ライフサイエンスコーパス: rising phase

1 ising phase, followed by a larger and slower rising phase.

2 ent that is followed by a slower and delayed rising phase.

3 The OFF Idelta component showed a rising phase.

4 ial components with time constants of 13 ms (rising phase), 70 ms and 350 ms (decaying phase).

5 total Ca2+ to the end-pools accelerated the rising phase and abbreviated the duration of the hump.

6 ouse brain and an increased action potential rising phase and greater excitability of hippocampal pyr

7 ibuted in part to lower amplification in the rising phase and in part to faster recovery kinetics.

8 ces are known to generate, respectively, the rising phase and the prolonged plateau phase of cerebell

9 very recording: monophasic EPSCs, with sharp rising phases and monoexponential decays, and multiphasi

10 rhodopsin in transgenic mice accelerates the rising phases and recoveries of flash responses by about

11 ist and directly elicited currents with slow-rising phases and submaximal amplitudes.

12 mmetrical shapes with accelerated synthetic (rising) phases and extended degradative (falling) phases

13 ted by two exponentials with means of 45 ms (rising phase) and 4408 ms (decaying phase) components.

14 (2+) signal revealed diverse kinetics of the rising phase, and hence various rates in the release of

15 ude at a given intensity of stimulus, faster rising phase, and shorter latencies than those of ISS-EP

16 ence of extracellular Ca(2+) showed a normal rising phase but a slower decay phase, resulting in long

17 within a few seconds, accompanied during its rising phase by a short burst of action potentials.

18 We further show that the rising phase depends on Ca(2+) feedback to the cyclase,

19 an approximate 20-y periodicity, including a rising phase during the 1990s that contributed to the pe

20 als, evoked calcium transients showed a fast-rising phase followed by a decay with a time constant of

21 ng sperm entry in vertebrate eggs has a slow rising phase followed by a sustained plateau.

22 iate [Ca2+]o bumps were biphasic with a slow rising phase followed by rapid amplification and inactiv

23 ransients composed of a small-amplitude fast rising phase, followed by a larger and slower rising pha

24 -140) divided by the area under curve of the rising phase (frames 24-40) in the fourth mode, i.e. the

25 ial EMG peaks were most prevalent during the rising phase in both wake and NREM.

26 The EPSC amplitude is enhanced, and its rising phase is accelerated under relatively low IR ligh

27 ysis corrects a previous assumption that the rising phase is determined entirely by activation rates.

28 outward current and a slow component to the rising phase of [Ca(2+)](i).

29 constrain recruitment of IP(3)Rs during the rising phase of a Ca(2+) puff and closure of IP(3)Rs dur

30 ansient tension rise when applied during the rising phase of a twitch contraction, the amplitude of w

31 hillock regions and propagating the initial rising phase of action potentials through axons.

32 iratory component that closely resembled the rising phase of burstlets.

33 s I, II, and IV, but not VSDIII, overlap the rising phase of charge moved, and even more for Ca(2+) r

34 ponse to light that suggests a defect in the rising phase of clock protein expression.

35 aP with 5 microm GABA resulted in a biphasic rising phase of current with time constants of 50-60 ms

36 annels; the first peak of the differentiated rising phase of depolarizations - attributed to the infl

37 he activation of NMDA receptors and that the rising phase of each calcium spike is coincident with a

38 We find the rising phase of global Ca(2+) signals is punctuated by a

39 4, WD25, WD36, or WD60) selected to span the rising phase of incubation and the transition from low t

40 The variability between the rising phase of individual sIPSCs was quantified by calc

41 The rising phase of LLDs shows characteristics of polyneuron

42 n to those from lactating rats in vitro, the rising phase of male bursts was more rapid, the decay ph

43 s consistently occurred during the days-long rising phase of multidien cycles of IEA.

44 ed by fitting an exponential function to the rising phase of responses to supramaximal serotonin to b

45 From analysis of the rising phase of rod-isolated flash responses we determin

46 hese bursting neurons fired primarily on the rising phase of SAM or the onset of unmodulated stimuli,

47 Standard deviation functions of the rising phase of simulated IPSCs accurately described dis

48 Unexpectedly, the rising phase of somatic APs is electrically indistinguis

49 The rising phase of such sIPSCs were multi-phasic, composed

50 rial dehydrogenases were synchronized to the rising phase of the [Ca(2+)](c) spikes.

