ライフサイエンスコーパス: right hemisphere

1 age 52.8 years, range 22-77; 63 females; 64 right hemispheres).

2 tes along dorsal and ventral pathways in the right hemisphere.

3 MOA) of SLF II, SLF III, and IFOF within the right hemisphere.

4 tors of recovery are at play in the left and right hemisphere.

5 hemisphere but to attention networks in the right hemisphere.

6 tained responses over the left than over the right hemisphere.

7 get-specific connections we obtained for the right hemisphere.

8 and dorsolateral prefrontal cortices of the right hemisphere.

9 al and subcortical limbic targets within the right hemisphere.

10 spectrum disorders group, especially in the right hemisphere.

11 gh normal or enhanced activity in the intact right hemisphere.

12 areas, and their anatomic homologues in the right hemisphere.

13 , and precuneus along the mesial wall of the right hemisphere.

14 on was significantly weaker in the undamaged right hemisphere.

15 belt ACFs, indicating neuroplasticity in the right hemisphere.

16 rontal lobes, mesolimbic connections and the right hemisphere.

17 hippocampal region were only observed in the right hemisphere.

18 is activation appeared to be stronger in the right hemisphere.

19 reased negativity of N170 amplitude over the right hemisphere.

20 ce recognition, and emotional prosody to the right hemisphere.

21 tion of spatial deficits after damage to the right hemisphere.

22 responses in a voice-sensitive region in the right hemisphere.

23 ferentially modulate voice processing in the right hemisphere.

24 asing signals than their counterparts in the right hemisphere.

25 I fiber pathways are better preserved in the right hemisphere.

26 unced age-related loss of gray matter in the right hemisphere.

27 arietal cortex and early visual areas of the right hemisphere.

28 ft hemisphere and a relative increase in the right hemisphere.

29 ination between stimuli observed only in the right hemisphere.

30 in control is not restricted to the left or right hemisphere.

31 gion and the inferior parietal lobule in the right hemisphere.

32 uage network, and a compensatory role of the right hemisphere.

33 ence, driven by a reduction in volume in the right hemisphere.

34 cortical thinning in prefrontal areas of the right hemisphere.

35 oxelwise comparisons of activity in left and right hemispheres.

36 ADC: left hemisphere, 1.18 x10(3)mum(2)/sec; right hemisphere, 1.17 x10(3)mum(2)/sec), anterior limb

37 5) demonstrated asymmetric left greater than right hemisphere (18)F-AV1451 uptake in three of five pa

38 ups had language function lateralized to the right hemisphere (54.5%) or dispersed bilaterally (27.3%

39 ctivation in both regions as well as reduced right hemisphere activation in the posterior temporal re

40 Right hemisphere activation in these two groups occurred

41 amage or healthy ageing results in increased right-hemisphere activation in homologous regions to tho

42 AgCC participants showing significantly more right hemisphere activations than controls or than indiv

43 sed, indicating that homologous areas in the right hemisphere actively contribute to language functio

44 ctivity only for contralateral memory items; right hemisphere activity reflected VSTM load regardless

45 hout the occipital and temporal lobes with a right hemisphere advantage.

46 results, with LSE capturing left hemisphere/right hemisphere affinity structure and ASE capturing gr

47 s that semantic activation is broader in the right hemisphere, affording it an advantage over the lef

48 For the right hemisphere, age [beta = -0.678, t(-3.087), P = 0.0

49 decreased fractional anisotropy (FA) in the right hemisphere and a subnetwork with increased mean di

50 sented on the left producing activity in the right hemisphere and accurate memory for items previousl

51 Patients who had damage to the left or right hemisphere and age-matched control participants re

52 ses in the left hemisphere, in 1 case in the right hemisphere and in 1 case bilateral.

53 nsight-related coarse semantic coding in the right hemisphere and internally focused attention preced

54 hen lexical information is introduced to the right hemisphere and must subsequently be transferred to

55 heory emphasizing a dominant function of the right hemisphere and others supporting an interhemispher

56 implanted with F98 glioblastoma cells in the right hemisphere and scanned 9-15 d later.

57 on a combination of visual expertise in the right hemisphere and semantics in the left hemisphere.

