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1 ophosphoglycan in the anterior midgut of the sandfly.
2 al for their survival and development in the sandfly.
3 r-borne disease occurred through the bite of sandflies.
4 asymptomatic participants were infectious to sandflies.
5 ations of two insect vectors: mosquitoes and sandflies.
6 nments encountered during transition between sandfly and vertebrate hosts.
7 tilize an arthropod vector (e.g., mosquitos, sandflies, and ticks) to spread throughout human populat
8 ria-infected mosquitoes, Leishmania-infected sandflies, and Trypanosoma-infected tsetse flies.
9 n mature infections and cotransmitted by the sandfly bite.
10 s that are recruited as a normal response to sandfly bites.
11 Leishmania infections specifically increased sandfly biting persistence and feeding on multiple hosts
12 e parasitic disease, is transmitted during a sandfly blood meal as the parasite is delivered into the
13                 As before, epidemics of this sandfly-borne disease occur periodically in India and el
14  and Uukuniemi groups) and from the mosquito/sandfly-borne phleboviruses.
15 rmal leishmaniasis were deemed infectious to sandflies by microscopy and 23 (88%) by qPCR.
16 maniasis, 42 (55%) were deemed infectious to sandflies by microscopy and 60 (78%) by qPCR before trea
17 proper management include inadequate vector (sandfly) control, no vaccine, and insufficient access to
18 rasites called Leishmania and transmitted by sandflies during blood meal.
19 hlebovirus genus, distinct from the existing sandfly fever and Uukuniemi virus groups, in the family
20              We show that the NSs protein of Sandfly Fever Sicilian virus (SFSV) allows the virus to
21  other phleboviruses-i.e., Punta Toro virus, sandfly fever Sicilian virus, or Frijoles virus-has no e
22 ts were exposed to 30-35 female P argentipes sandflies for 30 min.
23 te against the hydrolytic environment of the sandfly gut and/or the parasitophorous vacuole of host m
24 e is shaped by Leishmania development in the sandfly gut, described as a sequential differentiation o
25  infectious dogs (17%) accounted for >80% of sandfly infections.
26 V), which is transmitted by Phlebotomus spp. sandflies, is a major etiologic agent of aseptic meningi
27 s that it encounters during residence in the sandfly, its insect vector.
28  studies on salivary gland extracts from the sandfly Lutzomyia longipalpis identified SALO (salivary
29                             The phlebotomine sandfly, Lutzomyia longipalpis, a major vector of the Le
30 ain bacteria that are known to be members of sandfly midgut microbiota.
31 confined to life cycle stages present in the sandfly midgut.
32 e possibility that acoustic communication in sandflies might be more widespread than previously thoug
33 aniasis is only transmitted by blood-feeding sandflies, not through human remains.
34 ory-reared pathogen-free female P argentipes sandflies on their lower arms and legs.
35 een Leishmania parasites was demonstrated in sandflies over 10 years ago.
36 s of the enzyme that matched those seen in a sandfly pheromone extract.
37 i and transmitted by the bites of the vector sandfly Phlebotomus argentipes.
38 peat galectin expressed in the midgut of the sandfly Phlebotomus papatasi, is used by Leishmania majo
39 ciency virus (HIV) coinfection to the vector sandflies, Phlebotomus argentipes.
40 s, a parasitic disease transmitted by female sandflies, primarily affects impoverished populations in
41 ed to their mammalian hosts when an infected sandfly probes in the skin for a bloodmeal and injects t
42 s and transmitted through the bite of female sandflies, resulting in Leishmania replication in macrop
43  and aiding the parasite's attachment to the sandfly's hindgut.
44 report that a single molecule, maxadilan, in sandfly saliva can exacerbate infection with L. major to
45                                      Indeed, sandfly saliva markedly enhances the infectivity of L. m
46 se phleboviruses, namely, Rift Valley fever, sandfly Sicilian, and Toscana viruses.
47                   Accurate identification of sandfly species is critical for controlling and preventi
48  forms of Leishmania parasites adhere to the sandfly stomodeal valve, damaging this feeding valve and
49 thropods (for example, mosquitoes, ticks and sandflies) to humans and wild and domestic animals, with
50 t disease vectors (mosquitoes, tsetse flies, sandflies) to transmit parasites and disease.
51 a are kinetoplastid parasites that cause the sandfly-transmitted disease leishmaniasis.
52         Phlebotomus ariasi is one of the two sandflies transmitting the causative agent of zoonotic l
53 ully the passage of the parasite between its sandfly vector and different mammalian host species.
54 leishmaniasis can transmit L donovani to the sandfly vector and suggest that early diagnosis and trea
55  evaluate the relative infectiousness to the sandfly vector of patients with visceral leishmaniasis o
56 essed through time by xenodiagnosis with the sandfly vector, Lutzomyia longipalpis.
57 oa are introduced into a mammalian skin by a sandfly vector, whereupon they encounter increased tempe
58 . as promastigotes in the gut lumen of their sandfly vectors and as amastigotes in the phagolysosomal
59                                              Sandfly vectors transmit Leishmania through egestion of
60 ough the adaptive manipulation of one of its sandfly vectors, Lutzomyia longipalpis.
61 ptomatic individuals, who were infectious to sandflies, with a participant considered infectious if p