ライフサイエンスコーパス: rod photoreceptor

1 e photoreceptors (equivalent to rhodopsin of rod photoreceptors).

2 dual subcellular compartments of the retinal rod photoreceptor.

3 in three cell classes: Muller glia, cone and rod photoreceptors.

4 Rhodopsin is thus a functional biomarker for rod photoreceptors.

5 ntered transcriptional regulatory network in rod photoreceptors.

6 ein Dendra2 and expressing in Xenopus laevis rod photoreceptors.

7 bodies of ultraviolet-sensitive cones or in rod photoreceptors.

8 romote facultative heterochromatin in mature rod photoreceptors.

9 in, Dendra2, and expressed in Xenopus laevis rod photoreceptors.

10 bly when photons arrive rarely at individual rod photoreceptors.

11 f the XOPS-mCFP transgenic line, which lacks rod photoreceptors.

12 any of which are preferentially expressed in rod photoreceptors.

13 control of transgene expression in zebrafish rod photoreceptors.

14 hat CNG-modulin is expressed in cone but not rod photoreceptors.

15 ker for functional abnormalities in maturing rod photoreceptors.

16 l of transgene expression can be achieved in rod photoreceptors.

17 photoreceptors and only minimal contact with rod photoreceptors.

18 cal relevance of Mef2c expression in retinal rod photoreceptors.

19 could support Rhodopsin promoter activity in rod photoreceptors.

20 ds interactions in transgenic Xenopus laevis rod photoreceptors.

21 quirements for the death of kinesin-2-mutant rod photoreceptors.

22 ld-type as well as Nrl(-/-) mice, which lack rod photoreceptors.

23 rs and that it regulates the regeneration of rod photoreceptors.

24 r beta (ERRbeta) as selectively expressed in rod photoreceptors.

25 ld-type NRL that is able to convert cones to rod photoreceptors.

26 ing photosensitivity and OS morphogenesis of rod photoreceptors.

27 s likely triggers or stimulates the death of rod photoreceptors.

28 tions of avian vision rest on their cone and rod photoreceptors.

29 jor cause of human blindness is the death of rod photoreceptors.

30 mouse lacking its essential ArpC3 subunit in rod photoreceptors.

31 t the release of dopamine is defined only by rod photoreceptors.

32 ing degree of plasticity following rescue of rod photoreceptors.

33 harged linkers and expressed them in Xenopus rod photoreceptors.

34 otent stem cells (iPSCs) derived from murine rod photoreceptors.

35 sicles to selected membrane sites in retinal rod photoreceptors.

36 st notably a block in the differentiation of rod photoreceptors.

37 ursors in real time in single isolated mouse rod photoreceptors.

38 neurons operating immediately downstream of rod photoreceptors.

39 dendrites to form new synapses with healthy rod photoreceptors.

40 condary event resulting from degeneration of rod photoreceptors.

41 e inner segment and outer plexiform layer of rod photoreceptors.

42 cell processes, where they contact cone and rod photoreceptors.

43 peripapillary involvement resembles that of rod photoreceptors.

44 f-family transcription factor NRL to augment rod photoreceptors.

45 eurons of the retina that form synapses with rod photoreceptors.

46 receptor degeneration and dysfunction of the rod photoreceptors.

47 h by the morphology and number of integrated rod-photoreceptors.

48 in the form of a horizontal streak of higher rod photoreceptor (~80,000 rods mm(-2) ) and ganglion ce

49 Rod photoreceptors accomplish this task through the use

50 glial cells phagocytosed cell bodies of dead rod photoreceptors albeit at a lower frequency.

51 the end product of chromophore bleaching in rod photoreceptors, all-trans retinol, is part of a feed

52 ective subunit composition of the complex in rod photoreceptors allowed us to study the molecular und

53 However, rod photoreceptors also provide support that partially p

54 11-cis-retinol is not a useful substrate for rod photoreceptors although it is for cone photoreceptor

55 tween two siblings in the murine retina, the rod photoreceptor and bipolar interneuron.

56 ncodes visual information in dim light using rod photoreceptors and a specialized circuit: rods->rod

57 ncodes visual information in dim light using rod photoreceptors and a specialized circuit: rods-->rod

58 ions, which were biased toward production of rod photoreceptors and amacrine cell interneurons.

59 s required to produce the full complement of rod photoreceptors and amacrine cells in mouse.

