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1 s syringae and in response to treatment with salicylic acid.
2 y lithium borates fusion in combination with salicylic acid.
3 s sucrose, major amino acids, shikimate, and salicylic acid.
4 ber of soybean WRKY genes were responsive to salicylic acid.
5 ta, depending on levels of the plant hormone salicylic acid.
6 acid (ABA), ethylene, jasmonic acid (JA) and salicylic acid.
7 nology to manufacture anti-acne devices with salicylic acid.
8 efense signals, hydrogen peroxide (H2O2) and salicylic acid.
9 w research questions about the plant hormone salicylic acid.
10 cluding those regulated by jasmonic acid and salicylic acid.
11 ES1 or the local or systemic accumulation of salicylic acid.
12 h reinstated signaling by the immune hormone salicylic acid.
13 icantly improve electrochemical responses of salicylic acid.
14 hese were correlated with elevated levels of salicylic acid.
15 NFS1 expression was induced by pathogens and salicylic acid.
16 naling component of the defense phytohormone salicylic acid.
17 ut restricts the accumulation of both ET and salicylic acid.
18 nfluences pathogen defense responses through salicylic acid.
19 smate-glutamate conjugate that converts into salicylic acid.
20 ic reaction was induced by additional acetyl-salicylic acid.
21 riggered immunity (ETI) but independent from salicylic acid.
22 g loading in the FPLA-salicylic acid and PCL-salicylic acid 3D printed patches was 0.4% w/w and 1.2%
23 etics in the colonic epithelium with 5-amino salicylic acid, a PPAR-gamma (peroxisome proliferator-ac
24 owed that jasmonic acid (JA), jasmonoyl-Ile, salicylic acid, abscisic acid, and indole-3-acetic acid
25 ulescens on pathogen-induced ROS production, salicylic acid accumulation and downstream defence respo
26 l biosynthetic pathway positively influences salicylic acid accumulation and guarantees effective bas
27                                              Salicylic acid accumulation appeared as a generalized pl
28 al flagellin (flg22), and pathogen-inducible salicylic acid accumulation from PEN3 activity in extrac
29                                              Salicylic acid accumulation was triggered by high-growth
30 ns, high defence marker gene expression, and salicylic acid accumulation.
31 lutathione oxidation influences redox-driven salicylic acid accumulation.
32 ted topical emollients, corticosteroids, and salicylic acid along with oral retinoids, methotrexate,
33                  Hydrogen peroxide (H2 O2 ), salicylic acid and camalexin (a phytoalexin) levels were
34 ant showed elevated basal as well as induced salicylic acid and ethylene accumulation.
35 on in response to the pathogen suggests that salicylic acid and ethylene signaling pathways mediate r
36 e released from insect eggs, and they induce salicylic acid and H(2)O(2) accumulation, defense gene e
37 it shows variations in water use efficiency, salicylic acid and hydrogen peroxide concentrations, pho
38 precursor aminocyclopropanecarboxylic acid), salicylic acid and jasmonic acid (applied as methyl jasm
39  and herbivory-responsive pathways including salicylic acid and jasmonic acid also were up-regulated
40                                              Salicylic acid and jasmonic acid signaling pathway compo
41 ave elevated levels of two defense hormones: salicylic acid and jasmonic acid, and show increased res
42 s the action of the classic defense hormones salicylic acid and jasmonic acid.
43 NaCl, 1-aminocyclopropane-L-carboxylic acid, salicylic acid and methyl jasmonate) on the phytochemica
44                     Drug loading in the FPLA-salicylic acid and PCL-salicylic acid 3D printed patches
45 f Bs3 coincides with increased levels of the salicylic acid and pipecolic acid, two compounds that ar
46  pathogens would induce flavones to decrease salicylic acid and, hence, increase susceptibility.
47 n, coumarin, sulfadymethoxine, warfarin, and salicylic acid) and HSA molecules.
