ライフサイエンスコーパス: right ventricular

1 9); LAVA areas were smaller with atrial than right ventricular (12.3+/-10.5 versus 18.4+/-11.0 cm(2),

2 Bipolar scar was larger with atrial than right ventricular (31.7+/-18.5 versus 27.6+/-16.3 cm(2),

3 red with a control group of those undergoing right ventricular ablation only.

4 e respiratory distress syndrome treatment on right ventricular afterload and outcome.

5 Right ventricular afterload can be analysed in terms of

6 ty for ventricular tachycardia on programmed right ventricular and burst stimulation and spontaneousl

7 0.004), AIDA-positive status with both lower right ventricular and left ventricular ejection fraction

8 ular arrhythmias commonly originate from the right ventricular and left ventricular outflow tracts (O

9 Right ventricular and metabolic measurements were limite

10 es are associated with a steady reduction of right ventricular and pulmonary arterial pressures, towa

11 stable VTs and with pacing from the atrium, right ventricular apex, and an left ventricular branch o

12 , pulmonary vascular resistance (p = 0.008), right ventricular arterial elastance (p = 0.003), and ri

13 reimplant risk factors associated with early right ventricular assist device (RVAD) use in patients u

14 mplications, including bleeding, stroke, and right ventricular assist device implantation (P<0.01 for

15 Additionally, the right ventricular blood flow KEi(EDV) E/A ratio demonstr

16 nown whether there is an association between right ventricular blood flow kinetic energy (KE) and hea

17 Four-dimensional flow (4D flow) derived right ventricular blood flow kinetic energy assessment c

18 y ageing on tricuspid through-plane flow and right ventricular blood flow kinetic energy.

19 atrial capture threshold (4%), increases in right ventricular capture threshold (4%), and increases

20 Moreover, we showed strong alteration of right ventricular cardiomyocyte excitability.

21 Indeed, adult right ventricular cardiomyocytes from Delta 71 rats exhi

22 hort action potential duration compared with right ventricular cardiomyocytes from wild-type rats.

23 ed cardiomyopathy (1/250) and arrhythmogenic right ventricular cardiomyopathy (1/5,000) are probably

24 hycardia (CPVT) (n = 9 [8%]), arrhythmogenic right ventricular cardiomyopathy (ARVC) (n = 9 [8%]), an

25 s strain imaging, to identify arrhythmogenic right ventricular cardiomyopathy (ARVC) in adolescence i

26 Arrhythmogenic right ventricular cardiomyopathy (ARVC) is a familial he

27 Arrhythmogenic cardiomyopathy/arrhythmogenic right ventricular cardiomyopathy (ARVC) is an inherited

28 Arrhythmogenic right ventricular cardiomyopathy (ARVC) is an inherited

29 Arrhythmogenic right ventricular cardiomyopathy (ARVC) is associated wi

30 Arrhythmogenic right ventricular cardiomyopathy (ARVC) is associated wi

31 Arrhythmogenic right ventricular cardiomyopathy (ARVC) is associated wi

32 Arrhythmogenic right ventricular cardiomyopathy (ARVC), with skin manif

33 cardiomyopathy, specifically arrhythmogenic right ventricular cardiomyopathy (ARVC).

34 carditis has been reported in arrhythmogenic right ventricular cardiomyopathy (ARVC).

35 d functional abnormalities in arrhythmogenic right ventricular cardiomyopathy (ARVC).

36 etic variants associated with arrhythmogenic right ventricular cardiomyopathy and of an endomyocardia

37 ional Task Force Criteria for arrhythmogenic right ventricular cardiomyopathy diagnosis and data rega

38 rnational Task Force Criteria arrhythmogenic right ventricular cardiomyopathy diagnosis was reached o

39 Arrhythmogenic right ventricular cardiomyopathy diagnostic criteria had

40 thies occur infrequently; and arrhythmogenic right ventricular cardiomyopathy is rare.

