ライフサイエンスコーパス: sciatic nerve

1 xplaining pain along the distribution of the sciatic nerve.

2 econdary to compression or irritation of the sciatic nerve.

3 ecorded spike activity from the regenerating sciatic nerve.

4 lammatory macrophages, was upregulated in KO sciatic nerve.

5 in response to electrical stimulation of the sciatic nerve.

6 implant is sutured between the stumps of the sciatic nerve.

7 of circulating CX3CR1(+) monocytes into the sciatic nerve.

8 s following extraction of the Xenopus laevis sciatic nerve.

9 mes greater at the spinal column than at the sciatic nerve.

10 ronic constriction injury (CCI) model of the sciatic nerve.

11 nance of myelination and nodes of Ranvier in sciatic nerve.

12 , as occurs with conditioning lesions of the sciatic nerve.

13 of a polyethylene cuff placed around in the sciatic nerve.

14 he dorsal root ganglion (DRG) and the distal sciatic nerve.

15 ll proliferation is upregulated in Chd4-null sciatic nerve.

16 following chronic constriction injury of the sciatic nerve.

17 r after a chronic constriction injury of the sciatic nerve.

18 athologic changes in dorsal root ganglia and sciatic nerve.

19 s created by spared-nerve injury of the left sciatic nerve.

20 s recorded from isolated preparations of rat sciatic nerve.

21 les throughout the endoneurium of the mutant sciatic nerve.

22 ) meninges and in the epi/perineurium of the sciatic nerve.

23 olipid metabolites during myelination of the sciatic nerve.

24 -N-SH cells and on axonal transport in mouse sciatic nerves.

25 b weakness and impaired axonal conduction in sciatic nerves.

26 r roots, as well as in the femoral motor and sciatic nerves.

27 n in the conduction velocity along the adult sciatic nerves.

28 ssion of the T cell chemoattractant IP-10 in sciatic nerves.

29 sed numbers of large-diameter axons in their sciatic nerves.

30 doxycycline-treated compared with untreated sciatic nerves.

31 cell co-cultures and in vivo, in developing sciatic nerves.

32 epidermal nerve fibers and remyelination of sciatic nerves.

33 n the cerebrum, cerebellum, heart, liver and sciatic nerves.

34 Here, we report that sciatic nerve activation with electroacupuncture control

35 treatment also promoted remyelination of the sciatic nerve after crush.

36 A conditioning lesion of the sciatic nerve allows the central processes of dorsal roo

37 egeneration in dorsal root ganglia (DRG) and sciatic nerve and abundance of Schwann cells.

38 ere found in increased concentrations in the sciatic nerve and dorsal root ganglia of oxaliplatin tre

39 ve conduction velocity, nerve amplitude, and sciatic nerve and dorsal root ganglion morphology at 0.2

40 lso displayed a unique prolonged exposure in sciatic nerve and DRG.

41 ory components was evaluated in axons of the sciatic nerve and in spinal nerve axons after in vivo el

42 ssociated with a persistent M1 milieu in the sciatic nerve and in the regional and systemic lymphatic

43 having reliable signal acquisitions from the sciatic nerve and its branches such as the peroneal nerv

44 a small proportion of fibers that constitute sciatic nerve and its branches, but importantly breaks t

45 regeneration of growth-competent axons after sciatic nerve and spinal cord injury.

46 nction, intraepidermal nerve fiber loss, and sciatic nerve and spinal cord oxidative-nitrative stress

47 nduced by chronic constriction injury of the sciatic nerve and suppressed nerve injury-induced activa

48 e found an up-regulation of the HCAR2 in the sciatic nerve and the dorsal root ganglia in neuropathic

49 lly well in the lymph node, infection of the sciatic nerve and the rest of the nervous system was imp

50 which the first stimulus originated from the sciatic nerve and the second from the motor cortex.

