ライフサイエンスコーパス: salience

1 n subjective measures of color appearance or salience.

2 re most salient, and predicts their relative salience.

3 ces in cued approach behaviors and incentive salience.

4 eflects both expected value and motivational salience.

5 h modulations of the early effects of visual salience.

6 , commonly interpreted as "bottom-up" visual salience.

7 ronizes cortical theta and decreases sensory salience.

8 s as they viewed stimuli varying in semantic salience.

9 disrupts maintenance of conditioned stimulus salience.

10 xperienced stimuli acquiring aberrantly high salience.

11 associated with connectivity in the DMN and salience.

12 be important in attribution of motivational salience.

13 ions of striatum and cortex and also encodes salience.

14 non-social stimuli under conditions of equal salience.

15 tively maximizing communication distance and salience.

16 (RPE), in addition to other signals, such as salience.

17 ed for fixation than objects with low visual salience.

18 formance, similar to the effects of physical salience.

19 ory experience to signal areas of behavioral salience.

20 d learning by broadcasting information about salience [2, 3, 5, 7-19].

21 dopamine dysfunction, reward processing, and salience abnormalities in people at clinical high risk o

22 Our findings show that, whereas physical salience accelerates the speed of attentional selection,

23 ndings show stable individual differences in salience along a set of fundamental semantic dimensions

24 In addition to conscious goals and stimulus salience, an observer's prior experience also influences

25 itioned as an interface between motivational salience and behavioral output.

26 otor networks and negatively correlated with salience and cingulo-opercular networks.

27 cally gate auditory representations based on salience and context.

28 technology, appeared to have had widespread salience and effects.

29 ort these cognitive functions, including the salience and executive control networks.

30 ted by a correlation between individual face salience and face recognition skills.

31 have indicated that both increased physical salience and increased reward-value salience of a target

32 cal and neural underpinnings of value-driven salience and its effects on attention and behavior.

33 Stimulus salience and reward expectations influence this prioriti

34 othalamic peptide oxytocin in augmenting the salience and rewarding value of social stimuli, includin

35 alue of a conditioned stimulus (CS) into its salience and valence components.

36 eurochemical circuits that mediate incentive-salience and/or reward systems (dopamine, opioid peptide

37 rced behavior, such as motivation, incentive salience, and cost-benefit calculations.

38 lex data elements, feature heterogeneity and salience, and health analytics.

39 ciated with executive functioning, incentive salience, and interoceptive processing in cigarette deci

40 sory-motor, lateral sensory-motor, auditory, salience, and subcortical networks in participants with

41 eories, however, suggest that these forms of salience are intrinsically different, although the neura

42 after which it came to reflect motivational salience as movement onset neared.

43 ent planning, before reflecting motivational salience as movement onset neared.

44 it reflects expected value and motivational salience at different latencies.

45 nd cannabidiol (CBD) differentially regulate salience attribution and psychiatric risk.

46 ere sex differences on measures of incentive salience attribution and sensation-seeking behavior that

47 ships between multiple measures of incentive salience attribution and, based on these findings, we ge

48 ly, whereas intra-vHipp THC also potentiates salience attribution in morphine place-preference and fe

49 the dopamine-dependent process of incentive salience attribution.

50 ch behavior (to obtain an index of incentive salience attribution; 'sign-tracking'), and subsequently

51 consider the role of task goals or physical salience, but attention can also be captured by previous

52 outside the neuronal RF while we manipulated salience by varying the time between stimulus presentati

53 e inference to demonstrate how attention and salience can be disambiguated in terms of message passin

54 and emotional regulation (eg, sensorimotor, salience, central executive networks).

55 lt mode) and environmentally driven control (salience, cingulo-opercular) systems.

56 ty of human SN, which underpins value versus salience coding, and impulsive choice versus impulsive a

57 network in decisional impulsivity, while the salience-coding ventral SN network was associated with m

58 arrays for targets of either a high-physical-salience color or one of two low-physical-salience color

59 al-salience color or one of two low-physical-salience colors across three experimental phases.

60 ively higher for one of the two low-physical-salience colors, resulting in their RTs becoming as fast

61 with the object view, we suggest that visual salience contributes to prioritization among objects.

62 vealed that both groups exploited contextual salience cues in their gaze judgments, and that the aver

63 is of individual subjects' use of contextual salience cues).

