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1 ressed in freshly dispersed than in cultured sebocytes.
2 d synthesis in primary and transformed human sebocytes.
3 esteryl esters, and wax esters in OG-treated sebocytes.
4 will drive inflammatory gene expression from sebocytes.
5 y of antimicrobial peptide activity in human sebocytes.
6 AMP)-mediated innate immune defense of human sebocytes.
7 ic acid did not induce cytotoxicity to human sebocytes.
8 1, and stearoyl-CoA desaturase mRNAs in SZ95 sebocytes.
9 pression, in turn, stimulates lipogenesis in sebocytes.
10 and time-dependent decreases in viable SEB-1 sebocytes.
11 amma activation and COX-2 expression in SZ95 sebocytes.
12 can influence cytokine expression from human sebocytes.
13 ncreased PPARgamma reporter activity in SZ95 sebocytes.
14 entiation into interfollicular epidermis and sebocytes.
15 stimulates proliferation of undifferentiated sebocytes.
16 uman facial skin, human sebocytes, and SEB-1 sebocytes.
17 n the hair follicle infundibulum, and in the sebocytes.
18 matory changes were associated with abnormal sebocyte accumulation of lipid, defective sebum delivery
19 follicles into interfollicular epidermis and sebocytes and causes loss of LRC primarily through proli
20 ure, plus the active migration of individual sebocytes and cohorts of sebocytes, were measured.
21                      It is also expressed by sebocytes and contributes to the proliferation of this c
22 high extent before and after culture in both sebocytes and epidermal cells.
23 wed a subnormal level of FATP4 expression in sebocytes and exhibited abnormal development of both seb
24 ion by pilosebaceous epithelial cells (i.e., sebocytes and follicular keratinocytes) has been propose
25 y suppressing the NF-kappaB pathway in SEB-1 sebocytes and HaCaT keratinocytes.
26                                   Studies in sebocytes and human sebaceous glands indicate that agoni
27      Administration of CBD to cultured human sebocytes and human skin organ culture inhibited the lip
28 stem cell compartment and differentiate into sebocytes and interfollicular epidermis at the expense o
29 ndritic cells was opposite to this action on sebocytes and keratinocytes and did not correlate with t
30 and IL-13 drive lipid abnormalities in human sebocytes and keratinocytes through HSD3B1.
31 d cell cycle proteins were examined in SEB-1 sebocytes and keratinocytes.
32 P450c17, and steroidogenic factor 1 in SEB-1 sebocytes and sebaceous glands was compared to mRNA leve
33  is expressed by epidermal keratinocytes and sebocytes and serves as an antimicrobial protein that is
34 olism and one gene, PLIN2, expressed only by sebocytes and subcutaneous fat cells.
35 us glands IHH is expressed in differentiated sebocytes and the transcription factor GLI1 is activated
36 nd activity of PPARs in human skin and SEB-1 sebocytes and to assess the effects of PPAR ligands on s
37             Deleting K79 destabilizes K14 in sebocytes, and attenuates SGs and eyelid meibomian gland
38 ivated receptor-gamma, which is expressed in sebocytes, and contribute to secretory differentiation i
39       HSD3B1 enhances androgen production in sebocytes, and IL-4 and IL-13 drive lipid abnormalities
40 rmis in human skin, rat skin, cultured human sebocytes, and rat preputial cells.
41 otein-1 (SREBP-1) signaling pathway in SEB-1 sebocytes, and reduced inflammation by suppressing the N
42 1 was documented in human facial skin, human sebocytes, and SEB-1 sebocytes.
43 uclei and resistin in the cytoplasm of basal sebocytes, and stearoyl CoA desaturase in the cytoplasm
44 lti-modal lipid profiling, keratinocytes and sebocytes are implicated in lipid changes, which correla
45                                       Mature sebocytes are lost upon specific inhibition of the Jag2
46 he hair follicle-associated sebaceous gland, sebocytes are specialized lipid-producing cells that can
47 l peptides showed an age-related decrease in sebocyte area and increases in natural moisturizing fact
48                                  Analysis of sebocyte area, natural moisturizing factors, lipids, and
49 rt that 13-cis RA induces apoptosis in SEB-1 sebocytes as shown by increased Annexin V-FITC staining,
50 fense by inducing the expression of hBD-2 in sebocytes as well.