51 which the first stimulus originated from the sciatic nerve and the second from the spinal cord; and (

52 ing required to prevent axonal damage of the sciatic nerve and to terminate the P0106-125-specific im

53 mmunohistological staining were performed in sciatic nerve and/or L4-L5 DRG tissue for galanin, CGRP

54 ceptive neurons, increased CGRP release from sciatic nerves and DRGs, and a reduction in mechanical a

55 8 days for sciatic function index (SFI), and sciatic nerves and hind limb muscles were harvested for

56 howed presence of myc-tagged human SH3TC2 in sciatic nerves and lumbar roots in the perinuclear cytop

57 se reduces NMN accumulation in injured mouse sciatic nerves and preserves some axons for up to three

58 one marrow uptake and intense uptake in both sciatic nerves and right median nerve.

59 amined pathology and cell differentiation in sciatic nerves and ventral roots of the laminin-alpha2-d

60 DTI-MRN covered proximal (sciatic nerve) and distal (tibial nerve) nerve segments

61 e neurons (CSNs) in DRGs contributing to the sciatic nerve, and a decrease in their cold temperature

62 al day 7 onward in motoneurons, axons in the sciatic nerve, and axon terminals of the neuromuscular j

63 assays, immunohistochemistry with teased rat sciatic nerve, and immunoabsorption experiments.

64 uff is well-tolerated, delivers light to the sciatic nerve, and optically stimulates muscle in freely

65 turation, and functional recovery of injured sciatic nerves, and increased the ability of regeneratin

66 In sciatic nerves, application of extracellular QX-314 with

67 We used a chronic constriction injury of sciatic nerve as a model of neuropathic pain in male Spr

68 tivity, and morphological alterations in the sciatic nerve as evidenced by decrease in g-ratio; it al

69 r3 knockdown attenuates myelination in mouse sciatic nerves as well as in zebrafish.

70 e points in CMT2D mouse muscles, retina, and sciatic nerve, as well as in embryonic hindbrain.

71 logy in isolated hearts, retinal tissues and sciatic nerves, as well as bioelectronic cardiac sensing

72 Analysis of the sciatic nerve at 11 d after nerve crush showed that the

73 cord, and, during their apoptosis induced by sciatic nerve avulsion, nuclear and cytoplasmic 5-methyl

74 otyping after selectively silencing TRPV1(+) sciatic nerve axons by perineural injections of QX-314 a

75 se, live imaging of endosomal trafficking in sciatic nerve axons reveals disease-induced deficits in

76 , degenerating) nerve stumps on day 1 in the sciatic nerve axotomy model in rats.

77 l enrichment (EE) followed by a conditioning sciatic nerve axotomy that precedes a spinal cord injury

78 We evaluated the utility and efficiency of sciatic nerve block as an alternative method to relieve

79 nts who received ultrasound-guided popliteal sciatic nerve block for the relief of severe rest pain d

80 Ultrasound-guided popliteal sciatic nerve block provides effective pain control, whi

81 reveal major structural changes at proximal sciatic nerve branches or distal toe nerve fascicles at

82 Two-site stimulation of sciatic nerve bundles by MOSD induces precise extension

83 r was generated and delivered into the mouse sciatic nerve by a single injection immediately distal t

84 Here, we validate EIT imaging in rat sciatic nerve by comparison to micro-computed tomography

85 Infiltration of cytotoxic NK cells into the sciatic nerve by extravasation occurs within 3 days foll

86 vivo chromatin immunoprecipitation (ChIP) of sciatic nerve cells revealed a Sox10 binding site upstre

87 ng techniques, we have used a rodent partial sciatic nerve chronic constriction injury model (n = 5-8

88 Sciatic nerve conditioning at A-delta fiber strength, kn

89 elinated nerve fibers, specifically sural or sciatic nerve conduction velocities, but significantly i

90 Furthermore, treated mice showed increased sciatic nerve conduction velocities, improvement of myel

91 rmance, quadriceps muscle contractility, and sciatic nerve conduction velocities.

