ライフサイエンスコーパス: rot

1 in A (AntA), myxothiazol (Myx), or rotenone (Rot)).

2 the transcription factor repressor of toxin (Rot).

3 y on crystalline cellulose (reduced in brown rot).

4 kidney (HEK) 293 cell using electrorotation (ROT).

5 he transcription factor repressor of toxins (Rot).

6 aroma precursors also increased during noble rot.

7 on largely via inactivation of the repressor Rot.

8 plant cell walls, a characteristic of white rot.

9 aureus exoprotein expression (Sae) TCS, and Rot.

10 chitinases ChitA and ChitB during maize ear rot.

11 including increased incidence of blossom-end rot.

12 tional regulation of staphylococcal genes by Rot.

13 ral alternative for controlling banana crown rot.

14 es for cap repressors such as SaeR, CodY and Rot.

15 ng take-all and pythium and rhizoctonia root rots.

16 Rotational spinning in ROT-1' occurs at 97,000 s(-1) at 25 degrees C but is vir

17 In the presence of suitable reactants, ROT-2 acted as a catalytic machinery catalyzing a click

18 he self-assembly of the four-component rotor ROT-2 through metal translocation.

19 Rot (3 mg/kg/day) was infused subcutaneously to male Lew

20 which is slower than the overall in plane (D(rot) = 3.2 rad(2)/ns) for the lipid molecule.

21 ing (scRNA-seq) with Raman optical tweezers (ROT), a label-free single-cell identification and isolat

22 he promoter sequence of repressor of toxins (Rot), a master transcriptional regulator responsible for

23 n results from the Agr-mediated reduction in Rot activity rather than as a direct effect of the Agr s

24 We compared the transcriptional profile of a rot agr double mutant to that of its agr parental strain

25 bolomics approach, we demonstrate that noble rot alters the metabolism of cv Semillon berries by indu

26 ide host range for the ability to cause root rot and a very narrow host range for the ability to caus

27 Our data also indicate that Rot and agr have opposing effects on select target genes

28 ion of lignin has been well studied in white-rot and brown-rot fungi, but is much less well studied i

29 um rather than a dichotomy between the white-rot and brown-rot modes of wood decay.

30 usal agent of foliar blight, root rot, fruit rot and crown rot syndromes in squash (Cucurbita moschat

31 ich also contains non-lignin-degrading brown rot and ectomycorrhizal species.

32 racted in parallel lineages leading to brown rot and mycorrhizal species.

33 inned berries, was a common outcome of noble rot and red-skinned berry ripening.

34 To delineate the interaction of rot and sae and the contribution of sarT to hla expressi

35 of sarT to hla expression, an assortment of rot and sae isogenic single mutants, a rot sae double mu

36 Here we demonstrate that Rot and SaeR, the response regulator of the Sae TCS, syn

37 The SarA homologs Rot and SarT were previously shown to be repressors of h

38 In vitro, abcA expression depends on rot and sarZ regulators.

39 l pathogen that is responsible for maize ear rot and stalk rot diseases worldwide.

40 Until now, Rot and the Sae TCS have been proposed to work in opposi

41 e systemic resistance against Fusarium crown rot and to simultaneously improve tomato plant growth an

42 we have determined the crystal structure of Rot and used this information to probe the contribution

43 while limiting pythium and rhizoctonia root rots and fusarium root and crown rot in direct-seed syst

44 firmness or susceptibility to infestations, rots and mechanical damage, but limited information on t

45 patterns of LFF differed from those of wood-rotting and ectomycorrhizal taxa, likely reflecting cont

46 nization in intercellular spaces, leading to rotting and to the disruption of plant cell wall and mem

47 N(rot) and PSA were calculated with QikProp or Cerius2.

48 The relationship of rotatable bond count (N(rot)) and polar surface area (PSA) with oral bioavailabi

49 broad host-range pathogens that cause wilt, rot, and blackleg diseases on a wide range of plants.

50 scopy, then nondestructively isolated out by ROT, and finally RNA-sequenced.

51 le factors, including Agr, SarA, SarS, SarT, Rot, and MgrA.

