ライフサイエンスコーパス: scalp

1 ally affects actinically damaged skin of the scalp.

2 en-dependent, progressive hair loss from the scalp.

3 by applying weak electrical currents at the scalp.

4 nes from balding (BAB) and non-balding (BAN) scalp.

5 ortices, and no correlation was found on the scalp.

6 for the treatment of AKs on the forehead and scalp.

7 which the sensors can be placed close to the scalp.

8 emoving MeS and BeS from hair and underlying scalp.

9 uman brain non-invasively through the intact scalp.

10 han cryotherapy for thin AKs on the face and scalp.

11 small, constant electric current through the scalp.

12 ew examining the decontamination of hair and scalp.

13 of actinic keratosis located on the face or scalp.

14 dicative of cortical brain activity from the scalp.

15 nset, predominantly over bilateral posterior scalp.

16 ouble-planar gradiometers covering the whole scalp.

17 proximate flow of direct current (dc) in the scalp.

18 -69 years) were 2-fold higher for SCC on the scalp (0.38 [95% CI, 0.00-0.81] vs 1.07 [95% CI, 0.75-1.

19 (10 upper limb, 10 uterus, 5 craniofacial, 1 scalp, 1 abdominal wall, and 1 penile).

20 on the skin (vitiligo) and bald spots on the scalp (AA), which significantly impact patients' lives b

21 s a novel, low-risk therapeutic approach for scalp alopecias.

22 l or "pilar" cysts are commonly found on the scalp and are derived from the outer root sheath of the

23 ata (AA) involving hair loss over the entire scalp and body and is often difficult to treat.

24 emia developed widespread alopecia involving scalp and body hair within weeks after starting nilotini

25 Lesions are often localized to the scalp and can result in permanent scarring, disfiguratio

26 via 4 transducer arrays placed on the shaved scalp and connected to a portable medical device.

27 n spatial extent and duration of IEDs at the scalp and cortical levels using HD-EEG source-localizati

28 ve tumor densities of KCs were higher on the scalp and ear in men compared with women, and on the upp

29 th stage T2 multilesion angiosarcomas of the scalp and face that are larger than 10 cm demonstrate a

30 These scalp and intracranial data hence show that power-law sc

31 ase and the EPILEPSIAE project that includes scalp and intracranial recordings.

32 rant focused ultrasound (FUS) through intact scalp and skull.

33 with major features of aplasia cutis of the scalp and terminal transverse limb defects.

34 recordings that enable coverage of the full scalp and the full circumference of the forearm.

35 Here, we report through-scalp and through-skull fluorescence imaging of mouse ce

36 n times to recurrence were 365 days (face or scalp) and 274 days (trunk or extremities).

37 blindness, and necrotic plaques on her face, scalp, and hands.

38 ety of the qualified anatomical sites (face, scalp, and upper extremities) twice daily for 4 consecut

39 ash involving the body, extremities, face or scalp, and/or funisitis, presenting in the first week (<

40 ension placed on the pericranial muscles and scalp aponeurosis secondary to the underlying cervical s

41 16(INK4a), suggesting that DPCs from balding scalp are more sensitive to environmental stress than no

42 well-demarcated hairless fatty nevus on the scalp, benign ocular tumors, and central nervous system

43 biomarkers that will eliminate the need for scalp biopsies.

44 boundary was indistinguishable in the young scalp but prominent in the scalp of those aged >40 years

45 ng suffices to capture neural signals on the scalp, but recent studies posit that increasing sensor d

46 ata to show that population responses on the scalp can capture choice-predictive activity that builds

47 Cutaneous scalp changes were present in 5 patients.

48 icolor, folliculitis, atopic dermatitis, and scalp conditions such as dandruff.

49 t-related potential (ERP) over the occipital scalp contralateral to the sound's location.

50 Participants were randomized to scalp cooling (n = 119) or control (n = 63).

51 m baseline to chemotherapy cycle 4 among the scalp cooling and control groups.

52 A positive association between scalp cooling and reduced risk of hair loss would be dem

53 To assess whether a scalp cooling device is effective at reducing chemothera

54 Scalp cooling was done using a scalp cooling device.

55 Although scalp cooling devices have been used to prevent this alo

56 t of mild headache and 3 (2.8%) discontinued scalp cooling due to feeling cold.