48 tohormone signaling pathways, jasmonic acid, salicylic acid, and ethylene (ET), in TuMV-infected Arab
49 t hormones indoleacetic acid, jasmonic acid, salicylic acid, and gibberellic acid or by wounding, tem
50  amounts of active gibberellins, cytokinins, salicylic acid, and jasmonate compared with diploid indi
51 lasers and other agents including bleomycin, salicylic acid, and light-emitting diode have shown some
52 -inducing chemical tunicamycin, phytohormone salicylic acid, and pathogen infection.
53 ah1-2 background mutation, which could cause salicylic acid- and EDS5-independent mutant phenotypes.
54 defense response via cell wall modification, salicylic acid- and jasmonic acid-dependent pathways, re
55 itogen-activated protein kinases, as well as salicylic acid- and jasmonic acid-mediated defense signa
56  SCMV showed no obvious correlation with the salicylic acid- and jasmonic acid-related defense signal
57  AtWRKY40 (DMG402007388) that was induced by salicylic acid; and a jasmonate ZIM-domain protein 1 (DM
58 sis in two half-reactions: activation of the salicylic acid as an acyl-adenylate and ligation onto th
59                               By quantifying salicylic acid at the level of several nanograms in situ
60         This article explores the use of SDS-salicylic acid based micellar systems for their potentia
61 ere able to perform in situ determination of salicylic acid based on its electrocatalytic oxidation.
62 ise for cancer therapy, and a novel class of salicylic acid-based STAT3 dimerization inhibitors that
63 onocyclic aromatics (i.e., benzene, toluene, salicylic acid, benzyl alcohol, and phenol) can also und
64 re leaf yellowing of pp2a-b'gamma depends on salicylic acid biosynthesis via SALICYLIC ACID INDUCTION
65  CRWN proteins induces the expression of the salicylic acid biosynthetic gene ISOCHORISMATE SYNTHASE1
66 quantify selected disinfection byproducts of salicylic acid, bisphenol A, gemfibrozil, naproxen, dicl
67 aprolactone (PCL) filaments were loaded with salicylic acid by hot melt extrusion (HME) (theoretical
68 .g., phenol, 4-chlorophenol, 2-chlorophenol, salicylic acid, catechol, maleic acid, oxalate, and urea
69 f programmed cell death, and accumulation of salicylic acid, closely mimicking phenotypes observed pr
70  wild-type and two mutants with jasmonate or salicylic acid compromised immunities.
71 gnaling capacity but exhibit hyper-inducible salicylic acid concentrations and deregulated cell death
72 nin induction was positively correlated with salicylic acid content, but not that of trans-jasmonic a
73                                  Some of the salicylic acid-deficient Arabidopsis eds5 mutants have a
74 opeptidases TOP1 and TOP2 placed them in the salicylic acid dependent branch of ETI, with a current m
75 ato (Pst-AvrRpm1), which was associated with salicylic acid-dependent defense.
76                                 In addition, salicylic acid-dependent gene expression is similar in b
77 of powdery mildew infection, and neither the salicylic acid-dependent nor jasmonate-dependent pathway
78 especially, SRG3 were positive regulators of salicylic acid-dependent plant immunity.
79 ic acid (ABA) signaling and thereby suppress salicylic acid-dependent resistance.
80 oxygen species, which act by stimulating the salicylic acid-dependent signaling pathway of the plant
81 sal immunity is independent on stomatal- and salicylic-acid-dependent regulation.
82 tal results demonstrated that the amounts of salicylic acid differed statistically in normal, phytoen
83  gibberellic acid, methyl jasmonic acid, and salicylic acid differentially regulate the stability of
84  redox status triggered by the immune signal salicylic acid does not compromise the circadian clock b
85 rystallized from the melt in the presence of salicylic acid either generated from aspirin decompositi
86 tic interactions between the stress hormones salicylic acid, ethylene, jasmonic acid, and abscisic ac
87  Moreover, increased levels of oxylipins and salicylic acid favored closure of stomata in response to
88 as a nose-shape mask by FDM 3DP, but the PCL-salicylic acid filament was not.
