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1 ed hydrogen-bonded supramolecular complexes (rosettes).
2  neurons first assemble into a multicellular rosette.
3 -driven randomization of the monomers in the rosette.
4 ngement of its immediate neighbors to form a rosette.
5 axis extension, especially at the centres of rosettes.
6 e required for the formation and dynamics of rosettes.
7 lat sheets that sometimes interleave to form rosettes.
8 lso to induce and maintain stable epithelial rosettes.
9 namics and degradative functions of podosome rosettes.
10  multicellular structures called tetrads and rosettes.
11  marrow adipocytes and mesenchymal stem cell rosettes.
12 as blotches, strands, short white lines, and rosettes.
13  hydrogen-bonded sheets built from hexameric rosettes.
14 area predominantly assembled into multilobed rosettes.
15 ramework to characterize INM dynamics within rosettes.
16 and connects the basal poles of each cell in rosettes.
17 ium and the apicolateral cell membrane in NP rosettes.
18 within cell etxracts of Arabidopsis thaliana rosettes.
19 sitive cells in blood, forming RBC-leukocyte rosettes.
20  percentage of infected erythrocytes forming rosettes.
21 cells and leukocytes, forming characteristic rosettes.
22 nd contributed to the biogenesis of podosome rosettes.
23 omers induces an integrative self-sorting of rosettes.
24 coaggregates comprising a diverse mixture of rosettes.
25 ents in the ability to self-form into neural rosettes.
26 arge matrix-degrading superstructures called rosettes.
27 are delayed in sdk mutants, in particular in rosettes.
28 parasitemia were not correlated with P vivax rosetting.
29       Here, we have identified a new type of rosetting.
30 dwarfism, characterized by reduced growth in rosettes (6.5-fold), roots (4.3-fold), bolts (4.5-fold),
31 rther investigated how different leaves on a rosette acclimate to high light and show that younger le
32  orthorhombic tablets, acicular needles, and rosette aggregates which were identified as cerium oxala
33 mutations, the plants show upward curling of rosette and cauline leaves, in addition to early floweri
34 tion rate changed in different leaves of the rosette and correlated with leaf growth rate.
35        Mutants in SPEN3 and KHD1 had reduced rosette and leaf areas.
36 lopment of these methods, focusing on neural rosette and organoid approaches, and compare their relat
37 d erythrocytes at the schizont stage to form rosettes and in promoting merozoite invasion.
38 s, apatite blades intergrown among carbonate rosettes and magnetite-haematite granules, and is associ
39                                         Both rosetting and cytoadherence are mediated by the parasite
40                                              Rosetting and cytoadherence have been widely studied as
41 iates synchronicity of division and parasite rosetting and reveals that establishment and maintenance
42 on, neural precursor cells (NPCs), assembled rosettes, and differentiated neuronal cells.
43  increased adherens junction abundance, more rosettes, and glomerular expansion.
44 s mutated exhibited serrated leaves, compact rosettes, and, most significantly, short nondehiscent an
45          Here we demonstrate that within the rosette, angiotensin II evokes periodic Ca(v)3-dependent
46 graphitic inclusions in diagenetic carbonate rosettes, apatite blades intergrown among carbonate rose
47          We review recent data to understand rosette architecture and apply maxims derived from compu
48                    We measured the extent of rosette architecture at 22 degrees C and 28 degrees C in
49 us C5a increasing the retention of polarized rosette architecture in human neural progenitors after p
50                         Third, later derived rosettes are characterized by temporal instability in IN
51                                        These rosettes are polarized, transient epithelial structures
52                                Multicellular rosettes are transient epithelial structures that serve
53                                              Rosettes are widely used in epithelial morphogenesis dur
54  molecules involved in this so-called T-cell rosetting are important components of the immunological
55                    To do this, the projected rosette area of 710 worldwide distributed natural access
56 nation of fresh weight, sequential images of rosette area, and labeling of glucose in the cell wall.
57 orphophysiological traits, such as projected rosette area, transpiration rate, and rosette water cont
58 ed predominantly toward nonmalignant T cells rosetting around Reed-Sternberg cells provided meaningfu
59 ernberg cells and by most polyclonal T cells rosetting around Reed-Sternberg cells.