57 monstrated if 50% or more of patients in the scalp cooling group achieved treatment success, with the

58 nthracycline and taxane (106 patients in the scalp cooling group and 16 in the control group; 14 matc

59 No participants in the scalp cooling group received anthracyclines.

60 , 56.2%-75.4%) evaluable for alopecia in the scalp cooling group vs 0 of 16 patients (0%) in the cont

61 Of the 106 patients in the scalp cooling group, 4 (3.8%) experienced the adverse ev

62 1 month after the end of chemotherapy in the scalp cooling group.

63 To evaluate whether use of a scalp cooling system is associated with a lower amount o

64 Use of a scalp cooling system.

65 In previous studies of scalp cooling to prevent chemotherapy-induced alopecia,

66 py for early-stage breast cancer, the use of scalp cooling vs no scalp cooling was associated with le

67 reast cancer, the use of scalp cooling vs no scalp cooling was associated with less hair loss at 4 we

68 Scalp cooling was done using a scalp cooling device.

69 Scalp cooling was initiated 30 minutes prior to each che

70 anthracycline, or both, those who underwent scalp cooling were significantly more likely to have les

71 Of patients who underwent scalp cooling, 27.3% (95% CI, 18.0%-36.6%) reported feel

72 py cycle compared with those who received no scalp cooling.

73 characterized by congenital limb defects and scalp cutis aplasia.

74 Scalp data from these records were used to train a scalp

75 We propose that the limb and scalp defects might also be due to a vasculopathy in NOT

76 multiple scaly patches of alopecia underwent scalp dermoscopy, direct microscopic examinations, and m

77 mized, single-blind study, employing a split-scalp design, comparing the effectiveness and adverse ef

78 We investigated intracranial correlates of scalp-detected STW in 26 patients (14 women) undergoing

79 n D2 (PGD2), which is upregulated in balding scalp, differentially impacts on the proliferation of di

80 of oxidative stress on balding and occipital scalp DPCs.

81 of medical records as having been seen with scalp dysesthesia, 14 patients had cervical spine diseas

82 al spine disease may lead to the symptoms of scalp dysesthesia.

83 Scalp-ear-nipple (SEN) syndrome is a rare, autosomal-dom

84 entials in the subthalamic nucleus (STN) and scalp EEG (modified 10/20 montage) during sleep in human

85 Unilateral IEDs on scalp EEG (P = .001) and complete resection of brain reg

86 ere that KCs can be quasi-synchronous across scalp EEG and across much of the cortex using electrocor

87 Unilateral-only IEDs on preoperative scalp EEG and complete resection of IEDs on baseline ECo

88 We recorded scalp EEG and examined electrocortical dynamics time-loc

89 We visually marked STW segments in scalp EEG and selected stereo-EEG channels exhibiting no

90 High density scalp EEG and subdural ECoG recordings provide an opport

91 the ripple range have been identified in the scalp EEG as a promising non-invasive biomarker for epil

92 rties of interictal functional networks from scalp EEG can be estimated using a computer model and us

93 vity (ERN) is a well-established macroscopic scalp EEG correlate of error self-monitoring, but its ne

94 s over a 38 year age range (15-53 years) and scalp EEG data from healthy younger (20-30 years) and ol

95 als from two different datasets: the CHB-MIT Scalp EEG database and the EPILEPSIAE project that inclu

96 The computational results for a realistic scalp EEG database show a detection rate of 93.6% and a

97 (ECoG; subdural electrodes) and noninvasive scalp EEG during intensive care.

98 vapor and similar in size and flexibility to scalp EEG electrodes.

99 Applying this method to a dataset comprising scalp EEG from 38 people with epilepsy (17 with genetic

100 how that alpha-band activity, as measured by scalp EEG from human participants, varies with the speci

101 Elevated PAC is detectable using scalp EEG in PD patients off medications compared to on

102 ce with electrographic ictal onset zone from scalp EEG recording.

