ライフサイエンスコーパス: right heart

1 ansverse aortic arch, and compression of the right heart.

2 scular resistance, and increased load on the right heart.

3 owledge of left-sided physiopathology to the right heart.

4 sualizing interventional procedures from the right heart.

5 chronic cardiac damage, mainly involving the right heart.

6 The incidences of left and right heart abnormalities were 3.52 and 5.64 per 100 per

7 rtension by shunting oxygenated blood to the right heart and lungs.

8 rt, but its effects on the physiology of the right heart are incompletely understood.

9 use its development and consequences for the right heart are now seen as mainstay abnormalities that

10 oxidized lipids or fibrin deposition within right heart biopsy (RHB) specimens.

11 vival is possible, based on the principle of right heart bypass, whereby the ventricle pumps blood on

12 assessing laboratory, echocardiographic and right heart catheter based parameters to predict cardiac

13 led study, 60 patients with fibrotic IIP and right heart catheter confirmed PH were randomized 2:1 to

14 ccounting for deaths and withdrawals, paired right heart catheter data were available for analysis in

15 apillary pulmonary hypertension confirmed by right heart catheterisation, systolic blood pressure of

16 hould be referred to a specialist centre for right-heart catheterisation and pulmonary angiography.

17 d retrospectively all US veterans undergoing right heart catheterization (2007-2012) in the Veterans

18 In patients with right heart catheterization (30 HF-PH, 14 PVOD), similar

19 nary capillary wedge pressure </=15 mm Hg at right heart catheterization (allele frequency, 0.66; odd

20 ailure (OR, 3.7 [95% CI, 3.1-4.4]) and prior right heart catheterization (OR, 3.8 [95% CI, 3.4-4.3]).

21 57 patients with normal mPAP) who underwent right heart catheterization (RHC) and three-directional

22 and pulmonary hypertension (PH) diagnosed by right heart catheterization (RHC) are independent risk f

23 Right heart catheterization (RHC) must be performed in p

24 psy-proven sarcoidosis and SAPH confirmed by right heart catheterization (RHC) were identified from 1

25 6-min-walk testing, V/Q scanning, CTPA, and right heart catheterization (RHC) were prospectively obt

26 ure (mPAP) value of at least 25 mm Hg during right heart catheterization (RHC).

27 To define LV pressure-volume relationships, right heart catheterization and 3-dimensional echocardio

28 nts known or suspected to have PAH underwent right heart catheterization and adenosine stress MR imag

29 consent, 35 PAH patients were evaluated with right heart catheterization and cardiac magnetic resonan

30 In vivo measurements were assessed using right heart catheterization and cardiac MRI.

31 ved from 60 COPD patients who underwent both right heart catheterization and computed tomography in a

32 onsecutive patients, undergoing simultaneous right heart catheterization and DE.

33 consecutive participants who underwent both right heart catheterization and dual-phase dual-energy C

34 -17 years, 19 women) undergoing simultaneous right heart catheterization and echocardiographic imagin

35 In 50 patients with simultaneous right heart catheterization and echocardiographic imagin

36 II; Thoratec Inc; n=18) were evaluated with right heart catheterization and echocardiography preoper

37 Right heart catheterization and echocardiography were pe

38 upine cycle ergometer test with simultaneous right heart catheterization and echocardiography.

39 able patients who underwent first diagnostic right heart catheterization and from a prospective cohor

40 ly assessed with invasive procedures such as right heart catheterization and histopathology.

41 CMR) imaging within 24 hours of a diagnostic right heart catheterization and invasive measurement of

42 utinely underwent detailed reassessment with right heart catheterization and noninvasive testing at 3

43 All measurements, including data from right heart catheterization and norepinephrine requireme

44 nts undergoing clinically indicated left and right heart catheterization and same day research cardia

45 pulmonary edema, invasive techniques such as right heart catheterization and the sampling of alveolar

46 Hemodynamic variables obtained by right heart catheterization and transpulmonary thermodil

47 modynamic evaluation was performed by serial right heart catheterization and transthoracic echocardio

48 omboembolic pulmonary hypertension underwent right heart catheterization and-after digital subtractio

49 isk factors using systematic confirmation on right heart catheterization are lacking.

