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1 y and vaccine development against pathogenic Salmonellae.
2 lence by manipulating the mouse host and the salmonellae.
3 xpression of a variety of unrelated genes in salmonellae.
4 ted in equivalent virulence of Spv+ and Spv- salmonellae.
5 uired very recently, after speciation of the salmonellae.
6 tions elicited by typhoidal and nontyphoidal salmonellae.
7 y slightly attenuated relative to InvA+ Spv- salmonellae.
8 eased in vivo replication rate for wild-type salmonellae.
9 ancestor of all the contemporary lineages of salmonellae.
10 caused by groups A, B, C, and D nontyphoidal salmonellae.
11 ctions mediated by enteric pathogens such as salmonellae.
12 ved in typhoid toxin islets found in diverse salmonellae.
13 e important for gastrointestinal survival of salmonellae.
14  reagents suitable for serotyping strains of salmonellae.
15 morphine pellet followed by inoculation with salmonellae.
16  to kill the vast majority of nontransformed salmonellae.
17                             Of the serotyped salmonellae, 14% (21/152) were Salmonella enterica serov
18 difications reduce TLR4-signaling as part of Salmonellae adaptation to host environments.
19 n increased splenic infection with wild-type salmonellae after oral inoculation; however, Spv- salmon
20 aneously in wild-type and MsbB- strain 14028 salmonellae and accounts for about one-third of all of t
21 emonstrate specific binding affinity between salmonellae and cholesterol.
22 synthetic pathway is shared by virtually all salmonellae and must be maintained by selection, yet no
23 ease caused by the intramacrophage pathogens salmonellae and mycobacteria.
24  alters how macrophages recognize or process salmonellae and prevents the rapid onset of proinflammat
25   Intracellular pathogenic organisms such as salmonellae and shigellae are able to evade the effects
26 ncreased early (0 to 4 h) blood clearance of salmonellae and significantly decreased numbers of bacte
27 g challenge with the intracellular pathogens salmonellae and Staphylococcus aureus.
28        The long-standing association between Salmonellae and their animal hosts has resulted in the a
29 s complex in overall structure than those of salmonellae and Vibrio cholerae.
30                                          The salmonellae are a diverse group of bacteria within the f
31                                 Nontyphoidal Salmonellae are a major cause of life-threatening bacter
32       Upon interaction with epithelial cells salmonellae are able to elicit transepithelial signallin
33                                 Nontyphoidal salmonellae are among the leading causes of food-borne d
34                                              Salmonellae are associated with a wide spectrum of invas
35                             Non-subspecies I salmonellae are commensals of cold-blooded vertebrates a
36                                 Nontyphoidal salmonellae are enteric pathogens that cause acute gastr
37                                              Salmonellae are enterobacteria that have the unique abil
38                                              Salmonellae are gastrointestinal pathogens of man and an
39                                              Salmonellae are gram-negative bacteria that cause gastro
40                                 Nontyphoidal Salmonellae are highly prevalent food-borne pathogens.
41                                              Salmonellae are pathogenic bacteria that cause significa
42 ractory to boosting with orally administered salmonellae at 7 weeks.
43                           Mice infected with salmonellae become hypersusceptible to endotoxin.
44 ex, subspecies I, to which 99% of pathogenic salmonellae belong.
45 lt suggested an existing interaction between salmonellae, bile, and eukaryotic cell invasion.
46 during biofilm development, specifically how salmonellae bind to cholesterol, and suggest a target fo
47         We have previously demonstrated that salmonellae, but not Escherichia coli or Yersinia entero
48                                              Salmonellae can exist in an asymptomatic carrier state i
49                      These data suggest that salmonellae can sense and respond to bile to increase re
50                                              Salmonellae can use ethanolamine (EA) as a sole source o
51 o involve fewer virulence genes than that of Salmonellae, complex virulence-regulatory networks have
52 teric bacterial pathogens, including group B salmonellae, conjugates composed of the detoxified LPS o
53                                              Salmonellae coordinate SPI-1 expression with anatomical
54             The reasons why non-subspecies I salmonellae do not circulate in populations of warm-bloo
55 portant role in vivo in host defense against salmonellae during the early stages of infection.