58 -greater-than-parietal P3b topography in the right hemisphere and the highest P3a amplitude at fronta

59 g of the cortex of the lateral aspect of the right hemisphere and the medial aspect of the left, as w

60 and (3/15 Hz) responsivity prevailing in the right hemisphere and theta and gamma band (6/40 Hz) acti

61 the left visual field is represented in the right hemisphere and vice versa.

62 ws approximate symmetry between the left and right hemispheres and a net bias for upward-nasal motion

63 occipital and parietal areas of the left and right hemispheres and were short lived: they were observ

64 owed prolonged response profiles (all in the right hemisphere), and fast profiles (all but one in the

65 ns immediately surrounding the lesion in the right hemisphere, and also, surprisingly, in correspondi

66 al sensory motor cortex, particularly in the right hemisphere, and surrounding association areas (Bro

67 esentations are present in both the left and right hemisphere, and that the representations of the le

68 ital regions, the mesial frontal lobe of the right hemisphere, and the cuneus and precuneus in the le

69 greater extent when applied to left than to right hemisphere, and was more disruptive when applied c

70 at 40 962 homologous points in the left and right hemispheres, and the trajectory of change in asymm

71 to-parietal and default mode networks in the right hemisphere; and (iii) increased intrahemispheric c

72 have previously suggested that the left and right hemispheres are specialized for controlling differ

73 motor lateralization, in which the left and right hemispheres are specialized for different aspects

74 tion that increased activation of homologous right hemisphere areas supports aphasia recovery after l

75 osting of language in either the left and/or right hemisphere as assessed by a very high incidence of

76 ontal regions and the anterior insula of the right hemisphere as well as an expansive negative networ

77 ain damage as measured by weight loss of the right hemisphere at 22 days after HI and by gross and mi

78 in both temporal lobes, and rely more on the right hemisphere auditory regions, particularly right su

79 in nonspeech acoustic signals lateralize to right-hemisphere auditory areas, whereas rapid temporal

80 nspeech stimuli, and it is not known whether right-hemisphere auditory cortex is dominant for coding

81 Right-hemisphere auditory cortex was 100% more accurate

82 (left hemisphere) and orbitofrontal cortex (right hemisphere); bilateral precuneus, posterior cingul

83 ntralateral and ipsilateral responses in the right hemisphere but maintained or enhanced contralatera

84 with brain injuries or stroke in the left or right hemisphere, but not in the postcentral gyrus as th

85 pplied left hemisphere anodal-excitatory and right hemisphere cathodal-inhibitory tDCS, compared to s

86 (chi2 = 11.90, P = 0.003), especially in the right hemisphere (chi2 = 13.67, P = 0.001).

87 Heschl's gyrus, insula, and striatum in the right hemisphere, clearly different from the lesion patt

88 y more than control subjects, specifically a right hemisphere cluster encompassing the putamen, insul

89 d thicker cortical gray matter overall (left/right hemispheres: Cohen's d = 0.61/0.65), but focal thi

90 We observed higher metabolism in the right hemisphere compared to the left and a positive cor

91 was increased in APOE-e4 in a set of mostly right-hemisphere connections, including lateral parietal

92 e demonstrating consistently elevated within-right hemisphere connectivity.

93 as and prefrontal attention areas; increased right-hemisphere connectivity; reduced connectivity in t

94 A bias was also found in favor of the right hemisphere consistent with functional attentional

95 isual information across saccades underlying right-hemisphere constructional apraxia.

96 EEG activity over central electrodes in the right hemisphere, contralateral to the PA-induced, compe

97 control), acceleration time (associated with right hemisphere control), and speed equivalent to contr

98 during sham stimulation and stimulation to a right-hemisphere control brain region.

99 In contrast, right-hemisphere cortical regions involved in visuospati

100 resolving the activity imbalance with their right hemisphere counterparts, thus leading to persisten

101 showed pervasive reductions in 22q11DS (left/right hemispheres: d = -1.01/-1.02).

102 iparetic stroke patients with left (LHD) and right hemisphere damage (RHD).

