ライフサイエンスコーパス: rod

1 quantum dots) and shapes (e.g., spheres and rods).

2 ic dodecahedra, stacked clusters, and smooth rods).

3 COFs with valency 8 (cubes) and "infinity" (rods).

4 simulations to study unfolding of the talin rod.

5 a para-urethral steel rod, then removing the rod.

6 ttice array of concave hexagonal (CH)-shaped rods.

7 machine learning to optimize the biomimetic rods.

8 mplifying assumption that cells are straight rods.

9 e cross-sections, which turn them into rigid rods.

10 neralization in vitro, as observed in enamel rods.

11 and rods, but it was more pronounced in the rods.

12 owed by streptococci (26%) and Gram negative rods (18%).

13 ents), IgG4-related ophthalmic disease (IgG4-ROD) (5 scans, 3 patients), and granulomatosis with poly

14 on sole [S(472), M(523), M(536), L(559), and rod(511)] corresponding to the repertoire of transcripts

15 ly progressive degeneration of predominantly rods, accompanied by rhodopsin and blue cone opsin mislo

16 visual function as measured by HVF diameter, rod amplitude, flicker amplitude, and flicker implicit t

17 f blindness, ranging from rod dysfunction to rod and cone degeneration.

18 ast, Prph2R172W/Rom1+/- animals had worsened rod and cone function and exacerbated retinal degenerati

19 Prph2K/+/Rom1+/- mice had improved rod and cone function compared with Prph2K/+ as well as

20 ed overall Prph2 levels as well as decreased rod and cone function.

21 nal, the light-sensitive chromophore of both rod and cone opsin visual pigments.

22 ance the expression of the visual receptors, rod and cone opsins; inhibit the inflammatory reactions;

23 erations in visual processing common to both rod and cone pathways, these mechanisms are probably dow

24 ffects visual information processing in both rod and cone pathways.

25 e and regulation of the distinct isoforms of rod and cone PDE6.

26 Ca(2+) dysregulation is thought to cause rod and cone photoreceptor cell death.

27 The outer segments (OS) of rod and cone photoreceptor cells are specialized sensory

28 We applied ncRNA profiling to identify rod and cone photoreceptor CREs from wild-type and mutan

29 panin protein essential for the formation of rod and cone photoreceptor outer segments (OS).

30 Rod and cone photoreceptors convert light into electroch

31 In mammalian retinas, rod and cone photoreceptors form selective synaptic conn

32 Retinal rod and cone photoreceptors mediate vision in dim and br

33 Rod and cone photoreceptors of the retina are responsibl

34 gins when photons of the light interact with rod and cone photoreceptors that are present in the neur

35 P) is a highly expressed protein secreted by rod and cone photoreceptors that has major roles in phot

36 esponsive cell types, including Muller glia, rod and cone photoreceptors, and bipolar cells.

37 ongly correlated with epigenetic profiles of rod and cone photoreceptors, identified thousands of can

38 phore associated with the visual pigments of rod and cone photoreceptors.

39 phore associated with the visual pigments of rod and cone photoreceptors.

40 were plotted to summarise the topography of rod and cone pigment kinetics and descriptive statistics

41 ients (10/26) showed evidence of generalized rod and cone system dysfunction.

42 Rod and cone visual pigment synthesis rates in those wit

43 olerae and the benefits of switching between rod and curved shape have not been determined.

44 Relationships between double-Maddox rod and other tests were evaluated by calculating mean d

45 Furthermore, mmachc mutants bred to express rod and/or cone fluorescent reporters, manifested a reti

46 severely impaired self-association in mouse rods and analyzed mice of both sexes.

47 th time) at mesopic light levels, where both rods and cones actively respond to light.

48 lume, the most important differences between rods and cones are: (1) decreased transduction gain, ref

49 Extracellular activity from populations of rods and cones generate the negative-going a-wave, while

50 e functional role of GRK1 phosphorylation in rods and cones in vivo, we generated mutant mice in whic

51 l differences in the light responsiveness of rods and cones may result in part from differences in th

52 Rods and cones use intracellular Ca(2+) to regulate many

53 The function of dark-adapted GRK1-S21A rods and cones was also unaffected, as demonstrated by t

54 cessive LCA1 blindness, both of which affect rods and cones, but they cannot explain the selective lo

55 enough to exert gain-of-function defects in rods and cones.