103 Previous research has suggested that human scalp EEG recordings contain signals that reflect the ne

104 We evaluated spike ripples in scalp EEG recordings from a prospective cohort of childr

105 In human scalp EEG recordings, both sustained potentials and alph

106 ts, the onset of which was synchronised with scalp EEG recordings.

107 Time-resolved multivariate decoding of scalp EEG signals first revealed that both information-s

108 Beta (15-25 Hz) scalp EEG signals recorded over the motor cortex during

109 tices shows increases in 2-4 Hz power during scalp EEG STW, that STW are associated with a strong and

110 Scalp EEG was the most useful test for identifying patie

111 The classifications of scalp EEG were various, including non-localisible, later

112 y of extracting electrocortical signals from scalp EEG while performing sustained, physically demandi

113 conductance, respiration, eye tracking, and scalp EEG).

114 observed in clinically standard, continuous scalp EEG, and underlying depolarizations can spread wid

115 Confirming results from scalp EEG, responses to audiovisual speech were weaker t

116 We used simultaneous interictal scalp EEG-fMRI to evaluate its value for predicting long

117 uestion using fMRI and concurrently acquired scalp EEG.

118 ts high spatial resolution compared with the scalp EEG.

119 We analysed clinical semiology, the scalp EEG/SEEG findings and cortico-cortical evoked pote

120 hemorrhage who underwent continuous surface (scalp) EEG (sEEG) recording and multimodality monitoring

121 We recorded scalp-EEG (electrophysiology) in healthy participants an

122 brain networks were estimated in segments of scalp-EEG without interictal discharges (68 people with

123 For scalp-EEG, surprising events engage the same independent

124 First, using scalp-EEG, we found that both outright action-stopping a

125 Scalp electric potentials corresponding to the magnetic

126 We recorded scalp electrical brain activity in 4-6 months infants vi

127 es in the clustering coefficient observed in scalp electrodes over parietal-occipital regions.

128 currents are applied to the human brain via scalp electrodes.

129 ; Experiment 2) while recording EEG over 128 scalp electrodes.

130 ntocentral slow oscillations recorded in the scalp electroencephalogram (EEG) during rapid eye moveme

131 ive value of HFOs for developing epilepsy in scalp electroencephalogram (EEG) of children after a fir

132 om rare simultaneous human intrathalamic and scalp electroencephalogram (EEG) recordings from eight v

133 e of gamma during fast stopping and recorded scalp electroencephalogram and local field potentials fr

134 focal mesial temporal lobe seizures based on scalp electroencephalogram coherence features, lends wei

135 of logistic regression classifiers that used scalp electroencephalogram coherence properties as input

136 ising from the mesial temporal lobe based on scalp electroencephalogram network connectivity measures

137 ing any obvious signs of seizure activity on scalp electroencephalogram.

138 recordings with foramen ovale electrodes and scalp electroencephalogram.

139 structures that contribute minimally to the scalp electroencephalogram.

140 on, routine clinical interpretation of these scalp electroencephalograms failed to identify any of th

141 CE STATEMENT Sawtooth waves (STW) present as scalp electroencephalographic (EEG) bursts of slow waves

142 Here, we used pattern similarity analysis to scalp electroencephalographic (EEG) recordings during a

143 Scalp electroencephalography (EEG) and intraoperative el

144 of Brain Computer Interfaces (BCI) based on scalp electroencephalography (EEG) have demonstrated the

145 rdings while accessing global networks using scalp electroencephalography (EEG) in rhesus macaques.

146 Beta frequency oscillations in scalp electroencephalography (EEG) recordings over the p

147 Here, we assessed cortical excitability from scalp electroencephalography (EEG) responses to transcra

148 ty (FRN) is a commonly observed potential in scalp electroencephalography (EEG) studies related to th

149 his hypothesis, we analyzed intracranial and scalp Electroencephalography sleep recordings from pre-s

150 rain circuit models, magnetoencephalography, scalp electroencephalography, and even invasive electroe

151 cipitoparietal sources readily detectable in scalp electroencephalography.

152 ime point spatial similarity patterns of the scalp electrophysiological (EEG) activity of 30 human pa

153 Here, we recorded scalp electrophysiological (EEG) brain responses in 5-y-

154 extracted through decoding analyses from the scalp electrophysiological signal (EEG) with high tempor

155 e human brain using various neuroimaging and scalp electrophysiological techniques.