41 al variants that cause either arrhythmogenic right ventricular cardiomyopathy or dilated cardiomyopat

42 We identified arrhythmogenic right ventricular cardiomyopathy probands who met 2010 T

43 Of 501 arrhythmogenic right ventricular cardiomyopathy probands, 322 (64.3%) c

44 , DSG2, DSC2, and JUP) from 3 arrhythmogenic right ventricular cardiomyopathy registries in America a

45 P (55% versus 0% for PKP2, P<0.001), whereas right ventricular cardiomyopathy was present in only 14%

46 been variably associated with arrhythmogenic right ventricular cardiomyopathy.

47 ing dilated cardiomyopathy or arrhythmogenic right ventricular cardiomyopathy.

48 ic cardiomyopathy and none to arrhythmogenic right ventricular cardiomyopathy.

49 nt challenges associated with arrhythmogenic right ventricular cardiomyopathy/dilated cardiomyopathy

50 rt failure (HF) prevalence in arrhythmogenic right ventricular cardiomyopathy/dysplasia (ARVC/D) vari

51 ed in patients with inherited arrhythmogenic right ventricular cardiomyopathy/dysplasia, although the

52 s a specifier of outflow tract cells but not right ventricular cells, despite the failure of right ve

53 ribes normotensive patients with evidence of right ventricular compromise, whereas high-risk (massive

54 ed on echocardiography, and 40% reduction in right ventricular contractile function in isolated perfu

55 ture or tricuspid valve damage (Stage 3), or right ventricular damage or subclinical heart failure (S

56 nary artery vasculature damage), or Stage 4 (right ventricular damage).

57 ure, there was also significant reduction of right ventricular diameter as right ventricle free wall

58 Primary endpoint was the right ventricular diameter at the third rhythm analysis.

59 ltrasonographic images were obtained and the right ventricular diameter was measured.

60 d conduit functions is related to changes in right ventricular diastolic dysfunction.

61 This study identified evidence of abnormal right ventricular diastolic function in 29% of patients

62 centric left ventricular remodeling, greater right ventricular dilatation (base, 34+/-7 versus 31+/-6

63 These findings challenge the paradigm that right ventricular dilatation on ultrasound during cardio

64 itral regurgitation, pulmonary hypertension, right ventricular dilation and dysfunction, and tricuspi

65 Isolated right ventricular disease was seen in 13%, isolated left

66 Originally described as a right ventricular disease, ACM is increasingly recognize

67 nary artery vasculature damage (Stage 3) and right ventricular dysfunction (Stage 4).

68 are common in many lung diseases leading to right ventricular dysfunction and death.

69 cular diastolic dysfunction grade II or III, right ventricular dysfunction and pericardial effusions.

70 stenosis (AS), but the prognostic impact of right ventricular dysfunction has not been well studied.

71 goal-directed echocardiography in diagnosing right ventricular dysfunction in acute pulmonary embolis

72 intensivists' interpretations for evaluating right ventricular dysfunction in acute pulmonary embolis

73 The REDEFINE trial (Right Ventricular Dysfunction in Tetralogy of Fallot: In

74 Right ventricular dysfunction is an important and indepe

75 pulmonary embolism using imaging presence of right ventricular dysfunction is essential for triage; h

76 We have learned that right ventricular dysfunction may be a predictor of surv

77 ggesting that AVR should be discussed before right ventricular dysfunction occurs in severe AS.

78 ort class, use of multiple inotropes, severe right ventricular dysfunction on echocardiography, ratio

79 o four hierarchical groups: normal function, right ventricular dysfunction only (RV(dys)), left ventr

80 anced therapies being options for those with right ventricular dysfunction or unstable hemodynamics.

81 normal pressure profile to the low flow with right ventricular dysfunction profile.