92 athy remarkably ameliorated DPN by improving sciatic nerve conduction velocity and increasing thermal

93 mechanical hyperalgesia, cold allodynia, and sciatic nerve conduction velocity.

94 phagic flux in skeletal muscle 14 days after sciatic nerve constriction.

95 Electroacupuncture at the sciatic nerve controls systemic inflammation by inducing

96 irmed expression of Cx32 in lumbar roots and sciatic nerves correctly localized at the paranodal myel

97 O mice were subjected to neuropathic pain by sciatic nerve crash injury (SNI).

98 tion was impaired in CLU(-/-) mice following sciatic nerve crush and impaired regeneration nerve fibe

99 upregulated via MEK/ERK signaling and after sciatic nerve crush and Neto2(-/-) neurons from adult mi

100 nd conduction velocities consequent to acute sciatic nerve crush compared with wild-type control anim

101 Regeneration of axons was examined after sciatic nerve crush in pre- and symptomatic SOD1(G93A) m

102 kine IL-10 in terminating inflammation after sciatic nerve crush injury and promoting regeneration.

103 ution of inflammation and regeneration after sciatic nerve crush injury in mice.

104 Sciatic nerve crush injury in rats induced expression of

105 One day sciatic nerve crush injury triggered a robust increase i

106 Sciatic nerve crush injury triggers sterile inflammation

107 nd sensory recovery occurred in mice after a sciatic nerve crush injury, there was little return of m

108 in, retarded early axonal regeneration after sciatic nerve crush injury.

109 Male Sprague Dawley rats were subjected to a sciatic nerve crush under anesthesia and mechanical thre

110 Adult rats with sciatic nerve crush were immediately and systemically in

111 genic mice enhanced locomotor recovery after sciatic nerve crush, associated to an improvement in key

112 After sciatic nerve crush, functional recovery in vivo was ret

113 The resulting mice were challenged with sciatic nerve crush.

114 changes in wild-type and fat-1 mice after a sciatic nerve crush.

115 ation to augment neuroregeneration in both a sciatic nerve cut-and-repair and rat hindlimb transplant

116 Rats undergoing sciatic nerve cut-and-repair and treated with either loc

117 Sciatic nerve CuZnSOD content in SynTgSod1(-/-) mice was

118 liminary in vivo studies in a critical-sized sciatic nerve defect in Wistar rats confirmed conduit su

119 ponse to the loss of CCR2, injured Ccr2(-/-) sciatic nerves demonstrate prolonged expression of neutr

120 jected to unilateral partial ligation of the sciatic nerve developed significant mechanical and therm

121 TAB at a s.c. injection site remote from the sciatic nerve did not result in prolonged sensory-specif

122 erformed miRNA expression profiling of mouse sciatic nerve distal segment after crush injury.

123 after unilateral constriction injury to the sciatic nerve (DMG) and in the contralateral control leg

124 ncreased 24 h after oxaliplatin treatment in sciatic nerve, DRGs, or spinal cord tissue as revealed b

125 nt can be applied to measure excitability of sciatic nerves due to a stimulation of the footpad in co

126 d region (3'UTR) landscapes after unilateral sciatic nerve entrapment (SNE) injury in rats.

127 Sciatic nerve entrapment (SNE) injury was used to develo

128 ng in a rat neuropathy induced by unilateral sciatic nerve entrapment (SNE).

129 was injected directly into crush-injured rat sciatic nerves, ERK1/2 phosphorylation was observed in m

130 Both autoradiography analysis of sciatic nerves excised from injured rats as well as whol

131 ties vanished, and the ultrastructure of the sciatic nerve exhibited numerous tomacula and remyelinat

132 muscle with and was seen tracking along the sciatic nerve, explaining pain along the distribution of

133 l proteins at the nodes of Ranvier of teased sciatic nerve fibers.