156 vidence from a study employing pharmacology, scalp electrophysiology and computational modeling (N =

157 Erosive pustular dermatosis of the scalp (EPDS) is an inflammatory skin condition that deve

158 oped at flexural regions, genitalia, and the scalp, especially the psoriasiform lesions.

159 Here, we recorded high-density scalp event-related potentials (ERPs) while participants

160 anti-CD133 antibody treatment of human fetal scalp explants depresses beta-catenin and E-cadherin mem

161 laxation time T1 of brain tissue, blood, and scalp fat at B0 and Bp, and cerebrospinal fluid at B0.

162 on host status and body sites, including the scalp, feet, and groin.

163 concentrations, increased hair synthesis in scalp follicle organ culture and advanced mouse pelage h

164 les in vivo, mirroring eyelash behavior, and scalp follicles contain bimatoprost-sensitive prostamide

165 Thus, bimatoprost stimulates human scalp follicles in culture and rodent pelage follicles i

166 ysis identified 3 relevant receptor genes in scalp follicles in vivo.

167 n over 6,000 Latin Americans for features of scalp hair (shape, colour, greying, balding) and facial

168 rom oxidative stress/damage within the human scalp hair bulb.

169 Using our in vitro model of primary human scalp hair follicle melanocytes, we showed that ATM expr

170 Human scalp hair follicles (hHF) harbour several epithelial st

171 Finally, in organ-cultured human scalp hair follicles as well as in patients undergoing c

172 air follicle response to chemotherapy, human scalp hair follicles cultured ex vivo were treated with

173 et al. report that transplantation of human scalp hair follicles onto chemotherapy-treated immunodef

174 ct of the aging dermal environment on female scalp hair follicles remains unclear.

175 f2 in protecting human organ function (i.e., scalp hair follicles) against redox insult.

176 he epithelial stem cell (eSC) niche of human scalp hair follicles, during the inflammatory permanent

177 n a uniquely accessible human (mini-) organ: scalp hair follicles.

178 Human scalp hair has attractive features in clinical studies b

179 Amino acid profiles of scalp hair of 27 Jordanian subjects (15 diabetes mellitu

180 mino-acid-specific isotope ratio analysis of scalp hair of American individuals to predict soft biome

181 ial amino acid Ile were more abundant in the scalp hair of diabetic patients compared to the hair of

182 The scalp hair of each donor was washed, dried, homogenized

183 Incomplete scalp hair recovery was more frequent in the EC-T than E

184 Keratin protein is the major component of scalp hair shaft material and it is composed of 21 amino

185 These include novel loci for scalp hair shape and balding, and the first reported loc

186 EDAR370A has been associated with increased scalp hair thickness and changed tooth morphology in hum

187 Scalp hair was collected at baseline and week 20 for mea

188 urbance in childhood and had lifelong sparse scalp hair with normal facial hair.

189 data presented here, obtained directly from scalp hair, implies escalating coca and alcohol ingestio

190 e with central visual disturbance and sparse scalp hair.

191 strophies by the universally thin and sparse scalp hair.

192 tagen transformation of microdissected human scalp HFs can be observed in organ culture, it permits t

193 mis, we investigated in organ-cultured human scalp HFs whether TRH (30 nM), TSH (10 mU ml(-1)), thyro

194 Angiosarcoma of the head, neck, face and scalp (HNFS) was associated with a high tumor mutation b

195 ent of seborrheic dermatitis of the face and scalp in adults?

196 agen membrane discs were implanted under the scalp in diabetic (streptozotocin-induced) and control r

197 ggesting that deficiency of DPC from balding scalps in fostering vascularization around the hair foll

198 Greater scalp involvement (>25%) was associated with greater imm

199 produced by healthy hair follicles when the scalp is lightly pressed.

200 angiosarcoma (>/=20 cm) involving 80% of the scalp, left forehead, and left cheek, with no evidence o

201 radiological features of IPEH involving the scalp, localized on the left side of the skull and in th

202 The short latency and posterior scalp location of the effect suggest that perceptual his

203 ression (LMER) models at each time point and scalp location spanning a 3-s interval before, during, a

204 EG responses were found in a centro-parietal scalp location, whose slope depended on change size, con

205 ent predictors were ulceration, mitoses, and scalp location.