82 Screening for right ventricular dysfunction using goal-directed echoca

83 T-proBNP value was elevated (910 pg/mL), and right ventricular dysfunction was moderate/severe in 55%

84 pulmonary vascular resistance (PVR) >4 WU or right ventricular dysfunction were excluded.

85 New York Heart Association functional class, right ventricular dysfunction, and atrial fibrillation (

86 versus 61+/-7 and 61+/-7 mm, P<0.0001), more right ventricular dysfunction, increased epicardial fat

87 slightly higher in patients with HF-PH with right ventricular dysfunction, pulmonary vascular remode

88 ion was 31%, and 60% had moderate or greater right ventricular dysfunction.

89 graphic indicators of worse hemodynamics and right ventricular dysfunction.

90 ed cardiomyopathy (DCM, 49%), arrhythmogenic right ventricular dysplasia (ARVD, 17%), postmyocarditis

91 ibed the arrhythmic course of arrhythmogenic right ventricular dysplasia/cardiomyopathy (ARVD/C).

92 Right ventricular ejection fraction >54%, 37-54%, and <3

93 RV dysfunction was defined as right ventricular ejection fraction </=45%.

94 Right ventricular ejection fraction <45% was associated

95 c output (+2,021 +/- 956 mL; p = 0.002), and right ventricular ejection fraction (+7.6% +/- 1.5%; p =

96 ass (-0.13 g/m(2) [-1.6 to 1.3], P=0.86), or right ventricular ejection fraction (-0.23% [-1.2 to 0.8

97 xamined the incremental value of considering right ventricular ejection fraction for the prediction o

98 action was 32+/-12% (range, 6-54%) with mean right ventricular ejection fraction of 48+/-15% (range,

99 0) while right ventricular stroke volume and right ventricular ejection fraction were decreased (p =

100 wer pulmonary arterial compliance, depressed right ventricular ejection fraction, and shorter life ex

101 entricular ejection fraction, and especially right ventricular ejection fraction-associated with prog

102 r clinical and imaging covariates, including right ventricular ejection fraction.

103 ferior leads with T-wave inversion, left and right ventricular ejection fraction.

104 of late gadolinium enhancement, and left and right ventricular ejection fractions.

105 n) underwent combined endocardial-epicardial right ventricular electroanatomical mapping and ablation

106 mm Hg; 95% CI: 1.29 to 1.76; p < 0.001), and right ventricular end-diastolic area (HR: 1.04 per cm(2)

107 Fluid loading increased right ventricular end-diastolic volume (+31 +/- 13 mL; p

108 Diuretic treatment decreased right ventricular end-diastolic volume (-84 +/- 11 mL; p

109 Right ventricular end-diastolic volume (RVDV) was 262 mL

110 t ventricular ejection fraction, and indexed right ventricular end-diastolic volume resulted in signi

111 t ventricular ejection fraction, and indexed right ventricular end-diastolic volume.

112 m(2): -3.46 mL/m(2) [-5.8 to -1.2], P=0.003, right ventricular end-diastolic volume/m(2): -4.2 mL/m(2

113 re (-7 +/- 1 mm Hg; p < 0.001) and increased right ventricular end-systolic elastance (+0.72 +/- 0.2

114 tricular arterial elastance (p = 0.003), and right ventricular end-systolic volume (p = 0.020) while

115 e (RVDV) was 262 mL (RVDV/BSA, 164 mL/m(2)); right ventricular end-systolic volume (RVSV), 198 mL (RV

116 correlated with the right atrial volume than right ventricular end-systolic volume in AF-TR (P<0.001)

117 ependent MRI predictors of death (P < 0.01): right ventricular end-systolic volume index adjusted for

118 Percentage-predicted right ventricular end-systolic volume index can identify

119 Percentage-predicted right ventricular end-systolic volume index independentl

120 nts and Main Results: A percentage-predicted right ventricular end-systolic volume index threshold of

121 othelia in formalin-fixed, paraffin-embedded right ventricular endomyocardial biopsies is diagnostic

122 levated left- and right-sided pressures, and right ventricular enlargement were independently predict

123 Right ventricular failure (RVF) is a cause of major morb

124 pulmonary perfusion, ultimately resulting in right ventricular failure and dilation.