103 e to movement control, showing that left and right hemisphere damage produce different effects on mov

104 fying features of movement trajectory, while right hemisphere damage produces deficits in achieving a

105 We now propose that left and right hemisphere damage should also produce different de

106 individuals with left hemisphere damage and right hemisphere damage whereas the Multistep Object Use

107 lesional arm of stroke patients with left or right hemisphere damage, provided a critical test of our

108 rrors in movement extent were greatest after right hemisphere damage.

109 our predictions, patients with left, but not right, hemisphere damage showed reduced modulation of ac

110 Following right-hemisphere damage, a specific disorder of motor aw

111 measured attention and motor deficits in 44 right hemisphere-damaged patients with a first-time stro

112 f Kurzban et al. in light of our findings on right-hemisphere-damaged patients, who show increasing a

113 This dissociation was more evident in the right hemisphere, demonstrating functional lateralizatio

114 Despite the complete loss of her right hemisphere (di- and telencephalon) at birth, the p

115 However, a right-hemisphere difference was found in the N1 amplitud

116 tion in the discordant context, and, for the right hemisphere, discordant context information actuall

117 possibility of light-based interventions for right hemisphere disorders of spatial attention.

118 gion of the anterior cingulate cortex in the right hemisphere displayed greater thickness in SuperAge

119 Therefore, right hemisphere dominance during stimulus-driven shifts

120 LeVF) bias due to earlier reports suggesting right hemisphere dominance for faces, or would show an u

121 Right hemisphere dominance for visuospatial attention is

122 attended location produced a more widespread right hemisphere dominance in frontal, parietal, and tem

123 The anatomy and right hemisphere dominance of neglect follow from the an

124 volved in tutor song memory, while there was right hemisphere dominance of neuronal activation in HVC

125 (i.e., greater likelihood of bilaterality or right hemisphere dominance) in this cohort compared with

126 Here we show strong right-hemisphere dominance for coding the speech envelop

127 found that good readers indicated consistent right-hemisphere dominance in auditory cortex for all me

128 The right-hemisphere dominance model introduces a functional

129 frontoparietal network that conforms to the right-hemisphere dominance model.

130 o competing models: the orientation bias and right-hemisphere dominance models.

131 esponse involves the coordinated action of a right hemisphere dominant ventral frontoparietal network

132 lective attention is widely considered to be right hemisphere dominant.

133 atial attention in both visual fields evoked right-hemisphere dominant activity in temporoparietal ju

134 nd occipitotemporal regions (for faces), and right hemisphere dorsolateral prefrontal regions during

135 d that reduced normalized streamlines in the right-hemisphere dorsolateral prefrontal cortex-sensorim

136 stimulation, brain activity increased in the right hemisphere during negative emotion and was localiz

137 In contrast, the NN uniquely engage the right hemisphere during the M400.

138 effective connectivity between the left and right hemispheres during repetition of auditory and visu

139 By contrast, left-side onset patients (LPD; right hemisphere dysfunction) would show impaired global

140 sus right side of the body (LPD, predominant right-hemisphere dysfunction; RPD, predominant left-hemi

141 The load dependence of right hemisphere effects argues that memory-dependent an

142 the current prevailing theory suggests that right hemisphere engagement is ineffective or even malad

143 robust, emphasizing the contribution of the right hemisphere, especially the frontal lobe, to readin

144 te an electrophysiological response over the right hemisphere exactly at 1.2 Hz (6 Hz/5).

145 The dominant right hemisphere exhibited early visual discrimination b

146 ine whether local grey matter volumes in the right hemisphere explained additional variance in langua

147 recuneus was maintained across the lifespan (right hemisphere: F = 7.69, P < 0.001; left hemisphere:

148 Only patients with frontal damage in the right hemisphere failed to correct for this discrepancy

149 ed system can successfully reorganize to the right-hemisphere following left-hemisphere brain damage.

150 en reading span scores and activation in the right hemisphere for both types of ambiguous words sugge

151 ient sensory-motor behaviours, favouring the right hemisphere for fight-or-flight processes and the l

152 ately stronger in the left compared with the right hemisphere for place but not for voicing or manner

153 ing and accounting for limb dynamics and the right hemisphere for stabilizing limb position through i

154 emisphere for linguistic probes and over the right-hemisphere for non-linguistic probes.