206 ctroencephalogram (EEG) was recorded from 19 scalp locations from 371 subjects ranging in age from 5

207 creases in sleep slow wave activity power at scalp locations matching their seizure focus.

208 -old girl presenting with a 6-cm protuberant scalp mass, which had doubled in size since birth.

209 he TMS coil over the prefrontal cortex using scalp measurements.

210 mplitude at frontal and central sites at the scalp midline.

211 sorder characterized by cutis aplasia of the scalp; minor anomalies of the external ears, digits, and

212 es showing that weak currents applied to the scalp modulate neural processing.

213 nstrate that a powerful magnet placed on the scalp modulates normal brain activity and induces behavi

214 , Optically Pumped MEG (OP-MEG) allows for a scalp mounted system that provides measurements within m

215 (n = 19), larynx (n = 2), penis (n = 1), and scalp (n = 1).

216 absent TILs]), and anatomic site other than scalp/neck (0.1 [0.01-0.6] for scalp/neck vs trunk/pelvi

217 te other than scalp/neck (0.1 [0.01-0.6] for scalp/neck vs trunk/pelvis), and BRAF+ melanoma was asso

218 We identified 25 scalp-negative mesial temporal lobe seizures in 10 patie

219 the detector raised seizure alarms, 80% had scalp-negative mesial temporal lobe seizures.

220 Our scalp-negative seizure detector has clear clinical utili

221 data from these records were used to train a scalp-negative seizure detector, which consisted of a pa

222 As such, little is known about scalp-negative seizures and their role in the natural hi

223 a novel approach to non-invasively identify scalp-negative seizures arising from the mesial temporal

224 formance, this detector correctly identified scalp-negative seizures in 40% of patients, and correctl

225 al, currently, the only way to detect these 'scalp-negative seizures' is with intracranial recordings

226 encephalograms failed to identify any of the scalp-negative seizures.

227 e human brain via electrodes attached to the scalp of a participant.

228 bly pigmented follicles from the aging human scalp of healthy individuals (22-70 years).

229 able in the young scalp but prominent in the scalp of those aged >40 years, accompanied by reduced po

230 ion of intracranial electrodes together with scalp ones increased the average accuracy and specificit

231 has remained unclear whether the infra-slow scalp potential fluctuations in full-band electroencepha

232 mparable effect occurred at the level of the scalp potential in humans.

233 The current study measured scalp potential when subjects played a series of fair co

234 vioural loss aversion can be predicted using scalp potentials (EEG) remains unclear.

235 ionship of infra-slow fluctuations (ISFs) in scalp potentials and BOLD signals.

236 sociation between article predictability and scalp potentials approximately 300 to 500 ms after artic

237 ecific association indicates that infra-slow scalp potentials are directly associated with the endoge

238 Interestingly, at time of choice, scalp potentials continue to appear parametrically modul

239 We further found that the difference in the scalp potentials from the above two conditions could pre

240 mics of fast (1-100 Hz) oscillations and the scalp potentials per se exhibit fluctuations in the same

241 etween prenominal article predictability and scalp potentials, we conducted a wide-ranging explorator

242 lated signals that manifest with similar EEG scalp projections.

243 Electrodes placed on the scalp record voltage potentials resulting from current f

244 and female humans (N = 234), we investigated scalp-recorded beta-band activity during the stop-signal

245 a initiative, we used source localization of scalp-recorded EEG resting data to examine the neural co

246 it unlikely to be a direct generator of the scalp-recorded EEG, these covariational patterns appear

247 Evidence suggests that scalp-recorded occipital alpha-band (8-13 Hz) oscillatio

248 onsolidation during sleep is correlated with scalp-recorded spindles and downstates/upstates, but HC-

249 eature-selective information processing with scalp recordings of electroencephalography (EEG).

250 using relatively inexpensive and noninvasive scalp recordings.