52 pression of a key transcriptional regulator, Rot, and of several important exoproteins and surface fa

53 lts showed that coatings are able to prevent rot appearance in every sample, corroborating with the a

54 ct wild C. elegans population states in both rotting apples and reconstructed microbiomes: alpha-Prot

55 fusion due to a steeper length dependence, D(rot) approximately L(-)(3) versus D(trans) approximately

56 es without significant lignin removal (brown rot) are polyphyletic, having evolved multiple times fro

57 Dielectrophoresis (DEP) and electrorotation (ROT) are two electrokinetic phenomena exploiting nonunif

58 The role of Agr and Rot as regulators of ssl expression was observed across

59 Raman optical tweezers (ROT) as a label-free technique plays an important role i

60 During noble rot, B. cinerea induced the expression of key regulators

61 e the primary virulence determinants of soft rotting bacteria such as the potato pathogen, Pectobacte

62 been classified as either white rot or brown rot, based on the ability (in white rot only) to degrade

63 nokaryotic strains PC9 and PC15 of the white rot basidiomycete Pleurotus ostreatus.

64 The wood-rotting basidiomycete Schizophyllum commune was investig

65 Ca deficiency disorders such as blossom-end rot (BER) in tomato (Solanum lycopersicum) fruit may be

66 Blossom-end rot (BER) in tomato fruit (Solanum lycopersicum) is beli

67 Furthermore, blossom-end rot (BER) in tomato may be linked to changes in CAX acti

68 Evaluation of Rot binding to different activated and repressed promote

69 ffected by diseases such as sclerotinia stem rot, blackleg, and alternaria black spot resulting in si

70 benular nuclei in JP17 rats; the rat OT-bNP (rOT-bNP) transgene was not expressed in either line.

71 nvestigate this question, we determined that Rot bound to the protease promoters, and we observed tha

72 To assess the generality of the white-rot/brown-rot classification paradigm, we compared the g

73 The function of Rot, but not the transcription of rot, is regulated by t

74 llulose is a distinguishing feature of brown-rot, but the biochemical mechanisms and underlying genet

75 of a major pleiotropic transcription factor, Rot, by blocking its translation.

76 c electrostatic charge profile suggests that Rot can orient the WHTH domain to bind DNA.

77 ic resistance in tomato plants against crown rot caused by a soilborne pathogen, Fusarium oxysporum f

78 h southern leaf blight and anthracnose stalk rot caused by Cochliobolis heterostrophus and Colletotri

79 Apple bitter rot caused by Colletotrichum species is a growing proble

80 results of this work demonstrated that noble rot causes a major reprogramming of berry development an

81 protein production and pathogenicity in soft rot-causing Erwinia carotovora subsp. carotovora.

82 functional homologs of rsmB(Ecc) in non-soft-rot-causing Erwinia species, we cloned the rsmB genes of

83 e miPSMA scores that were obtained using the RoT CF were compared with those obtained using the actua

84 sequence of carbohydrate removal among brown rot clades.

85 assess the generality of the white-rot/brown-rot classification paradigm, we compared the genomes of

86 Given the qualitative nature of the 'brown rot' classifier, we aimed to quantify and compare the te

87 us and the causal agent of anthracnose stalk rot, Colletotrichum graminicola.

88 es insight into a precise mechanism by which Rot controls virulence factor regulation in S. aureus.

89 re compared, and a simplified rule-of-thumb (RoT) correction factor (CF) was derived for lesions at v

90 ordant between the corrected dataset and the RoT dataset for 205 of 232 lesions (88.4%).

91 Ascomycota fungi are causing extensive soft rot decay at all sites regardless of climate and local e

92 Brown rot decay removes cellulose and hemicellulose from wood-

93 Rot did not affect the State 4Deltapsi of RBM and rat li

94 effects, suggesting for the first time that Rot differentially interacts with target promoters.

95 Surprisingly, we observed that Rot directly interacts with and activates the ssl promot

96 A. euteiches causes root rot disease in legumes and is a limiting factor in legum

97 White root rot disease is threatening rubber tree (Hevea brasiliens

98 is pv. campestris, the causal agent of black rot disease of Brassica plants, possesses a specific sys

99 campestris (Xcc), the causal agent of black rot disease of Brassicaceae.

100 nic enterobacterium responsible for the soft rot disease of many plants of economic importance.

101 adantii is a plant pathogen that causes soft rot disease on vegetable and potato crops.

