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1 xposed to visible light and photosensitizer (rose bengal).
2 nglet oxygen sensitizer ([Ru(bpy)(3)](2+) or Rose Bengal).
3 nglet oxygen generation efficiency over free rose bengal.
4 ere varied to alter the fraction of adsorbed rose bengal.
5 of efficient photodynamic therapies based on rose bengal.
6 drazines in visible light, photocatalyzed by rose bengal.
7 ated photodynamically by the photosensitizer Rose Bengal.
8 s, which do not express mucins, stained with rose bengal.
9 of islands of stratified cells that excluded rose bengal.
10 PDAT was performed by applying a solution of rose bengal (0.1% or 0.2% RB in balanced salt solution)
11  irradiation); and Group 5, rose bengal PDT (rose bengal + 518 nm irradiation).
12 king (A0), or soaked with riboflavin (A1) or rose bengal (A2).
13  we show the in vitro photodynamic effect of rose bengal activated by intracellular generation of lig
14 d time to carotid artery occlusion following Rose Bengal administration and laser-induced arterial in
15         Separate measurements performed with rose bengal adsorbed on p-GaP surfaces pretreated with (
16 nd have better X-ray absorption ability than rose bengal alone.
17 groups: Group 1, no treatment; Group 2, 0.1% rose bengal alone; Group 3, 518 nm irradiation alone; Gr
18 illators are conjugated with photosensitizer rose bengal and arginylglycylaspartic acid (RGD) peptide
19 ts were provided in good yields, promoted by rose bengal and blue light in a single operation.
20 ico characterization of interactions between rose bengal and cationic poly(amidoamine) (PAMAM) and po
21 etal-free multicatalytic system comprised of rose bengal and cesium carbonate allowed the efficient f
22                                              Rose bengal and chlorin e6, photosensitizers (PSs) that
23 ts show that the strong affinity of PPI-5 to Rose Bengal and erythrosine B is attributed to the good
24 elayed carotid artery occlusion times on the rose bengal and ferric chloride thrombosis models.
25 ation of the mechanism of the ocular surface rose bengal and fluorescein staining that occurs in this
26 l retinal artery by intravenous injection of rose bengal and green laser irradiation of the artery.
27 hosphorescence decay of the triplet state of rose bengal and its quenching by ferricyanide.
28 Retinal vascular occlusion was introduced by rose Bengal and laser photocoagulation on chimeric mice
29                  It was also noted that both rose bengal and lissamine green B treatments slightly pr
30 tures were incubated for 5 minutes with 0.1% rose bengal and photographed.
31 noparticles was developed, which showed that Rose Bengal and Rapa have high non-specific encapsulatio
32 ravenous injection of the photosensitive dye rose bengal and skull irradiation with a beam of focused
33  43 nm were prepared and functionalized with rose bengal and sulforhodamine B by a ligand-exchange pr
34                                 In contrast, rose bengal and the ATP-regulated potassium channel anta
35 e-doped upconversion nanoparticle-conjugated rose bengal and triphenylphosphonium (LD-UCNP@CS-Rb-TPP)
36  the presence of a sensitizer (riboflavin or rose Bengal) and O(2), then incubated with a Cys-contain
37 et oxygen yield of a common photosensitizer (Rose Bengal) and the theoretical electric field enhancem
38 itive to peroxide but not methyl viologen or Rose Bengal, and GPXs, APX, and MSRA2 genes (encoding gl
39              Vital dyes such as fluorescein, rose bengal, and lissamine green B, among others, have b
40 ein mouse models with the photosensitive dye Rose bengal, and monitored plaque formation in real time
41  synthetic photosensitizers (methylene blue, rose bengal, and nitrite) and two model natural photosen
42 les for the photosensitizers methylene blue, Rose Bengal, and tetraphenylporphine.
43 n the DG, the VGLUT inhibitors Congo Red and Rose Bengal, and the mGlu2/3 agonist LY354740, also redu
44 sis in wild-type (WT) and Apoh-/- mice using rose bengal- and FeCl3-induced carotid thrombosis, laser
45                                              Rose bengal- and riboflavin-mediated photodynamic therap
46 0.1% rose bengal; Group III, MRSA with 0.03% rose bengal; and Group IV, MRSA with 0.1% riboflavin.