56 n the retina, including the photon-sensitive rods and cones.

57 Muscle biopsy shows rods and fiber type disproportion.

58 see text] is the density difference between rods and fluid, mu is the fluid's dynamic viscosity, g i

59 The reduction in rods and red/green cones correlated with defects in phot

60 l phenotypes, including decreased numbers of rods and red/green cones, whereas blue and UV cones were

61 atical representation of geometrically exact rods and the generalization of DNA basepair step paramet

62 oexistence of different metal sizes in their rods and therefore assume various metal sequences.

63 thy controls (v = 0.079 SD 0.024 min(-1) for rods and v = 0.206 SD 0.069 min(-1) for cones).

64 ynoptophore targets, 118 using single-Maddox rod, and 43 using fundus photography.

65 system can produce spherical nanoparticles, rods, and finally octahedral nanoparticles by using our

66 o different sets of cones, H4 feed back onto rods, and only H1 store and release the inhibitory neuro

67 esses, consistent with the absence of enamel rods, and underwent progressive cell pathology throughou

68 We examined associations between rod- and cone-mediated vision and HRF plus smaller hyper

69 receptors, identified thousands of candidate rod- and cone-specific CREs, and identified motifs for r

70 tors accompanied by marked decreases of both rod- and cone-specific gene expression.

71 one-specific CREs, and identified motifs for rod- and cone-specific TFs.

72 Once the rods are fixed with desirable orientations in a bimorph

73 s result in a Class B phenotype where mutant rods are retained in some retinal regions but show sever

74 models that treat the DNA helices as elastic rods are used to evaluate the local loss of stiffness at

75 demonstrate practical application of HD-CNTf rod arrays, detection of DA in human biological fluids a

76 In rods, arrestin-1 moves from the inner segments and cell

77 s a similar or higher encounter rate between rods as compared to the encounter rate between (equal vo

78 ahedral nanoparticles by using our optimized rods as seeds.

79 velopmental state, and potentiation of rod - rod bipolar cell signaling following rod photoreceptor d

80 ipts were previously shown to be enriched in rod bipolar cells (BCs), secondary neurons of the retina

81 PKCalpha-dependent phosphoprotein in retinal rod bipolar cells (RBCs).

82 d at GABAergic synapses on axon terminals of rod bipolar cells (RBCs).

83 LRRTM4 is expressed specifically by rod bipolar cells; eliminating it in mouse retina pertur

84 rea and sarcomeres are disorganized, contain rod bodies, and have longer thin filaments.

85 of GRK1 in regulating the dark adaptation of rod but not cone photoreceptors.

86 uter nuclear layer thickness; and peripheral rod, but not cone number, was reduced.

87 Vibrio cholerae typically exists as a curved rod, but straight rods have been observed under certain

88 This asymmetry was observed in cones and rods, but it was more pronounced in the rods.

89 of dysfunction and degeneration of retained rods by serially evaluating patients.

90 ion in the cylindrical part of the cell: the Rod complex and the class-A penicillin-binding proteins

91 While the Rod complex governs rod-like cell shape, aPBP function i

92 othesized to either work in concert with the Rod complex or to independently repair cell-wall defects

93 shape in Escherichia coli is the processive 'Rod complex'.

94 -particle tracking of the transpeptidase and Rod-complex component PBP2, we found that PBP2 binds to

95 d localization experiments of other putative Rod-complex components provide evidence that none of tho

96 correlations do not require curvature-based Rod-complex initiation but can be attributed to persiste

97 cell-wall architecture provides the cue for Rod-complex initiation, either through direct binding by

98 ught to govern formation and localization of Rod complexes based on local cell-envelope curvature.

99 Reduced mixed rod-cone a- and b-wave amplitudes were observed in both

100 h-frequency signals (1) does not require the rod-cone Cx36 gap junctions as has been proposed in the

101 Progressive rod-cone degeneration (PRCD) is a small protein localize

102 Rod-cone degenerations, for example, retinitis pigmentos

103 tis pigmentosa (RP), the most common form of rod-cone dystrophy, is caused by greater than 3100 mutat

104 RGCs respond to rod/cone input through the retinal circuitry, however, a

105 racterization of two recently reported rigid-rod conjugated polymers that do not contain single bonds

106 ASB9 and CUL5 behave as rigid rods, connected through a hinge provided by ELOB/C trans

107 surface was studied using crystal truncation rod (CTR) X-ray diffraction coupled with density functio

108 cle biopsies showed multiminicores, nemaline rods, cytoplasmic bodies, caps, central nuclei, rimmed f

109 In contrast, rod dark adaptation following exposure to bright bleachi

110 ecovery of rhodopsin to the ground state and rod dark adaptation.