89                                         FPLA-salicylic acid filament was successfully printed as a no
90       FZL and the defense signaling molecule salicylic acid form a negative feedback loop in defense
91 delic acid, phthalic acid, benzoic acid, and salicylic acid) from aqueous samples.
92 ucoside (SAG), while UGT74F2 forms primarily salicylic acid glucose ester (SGE).
93 rmation of different products: UGT74F1 forms salicylic acid glucoside (SAG), while UGT74F2 forms prim
94  Effect of foliar pre-harvest application of salicylic acid, glycine-betaine complex and seaweed extr
95               As one type of plant hormones, salicylic acid has recently been found to be one of pivo
96 -H carboxylation reaction of phenols to give salicylic acids has been developed.
97 dodecyl sulphate (SDS) and the phenolic acid salicylic acid have been studied at several temperatures
98                                              Salicylic acid helps the micellization of SDS, both by i
99 er than typical honeydew honeys); and 0.054% salicylic acid (higher than previous estimates).
100 isic acid, or autophagy, but associates with salicylic acid homeostasis and signaling.
101 with plant defense-related processes such as salicylic acid homeostasis.
102  development, e.g., flagella, prophages, and salicylic acid hydroxylase.
103 tible host (efficient in the accumulation of salicylic acid).IMPORTANCE High diversity of within-host
104 smonic, (+)-7-iso-jasmonoyl-L-isoleucine and salicylic acid in certain parts of the host plant.
105  apart from exceptionally elevated levels of salicylic acid in lazy1.
106 devices for sensitively in situ detection of salicylic acid in tomato leaves with the sample volume o
107 cumulation of abscisic acid, jasmonates, and salicylic acid in wild type; in irAGO4 plants, infection
108  the med14 mutation significantly suppresses salicylic acid-induced defense responses, alters transcr
109 ination and degradation through formation of salicylic acid-induced NPR1 condensates (SINCs).
110 a depends on salicylic acid biosynthesis via SALICYLIC ACID INDUCTION DEFICIENT2 and bears the hallma
111 nce they are lost in isochorismate synthase1/salicylic acid induction deficient2 and nonexpressor of
112 R) and full oomycete resistance, but not for salicylic acid induction or bacterial resistance.
113 s non-expressor of pathogenesis related1 and salicylic acid induction-deficient2 increased hybrid see
114 ut not in the salicylate biosynthesis mutant Salicylic acid induction-deficient2, indicating that sal
115 luding the predominant isoform SUPPRESSOR OF SALICYLIC ACID INSENSITIVE2.
116 e background of the well-known suppressor of salicylic acid-insensitive2 (ssi2-2) mutant to confirm t
117 igh similarity to AtSSI2/FAB2 (Suppressor of Salicylic acid-Insensitivity2/Fatty Acid Biosynthesis2),
118  and is responsible for the incorporation of salicylic acid into the mycobactin siderophores.
119 ependent on, the foliar defense phytohormone salicylic acid is required to assemble a normal root mic
120 ts, suggesting that the hyperaccumulation of salicylic acid is unlikely to be responsible for dwarfis
121 ll three canonical defense hormone pathways (salicylic acid, jasmonate, and jasmonate/ethylene pathwa
122 e result of a subtle transient aberration of salicylic acid-jasmonic acid homeostasis during developm
123                                              Salicylic acid levels are similar in both types of plant
124 ecreased abscisic acid levels, and increased salicylic acid levels at the early stages of infection.
125                       Intriguingly, elevated salicylic acid levels did not contribute to the HR-like
126                       Upon fungal challenge, salicylic acid levels were triggered in EV samples, whil
127 sorghum plants, however, SCA feeding-induced salicylic acid levels were unaltered in the sorghum tole
128 crease in plant hydrogen peroxide (H2O2) and salicylic acid levels.
129 ic stress that coincides with an increase in salicylic acid levels.
130 tutive hypersensitive response with elevated salicylic acid levels.