60 tensin II and highlight a novel role for the rosette as a facilitator of cell communication.
61                                KV utilizes a rosette as a prerequisite before forming a lumen surroun
62 d immunosorbent assay, flow cytometry, and a rosetting assay.
63                                              Rosetting assays using CD236R knockdown normocytes deriv
64 lectrostatic interaction that can also guide rosette association allows helicoidal growth of supramol
65                        Our results establish rosette-based CE as an evolutionarily conserved mechanis
66  the olfactory epithelium in adult mice, and rosette-bearing cells often have free centrioles in addi
67  atypical of the complex multi-element spine rosettes borne by most chancelloriids and N. pugio may s
68 ant architecture by increasing the number of rosette branches and reducing inflorescence height.
69 h rate, flowering time, main stem branching, rosette branching, and final plant height and observed s
70 ivator knockout did not affect the extent of rosetting, but almost completely abrogated T-cell activa
71  into dynamic extracellular matrix-degrading rosettes by distinct G protein-coupled receptor agonists
72    Formation and resolution of multicellular rosettes can drive convergent extension (CE) type cell r
73 degradation, due to a disruption of podosome rosettes caused by myosin-IIA overassembly, and a myosin
74 ports the "hexamer of trimers" model for the rosette cellulose synthesis complex that synthesizes an
75 cally place their cytokinetic bridges at the rosette center, where Rab11-associated vesicles transpor
76         Comparison of multiple reconstructed rosette colonies highlighted the variable nature of cell
77 al processes underlying the morphogenesis of rosette colonies in the choanoflagellate Salpingoeca ros
78 ered the presence of elongated cells in some rosette colonies that likely represent a distinct and di
79 ls in S. rosetta with that of multicellular 'rosette' colonies and collar cells in sponges, we recons
80 tory cycle of HPR1-T335D-complemented hpr1-1 rosettes compared to all other HPR1-containing lines.
81  of approximately up to 60 adult Arabidopsis rosettes concurrently.
82                                       Neural rosettes contain NSCs with strong epithelial polarity an
83 l energy per monomer and was consistent with rosette CSC morphology.
84  In summary, the multifaceted data support a rosette CSC with 18 CESAs that mediates the synthesis of
85 e space below an average FF-TEM image of the rosette CSC.
86 but its common ancestor with seed plants had rosette CSCs and a single CESA gene.
87           The moss Physcomitrella patens has rosette CSCs and seven CESAs, but its common ancestor wi
88                             We find that the rosette defect of one mutant, named Rosetteless, maps to
89 isruption of multiple CRFs results in larger rosettes, delayed leaf senescence, a smaller root apical
90 ric embryonal brain tumors with multilayered rosettes demonstrate a unique oncogenic amplification of
91         Rosetteless protein is essential for rosette development and forms an extracellular layer tha
92      Overall, our biophysical perspective on rosette development complements previous genetic perspec
93 at, together, activate, enhance, and inhibit rosette development in the choanoflagellate Salpingoeca
94  with extensive wall ingrowths varied across rosette development in three ecotypes displaying differi
95 t living relatives of animals, multicellular rosette development is regulated by environmental bacter
96              Unexpectedly, the initiation of rosette development requires bacterially produced small
97                               To investigate rosette development, we established forward genetics in
98  set of structural requirements for inducing rosette development.
99  demonstrate its necessity for multicellular rosette development.
100 (ros)) of Arabidopsis (Arabidopsis thaliana) rosettes displays a genuine circadian rhythmicity with a
101 ploids, but not octaploids, generated larger rosettes due to delayed flowering.
