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1 in the mice at one minute after the loss of righting reflex.
2 curs at the point of dexmedetomidine loss of righting reflex.
3 , sleep onset and anesthetic-induced loss of righting reflex.
4 A(A) receptors, for propofol-induced loss of righting reflex.
5 of adrenalectomy on ethanol-induced loss of righting reflex.
6 uced the duration of ethanol-induced loss of righting reflex.
7 drugs did not alter the duration of loss of righting reflex.
8 t concentrations associated with loss of the righting reflex.
9 of alcohol on locomotor activity or loss-of-righting reflex.
10 ncoordination or ethanol-induced loss of the righting reflex.
11 esia, expressed as decreased time to loss of righting reflex (-0.59), increased burst-suppression rat
12 with alcohol on locomotor activity, loss-of-righting reflex (a measure of alcohol sedative actions),
13 he potency for ketamine to provoke a loss-of-righting reflex, a behavioral correlate of hypnosis, was
14 erature, appearance of the fur and whiskers, righting reflex, acoustic startle, eye blink, pupil cons
16 social isolation on the PTB-induced loss of righting reflex and on the decrease of telencephalic All
17 se to injection of pentobarbital and loss of righting reflex and response to tail clamp stimulus in m
18 tive steroids in the ethanol-induced loss of righting reflex and the source of ethanol-induced elevat
19 ificant motor deficits (e.g., grip strength, righting reflex and touch escape) in bf mutants, worseni
20 and the key mechanisms involved in dragonfly righting reflexes and to develop physics-based models fo
21 ts of alcohol on locomotor activity, loss of righting reflex, and in an acute intoxication severity s
22 s are characterized by wild running, loss of righting reflex, and tonic flexion and extension, and ar
26 KO and WT mice did not differ in loss of righting reflex, but mutant mice displayed a delayed ons
27 f N(2)O to CO(2) lowered the time to loss of righting reflex by 10.3% (P<0.001), lead to a lower P(a)
29 ensory (less pain responsiveness and failing righting reflex) deficits that coincided with global low
31 , and a decrease in duration of, the loss of righting reflex following dexmedetomidine; hypnotic resp
36 potency than N/OFQ in eliciting loss of the righting reflex in mice and produced a long-lasting effe
41 nd evaluated at one minute after the loss of righting reflex in the mice, which was about two minutes
42 hortened alcohol-induced sleep time (loss of righting reflex) in rats that were given ethanol intraga
43 ever, the latency to ethanol-induced loss of righting reflex increased and the duration decreased in
45 40%), expressed by shorter times in loss of righting reflex (LORR) in response to a sedative dose of
46 hree screens of biological activity: loss of righting reflex (LORR) in tadpoles, enhancement of agoni
47 e was a reduction in the duration of loss of righting reflex (LORR) to a high dose of ethanol in the
48 Our data suggest that latency to recover the righting reflex may be an inadequate measure of injury s
49 concentration of ethanol causing loss of the righting reflex of rats was 82.3+/-2.9 mM, whereas media
52 asured isoflurane effects on time to loss of righting reflex, onset of continuous slow wave activity,
54 TBI increased the time to the return of the righting reflex, reduced grip force, altered sociability
59 ion (loss of movement) and hypnosis (loss-of-righting reflex), TASK knock-out mice showed a modest de
62 ificantly inhibits the PXR-activated loss of righting reflex to 2,2,2-tribromoethanol (Avertin) in vi
63 the ethanol dose-response curve for loss of righting reflex to lower concentrations and that this ef
64 pe is characterized by wild running, loss of righting reflex, tonic flexion, and tonic extension in r
65 y to isoflurane- or diazepam-induced loss of righting reflex was decreased at 12 and 24 h after EtOH
67 alpha-THP levels and the duration of loss of righting reflex were obtained regardless of finasteride
68 L-Name, by itself, did not produce a loss of righting reflex, while 6 out of 9 rats pretreated with L