ライフサイエンスコーパス: scald

1 y genes associated with the response to leaf scald.

2 canopy can decrease fruit quality due to sun scalding.

3 mediately subjected to either sham or dorsal scald 28% total body surface area burn injury.

4 nflammation, as typified by murine models of scald and arthritis.

5 wenty-four different samples composed of ten scald and ten contact burns and four healthy samples, su

6 fluid balance were measured before and after scalding and resuscitation.

7 alty points given for adverse events such as scalds and breakability.

8 SCALD appears to be the mouse ortholog of the human prot

9 t mMCP-5 or human neutrophil elastase to the scalded area increases epidermal injury with subsequent

10 dase activity in vanilla beans compared with scalded beans.

11 eceived a 40% total body surface area (TBSA) scald burn and fluid resuscitation using the Parkland fo

12 d a 60% total body surface area third-degree scald burn and were randomly divided to receive either 4

13 d a 60% total body surface area third-degree scald burn and were randomly divided to receive either r

14 muscles of rats after a full-thickness skin scald burn covering 20% of the total body surface area,

15 A second-degree epidermal scald burn in mice elicits an inflammatory response medi

16 o sham or burn injury consisting of a dorsal scald burn injury covering 30% of total body surface are

17 very of myofibre area and volume following a scald burn injury in mice.

18 ein, we use our clinically relevant model of scald burn injury in young and aged mice to determine wh

19 Alcohol administered 30 minutes before a 15% scald burn injury.

20 Using a scald burn model in mice, we find that this part of the

21 1) and the recently updated Walker-Mason rat scald burn model, we demonstrated that Pseudomonas aerug

22 om male rats subjected to a 40% third-degree scald burn or sham burn.

23 A 20% full-thickness scald burn was used in C57BL/6 mice.

24 ) and adults (11 weeks-old), received either scald burn, or sham procedure.

25 show decreased injury after a second-degree scald burn, whereas mice lacking the MC-restricted trypt

26 d subjected to a 30% total body surface area scald burn.

27 ged in age from 3 to 18 years, with flame or scald burns covering more than 40% of their body surface

28 The scald caused an initial 30% reduction in cardiac output,

29 Leaf scald, caused by Xanthomonas albilineans, is a severe di

30 essing pV1 sensed heat normally but suffered scald damages in a hot environment.

31 n Xanthomonas albilineans, which causes leaf scald disease of sugarcane and is also pathogenic to cor

32 In testis, SCALD expression is restricted to pachytene spermatocyte

33 se two extremes - intense food-poor cold and scalding food-rich oases.

34 es a membrane-bound enzyme that we designate SCALD, for short-chain aldehyde reductase.

35 We expressed SCALD in bacteria, purified it extensively, and studied

36 notypes was evaluated for resistance to leaf scald in field conditions for 2 years, followed by a 1-y

37 line dextran) were infused 30 mins after the scald injury at a rate to restore and maintain the basel

38 Using a well-established murine scald injury model with BALB/c and BALB/c CD1d knockout

39 reased mortality was explored using a murine scald injury model.

40 mice indicated a significant reduction after scald injury whereas mMCP4(-/-) mice showed no significa

41 The initial baseline period was followed by scald injury, and three different treatment regimens wer

42 g a standardized rat model of full thickness scald injury, left ventricular pressures were recorded i

43 rs later given a ~ 12.5% TBSA full thickness scald injury.

44 SCALD is also expressed in four layers of the retina, in

45 Northern blot analysis indicates that SCALD is expressed most abundantly in mouse liver and te

46 In the liver of mice, SCALD is suppressed by fasting and induced by refeeding,

47 des in addition to retinals, we propose that SCALD may be induced by SREBP in liver and other tissues

48 of human MC chymase restores burn injury to scalded mMCP-4-deficient mice but not to mMCP-5-deficien

49 unds, were created by a standardized porcine scald model.

50 gested for delaying ripening and superficial scald of 'Bartlett' pears during the long-term cold stor

51 The superficial scald on pear peels was only observed on fruit coated by

52 days and 14 days after 50% body surface area scald or sham injury.

53 ent as a burn model with full-thickness skin scald over 30% total body surface area.

54 entified, significantly associated with leaf scald resistance.

55 owed some disruption of the epidermis at the scald site in the protected strains suggesting the possi

56 Staphylococcal scalded skin syndrome (SSSS) is an exfoliative dermatiti

57 an, and its generalized form, staphylococcal scalded skin syndrome (SSSS), is a frequent manifestatio

58 wn to be a causative agent of staphylococcal scalded skin syndrome (SSSS).

59 s (DRESS), eczema herpeticum, staphylococcal scalded skin syndrome and inherited ichthyosis.

60 oliative toxin A (ETA) causes staphylococcal scalded skin syndrome which is characterized by a specif

61 xfoliative toxins (ETs) cause staphylococcal scalded skin syndrome, a disease characterized by specif

62 pneumonia, burn infections, bacteremia, and scalded skin syndrome.

63 the symptoms associated with staphylococcal scalded skin syndrome.

64 Staphylococcal scalded-skin syndrome is an exfoliative dermatitis chara

65 diseases bullous impetigo and staphylococcal scalded-skin syndrome, pathogenesis depends on cleavage

66 ering in bulbous impetigo and staphylococcal scalded-skin syndrome.

67 d are the causative agents of staphylococcal scalded-skin syndrome.

68 y mixture efficacy against rhynchosporium or scald symptoms caused by the splash-dispersed pathogen R