60 Retinal responses to photons originate in rod photoreceptors and are transmitted to the ganglion c

61 rmal retinal development led to apoptosis of rod photoreceptors and bipolar (BP) interneurons, wherea

62 eting to knock down Rac1 expression in mouse rod photoreceptors and found protection against light-in

63 is deficit resulted from a reduced number of rod photoreceptors and inner nuclear layer cells.

64 lity in the mouse that causes the absence of rod photoreceptors and is the mouse counterpart of 1 typ

65 eotide-gated (CNG) channels are expressed in rod photoreceptors and open in response to direct bindin

66 Transient responses utilize input from rod photoreceptors and output by the classical neurotran

67 ly documented the selective vulnerability of rod photoreceptors and rod-mediated (scotopic) vision in

68 Finally, we dissected the roles of rod photoreceptors and RPE for dark adaptation of M-cone

69 t vision in mammals depends fundamentally on rod photoreceptors and the well-studied rod bipolar (RB)

70 blotting that SynCAM 1 is expressed on mouse rod photoreceptors and their terminals in the outer nucl

71 gulators of Cav1.4 channel function in mouse rod photoreceptors and thus synaptic activity.

72 To determine how aging affects rod photoreceptors and to test the retinoid-deficiency h

73 is widely expressed, including expression in rod photoreceptors, and encodes a 75 kDa protein of the

74 datasets revealed predominant expression in rod photoreceptors, and immunostaining demonstrated RIMS

75 for healthy and putative early degenerating rod photoreceptors, and revealed the loss of MALAT1 expr

76 , and retinal microglia responses to induced rod photoreceptor apoptosis.

77 ydrophobic 11-cis retinal to the interior of rod photoreceptors appears to be retarded by transit acr

78 tion to vesicle release at synaptic ribbons, rod photoreceptors are capable of substantial slow relea

79 Rod photoreceptors are electrically coupled through gap

80 Rod photoreceptors are exquisitely sensitive light detec

81 that affect calcium homeostasis (Ca(2+)) in rod photoreceptors are linked to retinal degeneration an

82 tal-lined cups acting as macroreceptors, but rod photoreceptors are positioned behind these reflector

83 The outer segments of vertebrate rod photoreceptors are renewed every 10 d.

84 However, as rod photoreceptors are saturated in bright light, the co

85 Rod photoreceptors are specialized neurons that mediate

86 hat for light-induced retinopathies in mice, rod photoreceptors are the primary site of toxic retinoi

87 ate retina, light responses generated by the rod photoreceptors are transmitted to the second-order n

88 In dim light, rod-photoreceptors are active, but colour vision is impo

89 RNA levels of the mouse ortholog (Plk1s1) in rod photoreceptors, as well as its decreased expression

90 sion when an ancestral cone evolved into the rod photoreceptor at an unknown stage preceding the last

91 g pathway, resulted in the overproduction of rod photoreceptors at the expense of Muller glial cells.

92 This mechanism may explain how the rod photoreceptor balances the quantity of discs added a

93 gned to be perceptible to the cone-, but not rod-, photoreceptor based visual systems.

94 ines forming predominantly before birth, and rod photoreceptors, bipolars, and Muller glia differenti

95 demarcation of POS tips is not intrinsic to rod photoreceptors but requires activities of the RPE as

96 id vision loss and near complete ablation of rod photoreceptors by day 12.

97 cance of PI3K was investigated in vertebrate rod photoreceptors by deleting its regulatory p85alpha p

98 Conditional deletion of PTP1B in rod photoreceptors by the Cre-loxP system resulted in en

99 rochemistry were observed after the onset of rod photoreceptor cell death.

100 lpain activity as a key event during primary rod photoreceptor cell death.