51 iliary beta subunits are responsible for the rising phase of the action potential in cardiac muscle,

52 iliary beta subunits are responsible for the rising phase of the action potential in cardiac muscle,

53 (+) channels (VGSCs) are responsible for the rising phase of the action potential in excitable cells,

54 e was also significant Ca2+ entry during the rising phase of the action potential.

55 ients at the axon initial segment during the rising phase of the AP, suggesting complex effects on ch

56 This activity also correlated with the rising phase of the arterial blood pressure wave consist

57 llations by allowing Ca(2+) entry during the rising phase of the Ca(2+) spikes and by providing an OF

58 The rising phase of the Ca(2+) transient was correlated with

59 dback ( approximately 2-4 microm) shapes the rising phase of the Ca2+ signal by acting to co-ordinate

60 y declined (to about 60% of peak) during the rising phase of the cell-wide Ca2+ transient before incr

61 The rising phase of the center response was slower for large

62 ctivation of cone transduction; that is, the rising phase of the cone response per bleached rhodopsin

63 Background light did not affect the rising phase of the dim flash response, a measure of the

64 ERG recordings demonstrated a delay in the rising phase of the ERG b-wave, larger photopic b-wave a

65 of recurrent IPSPs did not occur during the rising phase of the evoked EPSP, but > 3.0 ms after the

66 bserved between monosynaptic g GABAA and the rising phase of the evoked EPSP/EPSC.

67 er BDZ receptor ligand upon the slope of the rising phase of the evoked population excitatory postsyn

68 phase of jumps occurs on average during the rising phase of the firing rate of the looming-sensitive

69 the effect and induced a speeding up of the rising phase of the flash response, similar to the actio

70 mutant subunit was expressed with GluK4, the rising phase of the glutamate steady state concentration

71 ar dendrites, Ret-RGS1 should accelerate the rising phase of the light response of the ON bipolar cel

72 -dependent potentiation and the delay in the rising phase of the mutant ACh response suggest that the

73 The onset of the rising phase of the photoresponse was significantly dela

74 umber of IP(3)Rs for 19 ms, representing the rising phase of the puff.

75 sets of feedforward IPSPs coincided with the rising phase of the pyramidal cell EPSP in > 80 % of pai

76 We show that the rising phase of the response depends on Ca(2+) feedback,

77 "deceleration-acceleration" notch during the rising phase of the response, as a signature of fast int

78 d attention was focused on the initial rapid rising phase of the responses.

79 molecule (Rh*) has a smaller slope than the rising phase of the rod response per Rh*, perhaps becaus

80 e rapid, the decay phase was slower, and the rising phase of the spike after hyperpolarization was fa

81 entry was nearly completely confined to the rising phase of the spike: only approximately 25% more s

82 ted reaction-diffusion of [Ca2+]i during the rising phase of the transient (first 30 ms after initiat

83 colliculus during the middle portion of the rising phase of the whisker movement protraction elicite

84 The earliest rising phase of this 'fractional response per unit inten

85 The rising phases of transients obtained from global illumin

86 sence of Na-current after, the regenerative (rising) phase of the action potential.

87 The resulting gating current exhibited a rising phase similar to that measured in voltage-depende

88 (2) slIPSCs frequently had an notched rising phase suggestive of summated IPSCs resulting from

89 smaller events exhibited a stammer in their rising phase that is interpreted as a brief pause in por

90 eases of [Ca(2+)](m) characterized by a fast rising phase that was followed by a slow decay.

91 t prolong the response decay, but caused its rising phase to become slightly sigmoidal by giving rise

92 that LCRs impart a nonlinear, exponentially rising phase to the DD later part, which exhibited beat-

93 amp methods were optimized for the fast MEPC rising phase using custom electronics.

94 prolonged in the presence of CPA, while the rising phase was unaffected.

95 a biphasic pattern consisting of an initial rising phase with a median half-time ( SD) of 23 15 h an

96 a biphasic pattern consisting of an initial rising phase with a median half-time (+/-SD) of 23 +/- 1

97 tail currents at -40 mV displayed an initial rising phase with tau = 10 msec, followed by a slow mult