125 In addition, Poly(I:C) also reduced right ventricular failure in established pulmonary hyper

126 ermine if patient survival and mechanisms of right ventricular failure in pulmonary hypertension coul

127 Additional right ventricular failure predisposed to futility (hazar

128 n the pulmonary arteries, often resulting in right ventricular failure with shortness of breath and s

129 ing acute pulmonary vascular dysfunction and right ventricular failure.

130 d, TR can progress and result in progressive right ventricular failure.

131 tality, renal replacement therapy, or severe right ventricular failure.

132 swings (obliteration during inspiration) in right ventricular filling and pulmonary perfusion, ultim

133 Despite growing interest in right ventricular form and function in diseased states,

134 ht ventricular cells, despite the failure of right ventricular formation in Hand2-null mice(4).

135 In 12 sheep, 8 weeks of right atrial and right ventricular free wall (DDD) pacing lead to LV dila

136 rial strain, and peak longitudinal strain of right ventricular free wall (odds ratios: 1.45 [95% conf

137 icuspid annular plane systolic excursion and right ventricular free wall strain).

138 ic excursion (13%) (P<0.05 for all), but not right ventricular free wall strain.

139 tricular size (94% versus 80%; P=0.001), and right ventricular function (87% versus 73%; P=0.006).

140 tricuspid regurgitation velocity; and worse right ventricular function (tricuspid annular plane syst

141 paucity of data regarding characteristics of right ventricular function - namely contractile and lusi

142 ing signs of abnormal diastolic and systolic right ventricular function and compression of the atriov

143 left ventricular longitudinal strain (LVLS), right ventricular function and right ventricular systoli

144 3) developing standard methods for assessing right ventricular function and, hopefully, its coupling

145 hile LA compliance, LA reservoir strain, and right ventricular function decreased with increasing AF

146 d pulmonary arterial compliance, and reduced right ventricular function.

147 alveolar counts (RACs), vessel density, and right ventricular hypertrophy (RVH).

148 t ventricular systolic pressure measurement, right ventricular hypertrophy, and pulmonary distal arte

149 proving right ventricular systolic pressure, right ventricular hypertrophy, cardiac fibrosis, and vas

150 ulmonary hypertension that directly leads to right ventricular hypertrophy, decompensated right-sided

151 PE + SU produced right ventricular hypokinesis, dilation, and hypertrophy

152 some of these anomalies are partially due to right ventricular insufficiency, recent data support a m

153 % subendocardial, 71% transmural), including right ventricular LGE (96%).

154 Finally, CMR-derived right ventricular mass showed considerable heritability

155 gle x 42.7 + log(10) ventricular mass index (right ventricular mass/left ventricular mass) x 7.57 + b

156 Right ventricular measurements were also correlated for

157 used to create a three-dimensional model of right ventricular motion.

158 le overestimation resulted from inclusion of right ventricular myocardium (n=37; 38.1%), LV trabecula

159 flow tract myocardium specification, whereas right ventricular myocardium was specified but failed to

160 ss of myocytes and fibrofatty replacement of right ventricular myocardium; biventricular involvement

161 Right ventricular NI PRSW moderately correlated to CI fo

162 catheter ablation for arrhythmias beyond the right ventricular OT a feasible option for cure-indeed a

163 homograft (41%), bioprosthesis (30%), native right ventricular outflow tract (RVOT) (27%) and other (

164 aracterized overall and according to type of right ventricular outflow tract (RVOT) anatomy.

165 There was no difference in E12 in the right ventricular outflow tract compared with the right-

166 (PPVI) has become an important treatment of right ventricular outflow tract dysfunction.