101 er, the P23H protein failed to accumulate in rod photoreceptor cell endoplasmic reticulum but instead

102 ne zipper (Nrl) is a critical determinant of rod photoreceptor cell fate and a key regulator of rod d

103 The removal of ARL13B in adult rod photoreceptor cells after maturation of OS resulted

104 player in prenylated protein trafficking in rod photoreceptor cells and establishes the potential ro

105 This constitutive activity can desensitize rod photoreceptor cells and lead to night blindness.

106 e Phlp1 gene in mouse (Mus musculus) retinal rod photoreceptor cells and measured the effects on G-pr

107 Mature rod photoreceptor cells contain very small nuclei with t

108 Rod photoreceptor cells depend completely on the output

109 Retinal rod photoreceptor cells have double membrane discs locat

110 This rule is broken by rod photoreceptor cells of nocturnal mammals, in which t

111 odopsin is the G protein-coupled receptor in rod photoreceptor cells that initiates vision upon photo

112 iated protein complex, and that apoptosis of rod photoreceptor cells triggered by protein mislocaliza

113 Rod photoreceptor cells were more sensitive to loss of i

114 known about the function of DICER1 in mature rod photoreceptor cells, another retinal cell type that

115 in the rhodopsin gene, which is expressed in rod photoreceptor cells, are a major cause of the heredi

116 The cGMP phosphodiesterase of rod photoreceptor cells, PDE6, is the key effector enzym

117 in these cilia, as well as in cilia of mouse rod photoreceptor cells, was reduced significantly when

118 To determine the importance of ARL3 in rod photoreceptor cells, we generated transgenic mice ex

119 he regulation or function of these lipids in rod photoreceptor cells, which have highly active membra

120 r termination of photoactivated rhodopsin in rod photoreceptor cells.

121 pressing exclusively the mutant rhodopsin in rod photoreceptor cells.

122 transcriptional regulator of homeostasis in rod photoreceptor cells.

123 hoinositide 3-kinase/Akt survival pathway in rod photoreceptor cells.

124 membranes rather than to plasma membranes of rod photoreceptor cells.

125 rhodopsin and protects IR phosphorylation in rod photoreceptor cells.

126 roles of Muller glia in the phagocytosis of rod photoreceptor cells.

127 TN3 associate in the outer segments of mouse rod photoreceptor cells.

128 ceptor-bound protein 14 (Grb14) may modulate rod photoreceptor cGMP-gated channels by decreasing chan

129 ient retinae; CrxGFP is a marker of cone and rod photoreceptor commitment.

130 representing all known neural retinal cells: rod photoreceptors, cone photoreceptors, Muller glia, bi

131 are exclusively cone photoreceptors and not rod photoreceptors, confirming that ThrbCRM1 progenitor

132 The outer segment (OS) of rod photoreceptors consist of a highly modified primary

133 Rod photoreceptors consist of an outer segment (OS) and

134 Rod photoreceptors contribute to vision over an approxim

135 In rod photoreceptors, deactivation of the light-activated

136 ases of the eye and is associated with early rod photoreceptor death followed by secondary cone degen

137 Rod photoreceptor death is characterized by apoptotic fe

138 to an indirect 'bystander effect' caused by rod photoreceptor death or a direct role for AIPL1 in co

139 Signaling of single photons in rod photoreceptors decreases the concentration of the se

140 BACKGROUNDIn retinitis pigmentosa (RP), rod photoreceptors degenerate from 1 of many mutations,

141 unfolded protein response (UPR), leading to rod photoreceptor degeneration and autosomal dominant re

142 hown that her9 is upregulated during chronic rod photoreceptor degeneration and regeneration in adult

143 ptor cells after cyclic light-mediated acute rod photoreceptor degeneration in a transgenic P23H muta

144 RIP1 kinase significantly prevented cone and rod photoreceptor degeneration in Irbp(-/-) mice.

145 Our previous observations suggest that rod photoreceptor degeneration in retinas lacking AIPL1

146 f rod - rod bipolar cell signaling following rod photoreceptor degeneration.

147 reduced visual function accompanied by slow rod photoreceptor degeneration.

148 entially suggested a novel role of MALAT1 in rod photoreceptor degeneration.

149 adult rods into cones, via knockdown of the rod photoreceptor determinant Nrl, could make the cells

150 ic zebrafish experience a continual cycle of rod photoreceptor development and degeneration throughou

151 er transcription factor NRL is essential for rod photoreceptor development and functional maintenance

152 PKC-gamma as isoforms that are essential for rod photoreceptor differentiation in mouse retinas.

153 The process by which rod photoreceptor discs are formed has been controversia

154 study provides 3D data of nascent and mature rod photoreceptor disk membranes at unprecedented z-axis

155 endoplasmic reticulum but instead disrupted rod photoreceptor disks.