51 ubset of differentiated keratinocytes called sebocytes, as demonstrated by Northern blot analysis, in
52       These molecules are shown to influence sebocyte behavior through two distinct mechanisms: the i
53  fibrate, induced lipid droplet formation in sebocytes but not epidermal cells.
54 ted receptor gammal mRNA was demonstrated in sebocytes, but not in epidermal cells; it was more stron
55  cycle arrest and induces apoptosis in SEB-1 sebocytes by a RAR-independent mechanism, which contribu
56                         We hypothesized that sebocyte cell culture systems lack activators of the per
57 he Hedgehog pathway thus plays a key role in sebocyte cell fate decisions and is a potential target f
58 is and immunofluorescence of an immortalized sebocyte cell line (SZ95) revealed the presence of hista
59 uce lipogenesis and proliferation of a human sebocyte cell line in vitro.
60             Galectin-12 knockdown in a human sebocyte cell line reduced lipogenesis and decreased the
61 gene is induced by TLR2 signaling in a human sebocyte cell line.
62                            Cooling disrupted sebocyte cell membranes, alkaline phosphatase activity,
63 performance liquid chromatography, untreated sebocytes contained 6.27 (+/-0.73) nmol squalene per 10(
64 athways of differentiation in adipocytes and sebocytes could be similar and therefore further underst
65 le by western blotting in the supernatant of sebocyte culture incubated with each FFA, but not with a
66             The supernatant of FFA-incubated sebocyte culture showed antimicrobial activity against P
67  show the presence of histamine receptors on sebocytes, demonstrate how an antagonist to these recept
68 pidermis; however, the signals that regulate sebocyte development are poorly understood.
69  the Hedgehog pathway selectively suppressed sebocyte development, Hedgehog pathway activation led to
70 aceous glands expressed molecular markers of sebocyte differentiation and were functional, secreting
71 1 signaling axis as the primary regulator of sebocyte differentiation in mouse homeostatic skin.
72  PTCH1 and IHH are up-regulated during human sebocyte differentiation in vitro and inhibition of hedg
73                                  Very little sebocyte differentiation occurs, however, in primary or
74                          Departing from four sebocyte differentiation stages generated by unsupervise
75                                      Ectopic sebocyte differentiation was another hallmark of the phe
76  initial wave of sebaceous gland duplication sebocyte differentiation was inhibited.
77  As lipogenesis is key to both adipocyte and sebocyte differentiation we hypothesize that sebocytes f
78        Thus, Notch activity promotes correct sebocyte differentiation, and is required to restrict pr
79 mis Blimp1 is important for keratinocyte and sebocyte differentiation, its role in dermal fibroblasts
80 ltiple aspects of skin physiology, including sebocyte differentiation, keratinocyte proliferation, ep
81 esses hair differentiation and gives rise to sebocyte differentiation.
82 a distinct role of long chain fatty acids in sebocyte differentiation.
83 sociation between galectin-12 expression and sebocyte differentiation.
84   The hallmark of sebaceous epithelial cell (sebocyte) differentiation is the accumulation of fused n
85 palmitic acid, or oleic acid (OA) with human sebocytes dramatically enhanced their expression of huma
86                        We found that SGs and sebocytes expressed IL-4 receptor and produced high leve
87 sebocyte differentiation we hypothesize that sebocytes follow a similar program of differentiation to
88 y, antihistamines could potentially modulate sebocyte function directly.
89                                              Sebocyte function was analyzed by histological analysis
90 tween mitochondrial dysfunction and abnormal sebocyte function within sebaceous and modified SGs thro
91     When overexpressed in immortalised human sebocytes, GATA6 triggers a junctional zone and sebaceou
92  ribosomal RNA gene sequencing, whereas host sebocyte gland area, skin lipids, natural moisturizing f
93 ys are involved in the specific processes of sebocyte growth and development.
94  the roles of specific retinoid receptors in sebocyte growth and differentiation, by testing the effe
95 etinoid X receptors differ in their roles in sebocyte growth and differentiation: (i) retinoic acid r
96 xpressed TNF receptor ligands, which limited sebocyte growth by repressing Notch signaling pathway.
97 ow that deletion of Acbp in mouse results in sebocyte hyperplasia and sparse, matted hair with a grea
98 ected MPZL3 protein in the keratinocytes and sebocytes in the skin.
99 sphatase activity, and significantly reduced sebocyte lipid content.
100 ose and lipid metabolism, thereby inhibiting sebocyte lipogenesis.
101 uced transcription of SREBP target genes and sebocyte lipogenesis.