56 as essential to xenophagic clearance and the Salmonellae effector SopF that inhibits bacterial cleara
57    Genome comparisons of the closely related salmonellae emphasize the insights that can be gleaned f
58                                              Salmonellae encode two virulence-associated TTSS.
59                                              Salmonellae encode two virulence-associated type III sec
60             During infection of their hosts, salmonellae enter intestinal epithelial cells.
61                                              Salmonellae establish infection and avoid clearance by t
62                           We have shown that salmonellae form bile-mediated biofilms on human gallsto
63         We have previously demonstrated that salmonellae form biofilms on human gallstones and choles
64 ge, and we have previously demonstrated that salmonellae form biofilms on human gallstones in vitro.
65                               In our assays, salmonellae formed full biofilms on the surfaces of gall
66 tion into susceptible BALB/c mice, wild-type salmonellae grew at the expected rate of approximately 1
67                                              Salmonellae have been shown to be competent for conjugat
68                    As enteric pathogens, the salmonellae have developed systems by which they can sen
69      Previous studies on the O-Ag capsule of salmonellae have focused primarily on its role in bacter
70                                      Diverse salmonellae have the potential to cause disease and may
71 l drive a revolution in the understanding of Salmonellae in many different niches that are critical f
72 ce, epidemiology, and genetic relatedness of salmonellae in nondomestic birds.
73 mine if the spv genes affected the growth of salmonellae in nonphagocytic cells, an invA::aphT mutati
74 var Typhi (S. Typhi) differs from most other salmonellae in that it causes a life-threatening systemi
75 hesis for the creation of lactose-fermenting salmonellae in the environment is presented.
76 ice survived asymptomatically with pbgA-lpxC salmonellae in their livers and spleens for months, but
77                 To gain further insight into salmonellae in these hosts, 22 Salmonella isolates from
78 ing infection of the gastrointestinal tract, salmonellae induce cytokine production and inflammatory
79 addition of 5'-methylthioadenosine increased Salmonellae-induced cell death.
80 ride, are important for host defense against Salmonellae infection.
81                                              Salmonellae initiate disease through the invasion of hos
82 d enzyme methods can cluster closely related salmonellae into epidemiologically relevant hierarchies.
83 fied ST313 lineage of invasive non-typhoidal Salmonellae (iNTS).
84 esistance to antimicrobial agents within the salmonellae is a worldwide problem that has been associa
85 inal study of antimicrobial resistance among salmonellae isolated from swine, we studied 484 Salmonel
86 or pathogenicity of several bacteria and for Salmonellae lacking components of AcrAB-TolC, expression
87                               In the case of salmonellae, many virulence factors are regulated via Ph
88      Specific factors unique to nontyphoidal salmonellae may also be important for stimulation of the
89 e a unique niche in which lactose-fermenting salmonellae may arise.
90                          Upon infection with salmonellae, mucosal expression of CCL2 is rapidly up-re
91                                        MsbB- salmonellae mutate extragenically to EGTA-tolerant deriv
92 ears, Streptococcus pneumoniae, nontyphoidal salmonellae (NTS), and Hib were the most frequently isol
93 ens of community-acquired BSI are nontyphoid salmonellae (NTS), Streptococcus pneumoniae, Escherichia
94                                          For Salmonellae, one such regulatory system is PhoP-PhoQ, wh
95 yphimurium or its flagella, but not by other salmonellae or S. typhimurium mutants unable to synthesi
96                     We isolated nontyphoidal salmonellae organisms from blood (n = 35), bone marrow (
97                                 Nontyphoidal salmonellae, particularly Salmonella enterica serovar Ty
98                                          The Salmonellae PhoP-PhoQ virulence regulators induce resist
99                                          The Salmonellae PhoQ sensor kinase senses the mammalian phag
100        In a previous study, we reported that salmonellae possess the ability to stimulate tumor necro
101 Escherichia coli or Yersinia enterocolitica, salmonellae rapidly induce TNF-alpha expression in these
102                                   Attenuated salmonellae represent attractive candidates for the deli
103 ted that CCL2(-/-) macrophages infected with salmonellae resulted in dysregulated cytokine production
104 sive bacterial infection caused by typhoidal Salmonellae (Salmonella enterica serovars Typhi and Para
105 of typhoid toxin produced by a non-typhoidal Salmonellae serotype.