155 ctivation that was not direction specific in right hemisphere frontal regions (FEF, SFG, MFG).

156 l cortex and/or the balance between left and right hemisphere functions.

157 tive Stroop test in 34 patients with frontal right hemisphere glioma.

158 Increased activation of the right hemisphere has been observed after left hemisphere

159 area in the left, and Wernicke's area in the right hemisphere) have been reported without evident rep

160 A right-hemisphere homolog (rVWFA) shows similarly positio

161 lated separately for Broca's area versus its right-hemisphere homolog and Wernicke's area versus its

162 phere homolog and Wernicke's area versus its right-hemisphere homolog.

163 ity of this left language region extended to right-hemisphere homologs was positively associated with

164 of correlated activity between the left and right hemispheres, however little is known about regiona

165 showed activation of the Broca's area in the right hemisphere in 3/4 cases of low grade gliomas (LGG)

166 We entrained the left versus right hemisphere in accordance to two different coupling

167 The role of the right hemisphere in aphasia recovery after left hemisphe

168 LD activation in the insula only; and in the right hemisphere in both the insular and temporal cortic

169 gyrus to the superior parietal lobule in the right hemisphere in healthy controls, at-risk mental sta

170 a pivotal role of the frontal cortex of the right hemisphere in limiting interference from an irrele

171 s between hemispheres rather than a dominant right hemisphere in the intact human brain.

172 are consistent with research implicating the right hemisphere in the representation of contextually r

173 ibute to hemispheric debates implicating the right hemisphere in therapy-driven language recovery.

174 The anterior insular cortex of the right hemisphere, in particular its dorsal subregion, wa

175 cal diseases or injuries that can affect the right hemisphere, including stroke, traumatic brain inju

176 mine whether local grey matter volume in the right hemisphere independently contributes to aphasia ou

177 had subcortical infarcts only and seven had right-hemisphere infarcts.

178 eurological syndrome following predominantly right hemisphere injuries and is characterized by both s

179 ostrocaudal white matter connectivity in the right hemisphere interferes with the maintenance of opti

180 lower spans are more likely to involve show right hemisphere involvement in the processing of the am

181 nduces a switch in spatial representation in right hemisphere IPS from contralateral to full-field co

182 key question is whether upregulation of the right hemisphere is adaptive for language recovery.

183 pe information at different time scales: the right hemisphere is thought to be specialized in process

184 categorize stimuli while the left-eye (i.e. right-hemisphere) is used to inspect novel items and ini

185 We studied 27 patients with acute right hemisphere ischaemic stroke and 24 neurologically

186 ate in an emotional empathy task after acute right hemisphere ischemic stroke.

187 cal measurements of CC size with left versus right hemisphere language activation in 74 normal subjec

188 Although older evidence suggested that right hemisphere language homologues compensate for dama

189 The right hemisphere language network seems to be important

190 , but fMRI may be more sensitive than IAT to right hemisphere language processing.

191 emporo-parietal junction was correlated with right hemisphere lateralization of language.

192 ure of spatial neglect and may relate to its right hemisphere lateralization.

193 ground, whereas the 4-8 Hz coupling remained right-hemisphere lateralized in both conditions.

194 ound that disruption of either region in the right hemisphere led to greater selection of both gains

195 Fourteen stroke patients with right hemisphere lesions and contralesional paralysis we

196 techniques, we explored the effect of acute right hemisphere lesions in 18 patients on perceived ang

197 ants, chronic aphasia after left rather than right hemisphere lesions, and the basis of partial recov

198 ction, which is more robust in patients with right-hemisphere lesions.

199 Alternatively, the right hemisphere may actively contribute to language fun

200 l neural processing of pitch in the left and right hemispheres may enable the audio-vocal system to d

201 eas of the left-hemisphere due to GBM in the right-hemisphere may be associated with poor-survival.