167 The 3-year mean right ventricular outflow tract echocardiographic gradie

168 abnormal electric activity in the epicardial right ventricular outflow tract may be beneficial in pat

169 increase in the risk of Ebstein's anomaly (a right ventricular outflow tract obstruction defect) in i

170 The prevalence of right ventricular outflow tract obstruction defects was

171 TOF is associated with various types of right ventricular outflow tract obstruction ranging from

172 ht ventricle was dilated without evidence of right ventricular outflow tract obstruction.

173 ile all others had no clinically significant right ventricular outflow tract obstruction.

174 activity were then evaluated in response to right ventricular outflow tract PVCs with fixed short, f

175 clinical entity of an isolated subepicardial right ventricular outflow tract scar serving as a substr

176 alve regurgitation in patients with repaired right ventricular outflow tracts.

177 or treatment for patients with dysfunctional right ventricular outflow tracts.

178 rdiography imaging of the appendage from the right ventricular outflow.

179 Right ventricular pacing (RVP) increases risk of atrial

180 own to occur among some patients who receive right ventricular pacing (RVP).

181 7, intraventricular conduction defect 5, and right ventricular pacing 5) referred for CRT in addition

182 high risk of developing HF in the setting of right ventricular pacing and to determine whether these

183 We then apply it to study right ventricular pacing induced electromechanical dyssy

184 and thus presumed to have a higher burden of right ventricular pacing, experienced an increased risk

185 h current leadless pacemakers are limited to right ventricular pacing, future advanced, communicating

186 Right ventricular pacing-induced dyssynchrony substantia

187 endurance training has been associated with right ventricular pathological remodeling and ventricula

188 ning arrhythmogenic cardiomyopathy, often of right ventricular predominance.

189 de B is up-regulated in both ventricles with right ventricular preference.

190 pulmonary embolism, fluid loading increased right ventricular preload and right ventricular stroke v

191 r stroke volume, whereas diuretics decreased right ventricular preload and right ventricular stroke v

192 ame amount of particulate intake, changes in right ventricular pressure and intimal thickening of pul

193 nocrotaline-treated rats developed increased right ventricular pressure and mass, along with right at

194 of elevated P(PL) on hemodynamics, left and right ventricular pressures and pulmonary vascular resis

195 es of the human disease, including increased right ventricular pressures, medial thickening, neointim

196 Future studies should assess the impact of right ventricular protective acute respiratory distress

197 lbuterol enhanced cardiac output reserve and right ventricular pulmonary artery coupling, reduced rig

198 ortic approach) alone or in combination with right ventricular (RV) (LVs+RV), BiV, and HB pacing was

199 sist device (LVAD) recipients, and increased right ventricular (RV) afterload may contribute.

200 nce (CMR) imaging is recommended to quantify right ventricular (RV) and left ventricular (LV) functio

201 Right ventricular (RV) and left ventricular (LV) volumes

202 ters were analyzed, as well as comprehensive right ventricular (RV) and left ventricular assessment o

203 iastolic function and valve hemodynamics and right ventricular (RV) assessment, as well as lung ultra

204 erizing structural changes of arrhythmogenic right ventricular (RV) cardiomyopathy are limited.

205 Assessment of right ventricular (RV) diastolic function is not routine

206 Right ventricular (RV) dilatation persisted at follow-up

207 smaller absolute but greater indexed LV and right ventricular (RV) dimensions as compared to males.

208 ing pandemic that confers augmented risk for right ventricular (RV) dysfunction and dilation; the pro

209 nation may contribute to long-term pulmonary right ventricular (RV) dysfunction in patients after sur

210 tricuspid regurgitation, timely detection of right ventricular (RV) dysfunction with conventional 2-d

211 LVEF is preserved or there is a concomitant right ventricular (RV) dysfunction.

212 Whether right ventricular (RV) ejection fraction (RVEF) can iden

213 s, aged 13.0+/-2.9 years, had higher indexed right ventricular (RV) end-diastolic (range 85-326 mL/m(

214 Right ventricular (RV) end-systolic dimensions provide i

215 is a degenerative arteriopathy that leads to right ventricular (RV) failure.