111 omerization-deficient arrestin-1 resulted in rod death.

112 me alterations were detected at the onset of rod degeneration compared with wild type mice, including

113 After primary rod degeneration concluded, caspase-3 activation appeare

114 e cells (CiPCs) into the subretinal space of rod degeneration mice (homozygous for rd1, also known as

115 , and in the rd1 and rd10 models for primary rod degeneration.

116 activity matched the progression of primary rod degeneration.

117 temperature range of 50-55 degrees C (myosin rod denaturation).

118 librium stresses acting among self-propelled rods destabilize motility-induced phase separation and f

119 e domain (CaMD) uniquely inserted within the rod domain.

120 cone-driven responses supplement the loss in rod-driven sensitivity to slow temporal variations.

121 d that the temporal processing properties of rod-driven vision in mice change significantly with ligh

122 glion cells) is sufficient for fast, mesopic rod-driven vision.

123 hoice, operant behavior assay to investigate rod-driven, temporal contrast sensitivity (TCS) in mice

124 use various forms of blindness, ranging from rod dysfunction to rod and cone degeneration.

125 ent features of STGD1 but eventually develop rod dysfunction, and specific missense variants may be a

126 retinitis pigmentosa in 14% (3/23) and cone-rod dystrophy (4%, 1/23).

127 amily with macular dystrophy, nine with cone-rod dystrophy (CORD), and three with retinitis pigmentos

128 -RD: retinitis pigmentosa (RP; 85.94%), cone-rod dystrophy (CORD; 10.94%), and Leber congenital amaur

129 sociated with human autosomal recessive cone-rod dystrophy, negatively regulates EV levels in the sen

130 ngenital amaurosis (LCA1), and dominant cone-rod dystrophy-6 (CORD6) affected RetGC1 activity and reg

131 5del associated with autosomal dominant cone-rod dystrophy.

132 Rods evolved from cone-like precursors through expressio

133 We know that rods evolved from cone-like precursors through the expre

134 ity-induced phase separation, self-propelled rods exhibit a variety of nonequilibrium phenomena, incl

135 The long rod facing the cytosol is mainly formed by the predicted

136 by analysing the kinetic differences between rod flash responses and recent voltage-clamp recordings

137 Most vertebrate eyes have rods for dim-light vision and cones for brighter light a

138 -gated protein switch based on cofilin-actin rod formation, occurring in stressed neurons in the Alzh

139 Even when followed for more than 20 years, rod function and structure at some retinal locations cou

140 pathway is sufficient to maintain continuous rod function and support cone photoreceptors as well alt

141 ationship supported the existence of a large rod function deficit which was also most prominent near

142 ut they cannot explain the selective loss of rod function in recessive CSNB.

143 test variability (rho = 0.81 [0.64-0.91] for rod-function anxiety and rho = 0.83 [0.68-0.92] for cone

144 aire has high marginal reliability (0.81 for rod-function anxiety, 0.83 for cone-function anxiety) an

145 usceptibility testing (AST) in Gram-negative rod (GNR) bacteremia is compelling; however, evidence su

146 In mouse and human retina, rods greatly outnumber cones and consume more energy ove

147 es than those present in straight- or curved-rod H. pylori.

148 ults show that the machine learning designed rods have 150% better buckling resistance than all the r

149 pically exists as a curved rod, but straight rods have been observed under certain conditions.

150 uence of intermediates, including filaments, rods, helices, and 2D rectangular plates, before transfo

151 7)O(12))(2)(Et(2)O)(MeCN)(2)] (2), as yellow rods in 42% isolated yield.

152 expend nearly twice as much energy as single rods in darkness, largely because they make more synapse

153 showed multiminicores and lobulated fibers, rods in half the patients, and no fiber type disproporti

154 tal sequences within crystals of metal-oxide rods in multivariate metal-organic framework-74 containi

155 s well as survival and function of cones and rods in patients with RPE65 mutations.

156 on rates as well as survival and function of rods in the Rpe65 R91W knockin (KI) mouse model of Leber

157 150% better buckling resistance than all the rods in the training database, i.e., better than the nat

158 binds to a POU motif in the promoter of the rod-inducing factor Nrl to repress its expression.