64 order cognitive network system including the salience, default mode, and central executive networks w

65 siform, somatosensory cortex, and thalamus), salience detection (anterior insula), and learning and m

66 nchored maturation of circuits important for salience detection and cognitive control, as well as dis

67 ed network consisting of regions involved in salience detection, attention, and task engagement as a

68 s and systems, particularly those supporting salience detection.

69 The set of reliable individual salience dimensions and their covariance pattern replica

70 The pattern was similar for ratings of salience, distress, personal relevance, global threat, a

71 Our results suggest that the contribution of salience dominates at higher levels of the visual system

72 signals within the brain and in controlling salience-driven behavior.

73 erior cingulate cortex (PCC) signal decision salience during foraging to motivate disengagement from

74 ponses indicative of positivity, reward, and salience during impression formation among men.

75 regions associated with visual attention and salience (e.g., precuneus, r = -0.35) to unhealthy relat

76 e regions of cortex and striatum and encodes salience (equal response to wins and losses).

77 Strikingly, salience-executive control between-network cohesion peak

78 stimuli with reward value can enhance their salience, facilitating their attentional selection.

79 ty typify natural sounds, they may differ in salience for communication behavior and engage distinct

80 The concept of self-management support had salience for patients, carers and specialist nurses alon

81 Here, we consider attention and salience from the perspective offered by active inferenc

82 linergic systems have been shown to increase salience/gain while suppressing extraneous information.

83 egulation of functional domains of incentive salience/habits, negative emotional states, and executiv

84 entive delay task in which stimuli varied by salience (high versus low) and valence (win versus loss)

85 fast RTs, the N2pc's for these low-physical-salience, high-value targets remained later than for hig

86 Consistent with the incentive salience hypothesis combining learned cue-reward associa

87 The social salience hypothesis posed an innovative solution to thes

88 The aberrant salience hypothesis proposes that striatal dopamine dysr

89 The 'variance is salience' hypothesis predicts that two-point correlation

90 ent coding principle leads to a 'variance is salience' hypothesis, and that this hypothesis accounts

91 en previously shown to vary as a function of salience (i.e., monetary reward/loss) in a spatial atten

92 This value-driven salience improves behavioral performance, similar to the

93 PE is known to support learning, the role of salience in learning remains less clear.

94 may be defined and the role of precision and salience in mediating the interactions between what is i

95 In turn, CS salience in the CE recruited these CS representations bo

96 ation, and thus decrease responses driven by salience in the RF.

97 e attention and facilitate memory, enhancing salience in ways that could underlie, for example, prefe

98 ts of location-based and object-based visual salience in young and older (65 + years) adults.

99 Our findings illustrate how salience, in terms of possible reward magnitude or proba

100 indings reveal a plausible mechanism for how salience, in terms of possible reward magnitude or proba

101 g, control of sleep and wakefulness, sensory salience including pain, and the physiology of correlate

102 bivariate correlation analysis and cognitive salience index calculations to disentangle the dynamics

103 Salience indices, measures of the most important words o

104 unclear, however, whether these two forms of salience interact with attentional networks through simi

105 This maintenance of salience is a necessary precursor for these stimuli to g

106 Finally, an attenuation of social salience is associated with social disorders, such as au

107 t misophonia is a disorder in which abnormal salience is attributed to particular sounds based on the

108 t such reward-related modulation of stimulus salience is conceptually similar to an "attentional habi

109 f these response changes in FEF suggest that salience is represented much earlier (<100 ms following

110 In brief, we suggest that salience is something that is afforded to actions that r

111 Thus, stimulus salience is the primary determinant of behavior in the S

112 ling is implicated may be caused by aberrant salience leading to a change in the nature of the inform

113 These results suggest that enhanced physical salience leads to faster attentional orienting, but valu

114 ateral VTA activity more closely reflected a salience-like signal.

115 Our results indicate salience-like signals can have similar downstream conseq

116 d porthole, suggesting that higher incentive salience made that cue more attractive.

117 ibility that expected value and motivational salience manifest at different latencies during movement

118 Finally, we show that bottom-up salience, manipulated through varying stimulus contrast,

119 emonstrated that optical blur reduces visual salience more for light than dark stimuli because it rem

120 ) and in reward responsiveness (subcortical, salience, motor/sensory).

121 ctors, the factors corresponded to incentive salience, negative emotionality, and executive function

122 al in the addiction cycle, namely, incentive salience, negative emotionality, and executive function,

123 in brain regions including a key node of the salience network (anterior insula) increased linearly ac

124 Regions of the salience network (dorsal anterior cingulate, insula, and

125 network [e.g., dorsolateral (dl)PFC], and a salience network (e.g., insula).