106 ococcus pneumoniae (eight) and non-typhoidal salmonellae (seven).
107                           Invasion-defective salmonellae still exhibited the Spv phenotype.
108 teria, while animals infected with wild-type salmonellae succumbed within 1 week.
109                In contrast, other pathogenic salmonellae, such as S. enterica serovars Typhimurium an
110 There is evidence of multidrug resistance in salmonellae that warrants vigilant monitoring and survei
111 ridization analysis revealed that, among the salmonellae, the fim gene cluster is present in all isol
112                                 Unlike other Salmonellae, the intracellular bacterial human pathogen
113         Compared to the OMs of the wild-type salmonellae, the OMs of the pbgA mutants had increased l
114 ribution of SPI-3 sequences varies among the salmonellae: the right end of the island, which harbors
115 al epithelial cells and are thought to allow salmonellae to enter and cross the intestinal epithelium
116 eature of salmonella pathogenesis and allows salmonellae to enter intestinal epithelial cells.
117 lial cell (IEC) lines, the capacity of these salmonellae to invade IECs, and the ability of the bacte
118 ich hilA and invasion genes are required for salmonellae to overcome a host clearance response elicit
119 , it was unclear which signals are sensed by salmonellae to promote PhoQ-mediated virulence.
120  viruses, but Campylobacter and nontyphoidal Salmonellae together account for about one fourth of cas
121                             After ingestion, salmonellae traverse the upper digestive tract and initi
122 eudomonas aeruginosa, Klebsiella pneumoniae, Salmonellae typhi, Candida albicans, Rhizopus stolonifer
123  Previous studies have shown that attenuated salmonellae utilized as vaccine vectors engender strong
124  promoter exhibited negligible impairment of Salmonellae virulence.
125 ealed that the TNFalpha-inducing activity of salmonellae was associated with flagellin, a major compo
126 rface fibre produced by Escherichia coli and salmonellae, was proposed on the basis of genetic eviden
127 nellae after oral inoculation; however, Spv- salmonellae were defective at increasing splenic infecti
128                       Furthermore, MMG-grown salmonellae were more resistant to acid stress and macro
129                                   InvA- Spv+ salmonellae were not significantly affected for splenic
130 ed with the wild-type strain, and InvA- Spv- salmonellae were only slightly attenuated relative to In
131 of T cells and B cells on the Spv phenotype, salmonellae were orally inoculated into nude and SCID BA
132                Overall, 70% (142/203) of the salmonellae were pansusceptible.
133                                              Salmonellae were recovered from nasal lymphoid tissues,
134         The results show that >200-fold more salmonellae were recovered in livers of the latter group
135            In the latter (heavily infected), salmonellae were seen within mononuclear cells, indicati
136             Furthermore, in contrast to most Salmonellae, which can infect a broad range of hosts, S.
137                                     However, salmonellae, which have a core that is chemically dissim
138            Unlike infections with most other Salmonellae, which result in self-limiting gastroenterit
139 rom challenge with a lethal dose of virulent salmonellae, with a dramatic reduction in bacterial numb
140 ions is known to directly kill intracellular salmonellae within macrophages.
141 nd possibly by inhibiting the replication of salmonellae within other macrophages.
142          The intricate web of interconnected salmonellae within this ecosystem underscores the import

 
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