131  flexible personalised-shape anti-acne drug (salicylic acid) loaded devices was demonstrated by two d
132 a strain Witches' Broom protein11 suppresses salicylic acid-mediated defense responses and enhances t
133 ral folates amenable for enhancement through salicylic acid-mediated elicitation, thereby holding a g
134 ns in diverse biological processes including salicylic acid-mediated immune response.
135 o been reported that Ca(2+) signals suppress salicylic acid-mediated plant defense through AtSR1/CAMT
136          CPK5 signaling resulted in enhanced salicylic acid-mediated resistance to the bacterial path
137 hydrogen peroxide, hypochlorite, dichromate, salicylic acid, melamine, and urea.
138                                              Salicylic acid modulates colonization of the root by spe
139 athogen-induced PME activity did not require salicylic acid or ethylene signaling, but was dependent
140                               In contrast to salicylic acid or its functional analog benzothiadiazole
141 rating enzymes) prevents full priming of the salicylic acid pathway and associated resistance by high
142         Key DEGs including PALs and PR-10 in salicylic acid pathway involved in SAR were significantl
143  contrast, biosynthesis and signaling of the salicylic acid pathway was downregulated.
144 are known to induce plant resistance via the salicylic acid pathway, whereas biting-chewing herbivore
145 nd inhibited cat2-triggered induction of the salicylic acid pathway.
146 induction of plant defense responses via the salicylic acid pathway.
147 o suggest cross talk between the flavone and salicylic acid pathways in Arabidopsis; in this way, pat
148  a pivotal role in regulating senescence via salicylic acid pathways.
149 enes in the abscisic acid, jasmonic acid and salicylic acid pathways.
150 utant alleles that revert the dwarf and high salicylic acid phenotypes of the high MEcPP containing m
151                                              Salicylic acid plays the role of a stabilizer, and gives
152                                 In addition, salicylic acid pre-treatment enhances the AtRbohD-mediat
153 dified or absent in N. caerulescens, whereas salicylic acid production in response to infection was r
154             NPR3 and NPR4, which function as salicylic acid receptors and adaptors of Cullin3-based E
155 ion of the chimera cause the accumulation of salicylic acid, reduced growth, and eventually lead to p
156 ired to negatively control the expression of salicylic acid-related defense genes, which have recentl
157 nodulation, including phosphorous supply and salicylic acid-related defense response.
158 ome activity, resulting in the inhibition of salicylic acid-related immune responses.
159 ropose PP2A-B'gamma age-dependently controls salicylic acid-related signaling in plant immunity and d
160 ct as precursors to aromatic amino acids and salicylic acid, respectively.
161 evealed that various phytohormone (auxin and salicylic acid) response genes are significantly altered
162 sis-Related1 (NPR1), the master regulator of salicylic acid responses, leading to the accumulation of
163  gene ontology terms related to immunity and salicylic acid responses.
164 eus and attenuates induction of jasmonic and salicylic acid-responsive genes.
165        StRDR1 was identified and shown to be salicylic acid-responsive.
166 dy was to evaluate the foliar application of salicylic acid (SA) (0.5, 1 and 2mM) or hydrogen peroxid
167 llae, hydrogen peroxide accumulation and the Salicylic acid (SA) - dependent pathway.
168  and assays for the hypersensitive response, salicylic acid (SA) accumulation and reactive oxygen spe
169 the responses to biotic stress by repressing salicylic acid (SA) accumulation and SA-mediated immunit
170 we demonstrate that SIZ1-mediated endogenous salicylic acid (SA) accumulation plays an important role
171 , use of NahG transgenic plants deficient in salicylic acid (SA) accumulation, measurement of endogen
172 ition, and reactive oxygen species (ROS) and salicylic acid (SA) accumulation.