102 The impact of Lys12 on progression of root rot disease, together with the finding that similar gene

103 t is responsible for maize ear rot and stalk rot diseases worldwide.

104 ectate component of plant cell walls in soft rot diseases.

105 n evolutionary shift from white-rot to brown-rot during which the capacity for efficient depolymeriza

106 Soft-rot Enterobacteriaceae (SRE), which belong to the genera

107 In this issue of Developmental Cell, Rot et al. (2014) show that muscle and the fracture call

108 promoters revealed that certain mutations on Rot exhibit promoter-specific effects, suggesting for th

109 factor (sigma(B)) has a repressive effect on rot expression during the postexponential phase of growt

110 A modest upregulation of rot expression was also observed in sarS-negative strain

111 An upregulation of rot expression was observed during the stationary phase

112 Fusarium crown rot (FCR) of wheat and barley, predominantly caused by t

113 um conicum were resistant to breakdown after rotting for extended periods or high-temperature acid tr

114 We named this ORF rot (for repressor of toxins) (GenBank accession no. AF1

115 The relative order of the values of k(rot) for different acyl groups parallels their reported

116 tational diffusion constants (D(trans) and D(rot)) for NRs were in good agreement with Tirado and Gar

117 diverse bacterial strains cultured from such rotting fruit allow C. elegans growth and reproduction w

118 r fruit fly larvae consume yeasts growing on rotting fruit and have evolved resistance to fermentatio

119 fungus Penicilium purpurogenum obtained from rotting fruit of the tree Averrhoa bilimbi growing in Sr

120 In their natural habitat of rotting fruit, the nematode Caenorhabditis elegans feeds

121 ian, the causal agent of foliar blight, root rot, fruit rot and crown rot syndromes in squash (Cucurb

122 bacteria of C. elegans' natural habitats of rotting fruits and vegetation to provide greater context

123 cumented in the literature, although ripe or rotting fruits play an important role as a food or brood

124 Loss of Rot function during the postexponential phase of growth

125 he contribution made by specific residues to Rot function.

126 Secreted basidiomycete white-rot fungal laccases orchestrate this with high thermodyn

127 d proteins (one third of the predicted white-rot fungal proteome) in a single experiment, as one of t

128 The use of white rot fungi (WRF) for bioremediation of recalcitrant trace

129 s a heme-containing enzyme produced by white-rot fungi and is part of the extracellular lignin degrad

130 specialized lignocellulolytic microbes-soft rot fungi and tunnelling bacteria-are active and degrade

131 White rot fungi efficiently degrade lignin, a complex aromatic

132 Some white-rot fungi have been reported to lack LiP when grown on d

133 apable of substantial lignin decay are white rot fungi in the Agaricomycetes, which also contains non

134 Root rot fungi of the Heterobasidion annosum complex are the

135 Brown-rot fungi such as Postia placenta are common inhabitants

136 revious lignin biodegradation studies, white rot fungi were used to produce functional biopolymers fr

137 kely catalysts of ligninolysis in many white-rot fungi, because they have the unusual ability to depo

138 has been well studied in white-rot and brown-rot fungi, but is much less well studied in bacteria.

139 The technology has focused on the white rot fungi, which have complex extracellular ligninolytic

140 ed multiple times from lignin-removing white rot fungi.

141 yketide synthase genes specific to the brown-rot fungi.

142 iomycetes, collectively referred to as white rot fungi.

143 d PFCAs and polyfluorocarboxylic acids, wood-rotting fungi should be evaluated as potential candidate

144 sized and the kinetics of laccase from white rot fungus adsorption and its direct electro-catalytic a

145 The white-rot fungus Ceriporiopsis subvermispora delignifies ligno

146 terium Pseudomonas putida GB-1 and the white-rot fungus Coprinellus sp. The rate of Mn(II) oxidation,

147 promote the degradation of Azo dye by white-rot fungus Irpex lacteus CD2 in the lignin/dye/fungus sy

148 ion of the orientation of laccase from white rot fungus on multi-walled carbon nanotube surface modif

149 a copper metalloenzyme produced by the wood-rot fungus Phanerochaete chrysosporium as an essential c

150 Comparisons with the closely related white-rot fungus Phanerochaete chrysosporium support an evolut

151 decomposition of carbamazepine by the white-rot fungus Pleurotus ostreatus in liquid culture compare

152 The white rot fungus Pleurotus ostreatus is able to completely rem

153 target screening for TPs formed by the white-rot fungus Pleurotus ostreatus.