50 Echocardiography and right heart catheterization are the principal modalities

51 All patients underwent right heart catheterization at baseline and were followe

52 underwent simultaneous echocardiography and right heart catheterization at rest and during exercise.

53 0 years; n=14 HFpEF; n=12 control) underwent right heart catheterization at rest, during supine exerc

54 Twenty-six patients underwent preoperative right heart catheterization before PTE.

55 d vital status for all patients referred for right heart catheterization between 1998 and 2014.

56 ve patients with heart failure who underwent right heart catheterization between 2000 and 2005.

57 Right heart catheterization confirmed constrictive physi

58 Laboratory, echocardiography, and right heart catheterization data collected from 205 pati

59 Right heart catheterization data from clinical records o

60 distress syndrome who had complete baseline right heart catheterization data from the Fluid and Cath

61 h heart failure, we retrospectively assessed right heart catheterization data in 162 consecutive pati

62 nship between screening echocardiography and right heart catheterization determinations of pressure,

63 Patients undergoing right heart catheterization for evaluation of pulmonary

64 Patients underwent right heart catheterization immediately followed by tran

65 The significance of right heart catheterization in critically ill patients i

66 cic echocardiography and following up with a right heart catheterization in patients in whom the righ

67 rt the routine use of fluid challenge during right heart catheterization in patients with risk factor

68 phic parameters and hemodynamics obtained by right heart catheterization in PH with AF.

69 valence of exercise-induced PH determined by right heart catheterization in scleroderma spectrum diso

70 cteristics of echocardiography compared with right heart catheterization in the determination of syst

71 We studied 233 patients assigned to right heart catheterization in the Vasodilation in the M

72 66.4 [9.9] years; 3.3% female) who underwent right heart catheterization in this cohort study, Td and

73 Fluid challenge at the time of right heart catheterization is easily performed, safe, a

74 In conclusion, right heart catheterization is necessary to confirm POPH

75 Right heart catheterization is the gold standard for ass

76 Pericardial tamponade was suggested by right heart catheterization measurements and diagnosed b

77 Using right heart catheterization measurements, mild PH was de

78 ents (simultaneously measured mPAP) and with right heart catheterization measurements.

79 operative transthoracic echocardiography and right heart catheterization measurements.

80 Invasive hemodynamic evaluation through right heart catheterization plays an essential role in t

81 ved in the PVDOMICS study, the comprehensive right heart catheterization protocol described here hold

82 progress in echocardiography and biomarkers, right heart catheterization remains the only test that c

83 We suggest that right heart catheterization should be reserved for patie

84 Right heart catheterization showed severe precapillary P

85 ations of variables collected during resting right heart catheterization that best predicted abnormal

86 ients with advanced heart failure undergoing right heart catheterization to assess cardiac transplant

87 sthoracic echocardiography further underwent right heart catheterization to confirm the diagnosis of

88 Subjects underwent right heart catheterization to define LV pressure-volume

89 went invasive haemodynamic measurements with right heart catheterization to define Starling and left

90 Sixty healthy subjects underwent right heart catheterization to measure age- and sex-rela

91 Left and right heart catheterization using MR guidance is feasibl

92 ditional baseline characteristics, including right heart catheterization variables, were not consiste

93 The confirmed PAH prevalence on right heart catheterization was 2.1% (95% confidence int

94 in beta-thalassemia patients as confirmed on right heart catheterization was 2.1%, with an approximat

95 Right heart catheterization was employed in one study to

96 Right heart catheterization was performed and serum uric

97 Left and right heart catheterization was performed in 7 swine wit

98 To investigate this hypothesis, right heart catheterization was performed in eight males

99 0.55 T (<1 degrees C heating) and MRI-guided right heart catheterization was performed in seven study

100 Right heart catheterization was performed using a pressu

101 h severe aortic stenosis and a preprocedural right heart catheterization were assessed.