173 ormation of tyloses, whereas treatments with salicylic acid (SA) and 1-aminocyclopropane-1-carboxylic
174 dogenous levels of jasmonates (JAs), but not salicylic acid (SA) and abscisic acid (ABA) increased in
175 onse, ceramide synthesis, JA, ethylene (ET), salicylic acid (SA) and abscisic acid (ABA) signaling.
176 nity that promotes production of the hormone salicylic acid (SA) and activation of defense gene expre
177 lant immunity that promote the production of salicylic acid (SA) and affect the expression of SA-depe
178 athways that separately engage ethylene with salicylic acid (SA) and cytokinin signaling during plant
179 ic plants showed increased concentrations of salicylic acid (SA) and higher levels of resistance to s
180 as to determine the effect of chitosan (CH), salicylic acid (SA) and hydrogen peroxide (H2O2) at diff
181 to the antagonistic interactions between the salicylic acid (SA) and JA defense pathways, efforts to
182  the normally antagonistic defence hormones, salicylic acid (SA) and jasmonic acid (JA) associated wi
183                       Expression analysis of salicylic acid (SA) and jasmonic acid (JA) biosynthesis
184  of DELLA on the archetypal defense hormones salicylic acid (SA) and jasmonic acid (JA) in Arabidopsi
185 ate plant defence signalling with a focus on salicylic acid (SA) and jasmonic acid (JA) pathways in r
186      Antagonism between the defense hormones salicylic acid (SA) and jasmonic acid (JA) plays a centr
187              The apparent antagonism between salicylic acid (SA) and jasmonic acid (JA)/ethylene (ET)
188       Here we show that the interconvertible salicylic acid (SA) and methylated SA (MeSA), well chara
189                                              Salicylic acid (SA) and pipecolic acid (Pip) levels were
190 the two immune-regulatory plant metabolites, salicylic acid (SA) and pipecolic acid (Pip), in the est
191                                          The salicylic acid (SA) and putrescine (Put) were sprayed (1
192 tress defense by the stress response hormone salicylic acid (SA) and the UPR, which is modulated by S
193 , yet the role of exogenous silicon (Si) and salicylic acid (SA) application has been largely unexplo
194 ) genes CBF1 and CBF2, and the repression of salicylic acid (SA) biosynthesis at warm temperature.
195 ts after inoculation reveal that several key salicylic acid (SA) biosynthesis genes are significantly
196 ion in the hybrids indicate decreases to the salicylic acid (SA) biosynthesis pathway and increases i
197 ant cells and causes a loss of ICS1-mediated salicylic acid (SA) biosynthesis.
198       We found that the plant immune hormone salicylic acid (SA) can trigger DNA damage in the absenc
199 hormonal signaling of jasmonic acid (JA) and salicylic acid (SA) compared with control.
200                         After observing that salicylic acid (SA) enhanced the accumulation of folates
201                                              Salicylic acid (SA) has long been implicated in plant re
202 as been shown that the crystal nucleation of salicylic acid (SA) in different solvents becomes increa
203 is identified the increased concentration of salicylic acid (SA) in the SV-treated plants after patho
204                                              Salicylic acid (SA) is a phytohormone regulating immune
205                                              Salicylic acid (SA) is a plant hormone that is critical
206                                              Salicylic acid (SA) is a signaling molecule utilized by
207                                              Salicylic acid (SA) is an important phytohormone mediati
208                                              Salicylic acid (SA) is an important plant hormone that i
209                                              Salicylic acid (SA) is central for the defense of plants
210 ignaling mediated by the phenolic metabolite salicylic acid (SA) is critical for the manifestation of
211                            The plant hormone salicylic acid (SA) is essential for local defense and s
212                                              Salicylic acid (SA) is the major metabolite and active i
213 pression of many genes bear the signature of salicylic acid (SA) mediated regulation, the breadth of
214 th 0.1 mM methyl jasmonate (MeJA) and 0.5 mM salicylic acid (SA) on quality parameters of lemon fruit
215                               The effects of salicylic acid (SA) or acetylsalicylic acid (ASA) treatm
216 emically during SAR signaling and locally by salicylic acid (SA) or its functional analog, benzo 1,2,
217  pathway and simultaneous down-regulation of salicylic acid (SA) pathway in guard cells.
