ライフサイエンスコーパス: saccade

1 ddle temporal area during the execution of a saccade.

2 RPE (+RPE) event that preceded the secondary saccade.

3 evaluation of stimulus values prior to first saccade.

4 nformation continuously available across the saccade.

5 ntent of that image, encouraging a secondary saccade.

6 the same, changed, or disappeared during the saccade.

7 xecution and lasting for up to 1 s after the saccade.

8 tion of a visual stimulus before and after a saccade.

9 ccompanied by interjection of a disconjugate saccade.

10 keys rapidly reported their decisions with a saccade.

11 al information is also available before each saccade.

12 stractor preceded the execution of the first saccade.

13 ve trans-saccadic retinal stimulation during saccades.

14 prevent them from performing large rightward saccades.

15 vironment to aid eye alignment at the end of saccades.

16 corrective vergence movements at the end of saccades.

17 during the fixation pauses in between large saccades.

18 pletion of vertical, horizontal, and oblique saccades.

19 hibitory errors in comparison to the correct saccades.

20 cluding the correlation between crowding and saccades.

21 uired the accumulation of information across saccades.

22 abilizing mechanism operates following small saccades.

23 tor center responsible for the generation of saccades.

24 idence for early information transfer across saccades.

25 ng their eye-centered representations across saccades.

26 Vision is drastically reduced at the time of saccades.

27 r colliculus (SC), and induces contralateral saccades.

28 entered projections of visual objects across saccades.

29 gence velocities observed during disjunctive saccades.

30 changes in object orientation that followed saccades.

31 ucidate how such value information modulates saccades.

32 enable rich and continuous perception across saccades.

33 We compared saccades (1.3 degrees -3.7 degrees ) initiated during in

34 ients with glaucoma had significantly slower saccades (602.9 +/- 50.0 ms versus 578.3 +/- 44.6 ms for

35 44.6 ms for controls, P = 0.009) and reduced saccade accuracy (direction bias = 7.4 +/- 1.8 versus 6.

36 This saccade adaptation typically follows an exponential time

37 colliculus (SC) sends error signals to drive saccade adaptation.

38 ror that drives adaptation, decreases during saccade adaptation.

39 re we examined the accuracy and precision of saccades aimed toward targets ranging from [Formula: see

40 y of neurons with visually evoked responses, saccade-aligned responses, and mixtures of both.

41 elopmental disorder characterized by reduced saccade amplitude and gaze stabilization deficits.

42 e fatigue during gaze stabilization, reduced saccade amplitude and velocity in the light, greater dis

43 ment patterns in which fixation duration and saccade amplitude are altered in response to the visual

44 We manipulated action costs by varying the saccade amplitude, and we dissociated in time and space

45 movement parameters were computed, including saccade amplitude, the spread of saccade endpoints (biva

46 This saccade and fixate pattern is similar to humans who use

47 d coupled to head yaw rotation to produce a "saccade and fixate" gaze pattern.

48 se in velocity and amplitude of both the eye saccade and head movement toward the target.

49 tudied in humans for its functional roles in saccade and visual processing, but less is known about i

50 ically supramarginal gyrus [SMG]) integrates saccade and visual signals to update grasp plans in addi

51 on of fixation in the presence of fixational saccades and (2) the biases and limitations of transsacc

52 from avoiding gaze-shifts in this procedure, saccades and blinks are inhibited prior to predictable r

53 The incidence and timing of saccades and blinks occurring from 450 ms before stimulu

54 e female cricketers had steadier gaze (fewer saccades and blinks) compared to female controls; (4) wh

55 on (HBO) that acts as a pacemaker for ocular saccades and controls the orientation of successive swim

56 ts with CTX executed more frequent multistep saccades and directional errors during the antisaccade t

57 both cell types respond similarly to visual saccades and display essentially identical speed tuning.

58 person perspective via self-directed motion (saccades and head turns).

59 ironments, which they naturally explored via saccades and head turns.

60 motor-mediated optimization of input across saccades and pupil dilation, the primate auditory system

61 There was a significant slowing of saccades and saccades became less accurate with worsenin

62 How rapid body saccades and smooth movement interact for simultaneous o

63 mis (OMV)] in the control of visually guided saccades and smooth-pursuit eye movements.

64 ations directed at the implant with repeated saccades and that the implant-induced responses were pre

65 large changes in many components of pursuit, saccades and visual motion processing as a function of t

66 y with the vergence component of disjunctive saccades and, based on modeling studies, are critically

67 al sensations as consequent on action (i.e., saccades) and implies that visual percepts must be activ

68 in sensitivity of neurons around the time of saccades, and provide a general framework to quantitativ

69 al suppression occurring prior to and during saccades, and the reduction in neural responses to visua

70 eptitiously while a saccade is underway, the saccade appears to be in error.

