ライフサイエンスコーパス: round cell

1 large multipolar cells and a group of small round cells.

2 h the histologic grade and the percentage of round cells.

3 Rac1 interfered with the ability of ExoT to round cells.

4 he apical cell and the shanks of the central round cells.

5 sponding more strongly to ECM alignment than rounded cells.

6 yer generates rapid shape transformations in rounded cells.

7 hout MreB exhibit an instability that favors rounded cells.

8 in severely stunted plants composed of small rounded cells.

9 l cell morphology, resulting in larger, more rounded cells.

10 s in this pathway lead to bent, branched, or round cells [2].

11 ) were included: 23 dendritic cell (40%), 21 round cell (37%), 2 dermal nests (4%), 2 combined (4%),

12 ic lbc and p115, increased the percentage of rounded cells 4-5-fold, and this was inhibited by C3.

13 of endothelial markers and the formation of round cells, a morphologic change characteristic of EHT.

14 atum basale, having one layer of columnar to round cells, a relatively flattened stratum spinosum and

15 y; pagetoid infiltration of dendritic and/or round cells; a nonspecific architectural pattern at the

16 ve use of systemic therapy for patients with round cell and pleomorphic LPS, which carry a high risk

17 Deleting these genes results in round cells and diminishes bud elongation in mutants tha

18 ar behavior, shifting back and forth between round cells and multibodied proplatelets.

19 hat VASP was localized at the cell cortex in round cells and redistributed to focal adhesions at the

20 ell division, resulting in plants with small rounded cells and fewer cells.

21 -differentiated/dedifferentiated, (b) myxoid/round cell, and (c) pleomorphic, based on morphologic fe

22 dedifferentiated, 144 (18%) myxoid, 81 (10%) round cell, and 64 (8%) pleomorphic histology.

23 ls, stellate cells, giant cells, radiate (or round) cells, and a variety of small cells.

24 0023, caused bulged cell poles, formation of rounded cells, and defects in polar localization of Wag3

25 chondroid metaplasia, lowering the number of rounded cells, and enhancing collagen fiber organization

26 Consequently, the rounded cells are suddenly presented with a several-fold

27 ic cells (especially roundish); and atypical round cells at the DEJ.

28 del, cell types strongly sort by shape, with round cells at the top of the colony and rod cells domin

29 read underwent osteogenesis, while unspread, round cells became adipocytes.

30 ng basal cells, with almost all cells having round cell bodies similar to rodent globose basal cells.

31 They have rounded cell bodies, have prominent nucleoli, attach poo

32 med long thin processes that extended from a round cell body.

33 y changed morphology characterized by a more rounded cell body and fewer dendrites than wild-type cel

34 four primary dendrites, a thick axon, and a rounded cell body and likely are made up of multiple typ

35 d mixed colonies consisting of diffuse small round cells characteristic of immature NK cells and othe

36 morphologic classification: dendritic cell, round cell, dermal nest, combined, and nonclassifiable t

37 esylation, prevented lovastatin-induced cell rounding, cell detachment and apoptosis, and stimulated

38 of FAK and AKT, sarcomeric disassembly, cell rounding, cell detachment from underlying matrix, and mo

39 In a multivariate analysis, only round-cell differentiation and low expression of p27 ind

40 posarcomas, even in neoplasms with few or no round-cell differentiation.

41 ells undergo the normal processes of mitotic rounding, cell elongation, polar ruffling, furrow ingres

42 Large, round cells expressing bone morphogenetic protein recept

43 stologically, these lesions consist of small round cells expressing variable levels of CD99 and speci

44 en and thicken uniformly, with the resultant rounded cell finally bursting.

45 that combines parallel plate confinement of round cells, fluorescence recovery after photobleaching,

46 s, YW_SL and LL_RQ, severely impaired single-round cell-free infectivity 10-fold or more relative to

47 re smaller size (HR = 0.7, P = 0.01), myxoid/round cell histologic subtype (HR = 0.3, P = 0.03) and t

48 mutant failed to completely demonstrate the round-cell histology that is seen in both Ewing's tumor

49 sed by coelomocytes in the nephridium and in round cells in the epidermis and in eggs.

50 d spermatids, and the appearance of numerous round cells in the epididymal lumen.

51 longated cells, it increased the motility of rounded cells in the tumor.

52 CD, SCs do not differentiate and appear as "rounded" cells in contact with axons.

53 se-activating protein-deficient form of ExoS rounded cells, indicating the ADP-ribosyltransferase dom

54 trol of division planes by the Min system in round cells is proposed.

55 Round cells lacking MinCDE were viable, but growth, morp

56 c or unresectable synovial sarcoma or myxoid round cell liposarcoma (confirmed by cytogenetics) expre

57 Myxoid round cell liposarcoma (MRCLS) is a common liposarcoma s

58 1 (NY-ESO-1) is a promising target in myxoid/round cell liposarcoma (MRCLS).