154 Zn, in wood being decayed by the model brown rot fungus Serpula lacrymans.

155 This is a soft-rot fungus that may be contributing to wood degradation.

156 tructure that the principal degrader, a soft-rot fungus, mobilized the king's highly (15)N-enriched n

157 n of 6:2 FTOH [F(CF2)6CH2CH2OH] by the white-rot fungus, Phanerochaete chrysosporium, was investigate

158 he secretome of Pleurotus ostreatus, a white rot fungus.

159 emediation of environmental hazards by white-rot fungus.

160 parative and functional genomics of the "dry rot" fungus Serpula lacrymans, derived from forest ances

161 the dominant oxidant during incipient white rot had a half-life under 0.1 s.

162 ciated with resistance to Phytophthora crown rot in an F(2) population (n = 168) derived from a cross

163 We finally characterized the impact of noble rot in botrytized wines.

164 l targets for MAS against Phytophthora crown rot in C. moschata.

165 thocyanins is a consistent hallmark of noble rot in cv Semillon berries.

166 ctonia root rots and fusarium root and crown rot in direct-seed systems.

167 eat confers resistance to both FHB and crown rot in diverse wheat backgrounds without yield penalty,

168 Cadophora species known to cause soft rot in polar environments were the most abundant Ascomyc

169 The transcriptional profiles of Rot in sigma(B)-positive and sigma(B)-negative strains i

170 lts provide further insight into the role of Rot in the regulatory cascade of S. aureus virulence gen

171 Resistance to Phytophthora crown rot in University of Florida breeding line #394-1-27-12

172 ere submitted to water loss, texture, color, rot index, and physic-chemical assays.

173 Noble rot-infected berries of cv Semillon, a white-skinned var

174 Rot infusion affected RLM little.

175 bind within the major groove, contributes to Rot interaction with target promoters.

176 says, we found that the repression of hla by rot is dependent on sae.

177 Our findings indicate that Rot is not only a repressor but a global regulator with

178 rough rotational diffusion and showed that D(rot) is an advantageous observable for monitoring bindin

179 Repressor of toxins (Rot) is known to be a global regulator of virulence gene

180 and discovered that the repressor of toxins (Rot) is part of this pathway.

181 unction of Rot, but not the transcription of rot, is regulated by the staphylococcal accessory gene r

182 ith encapsulated essential oil showed bitter rot lesions three times smaller than the ones treated wi

183 characterization is shared among these brown rot lineages, despite their diverse genomes and secretom

184 control programs, utilizes traps baited with rotting liver or a blend of synthetic chemicals such as

185 sumed sizeable crustaceans that sheltered in rotting logs.

186 data implicated the importance of sae in the rot-mediated repression of hla in S. aureus.

187 However, the mechanism by which Rot mediates gene regulation has remained elusive.

188 timicrobial activity was tested against root-rot microorganisms, among others.

189 a dichotomy between the white-rot and brown-rot modes of wood decay.

190 of growth likely involves degradation of the rot mRNA but not the inhibition of rot transcription.

191 We propose that the RNAIII-rot mRNA interaction plays a key role in agr regulation

192 omplementary to two G-rich stem loops of the rot mRNA translation initiation region (TIR).

193 tary stem loops, followed by the cleavage of rot mRNA.

194 Finally, we tested the rot mutant in a mouse catheter model of biofilm infectio

195 creted protease genes were up-regulated in a rot mutant, and we hypothesized that this regulation cou

196 the cysteine proteases, were increased in a rot mutant.

197 sing multiple different assays, and we found rot mutants in other strain lineages were also biofilm d

198 We show that a rot mutation returns wild-type virulence to an agr mutan

199 Unlike bunch rot, noble rot promotes favorable changes in grape berri

200 e majority of them cause the collar and root-rot of diverse plant species.

201 Fungal bunch rot of grapes leads to production of detrimental flavour

202 d most efficiently by fungi that cause white rot of wood.

203 ed by EPA in 1995 for control of postharvest rots of pome and citrus fruit, respectively, and are com

204 the vascular parenchyma, and maceration and rotting of the petiole and central bud.