134 ilized to assess the morphology of optic and sciatic nerves following treatment, and the morphology a

135 blished model of complete transection of the sciatic nerve for comparison, we observed similar but mo

136 ated a mouse model in which a portion of the sciatic nerve from one hind limb was transected at postn

137 and transmission electron microscopy of the sciatic nerve from one of the affected individuals revea

138 electron microscopy, we show that injury of sciatic nerves from mice of either sex triggers extensiv

139 f gene expression arrays of large myelinated sciatic nerves from pioglitazone-treated animals reveale

140 te was as effective as glucose in supporting sciatic nerve function, and was continuously released in

141 en assessed using von Frey filaments and the sciatic nerve functional index.

142 no significant long-term adverse effects on sciatic nerve functions.

143 supporting the re-innervation across a 10 mm sciatic nerve gap, with results close to that of the aut

144 through the loss of Limk1, results in faster sciatic nerve growth, and improved recovery of some sens

145 Single-cell RNA-sequencing of injured sciatic nerve identifies five macrophage subpopulations,

146 ting diodes embedded in a soft, circumneural sciatic nerve implant, powered and driven by a miniaturi

147 Ultrastructural analysis of adult sciatic nerve in GluN1- mice revealed increases in the d

148 ed low-voltage electrical stimulation of the sciatic nerve in live mice.

149 ord in females, but remains localized to the sciatic nerve in males (tier 3).

150 When injected proximal to the sciatic nerve in mice via intramuscular (i.m.) injection

151 by chronic constriction injury (CCI) of the sciatic nerve in mice, was related to both an increase i

152 eas western blotting detected PrP(Sc) in the sciatic nerve in one VV2 and one MV2K.

153 Transection of the sciatic nerve in the M83 Tg mice significantly delayed t

154 P was expressed throughout the length of the sciatic nerve in up to 50% of Schwann cells starting 2 w

155 Uninjured sciatic nerves in GluN1- mice did not demonstrate an inc

156 In response to crush injury, sciatic nerves in scLRP1(-/-) mice showed accelerated de

157 more than 100 O-GlcNAcylated proteins in rat sciatic nerve, including Periaxin (PRX), a myelin protei

158 Chronic constriction injury (CCI) of the sciatic nerve induced IL-33 production in the spinal cor

159 uency (0.2 or 3 Hz) stimulation (LFS) to the sciatic nerve induced long-term potentiation (LTP) of C-

160 Surprisingly, sciatic nerve-infiltrating CD4(+) cells had an expansion

161 ory neurons following direct intraganglionic sciatic nerve injection and intraperitoneal and intraven

162 orn would be altered by chronic constriction sciatic nerve injury (CCI).

163 peralgesia using a rat model of constriction sciatic nerve injury (CCI).

164 ys a critical role in neuropathic pain after sciatic nerve injury and bone cancer in rodents.

165 the mediodorsal thalamus (MD) to ACC, using sciatic nerve injury and chemotherapy-induced mouse mode

166 action against ER stress, in mouse models of sciatic nerve injury and found that ablation of the tran

167 pes in vitro and ameliorate a critical-sized sciatic nerve injury and its associated defects in a mur

168 of dorsal root ganglia (DRGs) neurons after sciatic nerve injury in the rat.

169 sures of allodynia in a chronic, neuropathic sciatic nerve injury model, but tolerance to morphine de

170 were protected from hypersensitivity in two sciatic nerve injury models.

171 Sciatic nerve injury triggered generation of two-chain S

172 tage-dependent anion channel 1 (VDAC1) after sciatic nerve injury triggers Schwann cell demyelination

173 Hyperalgesia gradually developed after sciatic nerve injury, and by the last day of testing, TH

174 Within hours of a sciatic nerve injury, the level of phosphorylated cofili

175 crease in ultrasonic, broadband clicks after sciatic nerve injury, which was reversed by THC, CBD, an

176 r mechanical hypersensitivity, or in partial sciatic nerve injury-induced mechanical allodynia.

177 ing the same population of neurons following sciatic nerve inoculation.