216 with high morbidity and mortality because of right ventricular (RV) failure.

217 The role of right ventricular (RV) fibrosis in pulmonary hypertensio

218 We report right ventricular (RV) filling and ejection abnormalitie

219 Knowledge of right ventricular (RV) function has lagged behind that o

220 ionship between parasympathetic activity and right ventricular (RV) function in patients with PAH, an

221 Right ventricular (RV) function is an important determin

222 Although it is known that right ventricular (RV) function is dependent on LV healt

223 ffect of angiotensin II receptor blockers on right ventricular (RV) function is still unknown.

224 Right ventricular (RV) function was determined by echoca

225 tensin II has been implicated in maladaptive right ventricular (RV) hypertrophy and fibrosis associat

226 Right ventricular (RV) impairment is postulated to be re

227 tely negative in 14 patients (10%), isolated right ventricular (RV) involvement was found in 58 (41%)

228 Additionally, right ventricular (RV) lead length was associated with d

229 Right ventricular (RV) maladaptation and failure determi

230 Right ventricular (RV) morphology has been associated wi

231 is an established therapy for dysfunctional right ventricular (RV) outflow tract conduits.

232 Right ventricular (RV) outflow tract obstruction (RVOTO)

233 Endocardial pacemaker leads and right ventricular (RV) pacing are well-known causes of t

234 Conventional right ventricular (RV) pacing, particularly RV apical pa

235 Right ventricular (RV) pacing-induced cardiomyopathy (PI

236 ivided into four left ventricular (LV) and a right ventricular (RV) segment on mid-ventricular short

237 , the decision to intervene is influenced by right ventricular (RV) size and function.

238 Controls had advanced liver disease, right ventricular (RV) systolic pressure <40 mm Hg, and

239 y of Fallot provides symptomatic benefit and right ventricular (RV) volume reduction.

240 inded quantification of left ventricular and right ventricular (RV) volumes was performed from standa

241 Patients were categorized by right ventricular (RV), left dominant (LD), or biventric

242 ove short-term computed tomographic-measured right ventricular (RV)-to-left ventricular diameter rati

243 Rationale: Cor pulmonale (right ventricular [RV] dilation) and cor pulmonale parvu

244 ar electroanatomical mapping and ablation of right ventricular scar-related ventricular tachycardia w

245 nselective (NS) His bundle pacing (HBP), and right ventricular septal capture in routine clinical pra

246 e differentiation between S-HBP, NS-HBP, and right ventricular septal capture morphologies by careful

247 We performed RNA sequencing on right ventricular septal endomyocardial biopsies prospec

248 ely differentiate between S-HBP, NS-HBP, and right ventricular septal pacing with a cumulative positi

249 us criteria for HFpEF (n=41) contrasted with right ventricular septal tissue from patients with HF wi

250 These leadless devices are self-contained right ventricular single-chamber pacemakers implanted by

251 ventricular size (96% versus 83%; P<0.001), right ventricular size (94% versus 80%; P=0.001), and ri

252 ssociated with a 10-20% increase in left and right ventricular size and a substantial increase in lef

253 strated improvement in functional status and right ventricular size and function as shown by echocard

254 ted echocardiogram as normal or abnormal for right ventricular size and function in patients with acu

255 natriuretic peptide serum concentration, and right ventricular size and function.

256 ventricular strain, left atrial strain, and right ventricular strain) are also discussed.