102                                              Rosette dynamics are regulated by both planar cell polar
103 xhibits a 6-fold symmetry and is known as a "rosette." Each CSC is believed to contain between 18 and
104          Embryonal tumours with multilayered rosettes (ETMRs) are aggressive paediatric embryonal bra
105           Embryonal tumors with multilayered rosettes (ETMRs) are highly lethal infant brain cancers
106           Embryonal tumors with multilayered rosettes (ETMRs) are primitive neuroectodermal tumors ar
107 ototropin-deficient mutants display impaired rosette evapotranspiration and leaf cooling at high temp
108                                              Rosette expansion and leaf movement exhibited a circadia
109 light signaling to the diurnal regulation of rosette expansion growth and leaf movement in Arabidopsi
110 stomatal development in cotyledons, promotes rosette expansion, and modulates guard cell mechanics in
111 eoretical model of the supramolecular barrel-rosette, favored by a network of intermolecular hydrogen
112 SAX-3 are localized to contracting edges and rosette foci and act to specify edge contraction during
113 oth the expulsion of apoptotic cells and the rosette formation among their neighbor cells.
114 ding defects in the outer limiting membrane, rosette formation and a reduction in functional acuity.
115 ignificant cellular rearrangement, including rosette formation and apical displacement of inner retin
116   We discuss and compare specific models for rosette formation and highlight outstanding questions in
117 eficient cells demonstrating abnormal neural rosette formation and neural progenitor cell proliferati
118 d CD58 knockout or CD2 blockade reduced both rosette formation and T-cell activation.
119 i and act to specify edge contraction during rosette formation and to mediate timely rosette resoluti
120 bind mouse RBCs suggesting a role for CIR in rosette formation and/or invasion.
121  cytoskeletal rearrangements responsible for rosette formation appear to be conserved.
122 genes, we show Fgfr2 is required for adrenal rosette formation by regulating adherens junction abunda
123  two factors interact with IGFBP7 to mediate rosette formation by the IRBC.
124 ere oxidative stress plays a key role in the rosette formation during the degenerative loss of CE.
125                                              Rosette formation has been studied in various developmen
126  that centrioles are amplified via centriole rosette formation in both embryonic development and turn
127 vestigate the function and factors affecting rosette formation in Plasmodium vivax.
128   We have identified a unique contributor to rosette formation in zebrafish Kupffer's vesicle (KV) th
129                              Agonist-induced rosette formation is blocked by pertussis toxin, depende
130 ens junction-mediated constriction, and that rosette formation underlies the maturation of adrenal gl
131                       Similarly, modeling of rosette formation with menadione (MN), led to phospho-Pa
132 binding of infected RBCs to uninfected RBCs (rosette formation), while antibodies targeting STEVOR in
133 D58 expression correlated with the extent of rosette formation, and CD58 knockout or CD2 blockade red
134 stored structural features, including neural rosette formation, and dampened the impact of infection
135 ional tension, a disruption of multicellular rosette formation, and defective convergent extension.
136 re frequently demonstrated lobular disarray, rosette formation, and hemorrhage than those with choles
137 blation of integrin beta1 abolishes the semi-rosette formation, preventing zippering and causing spin
138              Cdc42 activity is essential for rosette formation, whereas G12/13-mediated RhoA-ROCK sig
139 ulation and cellular transitions involved in rosette formation.
140 le of glycophorin C as a receptor in P vivax rosette formation.
141 C 4 region of CD236R significantly inhibited rosette formation.
142  the LD2,3 motifs of Hic-5, is necessary for rosette formation.
143 vadopodia and rosettes, which may facilitate rosette formation.
144 neage and embryonal tumors with multilayered rosettes fully recapitulate a neuronal lineage, while gr
145 computational network modeling to understand rosette function.
146 or family has been implicated in restricting rosette growth in response to stress.
147 loem companion cells caused strongly reduced rosette growth in the absence of wounding.
148               Here, we analyzed the leaf and rosette growth response of six Arabidopsis (Arabidopsis
149 nstructing a multiscale model of Arabidopsis rosette growth.
150              Lipid profiling revealed ala4/5 rosettes had perturbations in glycerolipid and sphingoli
151      Importantly, the IGFBP7-induced type II rosetting hampers phagocytosis of IRBC by host phagocyte
152                             In some studies, rosetting has been associated with malaria pathogenesis.
153                                Multicellular rosettes have recently been appreciated as important cel
154    Here, we study NSC dynamics within Neural Rosettes--highly organized multicellular structures deri
155 ential for the formation and organization of rosettes in active Src-transfected NIH3T3 fibroblasts an
156 sis and a framework for studying the role of rosettes in adult zona glomerulosa tissue maintenance an
157 e of individual invadopodia and invadopodial rosettes in CAFs.