71 han controls; (2) blinks and, in particular, saccades are associated with slower VRT regardless of th

72 s of eye positions acquired by goal-directed saccades are determined by the object's location.

73 he visual suppression.SIGNIFICANCE STATEMENT Saccades are known to produce a suppression of contrast

74 cephalography (MEG) recordings, we show that saccades are locked to the phase of visual alpha oscilla

75 Furthermore, although saccades are more frequent, their dynamics are more slug

76 Whether such learned compensatory saccades are optimal and generalize to more complex task

77 Saccades are rapid eye movements that orient the visual

78 dynamic environments.SIGNIFICANCE STATEMENT Saccades are the rapid, ballistic eye movements that we

79 Subjects were free to saccade around the screen and make a choice (via joystic

80 We estimated information in PFC about saccades as a function of ensemble size.

81 ht of sensory information around the time of saccades, as a result of signal dependent noise and of s

82 (Object-Match, Category-Match, and Category-Saccade associations) revealed signatures of explicit an

83 We evoked short-latency fixed vector saccades at low currents (<50 muA) in areas 45, 8aV, 8C,

84 Human subjects made horizontal saccades at will to two stationary saccadic targets sepa

85 re was a significant slowing of saccades and saccades became less accurate with worsening SAP sensiti

86 ification through dilated pupils with ocular saccades before an injection.

87 ed a delay-free, visual search task in which saccade behavior was unrestricted.

88 human performance in detecting fixations and saccades but fall short (50%) on detecting pursuit movem

89 d ventral higher-level sites the response to saccades (but not to external displacements) was suppres

90 e process was used, not only to generate the saccade, but also to provide input to the across-saccade

91 iority map of the extrafoveal space to guide saccades, but also a finer-grained priority map that is

92 w-level visual information processing across saccades by decoding the spatial frequency of a stationa

93 strictedly also toward locations to which no saccade can be executed.

94 functional connectivity with frontal cortex saccade centers.

95 mall saccades in ventrolateral FEF and large saccades combined with contralateral neck and shoulder m

96 dscaml1 mutant animals confirmed deficits in saccade-command signals (indicative of an impairment in

97 er neural signals related to updating across saccades contain information about stimulus features, or

98 Here, we present a novel saccade-contingent behavioral paradigm and investigate t

99 Saccade-contingent habituation might explain why we do n

100 d disturbance could be established through a saccade-contingent habituation to intra-saccadic displac

101 upper layers to support proactive inhibitory saccade control.SIGNIFICANCE STATEMENT Failures to inhib

102 monkeys (one male, one female) performing a saccade countermanding task.

103 nular cortical area, in monkeys performing a saccade-countermanding (stop signal) task.

104 p of the new fixation.SIGNIFICANCE STATEMENT Saccades create frequent discontinuities in visual input

105 lly, modeling data in two monkeys performing saccades demonstrated the generalization of PSID across

106 eccentricity, the probability of eliciting a saccade depends on its efficacy in reducing the foveal o

107 e same temporal interval without preparing a saccade did not affect performance.

108 pond to stimuli presented around the time of saccades differently than during fixation.

109 We observed a topography of saccade direction and amplitude consistent with findings

110 e additionally found that 3D orientation and saccade direction preferences aligned, particularly for

111 Moreover, cholinergic stimulation attenuated saccade direction selectivity in putative pyramidal neur

112 ade latency, and two measures of accuracy of saccades (direction bias and amplitude bias).

113 ion of cMRF neurons that, during disjunctive saccades, display a burst of spikes that are highly corr

114 In contrast, saccades do change for tasks such as object following an

115 Notably, these saccades do not preferentially target the prey location.

116 ffects of central vision loss on the optimal saccades during a face identification task, using a gaze

117 Catch-up saccades during steady visual tracking of the moving tar

118 Saccade dynamics start to become 'sluggish' at as low as

119 This redistribution is a consequence of saccade dynamics, particularly the speed/amplitude/durat

120 stimulation of the oculomotor vermis caused saccade dysmetria.

121 ertical gaze, slowed horizontal and vertical saccades, dysphagia, apathy, and progressive cognitive d

122 of alternating blocks of trials requiring a saccade either toward a large, high-luminance stimulus o

123 esponse to a fixated object began before the saccade ended, suggesting that this information is remap

124 eyes, we observed no benefit at their actual saccade endpoint.