47 on products of SQ incubated in solution with Rose Bengal as a photooxidizer were isolated by semiprep
48 of imidazo[1,2-a]pyridine heterocycles using rose bengal as a photoredox catalyst at room temperature
49 o-2,3-dihydrobenzofuran-2-carboxylates using rose bengal as a triplet photosensitizer at ambient temp
50                     In one type of particle, Rose Bengal as an efficient singlet oxygen ((1)O2) produ
51 idines with alcohols has been achieved using rose bengal as an organic photoredox catalyst at room te
52 ization in the presence of visible light and Rose Bengal as an organophotocatalyst.
53                                              Rose Bengal as an organophotoredox catalyst has been use
54 as been achieved using a catalytic amount of rose bengal as an organophotoredox-catalyst and tert-but
55 presence of the photooxidants riboflavin and Rose Bengal as well as the diffusible one-electron oxida
56 y transfer between excited- and ground-state rose bengal at the C-18 silica/solution interface.
57 chronically contused rat spinal cord using a rose Bengal-based phototoxic method.
58                                              Rose bengal, blue LEDs, KI, K(2)S(2)O(8), and DMSO are a
59 rmula: see text] inhibitor sulfasalazine and rose bengal, but not by system L inhibitor 2-aminobicycl
60 n the targeted artery, we photoactivated the rose bengal by illuminating the longitudinal hippocampal
61 tion include the use of metal-free, low-cost Rose Bengal catalyst and practical operation (ambient te
62 h MRSA strains was demonstrated (1) for both rose bengal concentrations under ambient and green LED i
63                      Herein, gadolinium (Gd)-rose bengal coordination polymer nanodots (GRDs) are rep
64 aggregation inhibition by ERB analogs except rose bengal correlated well to the inhibition of Abeta c
65                 We describe the synthesis of Rose Bengal-decorated silica-coated silver nanocubes (Ag
66 n, as indicated by increased permeability to rose bengal diagnostic dye.
67                  In vivo, the ophthalmic dye rose bengal displays profound antiviral effects against
68        The adsorption/desorption kinetics of rose bengal, distributed between a C-18 derivatized poro
69 ed manner using photodynamic thrombosis with rose bengal dye and thermal burns from an argon laser wi
70 ated HCLE cells showed significantly reduced rose bengal dye exclusion.
71 neas were treated on the incision walls with rose bengal dye followed by exposure to 514-nm laser rad
72 on surface barrier function were measured by rose bengal dye penetrance.
73 N ischemia was generated using laser-coupled rose Bengal dye photoactivation, and the infarct localiz
74 ity laser illumination of mice injected with Rose Bengal dye to induce photochemical injury in the re
75               HCLE cells were incubated with rose bengal dye to measure the role of MUC16 in ocular s
76 ed in adult rats by intravenous injection of Rose Bengal dye, followed by argon green laser treatment
77 nations: riboflavin/UV-A light (RF/UV-A) and rose bengal/green light (RB/green).
78 up I, MRSA control; Group II, MRSA with 0.1% rose bengal; Group III, MRSA with 0.03% rose bengal; and
79 olerance against stress induced by Paraquat, Rose Bengal, heavy metal, and the synthetic auxins 1-nap
80 ess conditions created by high light levels, rose bengal, high salt levels, and osmotic shock.
81 sfer observed for the pure-bromide CsPbBr(3)-rose Bengal hybrid (1.1 x 10(11) s(-1)).
82 othiocyanate, and rose Bengal), the CsPbI(3)-rose Bengal hybrid with the strongest binding showed the
83 ls at ambient temperature in the presence of rose bengal in acetonitrile under an oxygen atmosphere.
84                                    The 0.03% rose bengal in dark conditions showed complete inhibitio
85                        We then photoactivate Rose Bengal in specific, targeted blood vessels within t
86  green LED irradiation, and (2) for the 0.1% rose bengal in the dark.
87 pocampal artery in urethane anesthetized and rose bengal-injected mice.