158 ution, but not the formation, of tetrads and rosettes in Fgfr2 mutant limb-bud ectoderm.
159  the formation and resolution of tetrads and rosettes in the mouse embryo, possibly in part by spatia
160  primitive patterns such as neural tube-like rosettes in vitro.
161                        In conclusion, T-cell rosetting in HL is established by formation of the IS, a
162 at induces S. rosetta to form multicellular "rosettes." In this study, we report the identification o
163 reported the planar structure and femtomolar rosette-inducing activity of one rosette-inducing small
164 emical ecology between choanoflagellates and rosette-inducing bacteria, and provide a synthetic probe
165                             We find that the rosette-inducing bacterium Algoriphagus machipongonensis
166  one rosette-inducing small molecule, dubbed rosette-inducing factor 1 (RIF-1), produced by the Gram-
167  but synergizes with activating sulfonolipid rosette-inducing factors (RIFs) to recapitulate the full
168  femtomolar rosette-inducing activity of one rosette-inducing small molecule, dubbed rosette-inducing
169                                              Rosette-induction assays using synthetic RIF-1 stereoiso
170 -week-old plants enabled them to produce new rosette inflorescence stems.
171 sures, in agreement with progressive loss in rosette integrity at later developmental stages.
172 AX-3/Robo pathway cooperate to regulate, via rosette intermediaries, the intercalation of post-mitoti
173 ene core, as pivotal moieties for the barrel-rosette ion channel formation, and the activity of such
174 olecule that self-organizes to form a barrel rosette ion channel in the lipid membrane environment.
175 ation of a bacterially produced inhibitor of rosettes (IOR-1) as well as the total synthesis of this
176                                          The rosette is therefore a reversible pluripotent intermedia
177 he mechanism by which AHLs repress growth in rosettes is unknown.
178                                  In malaria, rosetting is described as a phenomenon where an infected
179                                              Rosetting is more common in vivax than falciparum malari
180 e hexagonal nature of the cellulose synthase rosette, it is assumed that the number of chains must be
181 or five 14-3-3 protein isoforms expressed in rosettes lacked circadian activation of K (ros) Two of t
182 ased numbers of inflorescences, reduction in rosette leaf photosynthesis and earlier fruit ripening.
183 1 displayed an increased number of secondary rosette-leaf branches.
184  novel cross talk existing between seeds and rosette leaves along with mutual effects between the Asp
185 es were obtained for mechanically inoculated rosette leaves and systemically infected cauline leaves
186 s antagonistic effect was blocked in younger rosette leaves by PBS3, a signaling component of the def
187 six Arabidopsis importin-alphas expressed in rosette leaves have an almost identical NLS-binding site
188 igns of early senescence in 6- or 7-week-old rosette leaves in the absence of any pathogen challenge,
189 s to reveal that SMM that was synthesized in rosette leaves of RNAi plants significantly contributed
190 rly, abcb19 mutants develop irregularly wavy rosette leaves that are less sensitive to blue light-med
191 d drought, blunted immune responses in older rosette leaves through the phytohormone abscisic acid si
192 a (i.e., the shoot apical zone versus mature rosette leaves) revealed that the antagonistic interplay
193            This ask1 mutant produces twisted rosette leaves, a reduced number of petals, fewer viable
194 AT) plants have longer hypocotyls and larger rosette leaves, but they also uniquely display early flo
195 ction of genes operating in the SMM cycle of rosette leaves, leading to elevated transport of SMM tow
196 o other common plant traits (e.g., number of rosette leaves, total volume occupied).
197 trolled by gibberellins (GAs) in Arabidopsis rosette leaves.
198 ession of Arabidopsis (Arabidopsis thaliana) rosette leaves.
