ライフサイエンスコーパス: seborrheic

1 tic papules and plaques that occur mainly in seborrheic areas.

2 Dandruff and seborrheic dermatitis (D/SD) are common hyperproliferati

3 Seborrheic dermatitis (SD) is a common skin disease char

4 ty, dietary pattern-derived PCA factors, and seborrheic dermatitis adjusted for confounders.

5 he epidemiologic associations of HTLV-I with seborrheic dermatitis and severe anemia in childhood.

6 Current topical treatment options for seborrheic dermatitis are limited by efficacy and/or saf

7 years) patients with a clinical diagnosis of seborrheic dermatitis for a 3-month or longer duration a

8 Seborrheic dermatitis is a prevalent chronic inflammator

9 s) and placebo or azoles in the treatment of seborrheic dermatitis of the face and scalp in adults?

10 f Malassezia globosa in healthy subjects and seborrheic dermatitis or atopic dermatitis patients.

11 63 individuals and revealing a pooled global seborrheic dermatitis prevalence of 4.38% (95% CI, 3.58%

12 Original investigations on seborrheic dermatitis prevalence were included after dup

13 ry outcome was the pooled estimate of global seborrheic dermatitis prevalence.

14 Current treatments for seborrheic dermatitis provide only temporary relief.

15 Skin manifestations ranged from atopic and seborrheic dermatitis to psoriasiform rash.

16 burden for dermatitis (atopic, contact, and seborrheic dermatitis), 0.29% for acne vulgaris, 0.19% f

17 ndicated for topical treatment of psoriasis, seborrheic dermatitis, and atopic dermatitis, whereas it

18 as cheilosis, angular stomatitis, glossitis, seborrheic dermatitis, and severe anemia with erythroid

19 atopic dermatitis, lichen planus, psoriasis, seborrheic dermatitis, and vitiligo.

20 with significantly higher incidence rates of seborrheic dermatitis, eczema, and persistent hyperrefle

21 ailed estimation of the global prevalence of seborrheic dermatitis, highlighting significant variabil

22 sitive associations with skin disorders (eg, seborrheic dermatitis, OR, 1.30; 95% CI, 1.15-1.46; psor

23 ent of erythema, scaling, and itch caused by seborrheic dermatitis, supporting further investigation

24 are associated with several diseases such as seborrheic dermatitis, tinea versicolor, folliculitis, a

25 highly effective anti-fungal medication for seborrheic dermatitis, to generate a novel compound, ket

26 a high fruit intake was associated with less seborrheic dermatitis, whereas high adherence to a "West

27 involved in disorders including dandruff and seborrheic dermatitis, which together affect >50% of hum

28 ," "atopic dermatitis," "acne," "vitiligo," "seborrheic dermatitis," "alopecia areata," and "lichen p

29 pattern in females was associated with more seborrheic dermatitis.

30 as pityriasis versicolor, folliculitis, and seborrheic dermatitis.

31 s and may be caused by eczema, psoriasis, or seborrheic dermatitis.

32 pants were included, of whom 636 (14.5%) had seborrheic dermatitis.

33 Other factors were not associated with seborrheic dermatitis.

34 tal antioxidant capacity are associated with seborrheic dermatitis.

35 lly infected and was not contact dermatitis, seborrheic eczema or hand eczema.

36 ed lesions (ROC = 0.823); and melanomas from seborrheic keratoses (ROC = 0.898).

37 Seborrheic keratoses (SKs) are common benign skin tumors

38 Seborrheic keratoses (SKs) are common, benign epithelial

39 treated AKs were included, and patients with seborrheic keratoses (SKs) were included as a comparator

40 enience sample of 40 skin lesions (8 nevi, 8 seborrheic keratoses, 7 basal cell carcinomas, 7 melanom

41 abnormalities, such as actinic keratoses and seborrheic keratoses, achieving 92% sensitivity and spec

42 basal cell carcinomas, 7 solar lentigines or seborrheic keratoses, and 3 actinic keratoses.

43 mpared with cSCC in situ, actinic keratoses, seborrheic keratoses, and normal skin.

44 pical nevi, melanocytic nevi, blue nevi, and seborrheic keratoses.

45 cases: keratinocyte carcinomas versus benign seborrheic keratoses; and malignant melanomas versus ben

46 Melanomas that clinically mimic seborrheic keratosis (SK) can delay diagnosis and adequa

47 ently classified as malignant melanoma (MM), seborrheic keratosis (SK), and benign nevi by a consulta

48 ratinocytic lesions including solar lentigo, seborrheic keratosis and lichen planus-like keratosis; d

49 in situ hybridization; and the expression in seborrheic keratosis and normal skin was very low.

50 eneration, osteoarthritis, osteoporosis, and seborrheic keratosis are strongly associated with aging,

51 keratoma, dermatofibroma, solar lentigo, and seborrheic keratosis were measured.

52 The presence of wild type p53 (seborrheic keratosis) or mutant p53 (cutaneous squamous

53 ll carcinoma and nonmelanocytic lesions (eg, seborrheic keratosis, lichen planus-like keratosis, basa

54 skin cancer, dermatophytosis, acne rosacea, seborrheic keratosis, or warts; 74.1% of the subjects re

55 cell carcinoma, and elongated rete ridges in seborrheic keratosis, supporting cross-modal's potential

56 Neel et al. have demonstrated that seborrheic keratosis, the most common of all skin tumors

57 Seborrheic keratosis-like melanomas can be dermoscopical

58 mplications of this study are not limited to seborrheic keratosis.

59 (n = 51 169) with a diagnosis of a nevus or seborrheic keratosis.

60 terized by multiple keratotic papules in the seborrheic regions of the body.

61 , and 14 (87.5%) had inflammatory lesions of seborrheic regions.