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1  the LD2,3 motifs of Hic-5, is necessary for rosette formation.
2 vadopodia and rosettes, which may facilitate rosette formation.
3 ulation and cellular transitions involved in rosette formation.
4 le of glycophorin C as a receptor in P vivax rosette formation.
5 C 4 region of CD236R significantly inhibited rosette formation.
6 g malaria, through the mechanism of enhanced rosette formation.
7 es spanning multiple pairs of cells leads to rosette formation.
8 y affect the frequency and directionality of rosette formation.
9 d tests their ability to inhibit erythrocyte rosette formation.
10 lted in an increase of sialic acid-dependent rosette formation.
11 ity, indicating an essential role for CR1 in rosette formation.
12 nditional knockout (PfDeltaCDP) promotes RBC rosette formation.
13 XRW motif, affect protein folding and/or CSC rosette formation.
14 oth the expulsion of apoptotic cells and the rosette formation among their neighbor cells.
15 ding defects in the outer limiting membrane, rosette formation and a reduction in functional acuity.
16 ignificant cellular rearrangement, including rosette formation and apical displacement of inner retin
17  accompanied by a reduction in multicellular rosette formation and axis elongation.
18   We discuss and compare specific models for rosette formation and highlight outstanding questions in
19 nduced downregulation of NID1 impairs neural rosette formation and integrity, likely contributing to
20 eficient cells demonstrating abnormal neural rosette formation and neural progenitor cell proliferati
21 ting the first four mitotic events disrupted rosette formation and prevented normal KV rounding.
22 d CD58 knockout or CD2 blockade reduced both rosette formation and T-cell activation.
23 i and act to specify edge contraction during rosette formation and to mediate timely rosette resoluti
24 bind mouse RBCs suggesting a role for CIR in rosette formation and/or invasion.
25 tructures align across multiple cells during rosette formation, and adherens junction proteins assemb
26 s, intra-acinar lymphocytes and eosinophils, rosette formation, and canalicular cholestasis yielded a
27 D58 expression correlated with the extent of rosette formation, and CD58 knockout or CD2 blockade red
28 s, the presence or absence of emperipolesis, rosette formation, and cholestasis in a blinded fashion
29 stored structural features, including neural rosette formation, and dampened the impact of infection
30 ional tension, a disruption of multicellular rosette formation, and defective convergent extension.
31        Portal and intra-acinar plasma cells, rosette formation, and emperiopolesis were features that
32 re frequently demonstrated lobular disarray, rosette formation, and hemorrhage than those with choles
33 e podJ gene are nonchemotactic, deficient in rosette formation, and resistant to polar bacteriophage,
34  cytoskeletal rearrangements responsible for rosette formation appear to be conserved.
35 otably, deficits in proliferation and neural rosette formation are rapidly reversed upon silencing on
36 calized to the leading zone are required for rosette formation, atoh1a expression, and primordium mig
37                                              Rosette formation by COS cells expressing a form of CD33
38 genes, we show Fgfr2 is required for adrenal rosette formation by regulating adherens junction abunda
39  two factors interact with IGFBP7 to mediate rosette formation by the IRBC.
40  stiffen global tissue mechanics and enhance rosette formation during morphogenesis.
41 ere oxidative stress plays a key role in the rosette formation during the degenerative loss of CE.
42 in, NR2F1, NR2F2, and IRX2 - in the onset of rosette formation, during spontaneous neural differentia
43                                              Rosette formation has been studied in various developmen
44 ut inflorescence height, flowering time, and rosette formation have greatly diverged between the nati
45  that centrioles are amplified via centriole rosette formation in both embryonic development and turn
46 cal regulator of cell spreading and podosome rosette formation in immature DCs.
47                  Eliminating Rpe65 prevented rosette formation in Nrl(-/-) retinas; supplementation o
48 vestigate the function and factors affecting rosette formation in Plasmodium vivax.
49 yield), which acts as bacterial inhibitor of rosette formation in S. rosetta.
50 aling was previously shown to be crucial for rosette formation in the pLLp.
51 n network to promote apical constriction and rosette formation in the pLLp.
52 hich is required for apical constriction and rosette formation in the pLLp.
53 d this appears to be linked to photoreceptor rosette formation in the rodless (cone-only) Nrl(-/-) re
54 roblast growth factor (Fgf) signals regulate rosette formation in the zebrafish posterior lateral lin
55   We have identified a unique contributor to rosette formation in zebrafish Kupffer's vesicle (KV) th
56 to Plasmodium falciparum-infected cells, and rosette formation is associated with severe malaria.
57                              Agonist-induced rosette formation is blocked by pertussis toxin, depende
58                            The potential for rosette formation is included, along with various juncti
59                            We also show that rosette formation is not a hallmark of retinoblastoma di
60 ne matrix sheaths are not, and photoreceptor rosette formation is observed.
61 h the inhibition of macrophage spreading and rosette formation, MacMARCKS mutant also inhibits integr
62 -coated sheep erythrocytes, and also induced rosette-formation of erythrocytes with human monocytes a
63 blation of integrin beta1 abolishes the semi-rosette formation, preventing zippering and causing spin
64                                     Instead, rosette formation reflects extensive cell-cell contacts
65 otic events during KV rounding coincide with rosette formation, spindle rotation and cell extrusion,
66  macrophages, (iii) is required for podosome rosette formation triggered by Hck, and (iv) is necessar
67 tDJ-1C and AtDJ-1E improved plant height and rosette formation under physiological conditions.
68 ens junction-mediated constriction, and that rosette formation underlies the maturation of adrenal gl
69                                              Rosette formation was analyzed in HCMV-infected and mock
70                                              Rosette formation was found to require de novo flagella
71               We went on to show that proper rosette formation was required for subsequent morphogene
72 hat OTX2 is highly expressed at the onset of rosette formation, when rosettes comprise no more than 3
73              Cdc42 activity is essential for rosette formation, whereas G12/13-mediated RhoA-ROCK sig
74 contrast to the conventional way of studying rosette formation, which involves induction of neuronal
75 binding of infected RBCs to uninfected RBCs (rosette formation), while antibodies targeting STEVOR in
76                       Similarly, modeling of rosette formation with menadione (MN), led to phospho-Pa
77 of immunized donors were separated following rosette formation with tetanus toxin (TT)-coupled immuno
78 mitive neuroectoderm, which is manifested by rosette formation, with consecutive differentiation into