59 confirmed synovial sarcoma (n=44) and myxoid round cell liposarcoma (n=8) were enrolled and received

60 ces apoptosis in dedifferentiated and myxoid/round cell liposarcoma cell lines, but not in either adi

61 myogenic and with neural differentiation and round cell liposarcoma, respectively.

62 two of eight; 3-65) for patients with myxoid round cell liposarcoma.

63 pressing advanced synovial sarcoma or myxoid round cell liposarcoma.

64 -differentiated liposarcomas, >90% of myxoid round cell liposarcomas, and >70% of pleomorphic liposar

65 gnostic factor in the spectrum of myxoid and round-cell liposarcomas has not been examined.

66 Forty-seven cases of myxoid and round-cell liposarcomas were examined.

67 predicts the clinical behavior of myxoid and round-cell liposarcomas, even in neoplasms with few or n

68 phic liposarcomas) and PIK3CA (18% of myxoid/round-cell liposarcomas, or MRCs).

69 DR was most frequent among pleomorphic and round cell LPS; the former recurred early (43% by 3 year

70 4d were ablated the embryo developed into a rounded cell mass covered with an epithelial cell sheet.

71 being associated with chronic sun damage and round cell melanoma with early age at onset and phototyp

72 Round cell melanomas were identified more often in the f

73 vels of YAP activation with smaller and more rounded cell morphologies were induced in hMSCs.

74 We introduce three complementary models of rounded cell morphologies with a prescribed excess surfa

75 ntiated, 9 (5%) with myxoid, and 4 (2%) with round cell morphology.

76 s with H218 or edg-3, but not edg-1, induces rounded cell morphology in the presence of serum, which

77 moeboid cells (poor collagenolytic activity, rounded cell morphology), were 5-fold more invasive in t

78 fficient to predict the location of blebs in rounded cells moving in a highly resistive environment.

79 Synovial sarcomas, round-cell/myxoid liposarcomas, clear-cell sarcomas and

80 anced or metastatic dedifferentiated, myxoid/round cell, or pleomorphic LPS incurable by surgery or r

81 grade (p = 0.03) and 61.5% had more than 5% round cells (p = 0.01).

82 gh grade (p=0.03) and 61.5% had more than 5% round cells (p=0.01).

83 The mutant virus induced a round-cell phenotype that accumulated in the G(2)/M comp

84 hibitor of viral DNA synthesis increased the round-cell phenotype.

85 , accompanied by a morphological change to a rounded cell phenotype and down-regulation of vimentin,

86 o maintain a straight sidewall and uniformly rounded cell poles.

87 Accumulation of rounded cells positive for MC markers close to the vesse

88 sarcomas, all high grade with pleomorphic or round cells rather than spindle cells, contained an unid

89 Regardless of their fate in vivo, all the round cells remained negative for smooth muscle markers,

90 e describe a mechanism through which coccal (round) cells rise to the upper surface of a community, l

91 vision planes, we investigated its role in a round-cell rodA mutant.

92 but highly aggressive undifferentiated small round cell sarcoma driven by a fusion between the tumor

93 ficiently induced Ewing's sarcoma-like small round cell sarcoma formation by these cells.

94 ighly aggressive and lethal subtype of small round cell sarcoma found predominantly in adolescents an

95 Ewing sarcoma is a primitive round cell sarcoma with a peak incidence in adolescence

96 Undifferentiated small round cell sarcomas (SRCSs) of bone and soft tissue comp

97 in a substantial portion of EWSR1-rearranged round cell sarcomas as an early and persistent clonal ev

98 acterized the spectrum of SVs in EWSR1-fused round cell sarcomas, including 173 ES and 104 desmoplast

99 n rhabdomyosarcoma, Ewing sarcoma, and other round cell sarcomas.

100 newly diagnosed and refractory or recurrent round cell sarcomas.

101 improved clinical outcomes in patients with round cell sarcomas.

102 s-RbAp48-hi) leads to cell size reduction, a rounded cell shape, decreased cellular protrusions, and

103 focused around the nucleus and resulted in a rounded cell shape.

104 This release is accompanied by cell rounding, cell swelling, and reorganization of the actin

105 ltered cellular morphology, producing small, round cells that closely resemble Cdc42-/- cells.

106 lso contain a third kind of cell: very small round cells that rapidly self-renew.

107 midpoint, prior to the formation of enlarged round cells that showed evidence of compromised cellular

108 Vincristine converted discoid platelets to rounded cells that extended pseudopods when chilled and

109 rs form irregular double or triple layers of rounded cells that lack zonula adherens.