126 d eight weaker circuits including within the salience network (insula seeds) and between temporal pol

127 k (left dorsolateral prefrontal cortex), and salience network (right anterior insula [rAI]).

128 iation networks [default-mode network (DMN), salience network (SAL), dorsal attention network, and fr

129 nectivity in two intrinsic networks [DMN and salience network (SAN)], and (iii) higher within-network

130 ventral seeds in LAI were correlated to the salience network (SN) and language network (LN) and anti

131 The salience network (SN) is dysregulated in many neuropsych

132 work (DN), frontoparietal network (FPN), and salience network (SN) while recording evoked responses i

133 s (RSNs) such as sensorimotor network (SMN), salience network (SN), and default-mode network (DMN)-an

134 n network (DAN), default-mode network (DMN), salience network (SN), and executive control network (EC

135 dynamic cross-network interactions among the salience network (SN), central executive network, and de

136 work (DN), frontoparietal network (FPN), and salience network (SN), in seven subjects undergoing inva

137 AN), the dorsal attention network (DAN), the salience network (SN), the somatosensory network (SMN) a

138 twork (CEN), default mode network (DMN), and salience network (SN).

139 e tested the hypothesis in core nodes of the salience network (the dorsal anterior cingulate cortex [

140 Specifically, diminished activity in the salience network and enhanced activity in a cortico-cere

141 and the anterior insula (AI) constitute the salience network and form as well the major cortical com

142 downregulation is reliant on input from the salience network and frontoparietal network, an importan

143 demonstrate a close relationship between the salience network and mesolimbic dopamine system, and the

144 is associated with aberrant iFC between the salience network and thalamus, and aberrant sensorimotor

145 n between nAChRs density/availability in the salience network and the heart rate variability in human

146 role of the sgACC, default mode network, and salience network as predictors of TMS response and sugge

147 Furthermore, these decreases in right insula/salience network connectivity correlated with baseline t

148 nthesis capacity was associated with greater salience network connectivity, and this relationship was

149 ces in three networks: DMN, ECN and anterior salience network connectivity, as well as a whole brain

150 release capacity was associated with weaker salience network connectivity.

151 ing that coordination of activity within the salience network dynamically tracks arousal.

152 Results revealed decreased right insula/salience network functional connectivity under MDMA.

153 taken in the same individuals to investigate salience network functional connectivity.

154 12 years since its initial description, the salience network has been extensively studied, using div

155 Stronger baseline theta-band rACC-rAI (salience network hub) connectivity predicted greater dep

156 ctivity between the default mode network and salience network in attempters and show that self-injure

157 ngs support roles for several regions of the salience network in the pathophysiology of FND.

158 ctional neurological disorder (FND), and the salience network is implicated in the pathophysiology of

159 ed with optimal noradrenergic signaling, the salience network is optimally able to engage the executi

160 ight insular disintegration (ie, compromised salience network membership) as a neurobiological signat

161 status and affective symptoms would map onto salience network regions and that patients with FND woul

162 of research, the essential functions of the salience network remain uncertain.

163 extent of traumatic axonal injury within the salience network strongly influenced the behavioural res

164 Deficits in the salience network suggest an impaired ability to process

165 three network modules interconnected by the salience network that displays reduced network coupling

166 tal gyrus/anterior insula to the rest of the salience network was associated with reduced beneficial

167 wn to be decreased in premature infants: the salience network with the superior frontal, auditory, an

168 auditory, and sensorimotor networks, and the salience network with the thalamus and precuneus network

169 rfaces with the anterior-cingulo-insular or "salience network" demonstrated to be transdiagnostically

170 The term "salience network" refers to a suite of brain regions who

171 ior insular cortex (AIC), a core hub of the "salience network" that is critical for perception of int

172 e anterior cingulate cortex that anchors the salience network, a system important for adaptive switch

173 These results suggest that the salience network, and its attention-control function, ar

174 r default-mode network (DMN), posterior DMN, salience network, and right central executive network (C

175 e network (DMN), frontoparietal network, and salience network, are key functional networks of the hum

176 c dopamine neurons may activate nodes of the salience network, but in vivo human research is required

177 covery, conceptualization, and naming of the salience network, highlighting aspects that may be unfam

178 arousal, and within-network cohesion in the salience network, indicating that coordination of activi

179 or frontal gyrus/anterior insula node of the salience network, which was targeted because our previou

180 In the salience network-centred system, patients had reduced as

181 , which correlated positively with decreased salience network-mediodorsal-thalamus iFC.

