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1 tality, renal replacement therapy, or severe right ventricular failure.
2 nary vascular resistance and, eventually, in right ventricular failure.
3 remodeling of the pulmonary vasculature, and right ventricular failure.
4 monary artery pressure, often culminating in right ventricular failure.
5 d right ventricular dysfunction and nine had right ventricular failure.
6 tor dysfunction, pulmonary hypertension, and right ventricular failure.
7 athophysiology, assessment and management of right ventricular failure.
8 rategies for the diagnosis and management of right ventricular failure.
9 ngle published practice guideline focused on right ventricular failure.
10 cardiothoracic surgery, require therapy for right ventricular failure.
11 ing acute pulmonary vascular dysfunction and right ventricular failure.
12 in patients with pulmonary hypertension and right ventricular failure.
13 Three animals that received L-NAME died of right ventricular failure.
14 lism-related mortality associated with acute right ventricular failure.
15 abnormally elevated pulmonary pressures and right ventricular failure.
16 entricular tachycardia, and (5) treatment of right ventricular failure.
17 d, TR can progress and result in progressive right ventricular failure.
18 icular unloading, protection of kidneys, and right ventricular failure.
19 lerance and early mortality due to systemic (right) ventricular failure.
20 to right atrial pressure, is a predictor of right ventricular failure after inferior myocardial infa
22 ulmonary hypertension, which can progress to right ventricular failure, an important cause of morbidi
27 so had a higher incidence of post-transplant right ventricular failure and overall mortality (P<0.05)
29 rue in transplant recipients who suffer from right ventricular failure and rejection and may undergo
37 tinely in their practice, but until recently right ventricular failure as a primary clinical entity r
38 y of beta-blockers in patients with isolated right ventricular failure because of pulmonary arterial
39 erioperative period were not attributable to right ventricular failure (chronic thromboembolic pulmon
40 bleeding, infection, neurologic events, and right ventricular failure continue to limit broader impl
41 Careful perioperative attention to avoid right ventricular failure from acutely elevated pulmonar
44 equences of impaired adrenergic signaling in right ventricular failure/hypertrophy (RVH) are poorly u
46 ermine if patient survival and mechanisms of right ventricular failure in pulmonary hypertension coul
47 w will focus on the pathophysiology of acute right ventricular failure in the critical care setting a
49 The most common serious adverse events were right ventricular failure (in 3% of patients in each gro
53 reliable hemodynamic threshold beyond which right ventricular failure is certain to occur, nor are t
55 g and discusses the pathophysiology of acute right ventricular failure, its differential aetiologies,
57 rapeutics, critical illness, intensive care, right ventricular failure, mitral stenosis, prostacyclin
58 increased pulmonary vascular resistance and right ventricular failure; morbidity and mortality remai
60 n our cohort died of their disease; however, right ventricular failure or sudden death was the sole c
62 ailure post-heart transplantation in 22, and right ventricular failure post-implantable left VAD in 1
65 e by using the search words right ventricle, right ventricular failure, pulmonary hypertension, sepsi
66 ions regarding the optimal method to predict right ventricular failure resurface, along with a modern
67 area ratio>0.6) of whom four patients had a right ventricular failure (right ventricular end-diastol
69 the likelihood of developing post-operative right ventricular failure (RV failure) in the setting of
71 Failure Risk Score was developed to predict right ventricular failure (RVF) after left ventricular a
72 URPOSE OF REVIEW: Pulmonary hypertension and right ventricular failure (RVF) in left ventricular syst
78 cted, overall and by shock etiology: left or right ventricular failure versus mechanical complication
83 understanding of the molecular mechanisms of right ventricular failure will lead to the development o
84 n the pulmonary arteries, often resulting in right ventricular failure with shortness of breath and s