218 nctions of defense genes associated with the salicylic acid (SA) pathway, including ENHANCED DISEASE
219 lants recognize insect eggs and activate the salicylic acid (SA) pathway.
220 ough it is well known that the plant hormone salicylic acid (SA) plays an essential role in defense,
221                            The plant hormone salicylic acid (SA) plays critical roles in plant defens
222 ochemical evidence supporting CATALASE2 as a salicylic acid (SA) receptor has finally emerged.
223  By contrast, under biological activation of salicylic acid (SA) signaling and hypersensitive PCD, Bi
224 crobe-associated molecular pattern-triggered salicylic acid (SA) signaling and infection-triggered et
225 tochondrial succinate dehydrogenase (SDH) in salicylic acid (SA) signaling was analyzed using two mut
226 ent in jasmonic acid (JA), ethylene (ET) and salicylic acid (SA) signaling.
227 is mutants impaired in jasmonic acid (JA) or salicylic acid (SA) signaling.
228                          In nox1 plants both salicylic acid (SA) synthesis and signalling were suppre
229 tion factors, induction of genes involved in salicylic acid (SA) synthesis, accumulation of free SA,
230 sochorismate synthase 1 (ICS1), required for salicylic acid (SA) synthesis, compromised gall formatio
231 njugates both indole-3-acetic acid (IAA) and salicylic acid (SA) to modulate auxin and pathogen respo
232 lent pathogens, and elevated accumulation of salicylic acid (SA) upon infection.
233                                              Salicylic acid (SA), a hormone essential for defense aga
234  to result from antagonism between auxin and salicylic acid (SA), a major regulator of plant defenses
235                                              Salicylic acid (SA), an essential regulator of plant def
236 ation, represses accumulation of the hormone salicylic acid (SA), an established regulator of plant i
237 , photosynthesis, regulation and response to salicylic acid (SA), and protein kinase leucine-rich rec
238 xpression of tomato defence genes related to salicylic acid (SA), and TD itself strongly induced the
239 odiphosphate (MEcPP) and the defense hormone salicylic acid (SA), as well as the high MEcPP but SA de
240                                   Effects of salicylic acid (SA), chitosan and combinative SA and chi
241 signalling components and in particular with salicylic acid (SA), hydrogen peroxide (H2O2), 6-benzyla
242 ation of defense genes that are regulated by salicylic acid (SA), jasmonic acid (JA) and ethylene (ET
243 ENE RESPONSE FACTOR 1 (ERF1)] clustered into salicylic acid (SA), jasmonic acid (JA), and ethylene (E
244                                    Levels of salicylic acid (SA), jasmonic acid, and abscisic acid in
245 ced by exogenous application of phytohormone salicylic acid (SA), methyl jasmonate (MeJA), phytopatho
246 xic acid (NA) and flumequine (FLU), but also salicylic acid (SA), natural organic matter (humic acid,
247 monas syringe pv. tomato (Pst) DC3000 in the salicylic acid (SA)- and nitric oxide (NO)-dependent pat
248 stance, biotrophic pathogens are resisted by salicylic acid (SA)- and reactive oxygen species (ROS)-d
249 nds in systemic acquired resistance (SAR), a salicylic acid (SA)-associated, broad-spectrum immune re
250 ciated with changes in cell wall defence and salicylic acid (SA)-dependent gene expression.
251 endent formation of lesions on leaves due to salicylic acid (SA)-dependent PCD, revealing roles for m
252 endent formation of lesions on leaves due to salicylic acid (SA)-dependent PCD.
253          We propose that PRR2 contributes to salicylic acid (SA)-dependent responses when challenged
254 florescences mainly involves accumulation of salicylic acid (SA)-inducible defense genes (ZmNAC, ZmHS
255                     The expression levels of salicylic acid (SA)-inducible genes were higher, but tho
256                                Plants deploy salicylic acid (SA)-mediated defences against biotrophs.
257  expression are associated with constitutive salicylic acid (SA)-mediated defense responses.