102  expression is restricted to differentiating sebocytes located in the suprabasal layers of the sebace
103 rget genes but also stimulated expression of sebocyte markers, suggesting that it may determine the d
104 tivation and long chain fatty acids finalize sebocyte maturation and are capable of stimulating epide
105 .1 mM stimulated significantly more advanced sebocyte maturation than any other treatment, including
106 It modulates the immunological repertoire of sebocytes, notably by upregulating PD-L1 and IL10.
107 turase in the cytoplasm of basal and luminal sebocytes of human scalp skin.
108 sheath and hair shaft and in the most mature sebocytes of the sebaceous gland and preputial, meibomiu
109 ages whereas IHH stimulates proliferation of sebocyte precursors.
110 erentiation of the sebaceous gland, with the sebocytes producing little or no sebum and undergoing ab
111 d hyperplasia resulted from expansion of the sebocyte-producing zone in sebaceous glands, with partic
112 he transcription factor GLI1 is activated in sebocyte progenitors, suggesting a paracrine signaling m
113                                    Increased sebocyte proliferation and aberrant expression of stem c
114 noleic acid and testosterone, and suppressed sebocyte proliferation via the activation of transient r
115        Depletion of HDAC8 and HDAC9 in human sebocytes resulted in an enhanced cytokine response to T
116  (termed 4.1) possesses activity to suppress sebocyte-specific expression and induce expression in th
117  sebocytes, we established a mouse line with sebocyte-specific expression of Cre recombinase.
118 s failed to elicit a PGE(2) response in SZ95 sebocytes stably expressing a dominant-negative PPARgamm
119 d receptor-gamma transcriptional activity in sebocytes stimulated with fatty acids.
120   Cooling produced characteristic changes in sebocyte structure and migration.
121 on and lipid metabolism, their expression in sebocytes suggests they may also play a similar role in
122 on of proinflammatory cytokine IL-8 in human sebocyte SZ95 cells.
123 lectin-12 is a lipogenic factor expressed in sebocytes that affects their differentiation and prolife
124 , the progressive transcriptional changes of sebocytes that lead to sebum production have never been
125 , lipogenesis assays were performed in SEB-1 sebocytes that were treated with PPAR ligands and isotre
126 amines were investigated for their effect on sebocytes, the major cell of the sebaceous gland respons
127 that IGF-1 transmits its lipogenic signal in sebocytes through activation of Akt.
128 in, further characterize the contribution of sebocytes to epidermal immunity, and demonstrate how cha
129 CL26 upregulation by IL-4 was reversed after sebocyte treatment with inducers of endoplasmic reticulu
130 450scc and P450c17 was demonstrated in SEB-1 sebocytes using radioimmunoassay.
131  histone H4 in the antimicrobial activity of sebocytes was confirmed by a specific neutralizing antib
132 f galectin-12, resistin, and SREBP-1 in SZ95 sebocytes was confirmed by Western blot analysis.
133             The sebaceous phenotype of SEB-1 sebocytes was confirmed using immunohistochemistry, Oil
134 the enhanced antimicrobial activity of human sebocytes was neutralized by anti-hBD-2 IgG.
135 d3, which is expressed exclusively in mature sebocytes, we established a mouse line with sebocyte-spe
136      Although not previously demonstrated in sebocytes, we report that 13-cis RA induces apoptosis in
137                  Using galectin-12-knockdown sebocytes, we showed that galectin-12 regulated the immu
138 lar infundibulum into sebaceous glands where sebocytes were found positive for the virus.
139                                         When sebocytes were incubated with an H-1 receptor antagonist
140       Acid-soluble protein extracts of SEB-1 sebocytes were separated by reverse-phase high-performan
141                                              Sebocytes were treated with IGF-1 and assayed for activa
142 ation of individual sebocytes and cohorts of sebocytes, were measured.
143                                              Sebocytes, which are characterized by lipid accumulation
144  upregulate the expression of hBD-2 in human sebocytes, which may enhance the disinfecting activity o
145 ecause genetic tools that allow targeting of sebocytes while maintaining intact epidermal lipids are
146 lays a unique role in the differentiation of sebocytes, while peroxisome proliferator-activated recep
147 )36 fatty acid translocase on the surface of sebocytes with anti-human CD36 IgG or blocking the NF-ka
148 depleting LRC and induces differentiation of sebocytes within the interfollicular epidermis.

 
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