199 osetting that involves direct interaction of rosetting ligands on IRBC and receptors on URBC, the IGF
200 scope and many more taste buds, patterned in rosette-like clusters, were found than previously report
201                                              Rosette-like stem cells erase constitutive heterochromat
202 ly, combined WNT and MEK inhibition supports rosette-like stem cells, a self-renewing naive-primed in
203 n shortening, establishing a transitory semi-rosette-like structure at the zippering point that promo
204     Invadosomes can reassemble into circular rosette-like superstructures, but the underlying signali
205     Together, our data provide an example of rosette-mediated postnatal tissue morphogenesis and a fr
206 ow that IE of the IT/R29 strain expressing a rosette-mediating PfEMP1 variant (IT4var09) cytoadhere i
207                       We established a novel rosetting model by coculturing HLA-II-matched peripheral
208  previously noted specificity and potency of rosette-modulating molecules, expand our understanding o
209 ally secreted extracellular matrix (ECM) for rosette morphogenesis and show that the interaction of t
210                                              Rosette nanotubes (RNTs) are a class of materials formed
211                                (15)N-labeled rosette nanotubes were synthesized and investigated usin
212 lts in disrupted adherens junctions, reduced rosette number, and dysmorphic glomeruli, whereas beta-c
213                                     Although rosetting occurs in all causes of human malaria, most da
214 ive cells are located lateral to the central rosette of exorhodopsin-positive cells.
215 ent transforms the amorphous epiblast into a rosette of polarized cells.
216  addition, global transcriptional changes in rosettes of amiCOX19 plants resembled those observed und
217 Quantitative proteomics of PG from senescing rosettes of PGM48 overexpression lines showed a dramatic
218  from a single cell-the zygote-multicellular rosettes of S. rosetta develop from a founding cell.
219 iomas and embryonal tumors with multilayered rosettes of the brain both display LIN28A positivity.
220 es known to regulate leaf size in developing rosettes of the hybrids, with the patterns of altered ex
221 nstrated centrilobular cholestasis and focal rosetting of hepatocytes, consistent with a cholestatic
222 ls and trailing cells, which form epithelial rosettes, or protoneuromasts.
223 hallenge going forward is to uncover how the rosette orchestrates the behavior of a functional networ
224 s separate entities; however, the ability of rosetting P. falciparum strains to cytoadhere has receiv
225                                              Rosetting phenomenon has been linked to malaria pathogen
226 d erythrocyte aggregation reminiscent of the rosetting phenomenon.
227 ocyst WNT signals drives the transition into rosette pluripotency by inducing OTX2.
228                           We identify here a rosette pluripotent state defined by the co-expression o
229 ion) by suppressing bud outgrowth from lower rosette positions under low R:FR.
230   Grafts of wild-type floral stems to mutant rosettes produce progeny with enhanced growth and altere
231 oflagellate Salpingoeca rosetta We find that rosettes reproducibly transition from an early stage of
232 eptors on URBC, the IGFBP7-mediated, type II rosetting requires two additional serum factors, namely
233 ring rosette formation and to mediate timely rosette resolution.
234  by cytokinetic bridges that position at the rosette's center.
235        A cellulose synthesis complex with a "rosette" shape is responsible for synthesis of cellulose
236  In seed plants, cellulose is synthesized by rosette-shaped cellulose synthesis complexes (CSCs) that
237 a well-established phenotyping need: imaging rosette-shaped plants.
238  cell migration and the self-organization of rosette-shaped sensory organs in the zebrafish lateral l
239 ow the distal wound response is regulated in rosettes, showing that both axial (shoot-to-root) and ra
240 IP2;1), a major plasma membrane aquaporin in rosettes, shows circadian oscillations and is correlated
241           CDA mutants display a reduction in rosette size and have fewer leaves compared with the wil
242 s, indicating cosegregation of the genes for rosette size and the germination trait.
243 o as radial organization, is associated with rosette size, presumably via mechanical constraints of t
244 nts showed different degrees of reduction in rosette size, thus confirming the role of these genes in
245                In adult plants, PGX3 affects rosette size.
246 nteraction to a negative one, as measured by rosette size.
247 ryza sativa) and petiole elongation in Rumex rosette species.
248 tubule acetylation, which increases podosome rosette stability and is sufficient to inhibit macrophag
249                                 Infection of rosette stage Arabidopsis with a virulent S. sclerotioru
250 ally provided by a Wnt/Fgf signaling system, rosettes still self-organize in the presence of Notch si
251                              IGFBP7-mediated rosette-stimulation was rapid yet reversible.
252 es in response to parasite stimulation, as a rosette-stimulator for Plasmodium falciparum- and P. viv
253 currence, and a multifunctional role for the rosette structure in activity-prolongation and coordinat
254 aMP3 and imaged zG cells within their native rosette structure.