102 s with PAH or ILD-associated PH confirmed by right heart catheterization were included in the study.

103 4, treadmill testing, echocardiography, and right heart catheterization were performed.

104 Simultaneous echocardiography and right heart catheterization were prospectively performed

105 resistance (PVR) > 400 dyn s cm(-5) based on right heart catheterization were randomized to treatment

106 ing transient RBBB pattern in lead V1 during right heart catheterization were studied.

107 clinic with transthoracic echocardiogram and right heart catheterization within 1 year.

108 All subjects with hemodynamics measured by right heart catheterization within 30 days before left v

109 e hypothesized that a fluid challenge during right heart catheterization would identify occult pulmon

110 ed to medical therapy with a sham procedure (right heart catheterization) versus medical therapy and

111 and left ventricular (LV) filling pressures (right heart catheterization) were measured under varying

112 ents who had coronary physiology assessment, right heart catheterization, and echocardiography perfor

113 not demonstrated any sustained benefits from right heart catheterization, and some studies have even

114 diac studies, including echocardiography and right heart catheterization, are key elements in the ass

115 sure product was also determined by means of right heart catheterization, as an index of the RV MVO2,

116 operation [n=6]) were evaluated monthly with right heart catheterization, CMR, and computed tomograph

117 utine invasive hemodynamic ramp testing with right heart catheterization, during which LVAD speeds we

118 al fibrillation included corticosteroid use, right heart catheterization, fungal infection, vasopress

119 ts were more than 15000 adults who underwent right heart catheterization, including 12232 in the Vete

120 iology, patient demographic characteristics, right heart catheterization, mechanical circulatory supp

121 ury during interventional procedures such as right heart catheterization, pacemaker implantation, inv

122 Diagnosis was confirmed by right heart catheterization, ventilation-perfusion lung

123 ce on the basis of PC-MRI in comparison with right heart catheterization-based measurements by a medi

124 s bag technique in adult patients undergoing right heart catheterization.

125 sure >35 mm Hg on echocardiogram underwent a right heart catheterization.

126 terminal telopeptide of type I collagen) and right heart catheterization.

127 anently implanted in all participants during right heart catheterization.

128 iratory rise in right atrial pressure during right heart catheterization.

129 onse to vasodilator testing require invasive right heart catheterization.

130 g hemodynamics underwent subsequent exercise right heart catheterization.

131 who were referred for a clinically indicated right heart catheterization.

132 thered data on IPAH patients who underwent a right heart catheterization.

133 -time MRI (MR fluoroscopy) to guide left and right heart catheterization.

134 ameters, transthoracic echocardiography, and right heart catheterization.

135 AH using Optical Coherence Tomography during Right Heart catheterization.

136 nits; or (3) inpatient status at the time of right heart catheterization.

137 ients with unexplained dyspnea who underwent right heart catheterization.

138 Twenty patients with PH underwent right heart catheterization: mean pulmonary artery press

139 study patients were divided into 2 groups by right heart catheterization: no PH (mean pulmonary arter

140 weeks after therapy; patients also underwent right-heart catheterization and LSM at these time points

141 as demonstrated by pulmonary hypertension at right-heart catheterization at days 21 to 35 and major r

142 Patients underwent right-heart catheterization at rest and during exercise

143 apillary wedge pressure waveform obtained by right-heart catheterization during 5 different loading c

144 diography were performed simultaneously with right-heart catheterization in 51 consecutive patients (

145 e anesthetized rat with a recently developed right-heart catheterization technique.

146 us Doppler echocardiographic examination and right-heart catheterization were performed in 44 patient

147 Right-heart catheterization, with its associated disadva

148 ss hemodynamic variables were measured using right-heart catheterization.