125 , including saccade amplitude, the spread of saccade endpoints (bivariate contour ellipse area), loca

126 piking activity preceded LFP activity in the saccade epoch.

127 esponses were remapped, appearing before the saccade even ended, and were not suppressed during maint

128 y 3 Hz), commencing approximately 1 s before saccade execution and lasting for up to 1 s after the sa

129 We show that the saccade/fixation cycle reformats the flow impinging on t

130 ature of the preceding RPE event: high vigor saccades followed +RPE events, whereas low vigor saccade

131 ades followed +RPE events, whereas low vigor saccades followed -RPE events.

132 f the preceding RPE event: the most vigorous saccades followed the largest +RPE, whereas the least vi

133 the largest +RPE, whereas the least vigorous saccades followed the largest -RPE.

134 A moving bar elicited sustained bouts of saccades following the bar, with surprisingly little smo

135 nment and uses it to compute the appropriate saccade for either eye.

136 additional time needed to program secondary saccades for correcting hypermetric errors, relative to

137 cted male rhesus macaque monkeys to initiate saccade-free smooth pursuit eye movements and injected a

138 Catch-up saccade frequencies and amplitudes were also similarly a

139 rgets and the motor metrics of memory-guided saccades from the spatial locations stored in WM, thus c

140 on of gaze events (e.g. fixations, pursuits, saccade, gaze shifts) while the head is free, and thus c

141 white-matter integrity of tracts between key saccade-generating regions, and that inhibition efficien

142 d the core cortical eye-movement network for saccade generation (frontal eye fields, posterior pariet

143 e time-specific contributions of pulvinar to saccade generation and decision making.

144 results bring into question extant models of saccade generation and support the possibility of a conc

145 l information-processing loops in optimizing saccade generation in dynamic environments.SIGNIFICANCE

146 ensitive to visual signals and that catch-up saccade generation is reset after a visual transient.SIG

147 rior colliculus, a major midbrain center for saccade generation, was examined to determine whether th

148 the time of, or after, the production of the saccades guided by tactile stimulation.

149 secondary saccade indicated that the primary saccade had experienced a movement error, inducing trial

150 Suppression of prepotent saccades has been shown to require proactive inhibition

151 It has been observed that saccades have a systematic tendency to fall short of the

152 world environments are actively explored via saccades, head turns, and body movements.

153 ropose a novel algorithm for tuning fixation saccades in flies.

154 that abnormalities in fixation, pursuit, and saccades in mTBI are the cause of post-concussive sympto

155 le of the cMRF in the control of disjunctive saccades in trained rhesus monkeys.

156 with findings in macaques and humans: small saccades in ventrolateral FEF and large saccades combine

157 Presence of the secondary saccade indicated that the primary saccade had experienc

158 ens of milliseconds in advance of the actual saccade, indicating the presence of a latent movement co

159 derpinning such behavior, saccade selection, saccade inhibition, and saccadic choice, in female and m

160 ect can be dissociated from motor effects on saccade initiation and execution.SIGNIFICANCE STATEMENT

161 ior colliculus (iSC) support such models for saccade initiation by relating variations in saccade rea

162 arameter in biologically plausible models of saccade initiation.

163 ameter for stochastic accumulation models of saccade initiation.SIGNIFICANCE STATEMENT The superior c

164 target is displaced surreptitiously while a saccade is underway, the saccade appears to be in error.

165 the following: (1) the cognitive control of saccades is achieved within key cortical saccadic brain

166 f the precision and accuracy of the smallest saccades is still lacking.

167 The neural control of disjunctive saccades is still poorly understood.

168 bivariate contour ellipse area), location of saccade landing positions, and similarity of fixations l

169 iate postsaccadic processing at the fovea on saccade landing.

170 However, catch-up saccades largely compensate for the tracking displacemen

171 eased odds of glaucoma; however, the AUC for saccade latency was only 0.635 compared to 0.914 for SVO

172 SVOP provided data on threshold sensitivity, saccade latency, and two measures of accuracy of saccade

173 ead displacements characterized by stepwise, saccade-like kinematics.

174 gration was determined by the metrics of the saccades made during training.

175 the target is foveated, microsaccades, tiny saccades maintaining the fixated object within the fovea

176 t the apparent richness of perception across saccades may be supported by the continuous availability

177 tion integration: they showed task-dependent saccade modulations and, during grasp execution, they we

178 sensitivity following two distinct regimes: saccade modulations counterbalance (whiten) the spectral

179 ss the cortical layers in the frontoparietal saccade network remains unknown because many of the area

180 e proactive inhibition in the frontoparietal saccade network.

