 ribosomal frameshifting is enhanced by torsional resista
131 e slippery sequence and stem-loop to promote ribosomal frameshifting is influenced by the flanking up
135 iae double-stranded RNA virus, programmed -1 ribosomal frameshifting is responsible for translation o
142 shift/slippage site, which is important for ribosomal frameshifting, is shown here to limit reverse
143 emonstrated that an evolutionarily conserved ribosomal frameshifting mechanism is used by simarterivi
148 ed exclusively as a Gag-Pol fusion either by ribosomal frameshifting or by read-through of the gag st
149 pathogenic RNA viruses and retroviruses use ribosomal frameshifting or stop codon readthrough to reg
150 unclear, a novel viral protein expressed by ribosomal frameshifting, PA-X, was found to play a major
151 ing mRNA elements that promote programmed -1 ribosomal frameshifting present a natural target for the
153 dictated by the frequency of a -1 programmed ribosomal frameshifting (PRF) event occurring in gag-pol
154 dictated by the frequency of a -1 programmed ribosomal frameshifting (PRF) event that occurs in gag-p
155 Coronavirus (SARS-CoV) employ programmed -1 ribosomal frameshifting (PRF) for their protein expressi
162 Translational control through programmed ribosomal frameshifting (PRF) is exploited widely by vir
163 ntification of reads flanking the programmed ribosomal frameshifting (PRF) signal at the genomic RNA
164 nse and activating a unique -2/-1 programmed ribosomal frameshifting (PRF) signal for the expression
165 show that CHIKV capsid modulates programmed ribosomal frameshifting (PRF) within the 6K/Transframe (
167 biochemical mechanisms, including programmed ribosomal frameshifting (PRF), which facilitates the pro
171 putative feline immunodeficiency virus (FIV) ribosomal frameshifting pseudoknot (PK) has been investi
172 s on killer virus maintenance, programmed -1 ribosomal frameshifting, resistance/hypersensitivity to
178 ere, we experimentally compared all known +1 ribosomal frameshifting sites in S. cerevisiae, includin
180 segmented genomes and viruses utilizing dual ribosomal frameshifting that we validate experimentally.
181 frames, the over-reading of stop codons via ribosomal frameshifting, the existence of an antizyme an
182 The SPEAR element enhances viral programmed ribosomal frameshifting, thereby expanding its functiona
185 A1 undergo highly efficient +1/-2 programmed ribosomal frameshifting to generate previously undescrib
186 mechanisms such as alternative splicing and ribosomal frameshifting to produce multiple distinct pro
187 Many pathogenic viruses use programmed -1 ribosomal frameshifting to regulate translation of their
188 isiae killer virus system uses programmed -1 ribosomal frameshifting to synthesize its gene products.
189 part of its life cycle, termed programmed -1 ribosomal frameshifting, to produce the required ratio o
190 e codons and/or the process of programmed -1 ribosomal frameshifting used by viruses to control their
191 distribution of recoding with a focus on the ribosomal frameshifting used for gene expression in bact
193 doknots in controlling the extent of -1-type ribosomal frameshifting, we determined the crystal struc
194 th sequences that trigger genuine programmed ribosomal frameshifting; we have experimentally confirme
195 ally mimic these RNA structures to induce +1 ribosomal frameshifting when annealed downstream of the
196 te tRNA slippage is the driving force for +1 ribosomal frameshifting while the presence of a 'hungry