149 ertension, but definitive diagnosis requires right-heart catheterization.

150 ansplant patients are followed with periodic right heart catheterizations (RHCs) to identify post-tra

151 nsplant patients, five of which had multiple right heart catheterizations allowing an assessment of c

152 ams (at 1, 2, 3, 4, 6, 9, and 12 months) and right heart catheterizations were performed after LVAD i

153 ly evaluated with serial echocardiograms and right heart catheterizations.

154 e), which reflects LV preload independent of right heart congestion and pericardial restraint, was si

155 lume was associated with higher incidence of right heart dilatation.

156 Right heart disease produces a substrate for AF maintena

157 This study sought to determine whether right heart disease promotes atrial arrhythmogenesis in

158 ne, and rats were studied 21 days later when right heart disease was well developed.

159 achieve this goal and assessed children with right heart disease.

160 tensive patients with PE but with indicia of right heart dysfunction (by biomarkers or imaging) const

161 Two readers evaluated signs of right heart dysfunction at CT pulmonary angiography, mea

162 On the basis of the findings of right heart dysfunction on echocardiograms, computed tom

163 on between clot burden measures and signs of right heart dysfunction.

164 valve replacement, moderate or severe TR and right heart enlargement are independently associated wit

165 l or percutaneous ASD closure in adults with right heart enlargement, with or without symptoms.

166 d more severe disease as indicated by recent right heart failure (OR, 3.3 [95% CI, 2.8-3.9]) or respi

167 Bisoprolol delayed time to right heart failure (P<0.05).

168 Acute right heart failure (RHF) after left ventricular assist

169 Our understanding of right heart failure (RHF) has lagged behind and many pro

170 amine reduces the incidence of postoperative right heart failure (RHF) in pediatric heart transplant

171 Background Early right heart failure (RHF) occurs commonly in left ventri

172 o associated with increased unadjusted early right heart failure (RHF).

173 n, retroperitoneal vascular constriction and right heart failure - has shown that serotonin and tachy

174 sidered when evaluating patients with severe right heart failure after PPM or ICD implantation.

175 of the pulmonary vasculature that results in right heart failure and death, are usually assessed with

176 ling of the pulmonary arteries, resulting in right heart failure and death.

177 relentlessly progressive disease leading to right heart failure and death.

178 nt, the disorder progresses in most cases to right heart failure and death.

179 a progressive disease that ultimately causes right heart failure and death.

180 arrowing of pulmonary arteries, resulting in right heart failure and death.

181 reased work of the right ventricle may cause right heart failure and liver congestion.

182 evealed a combination of left heart failure, right heart failure and moderate-to-severe tricuspid reg

183 c abnormalities that reflect the severity of right heart failure and predict adverse outcomes in pati

184 emodeling that can subsequently culminate in right heart failure and premature death.

185 cise intolerance, frequent hospitalizations, right heart failure and reduced survival.

186 lung inflammation, vascular remodeling, and right heart failure and reverses hypoxic pulmonary hyper

187 ic iron levels, this model developed PAH and right heart failure as a consequence of intracellular ir

188 One patient died from right heart failure at 3 months.

189 OF REVIEW: To review recent publications on right heart failure developing early and late after impl

190 have provided good evidence about predicting right heart failure early after LVADs, though how to pre

191 Patients with right heart failure from cor pulmonale were classified a

192 Patients with pulmonary hypertension and right heart failure have a high risk of clinical deterio

193 k of progressive tricuspid regurgitation and right heart failure in patients with moderate or lesser

194 Right heart failure is a cause of morbidity and mortalit

195 ure early after LVADs, though how to predict right heart failure late after LVAD is still unclear as

196 cyclin can be life-saving when perioperative right heart failure occurs due to exacerbation of pulmon

197 determined to be the direct cause of death (right heart failure or sudden death) in 37 (44%) patient

198 ents with PAH and 75.7% of those who died of right heart failure received parenteral prostanoid thera

199 evices is associated with improved outcomes, right heart failure remains a considerable challenge.