181 We show that even small saccades of just 14-[Formula: see text] are very effecti

182 saccadic information ramped up rapidly after saccade offset.

183 decoded at a later time-point, 151 ms after saccade offset.

184 re significant at a time window right before saccade onset.

185 may be important for detecting the onset of saccades or for signaling optical flow.

186 no increase in activity for either conjugate saccades or symmetric vergence.

187 we accounted for the presence of blinks and saccades, our group comparisons of VRT were virtually un

188 tial attention alone, even in the absence of saccade planning or a spatial cue, is sufficient to expl

189 We report that both saccade precision and crowded-target reports vary idiosy

190 s' oculomotor range during both fixation and saccade preparation.

191 Therefore, the output of the within-saccade process was used, not only to generate the sacca

192 This across-saccade process, in turn, helped to set the starting poi

193 to set the starting point of the next within-saccade process.

194 ade, but also to provide input to the across-saccade process.

195 Conversely, although most models of saccade programming are tightly coupled to underlying ne

196 d competition between motor point images for saccade programming, all of which cause further modulati

197 Stimulation affected saccade properties and target selection in a time-depend

198 saccade initiation by relating variations in saccade reaction time (SRT) to variations in such parame

199 Here, we show that saccades redistribute spatial information within the tem

200 functional connectivity with both prefrontal saccade regions (consistent with oculomotor input) and a

201 onclude that FEF signals govern the onset of saccade-related accumulation within the iSC, and that th

202 stead, FEF inactivation delayed the onset of saccade-related accumulation, emphasizing the importance

203 We next determined the exact location of the saccade-related CD neurons using the grid of penetration

204 y relevant visual targets and, subsequently, saccade-related neurons select the movements required to

205 encounters, while the magnitude and rate of saccades remained constant.

206 nvolved in the generation of the disjunctive saccades required to view objects in our 3D world.

207 Humans and many animals make frequent saccades requiring coordinated movements of the eyes.

208 ed either to generate or inhibit a prepotent saccade response.

209 ognitive functions underlying such behavior, saccade selection and inhibition, is a challenge.

210 nitive functions underpinning such behavior, saccade selection, saccade inhibition, and saccadic choi

211 esaccadic stimulus is transferred across the saccade so that it becomes quickly available and influen

212 unctional magnetic resonance paradigm, where saccades sometimes interrupted grasp preparation toward

213 tal and superior parietal) regions that show saccade-specific modulations during unexpected changes i

214 chanism for the long-held view that directed saccades support hypothesis-driven, constructive percept

215 Here we show that simulated visual saccades suppress ON-DSGCs, but not ONOFF-DSGCs.

216 oving animals, we show that simulated visual saccades suppress responses in ON-DSGCs but not ONOFF-DS

217 y be the underlying common neural basis of a saccade suppression deficit.

218 found that monkeys could generate predictive saccades synchronized to periodic visual stimuli when an

219 due to the very short timescale on which the saccade takes place.

220 whether peripheral information at a planned saccade target affects immediate postsaccadic processing

221 c attention, is considered to prioritize the saccade target and to enhance behavioral performance for

222 ual performance and neural responses for the saccade target are enhanced.

223 s prior to a saccade to start processing the saccade target before it lands in the foveola, the high-

224 to infer the spatial location of a rewarded saccade target in the presence of different forms of unc

225 esaccadic attention not only prioritizes the saccade target, but also automatically modifies its feat

226 he attended location rather than towards the saccade target.

227 nd to enhance behavioral performance for the saccade target.

228 tcome did so well before the presentation of saccade targets, indicating that decisions were made in

229 racy of eye-movements during a memory guided saccade task are related to fluctuations in the amplitud

230 and audiovisual distractors in a double-step saccade task to investigate if this stability mechanism

231 In this study, we used a memory-guided saccade task to temporally dissociate the visual epoch f

232 on discrimination task and a visually guided saccade task while we recorded from the caudal intrapari

233 en pain-free participants completed the anti-saccade task with dynamic facial expressions, specifical

234 hese two types of motivation with a rewarded saccade task, in patients with Parkinson's disease (PD).

235 With extensive training on the saccade task, these observers gradually acquired the abi

236 n macaque monkeys engaged in a memory-guided saccade task.

237 g monkeys during performance of an effortful saccade task.

238 FEF while macaques performed a memory-guided saccade task.

239 mulation of the dPul while monkeys performed saccade tasks toward instructed and freely chosen target

240 their behaviour associated with a change of saccade tasks.

241 l of several types of modified memory-guided saccade tasks.

242 ation are interspersed with non-compensatory saccades that abruptly shift gaze position.