156 Riggs CSNB demonstrates selective rod photoreceptor dysfunction and occurs due to mutation

157 Rod photoreceptor dysfunction is observed in Reep6-/- mi

158 Mutation of rod photoreceptor-enriched transcription factors is a ma

159 Cyclic nucleotide-gated (CNG) channels from rod photoreceptors exhibit a 3:1 stoichiometry of CNGA1

160 The 3D genome of rod photoreceptors exhibited inverted radial distributio

161 are characterized by the progressive loss of rod photoreceptors followed by loss of cones.

162 In the mammalian retina, rod photoreceptors form selective contacts with rod ON-b

163 n a diminished progenitor pool available for rod photoreceptor formation.

164 adult zebrafish retina continuously produces rod photoreceptors from infrequent Muller glial cell div

165 uscin and A2E, we analyzed RPEs and isolated rod photoreceptors from mice of different ages and strai

166 3K), which the authors have shown to protect rod photoreceptors from stress-induced cell death.

167 We demonstrate that the recovery of rod photoreceptors from the ambient saturating levels of

168 nder the control of Crx promoter can restore rod photoreceptor function and suppress cone gene expres

169 rate that ERRbeta is a critical regulator of rod photoreceptor function and survival, and suggest tha

170 To study whether cone or rod photoreceptor function was involved in the pathway t

171 opic threshold elevation led by worsening of rod photoreceptor function.

172 6beta expression, the db/db mice had reduced rod photoreceptor function.

173 Rhodopsin is the rod photoreceptor G protein-coupled receptor responsible

174 ound, Photoregulin3 (PR3) that also inhibits rod photoreceptor gene expression, potentially though Nr

175 Rod photoreceptors generate amplified, reproducible resp

176 Rod photoreceptors generate measurable responses to sing

177 In vertebrate eyes, the rod photoreceptor has a modified cilium with an extended

178 rial transport of rhodopsin is essential for rod photoreceptor health and function.

179 d by mutations in genes that are specific to rod photoreceptors; however, blindness results from the

180 BP removes all-trans-retinol from individual rod photoreceptors in a concentration-dependent manner.

181 outer segment (OS) and inner compartments of rod photoreceptors in a light-dependent manner thereby c

182 ses of retinal architecture indicated intact rod photoreceptors in all patients but abnormalities in

183 this hypothesis, we looked for transitioning rod photoreceptors in Blimp1 conditional knock-out (CKO)

184 Here, we show that rod photoreceptors in mice rely on glycolysis for their

185 onin receptor (mtnr1a) gene causes a loss of rod photoreceptors in retinas of developing Xenopus trop

186 ssessed the arrangements of retinal cone and rod photoreceptors in six nocturnal, three cathemeral an

187 a, exacerbated by the high O2 consumption of rod photoreceptors in the dark, is a primary cause of DR

188 Development of rod photoreceptors in the mammalian retina is critically

189 specification and functional maintenance of rod photoreceptors in the mammalian retina.

190 d thus assess the distribution of functional rod photoreceptors in the retina.

191 ain 3D visualization of the nascent disks of rod photoreceptors in three mammalian species, to gain i

192 phy (OCT) to measure light-driven signals of rod photoreceptors in vivo.

193 there was early-onset rapid degeneration of rod photoreceptors in young subjects with these ciliopat

194 ous mutants in vitro, and primarily affected rod photoreceptors in zebrafish mimicking cardinal featu

195 , Dendra2, and expressed in early developing rod photoreceptors, in which OSs are still cone-shaped.

196 mislocalizes to the plasma membrane (PM) of rod photoreceptor inner segments (ISs), and causes autos

197 GFP (+) retinas was mostly restricted to the rod photoreceptor inner segments, whereas GCAP1 immunofl

198 We find that retbindin is secreted by the rod photoreceptors into the inter-photoreceptor matrix w

199 The outer segment (OS) of the rod photoreceptor is a light-sensing cilium containing ~

200 hen a substantial fraction of rhodopsin in a rod photoreceptor is exposed to bright light, the rod is

201 Exocytosis from the rod photoreceptor is stimulated by submicromolar Ca(2+)

202 Temporal contrast detected by rod photoreceptors is channeled into multiple retinal ro

203 Specification of retinal rod photoreceptors is determined by several different tr

204 Bleaching adaptation in rod photoreceptors is mediated by apo-opsin, which activ

205 Vesicle release from rod photoreceptors is regulated by Ca(2+) entry through

206 d receptor, most abundant protein in retinal rod photoreceptors, is glycosylated at asparagines-2 and