257 diastolic volume (+31 +/- 13 mL; p = 0.004), right ventricular stroke volume (+23 +/- 10 mL; p = 0.00

258 diastolic volume (-84 +/- 11 mL; p < 0.001), right ventricular stroke volume (-40 +/- 6 mL; p = 0.001

259 icular end-systolic volume (p = 0.020) while right ventricular stroke volume and right ventricular ej

260 tics decreased right ventricular preload and right ventricular stroke volume without affecting mean a

261 ding increased right ventricular preload and right ventricular stroke volume, whereas diuretics decre

262 /m(2): -3.0 mL/m(2) [-4.5 to -1.5], P<0.001; right ventricular stroke volume/m(2): -3.8 mL/m(2) [-6.5

263 tion and to analyze their clinical value for right ventricular substrate delineation.

264 e detected between malformation indexes with right ventricular systolic and diastolic findings (P < .

265 associated with significant deterioration of right ventricular systolic function and greater tricuspi

266 Right ventricular systolic function was significantly lo

267 1.68; 95% CI: 1.12 to 2.51; p = 0.012), and right ventricular systolic pressure >=50 mm Hg (HR: 2.27

268 TMG >=8 mm Hg and right ventricular systolic pressure >=50 mm Hg were inde

269 n (TR) (7%, 35%, and 53%, respectively), and right ventricular systolic pressure (32 +/- 11, 45 +/- 1

270 The distribution of estimated right ventricular systolic pressure (eRVSP) was examined

271 uivalents (HR, 1.22), and higher peak-stress right ventricular systolic pressure (HR, 1.35), was asso

272 t ventricular volumes (p = 0.005) and higher right ventricular systolic pressure (p < 0.0001).

273 train (LVLS), right ventricular function and right ventricular systolic pressure (RVSP) was performed

274 asurements indicated modest increases in the right ventricular systolic pressure and right ventricle

275 Improved MR severity and reduced right ventricular systolic pressure at 30 days are assoc

276 MR grade and estimated right ventricular systolic pressure at 30 days were impr

277 xia-induced pulmonary hypertension judged by right ventricular systolic pressure measurement, right v

278 Following 3 months of altitude exposure, right ventricular systolic pressure was measured (solid-

279 lic equivalents; peak-stress MV gradient and right ventricular systolic pressure were 17+/-7 and 61+/

280 Society of Thoracic Surgeons score and right ventricular systolic pressure were 2+/-3 and 15+/-

281 Mean resting MV gradient and right ventricular systolic pressure were 8.5+/-3 and 39+

282 ated rats) palbociclib reverses the elevated right ventricular systolic pressure, reduces right heart

283 416/hypoxia/normoxia rat model, by improving right ventricular systolic pressure, right ventricular h

284 rk Heart Association functional class and on right ventricular systolic pressure, volumes, and dimens

285 etabolic equivalents, and higher peak-stress right ventricular systolic pressure, while invasive MV p

286 ration time associated with elevation of the right ventricular systolic pressure.

287 increased systemic blood pressure as well as right ventricular systolic pressure.

288 tractility and decreased systemic as well as right ventricular systolic pressure.

289 right ventricular systolic pressures in BERK-SS were hig

290 ) altitude increased rates of haemolysis and right ventricular systolic pressures in mice with SCD co

291 cially in the absence of methods to quantify right ventricular systolic pressures.

292 ttransplant formalin-fixed paraffin-embedded right ventricular tissue biopsies (14 positive for C4d a

293 Patients with symptomatic PE and right ventricular to left ventricular diameter ratio >=0

294 dverse events and a significant reduction in right ventricular to left ventricular diameter ratio and

295 At 48 hours post-BEC therapy, the right ventricular to left ventricular diameter ratio dec

296 endently predict pulmonary artery pressures, right ventricular-to-left ventricular (RV/LV) diameter r

297 e disease, when progressive dilation begins, right ventricular volume is the essential parameter to m

298 l of chronic pulmonary insufficiency causing right ventricular volume overload.

299 However, both LV and right ventricular volumes were significantly smaller in

300 zygous adult mice exhibit hyperplasia in the right ventricular wall.