251 mulator signals, population responses on the scalp reflect the influence of other decision-related si

252 e between 20-150 ms over left fronto-central scalp region.

253 ty, as indicated by activation levels across scalp regions (frontal, frontocentral, temporal, centrop

254 ye movement sleep over widespread, bilateral scalp regions.

255 from the anagen hair bulbs of affected human scalp remains unclear, oxidative stress sensing appears

256 tion and larger abnormalities in nonlesional scalp samples (ie, CXCL10 and KRT85).

257 patients with AA for comparison with normal scalp samples (n = 6).

258 osis and hypotrichosis, was also detected in scalp samples from affected individuals.

259 profiling of 27 lesional and 17 nonlesional scalp samples from patients with AA for comparison with

260 hen compared to those with intracortical and scalp seizures (50% and 25% death or severe disability,

261 undersampling has on the effective SNR of on-scalp sensors.

262 headaches are caused by spasm or tension of scalp, shoulders, and neck muscles inserted in the occip

263 In summary, aging female scalp shows striking structural and biological changes i

264 ion that weak electric fields applied to the scalp significantly affect neural processing in the prim

265 Alpha power at widespread scalp sites decreased with increasing acoustic detail (d

266 acies of 70% at ~105 ms, especially based on scalp sites over visual cortex, dropping to lower levels

267 mined the P3 component over central-parietal scalp sites that was elicited by the test probe at the e

268 clinical human hair research purposes, human scalp skin can be xenografted onto immunocompromised mic

269 RNA sequencing of the scalp skin from IFAP-affected individuals revealed a dra

270 sed expression of PADI3 in biopsy samples of scalp skin obtained from patients with CCCA.

271 We conclude that scalp spike ripple events identify disease and track wit

272 The primary outcome was the change in scalp surface area with hair loss over 24 weeks followin

273 th alopecia areata affecting at least 10% of scalp surface area, were eligible; 1 declined to partici

274 By assessing scalp surface EEG in conjunction with LORETA source esti

275 vides measurements within millimetres of the scalp surface.

276 A question that arises in developing on-scalp systems is: how many sensors are necessary to achi

277 nutes prior to each chemotherapy cycle, with scalp temperature maintained at 3 degrees C (37 degrees

278 initial transient reports of mild neck pain, scalp tingling and headache that were extinguished upon

279 Transcriptomic analysis of affected scalp tissue highlights overrepresentation of transcript

280 on technique that applies mA currents at the scalp to modulate cortical excitability.

281 imulation site across subjects, (ii) reduced scalp-to-cortical-target distance, and (iii) reduced var

282 nier offset responses differed in both their scalp topography and temporal dynamics.

283 ontralateral delay activity component with a scalp topography over somatosensory cortex contralateral

284 We sought to define the AA scalp transcriptome and associated biomarkers with compa

285 minal wall transplants, 1 patient received a scalp transplant, and 1 patient received a penile transp

286 Patients had half of their scalp treated with daylight photodynamic therapy and the

287 Two samples, a tea tree oil-based scalp treatment and a white lavender body lotion, exhibi

288 ions of varying sizes were identified on the scalp, trunk, and extremities and were reported to have

289 cia with topical 5% Minoxidil application on scalp two times a day.

290 EEG and localized current sources across the scalp using a surface Laplacian transform.

291 high-frequency activity is detectable at the scalp using electroencephalography (EEG).

292 al (BP), that can be recorded over the human scalp using electroencephalography (EEG).

293 ence of aplasia cutis congenita (ACC) of the scalp vertex and terminal limb-reduction defects.

294 timulation (rTMS) to the right PPC or to the scalp vertex.

295 In vivo, tDCS was delivered to intact mice scalp via surface electrodes.

296 Hair loss on the scalp was noted in 120 (96%), arms and legs in 52 (42%),

297 Whole hair follicles isolated from human scalp were cryopreserved by a slow-rate cooling medium a

298 ient-matched DPCs from balding and occipital scalp were cultured at atmospheric (21%) or physiologica

299 wsiness, hair loss, nausea, and dry or itchy scalp were reported while patients were receiving WBRT,

300 expression in vivo in full thickness dermal scalp wounds created in experimental K14.Cre (+) .Foxo1