207 ht-sensing molecule in the outer segments of rod photoreceptors, is responsible for converting light

208 , ultrastructural analysis demonstrates that rod photoreceptors lacking PRCD display disoriented and

209 Mutations primarily in genes expressed in rod photoreceptors lead to early rod death, followed by

210 rative disease, in which the death of mutant rod photoreceptors leads secondarily to the non-cell aut

211 mal RPE and neural retina but showed reduced rod photoreceptor light responses, indicating that lack

212 nduce the transformation of fibroblasts into rod photoreceptor-like cells.

213 rized by early loss of rod function and slow rod photoreceptor loss with a secondary decline in cone

214 m survival and, in some cases, expressed the rod photoreceptor marker, rhodopsin.

215 The rod photoreceptors may be more susceptible than cone pho

216 in mice with monoallelic Nampt deletion from rod photoreceptors, mice lacking SIRT3, and mice lacking

217 small RNA sequencing analysis, we identified rod photoreceptor miRNAs of the miR-22, miR-26, miR-30,

218 We generated a pan-retinal and rod photoreceptor neuron-specific conditional KO mouse l

219 reveals that many disc membranes in Prcd-KO rod photoreceptor neurons are irregular, containing fewe

220 mechanism by which rhodopsin mislocalizes in rod photoreceptor neurons is not well understood.

221 ly stages of diabetes are not exacerbated by rod photoreceptor O2 consumption in the dark.

222 d by exon 5 and is specifically expressed in rod photoreceptors of developing and mature retina.

223 n of PhLPs, we expressed this protein in the rod photoreceptors of mice and found that this manipulat

224 Rod photoreceptors of nocturnal mammals display a striki

225 enically expressed hAIPL1 exclusively in the rod photoreceptors of the Aipl1(-/-) mouse.

226 G protein-coupled receptor expressed in the rod photoreceptors of the eye, where it mediates transmi

227 the strength of electrical coupling between rod photoreceptors of the retina is regulated by the tim

228 ght blindness due to genetic deficits in the rod photoreceptors of the retina.

229 alpha) for rod transducin alpha (rTalpha) in rod photoreceptors of transgenic mice, which also expres

230 In its absence, rod photoreceptor outer and inner segment length was red

231 n by rhodopsin to a change in current at the rod photoreceptor outer segment plasma membrane.

232 ponsible for impeding their transport to the rod photoreceptor outer segment.

233 esence of vacuolar structures that distorted rod photoreceptor outer segments and became more promine

234 ve aldehyde all-trans retinal is released in rod photoreceptor outer segments by photoactivated rhodo

235 Imaging of rod photoreceptor outer-segment disc membranes by atomic

236 ng night (i.e., scotopic) vision in mammals, rod photoreceptor output is conveyed to ganglion cells (

237 outer segments (OSs) and connecting cilia in rod photoreceptors, overlapping with Rp1.

238 d cells, including bovine and Xenopus laevis rod photoreceptors, P/rds was robustly sensitive to endo

239 requisite component of the high-sensitivity rod photoreceptor pathway.

240 pathway in the TKO retina that originates in rod photoreceptors, potentially a rare subset of rods wi

241 Dysfunction or death of rod photoreceptors precedes cone loss in many retinal an

242 d pluripotent stem cells (iPSCs) from murine rod photoreceptors (r-iPSCs) and scored their ability to

243 In human retinal degeneration, rod photoreceptors reactively sprout neurites.

244 be partially reversed, with regeneration of rod photoreceptors recovering normal morphology (includi

245 ents were identified as the likely source of rod photoreceptor regeneration in the P23H retinas.

246 a, and c-myb in both retinal development and rod photoreceptor regeneration.

247 investigating the molecular determinants of rod photoreceptor regeneration.

248 duction of retinal ganglion cells (RGCs) and rod photoreceptors, respectively.

249 cluding Beclin1 systemically or Atg7 in only rod photoreceptors resulted in increased susceptibility

250 rer to ancestral pigments than the dim-light rod photoreceptor rhodopsin.