110 In vitro VIP-treated rNSCs changed from round cells to glia-like cells with processes that stain

111 r cell sarcoma (CCS), and desmoplastic small round cell tumor (DSRCT) are soft-tissue malignancies oc

112 Desmoplastic small round cell tumor (DSRCT) is a rare disease of children,

113 Desmoplastic small round cell tumor (DSRCT) is a rare sarcoma in adolescent

114 Desmoplastic small round cell tumor (DSRCT) is a rare, radiosensitive, yet

115 Desmoplastic small round cell tumor (DSRCT) is an abdominal malignancy in c

116 Desmoplastic small round cell tumor (DSRCT) is an aggressive, usually incur

117 Desmoplastic small round cell tumor (DSRCT) is defined by a chimeric transc

118 molecular pathogenesis of desmoplastic small round cell tumor (DSRCT), a fatal malignancy occurring p

119 Desmoplastic small round cell tumor (DSRCT), a rare sarcoma of adolescents/

120 an malignancies including Desmoplastic Small Round Cell Tumor (DSRCT), an aggressive cancer driven by

121 s translocation underlies desmoplastic small round cell tumor (DSRCT), which is noted for the abundan

122 One patient (desmoplastic round cell tumor [DSRCT]) achieved a confirmed partial r

123 ch with hepatoblastoma or desmoplastic small round cell tumor achieved a partial response; eight pati

124 leven of 14 patients with desmoplastic small round cell tumor had primary abdominal involvement.

125 e patients diagnosed with desmoplastic small round cell tumor with peritoneal involvement were enroll

126 ectodermal tumor, and one desmoplastic small round cell tumor).

127 eristic of intraabdominal desmoplastic small round cell tumor.

128 1 found in the aggressive desmoplastic small round cell tumor.

129 toneally in patients with desmoplastic small round cell tumor.

130 osarcoma protuberans, and desmoplastic small round cell tumor; one sarcoma predisposition syndrome wi

131 descriptively designated desmoplastic small round-cell tumor (DSRCT) are a distinctive, rare, poorly

132 Desmoplastic small round cell tumors (DSRCT) are a type of aggressive, pedi

133 including 173 ES and 104 desmoplastic small round cell tumors (DSRCT), to detect chromoplexy and eva

134 ilable in vitro model for Desmoplastic Small Round Cell Tumors (DSRCT), while having only minor effec

135 ng family tumors (EFT) or desmoplastic small round cell tumors (DSRCT).

136 f the hydra analysis framework to small blue round cell tumors (including rhabdomyosarcoma, synovial

137 SR1::WT1 is essential for desmoplastic small round cell tumors and upregulates the cyclin D-CDK4/6-RB

138 Notably, hydra analysis of all small blue round cell tumors revealed similar subtypes, characteriz

139 the diagnosis and therapy of malignant small round cell tumors that affect children, particularly in

140 9 osteosarcomas, 35 of 37 desmoplastic small round cell tumors, 2 of 3 synovial sarcomas, 4 of 4 leio

141 alveolar rhabdomyosarcomas, two desmoplastic round cell tumors, and one each of infantile fibrosarcom

142 ed with Ewing sarcoma or desmoplastic, small round cell tumors, which are other EWSR1-rearranged tumo

143 euroectodermal tumors and desmoplastic small round cell tumors.

144 omosomal translocation in desmoplastic small round-cell tumors (DSRCT), a rare but aggressive soft ti

145 ated tumors occurring in young people (small round-cell tumors).

146 s sarcoma family of tumours (ESFT) are small round cell tumours characterized by the non-random EWS-E

147 utants lacking CBK1 form large aggregates of round cells under all growth conditions and lack the abi

148 teration in Epa polysaccharide content, more round cells versus oval cells with OG1RF, decreased biof

149 The round cells were characteristic of an abortive viral inf

150 Spindle and round cells were loosely distributed with increased part

151 Sparse round cells were observed 21 days after plating onto dee

152 It was observed that when rounded cells were injected with either the integrin pep

153 e/Dead viability stain revealed that >90% of rounded cells were viable.

154 ation showed a clear change of morphology to round cells, which accompanies detachment from the surfa

155 ls that exit the neuroepithelium as isolated round cells, which then halt for a short period prior to

156 r, cells depleted of Rhb1 arrested as small, round cells with 1N DNA content, arrested more quickly i

157 In round cells with a long axis, such as those undergoing c

158 ) of neuroectodermal origin formed by small, round cells with limited neural differentiation, which a

159 toskeleton, since rho3 mutants exhibit large rounded cells with an aberrant actin cytoskeleton.

160 crease in the number of multinucleate cells, rounded cells with condensed ball-like nuclei, and cells

161 Wild type HA-p190 induced a phenotype of rounded cells with long, beaded extensions similar to th