205 LFPs of relay nuclei (the nucleus rotundus, ROT) of tectofugal pathway were also acquired.

206 The evaluation of Botrytis cinerea as noble rot on withered grapes is of great importance to predict

207 or brown rot, based on the ability (in white rot only) to degrade lignin along with cellulose and hem

208 ions due to extreme susceptibility to a root-rotting oomycete (Pythium spp), demonstrating that these

209 ve typically been classified as either white rot or brown rot, based on the ability (in white rot onl

210 less efficient wood-degraders such as brown rot or mycorrhizal fungi.

211 s suggest that wet heartwoods, regardless of rot or not, occur widely in living trees on various habi

212 the soil, previously being leached out from rotting PA-plants.

213 QDR) to different isolates of the plant root rot pathogen Aphanomyces euteiches, from a GWAS performe

214 tive response elicitor harpin (HrpN) of soft rot pathogen Erwinia chrysanthemi strains 3937 and EC16

215 Here we found that the soybean root rot pathogen Phytophthora sojae evades the soybean Resis

216 -dependent manner by infection with the root-rot pathogen Pythium irregulare.

217 even when immersed in inoculum from the root-rotting pathogen Nectria haematococca.

218 ion of ions and small molecules play in soft-rot pathogenicity.

219 Soft rot pathogens cause significant losses worldwide in frui

220 border cells of roots exposed to cotton root rot (Phymatotrichopsis omnivora), and the value of 7,4'-

221 ade with grapes infected and wilted by brown rot (Plasmopara viticola).

222 Rot plays a key role in regulating S. aureus virulence t

223 reconstruction from genomes of extant white-rot Polyporales, and evaluating their oxidative attack o

224 sition in S. lacrymans relative to the brown rot Postia placenta.

225 owed that turned green and also sprouting or rotting potato flesh contain high amounts of toxic solan

226 We show that insertion into the rot promoter by IS256 results in the derepression of cyt

227 ococcal lysates retarded the mobility of the rot promoter fragment and that the effect was reduced, b

228 nity purification of proteins binding to the rot promoter fragment, followed by N-terminal protein se

229 Primer extension analysis of the rot promoter revealed a number of discreet products.

230 Unlike bunch rot, noble rot promotes favorable changes in grape berries and the

231 dependent upon the presence of a functional Rot protein, and Rot was shown to be able to bind to the

232 dependent upon the presence of a functional Rot protein.

233 In Rot rat brain mitochondria (Rot-RBM) there was a 30-40%

234 -fold increase in O*2- or H2O2 generation in Rot-RBM oxidizing glutamate.

235 However, Rot-RBM required two times less Ca2+ to initiate permeab

236 In Rot rat brain mitochondria (Rot-RBM) there was a 30-40% inhibition of respiration in

237 oxygen species that increased 2-fold in the Rot-RBM.

238 The resulting energy of activation DeltaG()rot reflects the spatial requirement of the ortho substi

239 Transcriptional study has also shown that rot repressed sae, especially at the sae P3 promoter.

240 esults from the Agr system's inactivation of Rot repressor activity.

241 Rot (repressor of toxins), a SarA homologue, was previou

242 irst QTLs associated with Phytophthora crown rot resistance in C. moschata.

243 erived from backcrossing a Phytophthora root rot resistance locus from the donor parent Kingwa into t

244 ow efficient breeding for Phytophthora crown rot resistance through marker-assisted selection (MAS).

245 Mutations in rot restore in vitro toxin production to agr-negative st

246 Noble rot results from exceptional infections of ripe grape (V

247 activities correlate with hla expression in rot, rot sae, and rot sae sarT mutants of COL.

248 y mildew (Rmd-c), Phytophthora stem and root rot (Rps2), and an ineffective nodulation gene (Rj2) map

249 nt of rot and sae isogenic single mutants, a rot sae double mutant, and a rot sae sarT markerless tri

250 was not able to rescue the hla defect of the rot sae double mutant.

251 ngle mutants, a rot sae double mutant, and a rot sae sarT markerless triple mutant were constructed f

252 ate with hla expression in rot, rot sae, and rot sae sarT mutants of COL.

253 A rot sae sarT triple mutant was not able to rescue the hl

254 vities correlate with hla expression in rot, rot sae, and rot sae sarT mutants of COL.