178 Glycogen was present in sciatic nerve, its concentration varying directly with a

179 Here, we show that peripheral sciatic nerve lesion in adult mice leads to elevated lev

180 was to evaluate the functional relevance of sciatic nerve lesions in DPN, with the expectation of co

181 tic polyneuropathy (DPN) have found proximal sciatic nerve lesions.

182 e noxious stimuli, but subsequent to partial sciatic nerve ligation (PNL), KOs did not develop mechan

183 nitiation of neuropathic pain in the partial sciatic nerve ligation (PSNL) mouse model.

184 null mutant mice are protected from partial sciatic nerve ligation (PSNL)-induced neuropathic pain,

185 of behavioral hypersensitivity after partial sciatic nerve ligation (PSNL).

186 mplete Freund's adjuvant (CFA) or by partial sciatic nerve ligation (PSNL).

187 Neuropathic pain was induced by partial sciatic nerve ligation (which induces hypersensitivity t

188 mycin treatment of human SMA fibroblasts and sciatic nerve ligation in SMA mice provoked robust phosp

189 lations of murine models, we have shown that sciatic nerve ligation induces a re-emergence of immatur

190 tive properties in the capsaicin and partial sciatic nerve ligation models in mice.

191 R-neutralizing antibody to rats with partial sciatic nerve ligation resulted in a delayed onset of ne

192 We found that in mice with partial sciatic nerve ligation, TRPA1 silencing in nociceptors a

193 anscriptomic and spatial characterization of sciatic nerve macrophages (snMacs).

194 Ki 60 nM) and also completely reversed mouse sciatic nerve mechanoallodynia (in vivo, 3 mumol/kg, po)

195 in vitro findings in vivo using a transected sciatic nerve model.

196 We demonstrate that in sciatic nerve, myocilin is expressed in Schwann cells wi

197 In mouse sciatic nerve, myosin-1d is expressed along the axon and

198 cifically colocalize with SMIT1 and SMIT2 at sciatic nerve nodes of Ranvier and in axon initial segme

199 ed into partially denervated branches of the sciatic nerve of adult mice.

200 mary motoneurons in vitro and in vivo in the sciatic nerve of adult WT and mutant SBMA mice demonstra

201 The sciatic nerve of Ahr knockout mice exhibited both reduce

202 in vivo during the first postnatal week, the sciatic nerve of all 3 conditional KO animals displayed

203 cuff-mounted and acutely implanted onto the sciatic nerve of anaesthetized rats, the device conferre

204 pathic pain was induced by cuffing the right sciatic nerve of C57BL/6J mice.

205 tudied mitochondrial transport in the intact sciatic nerve of living mice and analyzed axonal mitocho

206 ice after chronic constriction injury of the sciatic nerve of the left hindpaw and observed a strikin

207 ticularly in the spinal cord, but not in the sciatic nerve of the PNS.

208 nodal expression of selected isoforms in the sciatic nerve of the transgenic mouse Oct6(DeltaSCE/beta

209 sociated protein tau, in the spinal cord and sciatic nerve of wild-type (WT) and SBMA mice at various

210 ssion was upregulated in the spinal cord and sciatic nerve of WT mice.

211 elow the incisures in the single mutants, in sciatic nerves of caspr(-/-)/caspr2(-/-) mice, these cha

212 oid precursor protein accumulated in ligated sciatic nerves of control and eribulin-treated mice, but

213 s found to be carbonylated and aggregated in sciatic nerves of dbdb mice.

214 We found that, compared with wild-type, sciatic nerves of Lama2(-/-) mice had a significant incr

215 Although the conduction velocity of sciatic nerves of mutant mice showed an 80% decrease, th

216 Sciatic nerves of myocilin null mice express reduced lev

217 Mtmr13 loss leads to axonal degeneration in sciatic nerves of older mice.

218 Electron microscopy of sciatic nerves of paclitaxel-treated mice showed reduced

219 Sciatic nerves of such mice showed thinner myelin of lar

220 s of demyelinated fibres in lumbar roots and sciatic nerves of treated Sh3tc2-/- mice.