159 odopsin was sequestered to the photoreceptor rod inner segment layer with a concomitant increase in p

160 changed to transform the flash response of a rod into that of a cone.

161 for uniformity, for recovery coefficients in rods larger than 2 mm, and for spillover ratios in the n

162 where l, A, and [Formula: see text] are the rods' length, aspect ratio (length divided by width), an

163 polyphiles, where glycerol-capped terphenyl rods lie on the gyroid surface while semiperfluorinated

164 nzyme levels that aPBPs do not contribute to rod-like cell shape but are required for mechanical stab

165 While the Rod complex governs rod-like cell shape, aPBP function is less well understo

166 ixing in a colloidal system of polydisperse, rod-like clay particles that is driven by particle repul

167 cal liposomes into elongated shapes, such as rod-like compartments.

168 de platelets and a nematic phase formed by a rod-like high-performance aramid.

169 ver, an increase in dystrophic microglia and rod-like microglia aligning to neurons harbouring tau pa

170 syndrome also displayed increased numbers of rod-like microglia.

171 ngle copy of Pex17p assemble to form a 20-nm rod-like particle.

172 -coil polymers bind more strongly than stiff rod-like polymers and that flexible polymers nucleate co

173 k of FimA with the Nte inserted in the pilus rod-like, antiparallel orientation, only depends on the

174 relatively spared from degeneration, and the rod loss phenotype was not obvious after metamorphosis.

175 Cone death generally follows rod loss regardless of the underlying pathogenic mutatio

176 [VA], contrast sensitivity), mixed cones and rods (low-luminance VA, low-luminance deficit, mesopic l

177 ERG cone and rod luminance response functions were recorded in 150 pa

178 ibiting IMPDH activity in living mice delays rod mass recovery.

179 tive vulnerability of rod photoreceptors and rod-mediated (scotopic) vision in early AMD, including d

180 opic) vision in early AMD, including delayed rod-mediated dark adaptation (RMDA) and impaired rod-med

181 ivity), or rods (scotopic light sensitivity, rod-mediated dark adaptation [RMDA]).

182 Functionally this is manifest as delayed rod-mediated dark adaptation and eventually as rod-media

183 mediated dark adaptation (RMDA) and impaired rod-mediated light and pattern sensitivity.

184 d-mediated dark adaptation and eventually as rod-mediated visual dysfunction in general.

185 Arrays of HD-CNTf rods microelectrodes were applied to detect neurotransmi

186 streak of higher rod photoreceptor (~80,000 rods mm(-2) ) and ganglion cell (~1,800 cells mm(-2) ) d

187 ovel form of recessive congenital TNNT1 core-rod myopathy.

188 rations to the rod shape, such as the curved rod, occur through manipulating the process of cell wall

189 pressure phases consisting of highly ordered rods of hydrofluorocarbons are presented based on theore

190 information on the condition of the control rods of pressurized water reactors.

191 cyclodeviation assessed using double-Maddox rods, of whom 105 were also assessed using fusible synop

192 ilar to that famously used to rotate a glass rod on the surface of a liquid crystal by synthetic mole

193 flavonoids also bind directly to unliganded rod opsin, enhancing its folding, stability, and regener

194 They bind to and stabilize unliganded rod opsin, which in excess accelerates degenerative proc

195 taining of liquid media showed Gram-positive rods or cocci, including some cocci in chains, suggestin

196 s up a new opportunity to design engineering rods or columns with superior buckling resistance such a

197 ment structure, but not functional rescue in rods or cones.

198 A bipedal robot is created by coupling the rod orientation with the alternating actuation of its tw

199 d plasmonic anisotropy allows control of the rod orientation, plasmonic excitation, and photothermal

200 , we asked whether the PRPH2 binding partner rod OS membrane protein 1 (ROM1) could serve as a phenot

201 ns such as PRPH2, the PRPH2 binding partner, rod outer segment membrane protein 1 (ROM1), and rhodops

202 protein lost the ability to oligomerize with rod outer segment membrane protein 1 (Rom1), but retaine

203 ted improvements in Prph2 protein levels and rod outer segment structure, but not functional rescue i

204 c system impairment and the affection of the rod pathway through the AII cells may explain and be par

205 osed in the past; and (2) a Cx36-independent rod pathway(s) (e.g., direct rod to OFF cone bipolar cel

206 g and in visual signal transmission from the rod pathway.