255 ase complex (CSC), visualized as a hexameric rosette structure.
256            Plant cellulose is synthesized by rosette-structured cellulose synthase (CESA) complexes (
257   Yet, emerging data indicate that in native rosette structures in situ, zG cells are electrically ex
258 iferation, and resulted in loss of polarized rosette structures in the aggregates.
259 ow a six-particle cellulose synthase complex rosette synthesizes microfibrils likely comprised of eit
260 ed by formation of the IS, and activation of rosetting T cells critically depends on the interaction
261  cells were CD25(-) provided that associated rosetting T cells expressed CD25.
262 s and showed IS formation with activation of rosetting T cells.
263 LH39, bHLH100, and bHLH101 developed smaller rosettes than wild-type plants in soil.
264 -signaling cells forming a single cell thick rosette that demarcates: domains of cell proliferation i
265 that CGCs could be connected to the multiple rosettes that arise from a single MF input.
266 Cs exist in the plasma membrane as six-lobed rosettes that contain at least three different cellulose
267 nal gland arrange in distinct multi-cellular rosettes that provide a structural framework for adrenal
268                                Unlike type I rosetting that involves direct interaction of rosetting
269                                          The rosette then activates MEK signals that induce lumenogen
270 ls in the adult adrenal cortex organize into rosettes through adherens junction-mediated constriction
271 es of the Arabidopsis (Arabidopsis thaliana) rosette throughout the vegetative stage of growth.
272 abolites was confined locally to the central rosette tissue.
273 oaches to leaves of the Arabidopsis thaliana rosette to characterize their protein degradation rate a
274                                Next, we used rosettes to investigate radial cell-to-cell transport of
275                                              Rosetting to P vivax asexual and sexual stages was evide
276 X-7, HMR-1 and DLG-1 function redundantly in rosette-to-epidermis attachment.
277 inactive regions of human chromosomes yields rosettes, topological domains and contact maps much like
278                       Evaluation of root and rosette transcriptomes revealed an early transcriptional
279 fect of deer exclusion on Alliaria came from rosette transitions, whereas the largest positive effect
280 scopy (TEM) images and image averages of the rosette-type CSC, revealing the frequent triangularity a
281 epiblast of the mouse blastocyst generates a rosette, undergoes lumenogenesis and forms the primed pl
282                                              Rosette vernalization increased seed germination in dive
283           We first tested whether effects of rosette vernalization persisted to influence seed germin
284 sion at different life stages in response to rosette vernalization.
285                    PAR-6 is localized to the rosette vertex and dendrite tips, and promotes DYF-7 loc
286 jected rosette area, transpiration rate, and rosette water content, were correlated with changes in t
287                               Using a strain-rosette, we demonstrate the existence of transverse stra
288 nd redox-related genes, whereas those of the rosette were related to the regulation of development an
289 adopodia dynamics and their coalescence into rosettes were also dependent on Rac1, formin, and myosin
290                                        These rosettes were preferentially disassembled in response to
291                            The vertex of the rosette, which becomes the dendrite tips, is attached to
292 ine and cyanuric acid units into a hexameric rosette, which brings together poly(A) triplexes with a
293                        Second, early forming rosettes, which are abundant with founder NSCs and corre
294                   In contrast, later derived rosettes, which are characterized by reduced NSC capacit
295 -actin fibers emanating from invadopodia and rosettes, which may facilitate rosette formation.
296 rganization of invadopodia into higher-order rosettes, which may promote the localized matrix degrada
297  cytoadherence of IT/R29 IE is distinct from rosetting, which is primarily mediated by NTS-DBL1alpha
298 , homochiral polymers comprised of hexameric rosettes with structural features that resemble nucleic
299 oadhere to microvascular endothelium or form rosettes with uninfected erythrocytes to survive in vivo
300  was induced by thrombin-activated platelets rosetting with neutrophils and was inhibited by anti-P-s

 
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