182 d us to tap early activity with the putative salience network.

183 mesolimbic dopamine system and the cortical salience network.

184 nt with a faster activation of the autonomic salience network.

185 connectivity between the hippocampus and the salience network.

186 l network that partially overlapped with the salience network.

187 effects of heroin abuse on cognition and the salience network.

188 ation-inhibition balance in key nodes of the salience network.

189 tive control systems: (i) cingulo-opercular "salience" network (SN) anchored in the right anterior in

190 disruption corresponded prominently to the "salience" network, the ventral striatal/ventromedial pre

191 functional integrity of a cingulo-opercular "salience" network.

192 "hub" areas of the default mode and cortical salience networks, respectively.

193 al cortex, particularly the default mode and salience networks.

194 frontoparietal executive, default mode, and salience networks.

195 rom the ventral attention, default mode, and salience networks.

196 ttention to sounds with different valence or salience (neutral, novel pre-habituated, threatening).

197 physical salience and increased reward-value salience of a target improve behavioral measures of atte

198 airport and a control population to test the salience of airport, and control population specific son

199 y bottom-up attention is graded by degree of salience of ambient events.

200 togenetic silencing of IPN neurons increases salience of and interaction with familiar stimuli withou

201 Oxytocin increases the salience of both positive and negative social contexts a

202 d it has been proposed that OT increases the salience of both positive and negative social cues.

203 ng actions by proposing that OT enhances the salience of both positive and negative social interactio

204 ns are offered to improve the visibility and salience of clinical significance in nursing science.

205 aimed at evaluating behavioral shifts in the salience of cocaine now vs money later, we found that ke

206 d activity of BLA neurons is to maintain the salience of conditioned stimuli that precede it.

207 or nonreinforcement causes reductions in the salience of conditioned stimuli, rendering these stimuli

208 gest that reward can increase the perceptual salience of environmental stimuli, ensuring that potenti

209 that PVH-OT neurons may act to convey social salience of environmental stimuli.

210 ndispensable for reappraising the behavioral salience of external cues.

211 heir gendered environment to investigate the salience of gender norms during adolescence for social m

212 n while also demonstrating the communicative salience of male USVs.

213 gender-neutral pronouns decreases the mental salience of males.

214 limbic system, in tracking context-dependent salience of old and new information.

215 ty and pupil size reflected task-conditional salience of old and new stimuli, but, unexpectedly, this

216 ler increases in the LPP as the motivational salience of pleasant images increased (exciting<affiliat

217 reases in LPP amplitudes as the motivational salience of pleasant images increased.

218 s from the body potentiates the motivational salience of reward cues through the recruitment of hedon

219 rons of the VTA encode strength of incentive salience of reward cues.

220 nsory processing, selectively increasing the salience of reward-associated stimuli.

221 This study highlights the speed and salience of social information relating to group dynamic

222 oal-directed attention) [1, 2], the physical salience of stimuli (stimulus-driven attention) [3-5], a

223 ipulating both the value-driven and physical salience of targets in a visual search task while record

224 manipulating the physical and value-related salience of targets in a visual search task, comparing t

225 to the NAc do not influence the motivational salience of the cue.

226 ck the ability to increase the physiological salience of the events that provide the convergent cross

227 In showing how visual salience of the focal actor may introduce unique effects

228 was due, in part, to variation in the visual salience of the focal actor: the body cam wearer is typi

229 amaging external stimuli, and to enhance the salience of the stimulus resulting in escape and avoidan

230 ies suggest that differences in the relative salience of the two pitch cues can be attributed to diff

231 Predictive models can enhance the salience of unanticipated input.

232 tion, it is not surprising that the acquired salience of visual items further modulates.

233 Neurons sensitive to the salience of visual stimuli are widespread throughout the

234 al stimulation and the abrupt appearance, or salience, of the presentation.

235 ity reflects expected value and motivational salience on different time scales during reach planning.

236 cortex but also diminished the influence of salience on visually guided behavior.

237 tion in real-world scenes is guided by image salience or by objects has been a matter of scientific d

238 no relationship between rsFC in the anterior salience or default mode networks with inflammation in e

239 isplay abnormalities related to motivational salience, or the ability of stimuli to elicit attention

240 forth as a likely system to evaluate social salience owing to its well-described role in motivation.

241 idance without recurrence to objects, visual salience predicted whether image patches were fixated or

242 nterior insula, a brain region implicated in salience processing and alertness, activations that are

243 ntion-emotion interactions, which could bias salience processing and associative learning in youth wi

244 d activation in key structures of reward and salience processing regions.