202 e of processing; attention processing in the right hemisphere; memory retrieval and semantic judgemen

203 Wistar rats were subjected to right hemisphere middle-cerebral artery occlusion and re

204 of patients with either lOFC (predominantly right hemisphere), mOFC/vmPFC, or dorsomedial prefrontal

205 ate long-latency causal interactions between right-hemisphere motor areas and the left M1 (lM1).

206 at (1) convergent gazes evoked both left and right hemisphere N170, while non-convergent gazes evoked

207 ial diffusivity profile of white matter in a right hemisphere network of white matter regions in keta

208 ral lobe perisylvian cortices, predominantly right-hemisphere occipital and occipitotemporal regions

209 ural differences were observed solely in the right hemisphere of patients with freezing of gait.

210 The right hemisphere of the brain was seen during injection

211 In the right hemisphere of the brain, the pericalcarine gyrus w

212 e given, structural adaptation in the intact right hemisphere of the brain.

213 Although the left and right hemispheres of our brains develop with a high degr

214 as injected intracerebroventricularly in the right hemisphere on postnatal day 6 at 30min prior to th

215 ) and grey matter density (in the unaffected right hemisphere) on language laterality.

216 thod to align FreeSurfer-registered left and right hemispheres onto a common template in order to cha

217 The bilateral centrocortical (right hemisphere p = 0.032, r(2) = 0.31, left hemisphere

218 bilateral corticoamygdaloid transition area (right hemisphere p = 0.032, r(2) = 0.38, left hemisphere

219 Maturation occurred earlier in the right hemisphere (P < .001) in several regions, includin

220 The greatest differences were in the right hemisphere (P = 0.006).

221 visual stimuli, via an asymmetric effect on right-hemisphere parieto-occipital alpha-power.

222 Previously, assessments of right-hemisphere patients with hemispatial neglect have

223 Here we examined the ability of right-hemisphere patients with neglect to maintain atten

224 left prefrontal language-related regions and right hemisphere pitch-related regions, which reflected

225 Results provide evidence that the right hemisphere plays a specific and important role in

226 gs suggest that the grey matter structure of right hemisphere posterior dorsal stream language homolo

227 ptica patients and human complement into the right hemisphere preferentially turned to the right at 7

228 ndings suggest that cortical thinning in the right hemisphere produces disturbances in arousal, atten

229 th ASD also showed underconnectivity between right-hemisphere pSTS, a region known for processing spe

230 Conversely, connection strengths between right hemisphere regions became weaker after training.

231 plore the joint involvement of occipital and right hemisphere regions in a visual-based phonological

232 showed that joint speech recruits additional right hemisphere regions outside the classic speech prod

233 with cortical thickness in two left and four right hemisphere regions, as follows: bilateral temporal

234 slexics showed less GMV in multiple left and right hemisphere regions, including left superior tempor

235 ructural adaptation in similar (overlapping) right hemisphere regions.

236 nstructional apraxia patients' damage to the right-hemisphere regions involved in remapping locations

237 is highly heritable and, specifically in the right hemisphere, regulated oligogenically with linkages

238 ions of the human brain, particularly in the right hemisphere, remains poorly understood.

239 phere represents the right side, whereas the right hemisphere represents both sides of the sensorium.

240 -4.40; 95% CI: -7.96, -0.84 for the left and right hemispheres, respectively).

241 position may be lateralized to the left and right hemispheres, respectively.

242 +/- 6.1 and 48.7 +/- 8.5 Hz for the left and right hemispheres, respectively.

243 ound that indicated size/distance, LO in the right hemisphere responded significantly more to the sma

244 ed behaviours involved partially overlapping right hemisphere reward circuit regions including putame

245 egmental) content (e.g., [1-3]), whereas the right hemisphere (RH) is more sensitive to prosodic (sup

246 rges under stimulus-guided attention: in the right hemisphere (RH), visual maps IPS0, IPS1, and IPS2

247 whereas in infants, it is lateralized to the right hemisphere (RH).

248 ther on patterns of left-hemisphere (LH) and right-hemisphere (RH) activation across individual parti

249 rongly related to structural deficits in the right hemisphere's locomotor network involving prefronta

250 subjects also received inhibitory rTMS over right hemisphere S1 and the vertex (control).