255 rbon--and is derived from an ancestral white rot saprotrophy in which both lignin and cellulose are d

256 of both ectomycorrhizal biotrophy and brown rot saprotrophy were accompanied by reductions and losse

257 ntary RNAs would be predicted to occlude the rot Shine-Dalgarno (SD) site and to block rot translatio

258 ceae are transitional between those of white rot species and less efficient wood-degraders such as br

259 ose, and they group close to the model white-rot species Phanerochaete chrysosporium in the PCA.

260 comycetes, which is reconstructed as a white rot species, and then contracted in parallel lineages le

261 ent and functional in both blackleg and soft rotting species of Erwinia.

262 nables the study of the variation in DEP and ROT spectra according to different parameters, such as t

263 to the SMF, no difference was noted between ROT spectra of unexposed and exposed yeast cells, which

264 We also compared the ROT spectrum and the extracted electrical characteristic

265 to dissect the genetics of Sclerotinia stem rot (SSR) [caused by Sclerotinia sclerotiorum (Lib.) de

266 leaf senescence, in turn, leads to charcoal rot, stalk lodging, and significant yield loss.

267 foliar blight, root rot, fruit rot and crown rot syndromes in squash (Cucurbita moschata), is a devas

268 in the Polyporales, which harbors many white-rot taxa, whereas MnPs and VPs are more widespread and m

269 three additional regulators (SarA, SarZ, and Rot) that bind to the overlapping promoter region of abc

270 y, focusing first on fusarium root and crown rot, then including take-all and pythium and rhizoctonia

271 239) because of the activity associated with rot::Tn917 mutant strains.

272 ium support an evolutionary shift from white-rot to brown-rot during which the capacity for efficient

273 the direction of entopallium to NCL and from ROT to entopallium.

274 Herein we designed a chip utilizing ROT to isolate a specific single cell.

275 dominately processing color information from ROT to NCL, relayed by entopallium, and blue could trigg

276 Thus S. aureus uses the transcription factor Rot to repress secreted protease levels in order to buil

277 Chronic infusion of rotenone (Rot) to Lewis rats reproduces many features of Parkinson

278 The DEP force and ROT torque depend on the respective polarizabilities of

279 ta suggest that the primary mechanism of the Rot toxic effect on RBM consists in a significant increa

280 on of the rot mRNA but not the inhibition of rot transcription.

281 f these loops are critical for inhibition of rot translation and suggest that this inhibition is affe

282 ses, we show that RNAIII does, indeed, block rot translation.

283 he rot Shine-Dalgarno (SD) site and to block rot translation.

284 we demonstrate that the production of ROS by ROT treated AML12 hepatocyte cells dissociates the Trx-A

285 (CI) generated ROS, in response to rotenone (ROT) treatment, is based on the ability of reduced Trx t

286 egies that employ reporters of transfection (RoT), TREE significantly improved the editing efficiency

287 nents, e.g. potential scaffolding cofactors (Rot/TROVE and SPFH/Band-7 modules) with their respective

288 unusual; the rotational transition state 9a-rot-ts connects directly to the orthogonal transition st

289 A more nuanced categorization of rot types is needed, based on an improved understanding

290 oteomic analysis of grapes infected by noble rot under withering conditions to identify possible mark

291 ground state decreased the expected DeltaG()rot values resulting in a minimization of their apparent

292 Rot was found to form a dimer, with each monomer harbori

293 he presence of a functional Rot protein, and Rot was shown to be able to bind to the seb promoter.

294 llin-resistant S. aureus strain deficient in Rot was unable to form a biofilm using multiple differen

295 scripts and protein production controlled by Rot, we observed that all the secreted protease genes we

296 rich loops of RNAIII and the G-rich loops of rot, we show that the sequences of these loops are criti

297 ciated with resistance to Phytophthora crown rot were detected on chromosome 4 (QtlPC-C04), 11 (QtlPC

298 )Ir with species of the type ROAr, RO2CMe or ROTs, where R possesses beta-C-H bonds (e.g., R = ethyl

299 s of cattle such as liver abscesses and foot rot, which have economically important consequences for

300 ate recurring consumption of crustaceans and rotted wood by large Late Cretaceous dinosaurs.