243 re often accompanied by rapid eye movements (saccades) that displace the desired object image relativ

244 Likewise, when making a downward saccade, the eyes converged to enable alignment with cro

245 For example, when making an upward saccade, the eyes diverged to be aligned with the most p

246 We show that the neurons encode, before the saccade, the information gain (reduction in decision unc

247 rast, when the target was changed during the saccade, the new target was decoded at a later time-poin

248 During head-initiated saccades, the eyes moved together in the head direction

249 itional classifications from a memory-guided saccade, they were indistinguishable from the rest of th

250 of male macaque monkeys performing a delayed saccade to a memorized spatial location.

251 ffers and could accept the offer by making a saccade to a peripheral target or reject the offer by br

252 It is known that attention shifts prior to a saccade to start processing the saccade target before it

253 Participants either made a saccade to the cue or maintained fixation while they dis

254 nating strabismus, either eye can be used to saccade to visual targets.

255 is interplay operates both within and across saccades to ensure that these eye movements are guided e

256 d brain potentials while human subjects made saccades to face stimuli.

257 otor control in a task in which monkeys made saccades to gather visual information relevant to a subs

258 Humans use saccades to inspect objects of interest with the foveola

259 eurons carrying CD signals discharged before saccades to ipsilateral as well as contralateral visual

260 ld, but leaving intact their ability to make saccades to targets presented alone.

261 or a saccadic eye movement, while inhibiting saccades to task-irrelevant stimuli, is crucial for acti

262 o fixate, yet participants continued to make saccades to the empty, but predictable, waypoint locatio

263 ntion, but existing SC models cannot predict saccades to visually complex real-world stimuli.

264 Just as saccading to a relevant stimulus can be an overt correla

265 Humans use rapid gaze shifts, known as saccades, to explore visual scenes.

266 ts (both male and female) were instructed to saccade toward a face or a house that, on different tria

267 en the animals were instructed to suppress a saccade toward a peripheral stimulus.

268 ion of this prepotent response in favor of a saccade toward a small, low-luminance stimulus.

269 Human subjects (both sexes) made saccades toward an image.

270 ent results demonstrate that CDt facilitates saccades toward good objects by serial inhibitory pathwa

271 e able to make rapid eye movements, known as saccades, toward visual targets almost as gracefully as

272 The variance of grid cell activity along saccade trajectories exhibits 6-fold symmetry across 360

273 We found that saccade trajectories systematically curved away from the

274 Here, we measured saccade trajectory curvature following the presentation

275 mulus remained present for ~200 ms after the saccade, transcending retinotopic specificity.

276 gs suggest important computational roles for saccade transients in the establishment of spatial repre

277 little is known about the spatial content of saccade transients.

278 sifier, whose performance was then tested on saccade trials.

279 el based on integrated motion cues simulates saccade trigger and dynamics.

280 , like remapping, is highly dependent on the saccade vector and the spatial arrangement of current an

281 These neurons are termed saccade-vergence burst neurons (SVBNs) to maintain consi

282 These combined saccade-vergence eye movements result in disjunctive sac

283 pattern for different types of eye movement (saccades, vestibulo-ocular reflex, vergence) and gaze-ho

284 ive or positive RPE events and observed that saccade vigor carried a robust signature of the precedin

285 We found that reaction time of the secondary saccade was affected in an orderly fashion by the magnit

286 (reduction in decision uncertainty) that the saccade was expected to bring for the following action.

287 During execution of the primary saccade, we probabilistically changed the position and c

288 sociated with initiation and cancellation of saccades, we found that beta-bursts occur too infrequent

289 Slower saccades were associated with increased odds of glaucoma

290 directly adjacent Frontal Eye Fields (FEF), saccades were only rarely evoked by the stimulation.

291 Optomotor saccades were tuned to the dynamics of panoramic image m

292 n, the reduced visibility around the time of saccades, which is important in mediating visual stabili

293 rs performed a task in which they executed a saccade while discriminating the motion of a cued visual

294 d task, but stimulation during memory-guided saccades, while influencing RTs and errors, did not affe

295 flies turned stochastically with stereotyped saccades, whose direction was biased upwind by the timin

296 in aligning temporally the initiation of the saccade with the visual suppression.

297 in temporally aligning the initiation of the saccade with the visual suppression.SIGNIFICANCE STATEME

298 vergence eye movements result in disjunctive saccades with a vergence component that is much faster t

299 patients with DN damage showed less precise saccades with longer latencies, and more frequent direct

300 isual information (spatial frequency) across saccades with magnetoencephalography.

301 CTX patients executed normally accurate saccades with normal main sequence relationships, indica