200 Despite improved outcomes, right heart failure remains a significant challenge to s

201 ention that left heart failure has received, right heart failure remains understudied both at the pre

202 perience progressive symptoms of dyspnea and right heart failure resulting in significant morbidity a

203 Twelve patients died or developed right heart failure secondary to pulmonary hypertension

204 cured from explanted hearts of patients with right heart failure served as novel comparison samples.

205 1) How is right heart failure syndrome best defined?

206 s with dyspnea, exercise intolerance, and/or right heart failure who have elevated pulmonary artery s

207 ped for treating patients with severe TR and right heart failure with prohibitive surgical risk.

208 Lower extremity edema, venous stenosis, right heart failure, and deep venous thrombosis occurred

209 The main causes of death included sepsis, right heart failure, and multiorgan failure.

210 nt with bisoprolol delays progression toward right heart failure, and partially preserves RV systolic

211 or adverse events included bleeding, stroke, right heart failure, and percutaneous lead infection.

212 nts included postoperative bleeding, stroke, right heart failure, and percutaneous lead infection.

213 Echocardiogram showed acute right heart failure, and pulmonary perfusion scan demons

214 ve clearer evidence now for predicting early right heart failure, and treating it in those patients w

215 antly left heart failure in combination with right heart failure, and tricuspid regurgitation; and (i

216 This leads to reduced cardiac output, right heart failure, and ultimately death.

217 rized by heightened ventricular interaction, right heart failure, and worsening pulmonary vascular di

218 t estimates for bleeding, stroke, infection, right heart failure, arrhythmias, and rehospitalizations

219 e pulmonary hypertension precipitating acute right heart failure, despite administration of milrinone

220 anifest pulmonary veno-occlusive disease and right heart failure, detectable at 8 months of age.

221 pertension, along with frequently associated right heart failure, is extremely challenging.

222 th respect to medical therapies for treating right heart failure, there is evidence for the use of bo

223 ar and biventricular assist devices, such as right heart failure, valvular regurgitation, cardiac arr

224 e progression of tricuspid regurgitation and right heart failure.

225 ere disorder of lung vasculature that causes right heart failure.

226 riuretic peptide levels, and the presence of right heart failure.

227 terized by pulmonary vascular remodeling and right heart failure.

228 ients with severe pulmonary hypertension and right heart failure.

229 n as a novel therapeutic strategy for PH and right heart failure.

230 te treatment strategies for PH and resultant right heart failure.

231 ypoxic pulmonary hypertension and ultimately right heart failure.

232 pulmonary arterial tree, eventually leads to right heart failure.

233 nt of pulmonary hypertension, and associated right heart failure.

234 in pulmonary vascular resistance leading to right heart failure.

235 -recognized but treatable etiology of severe right heart failure.

236 primary graft nonfunction or intraoperative right heart failure.

237 viduals present with dyspnoea or evidence of right heart failure.

238 n elevated pulmonary vascular resistance and right heart failure.

239 No deaths were associated with right heart failure.

240 TV) is increasing and results in intractable right heart failure.

241 y vascular resistance, eventually leading to right-heart failure and death.

242 ications (one constrictive pericarditis, two right heart failures without underlying infection, and o

243 systolic HF, CXL-1020 reduced both left and right heart filling pressures and systemic vascular resi

244 Syncope in PAH is associated with worsening right heart function and is an independent predictor of

245 hysiology may be useful during assessment of right heart function and pulmonary pressures before tran

246 f venous return and its interaction with the right heart function as it relates to mechanical ventila

247 autoimmune disease that can affect left and right heart function directly through inflammation and f

248 tative two-dimensional methods for assessing right heart function that are both well established and

249 In the postoperative period, changes in right heart function will depend on preexisting pulmonar

250 ine nonconsecutive patients with compromised right heart function, pulmonary hypertension, and severe

251 ching, without impairment of hemodynamics or right heart function.