207 , we do not yet understand how the different rod pathways contribute to the perception of temporal co

208 However, in daylight conditions, rod pathways exhibit high sensitivity to fast but not to

209 receptors is channeled into multiple retinal rod pathways that ultimately connect to cone photorecept

210 NA is expressed in human retina and identify rod PDE6 as a retina-specific substrate of FAT10 conjuga

211 ort the bacterial actin homolog MreB and the Rod PG synthesis complexes away from poles.

212 in the form of a horizontal streak of higher rod photoreceptor (~80,000 rods mm(-2) ) and ganglion ce

213 This constitutive activity can desensitize rod photoreceptor cells and lead to night blindness.

214 This rule is broken by rod photoreceptor cells of nocturnal mammals, in which t

215 hown that her9 is upregulated during chronic rod photoreceptor degeneration and regeneration in adult

216 f rod - rod bipolar cell signaling following rod photoreceptor degeneration.

217 6beta expression, the db/db mice had reduced rod photoreceptor function.

218 We generated a pan-retinal and rod photoreceptor neuron-specific conditional KO mouse l

219 reveals that many disc membranes in Prcd-KO rod photoreceptor neurons are irregular, containing fewe

220 ted melatonin receptor, is incompatible with rod photoreceptor survival.

221 a(v)1.4 L-type channels for the formation of rod photoreceptor synapses in the retina.

222 The severe impact on early rod photoreceptor viability may signify a previously und

223 nduce the transformation of fibroblasts into rod photoreceptor-like cells.

224 toreceptors using a mcu(-/-) zebrafish and a rod photoreceptor-specific Mcu(-/-) mouse.

225 ly documented the selective vulnerability of rod photoreceptors and rod-mediated (scotopic) vision in

226 BACKGROUNDIn retinitis pigmentosa (RP), rod photoreceptors degenerate from 1 of many mutations,

227 Temporal contrast detected by rod photoreceptors is channeled into multiple retinal ro

228 , ultrastructural analysis demonstrates that rod photoreceptors lacking PRCD display disoriented and

229 e is caused by mutations in RHO expressed in rod photoreceptors that provide vision in dim ambient li

230 datasets revealed predominant expression in rod photoreceptors, and immunostaining demonstrated RIMS

231 role of visual arrestin-1 oligomerization in rod photoreceptors, we expressed mutant arrestin-1 with

232 eurons of the retina that form synapses with rod photoreceptors.

233 any of which are preferentially expressed in rod photoreceptors.

234 receptor transcription factors Crx, Nrl, and rod phototransduction genes.

235 processing strategy for this class of rigid-rod polymer system to optimize their charge transport pr

236 , reactive oxygen species, and cofilin-actin rods, present numerous challenges in the development of

237 ptive attractions, even to the painful shock-rod, recruit mesocorticolimbic incentive-related circuit

238 In dim lights, rods relay relatively slow temporal variations.

239 etasurfaces, consisting of a graded array of rod resonators attached to an elastic substrate that, to

240 eurodevelopmental state, and potentiation of rod - rod bipolar cell signaling following rod photorece

241 ance deficit, mesopic light sensitivity), or rods (scotopic light sensitivity, rod-mediated dark adap

242 Rods settling under gravity in a quiescent fluid can ove

243 The Rod system establishes rod shape de novo by elongating PG at nonpolar regions.

244 ell-wall-synthesis machinery responsible for rod shape in Escherichia coli is the processive 'Rod com

245 Alterations to the rod shape, such as the curved rod, occur through manipul

246 ingly, the ugtP lytE mutant recovered normal rod-shape by acquiring mutations that decreased the expr

247 uses a geometry-specific defect in engulfing rod-shaped bacteria and ellipsoidal beads.

248 aled a new cofactor that may help a group of rod-shaped bacteria elongate and grow.

249 The cell morphology of rod-shaped bacteria is determined by the rigid net of pe

250 hase-separated occlusive compartments around rod-shaped bacteria leading to increased bacterial survi

251 uperatomic architectures that are built from rod-shaped Co(12)Se(16)(PEt(3))(10) and C(140) nanoclust

252 Macroscopic rod-shaped crystals show various photomechanical effects

253 In rod-shaped Escherichia coli, two conserved sets of machi

254 In the rod-shaped fission yeast Schizosaccharomyces pombe, symm

255 chromosomes reorganise into highly compact, rod-shaped forms, thought to consist of consecutive chro

256 mpacted in length by more than 100-fold into rod-shaped forms.