245 s, represent an attentional effect to facial salience rather than to their rewarding properties.

246 This salience reduction was stimulus-specific, long-lasting,

247 In our foraging tasks, salience refers to the difference between decision thres

248 Visual salience reflects features of the observer as well as th

249 jectively greater behavioral and attentional salience, regulating speed of simple color discriminatio

250 in the anterior insula cortex and targeting salience-related and attention-related frontoparietal ar

251 errors [20-24]-and to date, precisely which salience-related information the BF broadcasts is unclea

252 However, a neural source of salience remains elusive.

253 rocessing related to the default mode (DMS), salience/reward (SRS), and frontoparietal (FPS) subnetwo

254 Salience scores indicating the relative importance of ea

255 speed of attentional selection, value-driven salience selectively enhances its strength.

256 he first evidence in humans linking NPY with salience sensitivity of the NAc, raising the possibility

257 ss micturition loci (e.g., subregions of the salience, sensorimotor, and default networks) that were

258 magnitude consistent with a population-level salience signal, which was required for Pavlovian condit

259 related with an unsigned prediction error or salience signal.

260 aused pronounced and selective reductions of salience signals in prefrontal cortex but also diminishe

261 parietal cortex and simultaneously recorded salience signals in prefrontal cortex.

262 Salience signals were stronger in poor foraging contexts

263 causal role of parietal cortex in regulating salience signals within the brain and in controlling sal

264 gnals are localized in TS along with general salience signals, while VS dopamine reliably encodes rew

265 of neural resources according to behavioral salience.SIGNIFICANCE STATEMENT The present work demonst

266 tic activity with dorsal attention (DAN) and salience (SN) networks.

267 al NAc responses to high-salience versus low-salience stimuli were greater for Low-NPY subjects relat

268 egulation and the effects on brain incentive salience systems, such as dopamine.

269 ia dopamine-dependent, cue-driven, incentive salience systems.

270 2pc latencies were shorter for high-physical-salience targets, indicating faster attentional orientin

271 argets remained later than for high-physical-salience targets, instead eliciting significantly larger

272 ir RTs becoming as fast as the high-physical-salience targets.

273 ovide a reinforcement signal of motivational salience that invigorates adaptive behaviors by promotin

274 zed that the rapid presentation would reduce salience (the sudden appearance within the visual field)

275 e, more than passively representing value or salience, the signal appears to play a versatile and act

276 tential components sensitive to motivational salience-the Early Posterior Negativity (EPN), reflectin

277 ning in the basal ganglia with the incentive salience theory in a single simple framework, and it pro

278 We quantified the attribution of incentive salience through cue approach behavior and cue interacti

279 al variation in the attribution of incentive salience to both food- and social-related cues, which ha

280 Research on the attribution of incentive salience to drug cues has furthered our understanding of

281 re used to assess the unique contribution of salience to fixation selection in scenes.

282 -trackers; GTs) prone to attribute incentive salience to food reward cues.

283 amine dysregulation causes misattribution of salience to irrelevant stimuli leading to psychosis.

284 ition, the propensity to attribute incentive salience to reward cues can predict the propensity to ap

285 uch as the propensity to attribute incentive salience to reward cues that is modeled in rats by sign-

286 kers (STs)] are prone to attribute incentive salience to reward cues, which can manifest as a propens

287 mine neurons in the attribution of incentive salience to reward-paired cues, and underscore the conse

288 ndency of STs and GTs to attribute incentive salience to social reward cues as well as form a social-

289 mpared to GTs, STs attributed more incentive salience to social-related cues and exhibited prosocial

290 ster attentional orienting, but value-driven salience to stronger attentional orienting, underscoring

291 : control network, default mode network, and salience/ventral attention network.

292 e found that bilateral NAc responses to high-salience versus low-salience stimuli were greater for Lo

293 ventral hippocampus (vHipp) relays emotional salience via control of dopamine (DA) neuronal activity

294 Reconciling the salience view with the object view, we suggest that visu

295 al-functional decoupling was observed in the salience, visual and somatomotor networks.

296 dependence-related alterations in incentive salience/'wanting'.

297 y difference was not observed when the cue's salience was diminished after extinction learning.

298 In an attempt to identify a source of visual salience, we reversibly inactivated parietal cortex and

299 s show increased connectivity in the DMN and salience when neocortical Tau levels are low, whereas ab

300 ies and ignore the variation of motivational salience within these categories.