252 -term outcome; namely, exercise capacity and right heart function.

253 n leads to clinically significant changes in right heart function.

254 is inflammation is associated with decreased right heart function.

255 function, pulmonary vascular remodeling, and right heart function.

256 exercise testing, demonstrates that that the right heart has enormous contractile reserve, with a thr

257 ge demonstrating that diseases affecting the right heart have been shown to have the same clinical co

258 Clinical bedside evaluations and right heart hemodynamic assessments can alert clinicians

259 ative diastolic mitral inflow velocities and right heart hemodynamic data in 39 consecutive patients

260 The ECGs, echocardiograms, and right heart hemodynamic data were reviewed to determine

261 Improvements in right heart hemodynamics and exercise capacity were conf

262 Accurate noninvasive evaluation of right heart hemodynamics is an essential component of th

263 ciation class III HF underwent assessment of right heart hemodynamics, gas exchange, and first-pass r

264 n together for an accurate interpretation of right heart hemodynamics.

265 right ventricular systolic pressure, reduces right heart hypertrophy, restores the cardiac index, and

266 naling, and reversed vascular remodeling and right-heart hypertrophy in vivo.

267 amine associations with venous return to the right heart in individuals with chronic COPD and emphyse

268 ul navigation of the aorta, left atrium, and right heart, including detailed understanding of relatio

269 Conditions affecting the right heart, including diseases of the lungs and pulmona

270 rt the need for future studies on TR and the right heart, including whether concomitant treatment of

271 h global function and catheterization of the right heart indexes.

272 how these shock states perturb venous return/right heart interactions.

273 py, indicating that diastolic filling of the right heart is not passive.

274 e venous system and its interaction with the right heart may be more useful.

275 Among right heart metrics, RVESRI demonstrated the best test-r

276 al area were strongly connected to the other right heart metrics.

277 egistrants supported with dual inotropes and right heart monitoring had a higher risk of adverse even

278 n=5), tetralogy of Fallot (n=1), hypoplastic right heart (n=1), and common arterial trunk (n=1).

279 the authors discuss the emerging concepts of right heart pathobiology in PAH.

280 ority, 86 of 109 (79%), had CHD resulting in right heart pressure or volume overload.

281 Several large clinical databases with right heart/pulmonary catheterization data were analyzed

282 2.7 L/min/m2; RV % area change, 24 vs. 33%), right heart remodeling (right atrial area index, 17.0 vs

283 TV deformations and their association with right heart remodeling differ between AF-TR and left-sid

284 a in smooth muscle on pulmonary vascular and right heart responses to chronic hypoxia.

285 id valves (n=16) were studied in an in vitro right heart simulator.

286 erity using the Mastora system and evaluated right heart strain.

287 Significant right heart structural reverse remodeling takes place im

288 rtension is associated with abnormalities of right heart structure and function that contribute to th

289 Visualization of right heart structures was rated significantly (P < .05)

290 No significant changes were found in the right heart structures.

291 n, a patent foramen ovale, and free-floating right-heart thrombus are echocardiographic markers that

292 ses on the function of the venous system and right heart under normal and stressed conditions.

293 In combination with echocardiographic right heart variables, also available from routine echoc

294 protected from the effects of hypoxia on the right heart, vascular remodeling, and raised serum endot

295 specified cardiac targets were imaged from a right heart venue.

296 Mean contrast medium attenuation in the right heart was significantly (P < .001) higher in the s

297 the basal septal myocardium to re-enter the right heart where it is exchanged for a suture.

298 ficient attenuation for visualization of the right heart, while streak artifacts from high-attenuatio

299 esigned to optimize the visualization of the right heart with echocardiography with case examples wil

300 he interaction of the pathologically altered right heart with the anatomically-supposedly-normal left