251 at different time scales, with the left and right hemispheres sampling at short (25 ms; 40 Hz) and l

252 3 amplitude at left hemisphere, midline, and right hemisphere scalp locations was affected by the sta

253 In contrast, hV4 of the right hemisphere showed expanded response properties.

254 This may be important as right-hemisphere spatial abilities may underlie our abil

255 is to achieve optimal separation of left and right hemisphere ssSEPs.

256 Reading impairments after left or right hemisphere stroke are common yet receive little at

257 tudy on the perception of C-tactile touch in right hemisphere stroke patients (N = 59), revealing tha

258 esions were significantly more impaired than right hemisphere stroke patients without uncinate fascic

259 0 individuals with left neglect secondary to right hemisphere stroke.

260 l empathy in 30 patients with acute ischemic right hemisphere stroke.

261 ied functional connectivity in patients with right-hemisphere stroke and found a pattern of correlati

262 gical evidence that attention deficits after right-hemisphere stroke arise in part from hyper-excitat

263 Hemispatial neglect following right-hemisphere stroke is a common and disabling disord

264 sion behavior mapping analysis of a group of right-hemisphere stroke patients supported this hypothes

265 Sixteen right-hemisphere stroke patients were recruited, all of

266 using both visuospatial and verbal tasks in right-hemisphere stroke patients with anosognosia (n = 1

267 Right-hemisphere stroke patients with constructional apr

268 (FC) and ROI-to-ROI connectivity in subacute right-hemisphere stroke patients.

269 neuroimaging methods in 174 patients with a right-hemisphere stroke, we were able to identify three

270 PTN expression were on average higher in the right hemisphere, suggesting that asymmetric NPTN expres

271 These effects appeared in the right hemisphere, supporting lateralization and top-down

272 bservations: a first model proposes that the right hemisphere sustains production and comprehension o

273 of the contralesional hemisphere (i.e., the "right-hemisphere-take-over" theory).

274 One aphasic case displayed higher right-hemisphere tangle density despite greater left-hem

275 ecline in cortical thickness with age in the right hemisphere than in the left on the lateral surface

276 of nonadjacent patterns was stronger in the right hemisphere than in the left, and may reflect an ef

277 male and female birds, were stronger in the right hemisphere than in the left, and that right-side r

278 more precise and mature more rapidly in the right hemisphere than in the left.

279 posterior cortical network, particularly in right hemisphere, that prepares the saccade system for r

280 ispheres, contrary to the predictions of the right hemisphere theory of metaphor.

281 These results support the "brain's right-hemisphere" theory, which predicts that the right-

282 he differential contribution of the left and right hemisphere to executing a grasping movement with t

283 rs generally performed worse than those with right hemisphere tumors.

284 f 45 patients with unilateral strokes in the right hemisphere underwent cognitive testing for neglect

285 ace-, body- and scene-selective areas in the right hemisphere using moving and static stimuli.

286 ional relationship between the LC-NE system, right-hemisphere ventral attention network, and P300 EEG

287 (face) influences on neuronal responses in a right-hemisphere voice-sensitive region in the anterior

288 pressed the auditory evoked field, while the right hemisphere was sensitive to visual constraints onl

289 The length of the planum temporale in the right hemisphere was the main predictor of language late

290 Activity in the right-hemisphere was not correlated with damage in the l

291 vs cathodal) and electrode location (left vs right hemisphere) was tested in a series of separate sin

292 We obtained activation only in the right hemisphere Wernicke's area in 4/5 of the cases.

293 hat high-frequency activity increased in the right hemisphere when participants were biased toward ri

294 s within the left (P < 0.05), but not in the right, hemisphere when compared to controls.

295 ital gyrus was unilaterally activated in the right hemisphere while the cuneus was bilaterally activa

296 rds was associated with volume of pretherapy right hemisphere white matter and post-therapy grey matt

297 onal connectivity between IPS and FEF in the right hemisphere with early visual areas was stronger fo

298 waves are generally larger and lead from the right hemisphere with only moderate covariation of ampli

299 us pallidus and orbitofrontal regions of the right hemisphere (with the left hemisphere not analyzed

300 ences in tensor metrics between the left and right hemispheres within or between the two groups.