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1 t of saffron and its common substitutes i.e. safflower and calendula (n = 21) was employed for determ
2  to fraudulent admixture with allied plants "safflower and calendula".
3 on and detecting its common substitutes i.e. safflower and calendula.
4  skim milk and 50 g fat from high-oleic acid safflower and canola oils (monounsaturated fatty acid; M
5 chness of flavanones/chalcones glycosides in safflower and cinnamates/terpenes in calendula.
6  from whole almonds, almond oil, or a mix of safflower and corn oils.
7 genetic architecture of drought tolerance in safflower and highlight candidate loci for marker-assist
8 , and plant oils including caraway, parsley, safflower, and jojoba oils.
9 adulterants (i.e., saffron style, calendula, safflower, and rubia).
10 ysis demonstrates that natural pigments from safflower are more stable in sugar-based NADES than in w
11 lop DNA-based methods to detect and quantify safflower as an adulterant in saffron.
12  is the first successful attempt to quantify safflower by a DNA-based approach.
13 f incorporating cold-pressed sesame (C-SEO), safflower (C-SAO), and black cumin oils (C-BCO) into may
14 ng the genetic basis of drought tolerance in safflower (Carthamus tinctorius L.) is essential for dev
15 e of dayflower (Commelina communis) blue and safflower (Carthamus tinctorius) red in purple samples c
16 curbita), Sunflower (Helianthus annuus), and Safflower (Carthamus tinctorius), with a focus on their
17 ch: an extract of Carthamus oxyacantha (wild safflower), containing an array of spiro compounds, and
18 category) from other vegetable oils (canola, safflower, corn, peanut, seeds, grapeseed, palm, linseed
19 bsolute and relative sensitivities of 2pg of safflower DNA ( approximately 1.4 DNA copies) and 0.1% o
20                                     Coconut, safflower, evening primrose, and linseed oils marketed i
21 drought tolerance across 90 globally diverse safflower genotypes.
22  ( approximately 1.4 DNA copies) and 0.1% of safflower in saffron (Crocus sativus L.), respectively.
23 so proposed in the range of 0.1-20% (w/w) of safflower in saffron, which was successfully validated a
24       Additionally, four adulterants (style, safflower, madder and calendula) were reliably detected
25 ,-p-coumaroyl spermidine were characteristic safflower metabolites, whereas, calendulaglycoside C and
26 A; n-9 and n-3 rich), 3) a blend of corn and safflower oil (25:75) (CornSaff; n-6 rich), 4) a blend o
27 H versus TCA cycle flux was decreased in the safflower oil (43 +/- 8%) versus the control (73 +/- 8%,
28 on than after sunflower oil (P: = 0.003) and safflower oil (P: = 0.001) supplementation.
29 ary oils, conjugated linoleic acid (CLA) and safflower oil (SAF), on body weight and composition in o
30 eatment with 4 g/d of ethyl esters of either safflower oil (SAF; control), eicosapentaenoic acid (EPA
31 oils (no supplementation, JO, fish oil [FO], safflower oil [SO], and arachidonic acid [AA]) were fed
32  were consistent with complete absorption of safflower oil and the nonabsorbability of olestra and ca
33 sumption of triacylglycerol-esterified LA in safflower oil did not increase plasma concentrations of
34                        In both genotypes the safflower oil diet blunted insulin-mediated suppression
35 re supplemented with standard diet (STD, ~4% safflower oil diet) and OBD (10% safflower oil) for 2 mo
36 s when the fish oil diet was replaced by the safflower oil diet.
37                                         Pure safflower oil exhibited the highest total TPUFA (76.78%)
38 in-stimulated glucose disposal attributed to safflower oil feeding was a consequence of reduced glyco
39 iglyceride (TG) content was increased in the safflower oil group (7.3 +/- 0.8 micromol/g) compared wi
40 n insulin-stimulated glucose disposal in the safflower oil group was associated with a lower rate of
41            After ingestion of a 50-g load of safflower oil in capsules (to preclude oral exposure to
42 modified by incorporating varying amounts of safflower oil into the diet.
43                                         SAM (safflower oil mayonnaise) displayed the greatest color c
44 trol diet or isocaloric diets containing 27% safflower oil or 27, 13.5, and 8% menhaden fish oil.