258 y been identified to play important roles in salicylic acid (SA)-mediated defense signaling.
259 usceptibility could be rescued by activating salicylic acid (SA)-mediated defense.
260 TA3 (CAMTA123) serve as master regulators of salicylic acid (SA)-mediated immunity, repressing the bi
261           To determine this, decoding of the salicylic acid (SA)-mediated plant immunity signalling n
262 regulated by jasmonic acid (JA), whereas the salicylic acid (SA)-responsive pathogenesis-related gene
263 y cross-talk between jasmonic acid (JA)- and salicylic acid (SA)-signaling pathways.
264  signalling is associated with a decrease in salicylic acid (SA)-triggered immunity (SATI) in Arabido
265 and the stress-related hormones ethylene and salicylic acid (SA).
266 d that the interaction can be deregulated by salicylic acid (SA).
267  acid (AzA), glycerol-3-phosphate (G3P), and salicylic acid (SA).
268 ich are receptors for the signaling molecule salicylic acid (SA).
269 t on the accumulation of the defense hormone salicylic acid (SA).
270 r by the phytohormones jasmonic acid (JA) or salicylic acid (SA).
271 l, and the generation of the signal molecule salicylic acid (SA).
272 sitive feedback loop with the defense signal salicylic acid (SA).
273 sistant to downy mildew and accumulated more salicylic acid (SA).
274 iented approach that transforms a benzofuran salicylic acid scaffold into a highly potent (IC(50) = 3
275 mutants affected in the synthesis pathway of salicylic acid (sid2-2) and jasmonate perception (coi1).
276 f downstream responses mediated by jasmonate-salicylic acid signaling cross talk, is involved in the
277 ogs of PR1 and PR5, and other factors of the salicylic acid signaling pathway.
278 piration and gas exchange, as well as better salicylic acid signaling.
279       In rice, the critical regulator of the salicylic acid signalling pathway is OsWRKY45, a transcr
280 ddress challenges in both the naringenin and salicylic acid synthesis pathways.
281 cluding metabolic capacity, streptomycin and salicylic acid synthesis, and nitrification.
282 roach, three strongest inhibitors namely are salicylic acid, tannic acid and trans-cinnamaldehyde hav
283 The ref8 mutant accumulates higher levels of salicylic acid than the wild type, but depletion of this
284 e plant hormones ethylene, jasmonic acid and salicylic acid to control host defense against this path
285 ize catechol (a potent anticancer drug) from salicylic acid to inhibit lung, brain, and soft-tissue c
286 ng two bioactive molecules, either niacin or salicylic acid, to an omega-3 fatty acid.
287 ccelerated by up to threefold in response to salicylic acid treatment and challenges with mannitol.
288 pt level of OsGR3 was greatly increased with salicylic acid treatment but was not significantly affec
289 de, low temperature, heat, abscisic acid and salicylic acid treatments.
290 licitations in the form of methyl jasmonate, salicylic acid, ultraviolet B light, and wounding, chose
291                                            o-Salicylic acid undergoes ESPT only in the "antiaromatic"
292 on of the primary metabolite chorismate into salicylic acid via isochorismate.
293 mation of micelles of SDS in the presence of salicylic acid was a thermodynamically spontaneous proce
294                                              Salicylic acid was readily halogenated, which is evident
295 , active cytokinins, active gibberellin, and salicylic acid were detected in the root tips of these p
296                 Concentration of sinapic and salicylic acids were highest phenolic acids of pearl mil
297                    Subsequently, substituted salicylic acids were prepared by deprotection of the est
298 ssion upon application of phytohormones like salicylic acid which might be the key in fine-tuning the
299                                 Furthermore, salicylic acid, which is the major plant defense hormone
300  a punched hole of 1.5mm diameter to release salicylic acid with minor influence on continuous growth

 
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