251 removal of scaffolding proteins RIM1/2 from rod photoreceptor ribbon synapses causes a dramatic loss

252 Here we provide insight from studying rod photoreceptor ribbon-type active zones after disrupt

253 logical concentrations of IGF-1 can increase rod photoreceptor sensitivity in mammalian retinas.

254 for efficient enrichment of rhodopsin within rod photoreceptor sensory cilia, inhibited enrichment of

255 In rod photoreceptors, several phototransduction components

256 increasing the signal-to-noise ratio of the rod photoreceptor single-photon response in a transgenic

257 ously reported the identification of a novel rod photoreceptor specific isoform of Receptor Expressio

258 death was observed when retinas lacking the rod photoreceptor-specific Atg7 gene were coincubated wi

259 Rod photoreceptor-specific inactivation of numb in vivo

260 ated a pan-retina knock-out (Six3-Cre) and a rod photoreceptor-specific inducible conditional knock-o

261 toreceptors using a mcu(-/-) zebrafish and a rod photoreceptor-specific Mcu(-/-) mouse.

262 The lack of phenotype in pik3r1 KO rod photoreceptors suggests a redundant role in controll

263 uggesting that miRNAs play a primary role in rod photoreceptor survival.

264 ted melatonin receptor, is incompatible with rod photoreceptor survival.

265 a(v)1.4 L-type channels for the formation of rod photoreceptor synapses in the retina.

266 up on previous results, which indicated that rod photoreceptor synaptic ribbons lose their structural

267 Vision requires the generation of cone and rod photoreceptors that function in daylight and dim lig

268 dCVF) is an inactive thioredoxin secreted by rod photoreceptors that protects cones from degeneration

269 e is caused by mutations in RHO expressed in rod photoreceptors that provide vision in dim ambient li

270 nly observed during persistent activation of rod photoreceptors that saturate rods.

271 lation then likely triggers the apoptosis of rod photoreceptors that was observed.

272 Here we investigate in mouse rod photoreceptors the relationship between rhodopsin de

273 icularly how they are determined relative to rod photoreceptors, the cells that initiate vision in di

274 -induced suppression of glutamate release in rod photoreceptors, thereby driving ON-BC depolarization

275 cipated in the phagocytosis of dying or dead rod photoreceptors throughout the outer nuclear layer.

276 his high amplification system allows retinal rod photoreceptors to detect single photons of light.

277 n cascade and is critical for the ability of rod photoreceptors to function in low light conditions.

278 We have used mouse rod photoreceptors to investigate the generation of NADP

279 19 recognized the acylated N terminus of the rod photoreceptor transducin alpha (Talpha) subunit and

280 We present a comprehensive assessment of rod-photoreceptor transplantation across six murine mode

281 demonstrated restoration of vision following rod-photoreceptor transplantation into a mouse model of

282 ing adeno-associated virus in Xenopus laevis rod photoreceptors using a transgene and in ciliated IMC

283 ngth and motorless KIF17 constructs in mouse rod photoreceptors using adeno-associated virus in Xenop

284 The severe impact on early rod photoreceptor viability may signify a previously und

285 role of visual arrestin-1 oligomerization in rod photoreceptors, we expressed mutant arrestin-1 with

286 in P23H mice even when more than half of the rod photoreceptors were lost.

287 Rod photoreceptors were present in normal numbers, and t

288 Markers for amacrine, ganglion and rod photoreceptors were present in treated but not in co

289 Rod photoreceptors were recently shown to contact 'Off'

290 LKB1 and AMPK function in rod photoreceptors where their loss leads to aberrant ax

291 membrane of photosensitive outer segments of rod photoreceptors where they generate the electrical re

292 st all newly postmitotic N1-CKO cells became rod photoreceptors, whereas wild-type (WT) cells achieve

293 tatic regulation of ER function in mammalian rod photoreceptors, whereby miR-708 may help prevent an

294 ort hairpin RNA knockdown of nudC in tadpole rod photoreceptors, which leads to the inability of rod

295 Rod photoreceptors, which mediate dim light vision, rema

296 The responses of rod photoreceptors, which subserve dim light vision, are

297 toreceptors, promotes the differentiation of rod photoreceptors while preventing rods from adopting c

298 ontrast, disruption of LINC complexes within rod photoreceptors, whose nuclei are scattered across th

299 Dystrophic rod photoreceptors with truncated rod outer segments wer

300 ller glia results in reduced regeneration of rod photoreceptors without affecting injury-induced prol