257 In order to achieve this, a new class of rod-shaped molecular switches, suitable for formation of

258 Mature hiPSC-CMs showed rod-shaped morphology, highly organized sarcomeres, elev

259 In the rod-shaped Myxococcus xanthus cells, T4P localize at the

260 e transitions also vary with particle shape: rod-shaped particles show a partially reversible phase t

261 analyses showed that the ability to induce a rod-shaped structure was not required for viral replicat

262 and showed that the ability of gI to induce rod-shaped structures correlated well with the capabilit

263 und that gI could self-interact and that the rod-shaped structures failed to recognize glycoprotein E

264 a] 110 to 202) both failed to stain the long rod-shaped structures, suggesting the formation of a hig

265 rly boiling acid: Saccharolobus solfataricus rod-shaped virus 1 (SSRV1), at 2.8- angstrom resolution,

266 both in isolation and bound to S. islandicus rod-shaped virus 3 AcrID1 reveal that the anti-CRISPR pr

267 s well as a panel of unrelated S. islandicus rod-shaped viruses (SIRVs).

268 ar how these spherical spores germinate into rod-shaped, walled cells without preexisting PG template

269 Many of these mediate canonical transfer of rod signals from Aii cells to ON CBCs for night vision,

270 f NRL and the retinal TF CRX correlated with rod-specific ncRNA expression, whereas CRX alone favored

271 by presence or absence, respectively, of the rod-specific transcription factor (TF) Nrl Nrl-dependent

272 orks through the assembly of T-shape polymer-rod-sphere amphiphiles in the bulk state.

273 cts (OR 7.78; 95% CI, 2.69-22.35, P < .001), rod/spindle objects (OR 7.05; 95% CI, 2.11-23.59, P = .0

274 Absence of rod/spindle objects was associated with a BCVA of >=6/6

275 uggest that the intermediate states in talin rod subdomains are stabilized by force during unfolding,

276 free energy profiles for unfolding of talin rod subdomains.

277 Interestingly, Crb1 promotes rod survival under strong white light irradiation in a p

278 churin-dystroglycan transsynaptic complex at rod synapses.

279 e, is required for the molecular assembly of rod synapses; however, Ca(v)1.4 Ca(2+) signals are neede

280 The Rod system establishes rod shape de novo by elongating P

281 ated differences compared with double-Maddox rod test, when summing right and left eyes and when usin

282 phosphorylated AMELX, KI enamel lacks enamel rods, the hallmark component of mammalian enamel, and, u

283 d both the urethra and a para-urethral steel rod, then removing the rod.

284 copies of FimA assemble to the helical pilus rod through a mechanism termed donor strand complementat

285 x36-independent rod pathway(s) (e.g., direct rod to OFF cone bipolar cell synapses and/or glycinergic

286 ets of 16 motors on opposite ends of a stiff rod to two binders, each representing 16 motors, reduced

287 ye diseases in which initial degeneration of rods triggers secondary degeneration of cones, leading t

288 ionally isomeric COF (BP-COF-6), composed of rod units, were also synthesized using the same strategy

289 ression to buckling of 3D printed biomimetic rods using a polymeric ink.

290 the largest (of right or left) single-Maddox rod values were similar to double-Maddox values (mean di

291 Mean summed single-Maddox rod values were somewhat similar to double-Maddox values

292 tests, with their reliance on both cone and rod vision, may be a more comprehensive assessment of th

293 However, one difference is that, whereas a rod visual pigment remains stable in darkness, a cone pi

294 Center-Surround model of cone resilience and rod vulnerability.

295 lumn or spine assembles around the notochord rod which contains a core made of large vacuolated cells

296 tron-dense structures that we named "exocyst rods," which likely represent accumulated membrane prote

297 . cholerae cells tended to exist as straight rods, while genetically engineering cells to maintain hi

298 While there has been progress in elucidating rod wiring, molecular mechanisms used by cones to establ

299 ace of the Hfq hexamer as parallel, straight rods with B-DNA like conformational properties.

300 These diamond-like molecular rods with extraordinarily small thickness are among the

301 s the exceptional stiffness of these helical rods, with a persistence length of more than 200 angstro