221 were performed in dorsal root ganglia after sciatic nerve or dorsal column axotomy.

222 Like acute hypoxia, stimulation of the sciatic nerve or the nasal mucosa evoked greater increas

223 ic with streptozotocin displayed less severe sciatic nerve oxidative-nitrative stress and peripheral

224 n, whereas wild-type mice developed complete sciatic nerve paralysis due to massive CPNI.

225 ice, and no evidence of CGRP upregulation in sciatic nerve post-CCI was found.

226 d genes in non-neuronal cells located in the sciatic nerve (potentially representing Schwann cells (S

227 Transection of the sciatic nerve prior to hind-limb inoculation diminished

228 eported that myelin clearance in the injured sciatic nerve proceeds unhindered in the Ccr2(-/-) mouse

229 tion of 6 mul of 150 mum ATP into female rat sciatic nerve quadrupled the number of axons growing int

230 ined GSK3 activity show markedly accelerated sciatic nerve regeneration.

231 e (M6P), a scar reducing agent, to a site of sciatic nerve repair.

232 ize independently after complete and partial sciatic nerve resection, respectively.

233 ieved after pelvic exenteration with en bloc sciatic nerve resection.

234 roduced by placing a plastic cuff around the sciatic nerve resolved within several days when the cuff

235 ring rate and stimulus-evoked (brush, pinch, sciatic nerve) responses were markedly enhanced as were

236 Molecular interrogation of the sciatic nerve reveals that aged Schwann cells (SCs) fail

237 hsTNT showed a negative correlation with the sciatic nerve's FA (r = -0.52, P < 0.001), with a closer

238 The sciatic nerve's fractional anisotropy (FA), a marker of

239 ion in the leg along the distribution of the sciatic nerve, secondary to compression or irritation of

240 iation of pain along the distribution of the sciatic nerve should always be looked for if abnormaliti

241 t this time, ultrastructural analysis of the sciatic nerve showed de- and dysmyelination of fibers, w

242 Electron microscopic analysis of sciatic nerves showed a reduction in the number of neuro

243 rsisted in the dorsal root ganglia (DRG) and sciatic nerve (SN) for up to 72 hours.

244 The sciatic nerve (SN) showed increased nonuniform, internod

245 egrated proteomics and metabolomics from the sciatic nerve (SN), the lumbar 4/5 dorsal root ganglia (

246 ) direct current flowing from spinal cord to sciatic nerve [spinal-to-sciatic direct current stimulat

247 n response was identified in neural tissues (sciatic nerve, spinal cord) of streptozotocin diabetic r

248 sthetised, ventilated dogs were elicited via sciatic nerve stimulation (50 Hz; 200 ms duration; 1 con

249 TP) following conditioning by high-frequency sciatic nerve stimulation (HFS) at intensities recruitin

250 oration of direct muscle responses evoked by sciatic nerve stimulation to pretransection levels over

251 ns (50 Hz; 200 ms duration) via supramaximal sciatic nerve stimulation were used to manipulate metabo

252 This study investigated the association of sciatic nerve structural damage in 3 Tesla (3T) magnetic

253 -positive vesicular clusters in axons in the sciatic nerve suggest that this denervation results from

254 Four weeks after axonal crush of sciatic nerve, TDP-43 transgenic mice remained paralyzed

255 The infection spread to the sciatic nerve, the spinal cord, and the brain, causing p

256 K3-mediated CRMP2 inhibition was detected in sciatic nerves, thus revealing a fundamental difference

257 titative RT-PCR and Western blot analysis of sciatic nerve tissues showed that SC-Exo treatment rever

258 thanized, and blood, urine, spinal cord, and sciatic nerve tissues were collected.