88                                              Rose bengal is an anionic dye considered as a potential
89                                              Rose bengal is an organic anionic dye used to assess dam
90 d to these stratified cells, indicating that rose bengal is excluded from cells that lack negative ch
91                      The clinical utility of rose bengal is hampered by its short half-life, limited
92    We measured the binding of one inhibitor, rose bengal lactone (RBL), to kinesin (dissociation cons
93 odels were created in New Zealand rabbits by Rose Bengal laser-induced RVO.
94  2 carotid artery injury models (FeCl(3) and Rose Bengal/laser), fXII-deficient mice are more resista
95                                              Rose bengal-mediated PDT successfully inhibited the grow
96                                              Rose bengal-mediated PDT successfully inhibited the grow
97          Using focal photothrombosis via the Rose Bengal method, as well as excitotoxic NMDA lesions,
98 o occlusion of the carotid after exposure to rose bengal or FeCl3 and reduced platelet and fibrin acc
99 intervals up to 16 days post infection (dpi) rose bengal or lissamine green B was instilled in the le
100 ous HSV-1 and viral DNA in eyes treated with rose bengal or lissamine green B.
101 amples that are culture negative and contain rose bengal or lissamine green B.
102 n of dGuo produced 5-Lys-Sp exclusively when Rose Bengal or methylene blue was used to photochemicall
103 g antibody were the negative controls, while rose bengal or protoporphyrin IX with visible light were
104 boflavin + 375 nm irradiation); and Group 5, rose bengal PDT (rose bengal + 518 nm irradiation).
105                                              Rose bengal PDT improved BSCVA among eyes with Fusarium
106 r apical surfaces provide protection against rose bengal penetrance in vitro and suggest a role for m
107 l (PLP)-dependent threonine aldolases with a Rose Bengal photoredox catalyst.
108 as severe focal stroke injury was induced by Rose Bengal photosensitization.
109  DNA samples were extracted from brucellosis Rose Bengal plate test (RBPT) seropositive samples from
110  cell concentrations (8%RB and 3%RB), French rose bengal plate test with 4.5% cell concentration (4.5
111 id automated presumptive test (RAP), Mexican rose bengal plate tests with 8 and 3% cell concentration
112       We evaluated collagen cross-linking by rose bengal plus green light (RGX) in rabbit eyes and in
113 cetonitrile medium and a catalytic amount of Rose Bengal provided the corresponding phenyl, benzyl, o
114 agreement with a model photosensitization by rose bengal (RB(2-)) in deoxygenated aqueous solutions r
115                         The photosensitizers rose bengal (RB) and acridine orange (AO) were localized
116 d a phospholipid coating functionalised with Rose Bengal (RB) and/or 5-fluorouracil (5-FU), were asse
117   In this manuscript, we covalently attached Rose Bengal (RB) to the amphipathic peptide (AMP) C(KLAK
118       ARPE-19 cells preloaded with MC-540 or rose bengal (RB) were sublethally irradiated with green
119 rategy to boost the lymphotropic delivery of Rose Bengal (RB), a hydrosoluble chemotherapeutic, is pr
120 esence of (1) (1)O(2) and (*)OH sensitizers [rose Bengal (RB), perinaphthanone, and H(2)O(2)] and (2)
121 rcially available xanthene dyes as ATRP PCs: rose bengal (RB), rhodamine B (RD), and rhodamine 6G (RD
122 served human amniotic membrane, stained with Rose Bengal (RB), was placed over a full-thickness wound
123 hores namely Nile Red (NR), Eosin Y (EY) and Rose Bengal (RB).