45  were fed isocaloric diets containing either safflower oil or fish oil for 2 weeks before the start o
46     When rats were fed a diet containing 10% safflower oil or menhaden fish oil, the level of hepatic
47 igh-fat diets containing 27% fat from either safflower oil or safflower oil with an 8% fish oil repla
48 ially available tocotrienol supplements or a safflower oil placebo for 28 d.
49  PPAR-alpha null mice fed the fish oil diet, safflower oil plus fish oil, hepatic insulin resistance
50 tion period a salad dressing containing 21 g safflower oil providing 16 g LA/d was added to the subje
51 s demonstrated that high dietary corn oil or safflower oil rich in omega-6 fatty acids increased the
52 s were significantly lower after addition of safflower oil to the diet.
53  work investigates the extraction process of safflower oil using pressurized ethanol, and compares th
54 ; control birds were exposed via diet to the safflower oil vehicle only (24 pairs).
55 .8 and 31.7 +/- 1.9 mg x kg(-1) x min(-1) in safflower oil versus control and fish oil groups, respec
56                    To examine the effects of safflower oil versus fish oil feeding on in vivo intramu
57 taining 27% fat from either safflower oil or safflower oil with an 8% fish oil replacement (fish oil
58 ed by measuring absorption of well-absorbed (safflower oil) and poorly absorbed (olestra; calcium soa
59 (milk-derived) fat, but not polyunsaturated (safflower oil) fat, changes the conditions for microbial
60 .9 g/d n-3 PUFAs) or control oil (high-oleic safflower oil) for 12 wk in a randomized, double-blind,
61 t (STD, ~4% safflower oil diet) and OBD (10% safflower oil) for 2 mo and then subjected to coronary a
62 tion [HGP], P < 0.002 vs. wild-type mice fed safflower oil), whereas in contrast, in PPAR-alpha null
63  were pair-fed on diets consisting of 1) 59% safflower oil, 2) 59% menhaden fish oil, or 3) 59% carbo
64 n was used to study the effects of palm oil, safflower oil, a mixture of fish and safflower oil, and
65 lm oil, safflower oil, a mixture of fish and safflower oil, and olive oil on postprandial apolipoprot
66  a different fat: menhaden oil, herring oil, safflower oil, canola oil, coconut oil, or cocoa butter.
67 19, and 125 +/- 14 nmol x g(-1) x min(-1) in safflower oil, fish oil, and control, respectively) was
68 ic acid from herring oil, linoleic acid from safflower oil, linolenic acid from canola oil, lauric ac
69 ocaloric diets modified to include corn oil, safflower oil, or DFO (doses ranging from 0.75% to 6.00%
70 d a 5% lipid diet containing lard, lard plus safflower oil, or lard plus linseed oil for 10 weeks.
71  Animals were either untreated, treated with safflower oil, or treated with DMH-11C in safflower oil.
72 loid precursor protein transgenic mice fed a safflower oil-enriched ("Bad") diet used to accelerate p
73  resistance (HGP, P = NS vs. PPAR-alpha null safflower oil-fed mice).
74  mice fed the fish oil diet as compared with safflower oil-fed mice.
75                                          The safflower oil-fed rats were insulin resistant compared w
76 ns-11 and 38.5% trans-10,cis-12) or 4 g/d of safflower oil.
77 th safflower oil, or treated with DMH-11C in safflower oil.
78 thyl ester of either fish oil, olive oil, or safflower oil.
79        Maximum increases occurred 10 h after safflower oil; 14 h after cocoa utter, coconut oil, cano
80 ion than after sunflower oil (P: = 0.01) and safflower-oil (P: = 0.0003) supplementation.
81 TBARS was higher than with sunflower-oil and safflower-oil supplementation.
82 (CornSaff; n-6 rich), 4) a blend of flax and safflower oils (60:40) (FlaxSaff; n-6 and short-chain n-
83 s and detecting small quantities of corn and safflower oils in olive oil.
84 lower (SUN-HO), high-oleic and high-linoleic safflower (SAF-HO, SAF-HL) grapeseed (GRA) and borage (B
85 ant-derived adulterants of saffron including safflower, saffron style, calendula, rubia and turmeric
86      From 19 samples, three were positive to safflower, though at levels below the limit of detection
87 ts in saffron, i.e., Crocus sativus stamens, safflower, turmeric, and gardenia were investigated.
88                                              Safflower was selected as a case study because its aroma
89                                             (safflower) were successfully proposed.
90 fied as a marker to distinguish saffron from safflower, whereas calendula aroma was predominated by m