259 Local lidocaine was applied to the left sciatic nerve to block both orthodromic signals and anti

260 and its subsequent retrograde transport via sciatic nerve to DRG.

261 rve injury, we demonstrate that exposing the sciatic nerve to MBP(84-104) via endoneurial injection p

262 ent unilateral electrical stimulation of the sciatic nerve to mimic resistance exercise in the tibial

263 glycoprotein gp120 application onto the rat sciatic nerve to test the role of phosphorylated C/EBPbe

264 ontrol and injury conditions at 3 days after sciatic nerve transection (SNT).

265 d time course in a more severe injury model, sciatic nerve transection and reanastamosis.

266 Sciatic nerve transection induced upregulation of OPN an

267 ttern nor the number of neurons changed in a sciatic nerve transection model of neuropathic pain or i

268 as applied to bridge injured nerves in a rat sciatic nerve transection model.

269 ided into 5 groups, including naive control, sciatic nerve transection or repair, immediate transplan

270 As expected, sciatic nerve transection triggered WGA expression in NP

271 re was little return of motor function after sciatic nerve transection, because of the delay in motor

272 pacity to disseminate to the brain following sciatic nerve transection, indicating that wild-type reo

273 randomly assigned to four different types of sciatic nerve transection: Simple Transection (ST), Simp

274 Sciatic nerve transections and repairs were performed in

275 ) via electrical stimulation (e-stim) of the sciatic nerve twice a week for 8 weeks.

276 ication of its target mRNAs in vivo in mouse sciatic nerves using ribonomics showed an enrichment of

277 After CCI, upregulation of CD11b in sciatic nerve was less in GFAP-IkappaBalpha-dn mice comp

278 hronic constriction injury (CCI) of the left sciatic nerve was performed on wild type (WT) and GFAP-I

279 following chronic constriction injury of the sciatic nerve was rapidly and dose-dependently reversed

280 In anaesthetised C57-Black-6 mice, the left sciatic nerve was sectioned and repaired using 4 epineur

281 After control data collection, the sciatic nerve was transected and repaired and the rat wa

282 appeared that myelin sheath thickness in the sciatic nerves was not increased in BACE1(-/-)/Akt-DD mi

283 ted tomography (CT)-guided IRE of 11 porcine sciatic nerves was performed in nine pigs, and histopath

284 In addition, acute RFA of six porcine sciatic nerves was performed in six pigs that were harve

285 tokes Raman scattering (CARS) imaging of the sciatic nerve, we deciphered the spatiotemporal choreogr

286 of perineural HIV gp120 application onto the sciatic nerve, we found that pC/EBPbeta was triggered by

287 ata for 24-week-old BKS db/db and db/+ mouse sciatic nerve were analyzed to define significantly diff

288 conduction velocities of the trigeminal and sciatic nerves were decreased.

289 he cholesterol and sphingomyelin contents of sciatic nerves were greatly reduced and so was the numbe

290 as confirmed by histological analysis of the sciatic nerve which showed predominantly axonal damage:

291 l neurons and peripheral nerves, such as the sciatic nerve, which have provided most of our informati

292 ort the concept that HFS conditioning of the sciatic nerve, which leads to spinal LTP, is associated

293 r perianal abscess causing irritation of the sciatic nerve, which was diagnosed on MRI of the lumbosa

294 we observed galanin upregulation in DRG and sciatic nerve, which was less in GFAP-IkappaBalpha-dn mi

295 ignals were recorded simultaneously from the sciatic nerve with a 16-contact cuff electrode.

296 icipants underwent 3-T MR neurography of the sciatic nerve with a T2-weighed fat-suppressed sequence,

297 estigate this, we inoculated hamsters in the sciatic nerve with long-incubation-period strain 139H pr

298 l of strain interference, inoculation of the sciatic nerve with the drowsy (DY) strain of the transmi

299 ster of differentiation 45 expression in the sciatic nerve, with no difference between genotypes.

300 We cared for a child with sciatic nerve zoster who had severe pain over the lower