124                                              Rose bengal (RB, 0.1%) was applied to deepithelialized c
125 the vesicular glutamate transport inhibitor, Rose Bengal, reduced astrocytic glutamate release, sugge
126 mine whether exclusion of negatively charged rose bengal requires a negative charge at the cell surfa
127 pulate singlet and triplet excited states of rose Bengal, respectively, the mixed halide perovskites
128 zed separately by six triphenylmethane dyes (rose bengal, rhodamine B, crystal violet, ethyl violet,
129        Bacterial suspensions were mixed with rose bengal, riboflavin, or water according to experimen
130 hB), rhodamine isothiocyanate (RhB-NCS), and rose Bengal (RoseB)-contain an increasing degree of pend
131 bilised microbubbles (MBs), decorated with a Rose Bengal sensitiser, for SDT-based treatment of a pan
132 pression; however, HCLE cells incubated with rose bengal showed that exclusion of the dye was signifi
133                                 In contrast, rose bengal significantly decreased the infectious virus
134 erated epi-off CXL using both riboflavin and rose bengal significantly increases resistance to enzyma
135 ; however, there was no surface keratin, and rose bengal stain results were negative.
136 nifera (testate protists), including 'live' (Rose Bengal stained) and dead tests, in 5 cores (0-1 cm
137 of the eyes treated with NGF plus DHA showed rose bengal staining 30 days after PRK, compared with 50
138                        GK rats had increased rose bengal staining and cornea fragility.
139 ased epithelial proliferation, and decreased rose bengal staining compared with NGF, DHA, or vehicle
140 The current study was undertaken to evaluate rose bengal staining in a human corneal-limbal epithelia
141         The effect of hyperosmotic stress on rose bengal staining in vitro was evaluated by increasin
142 at mucins have a protective role, preventing rose bengal staining of normal ocular surface epithelial
143 tear production, tear film breakup time, and rose bengal staining score were determined.
144 eakup time were significantly decreased, and rose bengal staining was significantly increased.
145 ye diagnosis (Lactoplate, Schirmer test, and Rose Bengal staining), even when the other test measures
146 chirmer testing with and without anesthesia, rose bengal staining, central corneal sensitivity, nucle
147                     STZ rats showed stronger Rose Bengal staining, decreased tear secretion, slightly
148                       All parameters, except rose bengal staining, deteriorated significantly after s
149  mucin layer were assessed via LC-biotin and Rose Bengal staining, respectively.
150 covery after surgery in goblet cell density, rose bengal staining, Schirmer test values without anest
151       The starry sky pattern correlated with rose bengal staining.
152 ier function, as evidenced by an increase in rose bengal staining.
153 ith dry eye, may cause MAM release, allowing rose bengal staining.
154                             We evaluated the Rose Bengal test (RBT) using two protocols, four commerc
155 aggregating to 577 were also tested with the Rose Bengal Test (RBT).
156              The analysis includes serology (Rose Bengal Test and indirect Enzyme-Linked Immunosorben
157 ure to the three zoonoses using serological (Rose Bengal test for Brucella spp., ELISA for C. burnett
158 silica nanoparticles with a photosensitizer, Rose Bengal, tethered to their surface.
159 (-2) hour(-1) bar(-1) and 94.5% rejection to Rose Bengal) that surpasses the permeability-selectivity
160 rhodamine B, rhodamine B isothiocyanate, and rose Bengal), the CsPbI(3)-rose Bengal hybrid with the s
161           Cores were sliced and stained with rose Bengal to detect live specimens of foraminifera.
162                A total of 44% and 78% of the rose bengal-treated and untreated eye samples, respectiv
163 itive than culture for detection of HSV-1 in rose bengal-treated eyes, in that 74% of rose bengal-tre
164  in rose bengal-treated eyes, in that 74% of rose bengal-treated samples were positive by PCR compare
165 e presence of the nonhazardous photocatalyst Rose Bengal under irradiation of visible light.
166 the ion concentration (Ca+2 and Mg+2) in the rose bengal uptake assay.
167  reduced the cellular area protected against rose bengal uptake.
168                                              Rose bengal was also effective in dark and ambient condi
169                                 In addition, rose bengal was used as the photosensitizer for membrane
170        Herein, riboflavin, anthraquinone, or Rose Bengal were allowed to react with the canonical nuc
171 rescein analogs (ethyleosin, phloxine B, and rose bengal) were relatively potent inhibitors of bindin
172 tors, Erythrosin B, Eosin Y, Phloxine B, and Rose Bengal, were determined.
173 ia, only methylene blue with E. coli K12 and rose bengal with C. jejuni showed an enhancing effect.

 
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