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1 eous glands to induce holocrine secretion of sebum.
2 icrobial action of free fatty acids in human sebum.
3 he sebaceous gland responsible for producing sebum.
4 l therapy, no agents are available to reduce sebum.
5 ith a significant role in the composition of sebum.
6 PCNA, and correspondingly high production of sebum.
7 ecretes a unique mixture of lipids, known as sebum.
8 ple treatments caused greater suppression of sebum.
9 cies and, for example, in vernix caseosa and sebum.
10 excessive loss of lipids through the skin as sebum.
11 arget given its central role in formation of sebum, a key factor in acne pathogenesis.
12 identify changes to lipids in PD observed in sebum, a non-invasively available biofluid.
13                           As a way to reduce sebum and its interference with the process of follicula
14 onic and is therefore enriched in the acidic sebum and paracellular stratum corneum lipids containing
15 nt to effective antimicrobial penetration in sebum and selective antimicrobial action in the skin are
16 ed from a coacervate vehicle into artificial sebum and the coacervate's composition and properties.
17 d, with the sebocytes producing little or no sebum and undergoing abnormal cornification.
18 of entrained agents from the coacervate into sebum and, consequently, higher bioavailability on the s
19 ate is the most abundant fatty acid in human sebum, and among hair-bearing animals is restricted to h
20 c sweats (donor solutions), the influence of sebum, and the characterization of diffusion parameters
21 ttability tests revealed that the artificial sebum application significantly reduced the static conta
22 aceous glands (SG) and their oily secretion (sebum) are indispensable for maintaining skin structure
23  consisting of desquamated keratinocytes and sebum, are central to its pathogenesis.
24  which naturally accumulate in epidermis and sebum, as autoantigens presented by CD1a.
25 TSLP and T cells regulated sebum release and sebum-associated antimicrobial peptide expression in the
26 in, TSLP expression correlated directly with sebum-associated gene expression.
27                             This study shows sebum can be used to identify potential biomarkers for P
28 skin hormone quantification through adhesive sebum collection tape followed by analysis through mass
29                                 MSI for both sebum components and psychoactive substances revealed a
30  sebum production, because over 80% of human sebum components contain fatty acids.
31 , through a variety of cellulosic coacervate/sebum composite barriers prepared on cell culture insert
32 ns to the hair, likely caused by an abnormal sebum composition also described in a mouse model of FA2
33 igh adsorption for squalene, suggesting that sebum composition is responsible for the observed anti-i
34 lterations in sebaceous gland activation and sebum composition.
35      Moreover, gender-related differences in sebum content are not associated with altered SC hydrati
36  body temperature, pH, humidity/hydration or sebum content.
37 significant for SC glycerol content than for sebum content.
38 onsteroidal androgen receptor antagonist for sebum control and treatment of androgenetic alopecia.
39 al sebocyte accumulation of lipid, defective sebum delivery at the skin surface, and the up-regulatio
40                      Finally, we discuss how sebum-derived volatiles intersect with human health and
41                            Overproduction of sebum, especially during adolescence, is causally relate
42                                              Sebum excretion rate and auto-fluorescence from follicul
43                Medical therapies that reduce sebum excretion result in clinical improvement of acne.
44                                              Sebum excretion was eliminated for several weeks, and de
45    In this study, we examined the effects of sebum FFAs on the antimicrobial peptide (AMP)-mediated i
46                      These data suggest that sebum FFAs upregulate the expression of hBD-2 in human s
47                                      Various sebum free fatty acids (FFAs) have shown antibacterial a
48                                              Sebum from these mice contained a reduced level of type
49 he low ice adhesion a consequence of the fur sebum (hair grease).
50                              In recent years sebum has been shown to contain endogenous metabolites d
51 ich adipose loss can be achieved by means of sebum hypersecretion and uncover a role for adaptive imm
52 producibility of mass spectrometry data from sebum in relation to both storage temperature and length
53                   The influence of synthetic sebum in the permeation of metal(loid)s was also investi
54                              The presence of sebum increased lag times for copper permeation.
55                     We assessed here whether sebum influences stratum corneum hydration or permeabili
56                                              Sebum is a biofluid excreted by sebaceous glands in the
57 hypotheses that may partly explain why human sebum is so unique.
58 nt than for liquid samples and indicate that sebum is suitable for remote and at home sampling prior
59 irst insights into a potential link of lower sebum levels and lipid content and thus reduced skin moi
60 ion and is EGFR dependent, as gland size and sebum levels return to normal values after crossing to t
61             Although a mixture of synthetic, sebum-like lipids (sterol/wax esters, triglycerides) did
62  well as in the formation and composition of sebum, likely by regulating the trafficking of fatty aci
63              Widespread alterations to serum-sebum lipid relationships were observed in COVID-19 posi
64  level of type II diester wax, a major mouse sebum lipid species, and showed perturbations in mass sp
65  There was also a marked correlation between sebum lipids and the immunostimulatory hormone dehydroep
66  serum metabolites, salivary metabolites and sebum lipids are studied for the first time.
67 atty acids necessary for proper synthesis of sebum lipids.
68 ndicates that an additional component of the sebum may be required to improve bacterial clearance.
69            Recent studies also indicate that sebum may have important immunomodulatory activities and
70 haracterized, for the first time, meibum and sebum of Sdr16c5/Sdr16c6-null (DKO) mice using high-reso
71                                              Sebum on the skin surface was naturally fluorescent and
72 cooling damaged sebaceous glands and reduced sebum output for 2 weeks, with minimal injury to surroun
73                                     TrueMass Sebum Panel analyses showed a reduction in saturated and
74                                              Sebum performed well (sensitivity 0.92; specificity 0.84
75             Sebaceous glands are specialized sebum-producing epithelial cells that promote skin barri
76 nes or fibrates had significant increases in sebum production (37 and 77%, respectively) when compare
77 lands in mature mice, leading to exacerbated sebum production and accumulation on the skin surface.
78               flake homozygotes show reduced sebum production and are unable to synthesize the monoun
79 nd could be a desirable approach to reducing sebum production and improving acne.
80 sites, and SC hydration correlates with both sebum production and SC glycerol content, but the correl
81  of melanocortins in the regulation of human sebum production and support the use of the rat preputia
82 ndicate that PPARs play a role in regulating sebum production and that selective modulation of their
83 abolizing enzymes is important in regulating sebum production because these enzymes can convert weak
84 for melanocortins in the modulation of human sebum production has not been established.
85 Changes in sebaceous gland (SG) function and sebum production have been reported during aging.
86 criptional changes of sebocytes that lead to sebum production have never been characterized in detail
87 and to assess the effects of PPAR ligands on sebum production in patients.
88 asing our understanding of the regulation of sebum production is a milestone in identifying alternati
89                                       Excess sebum production is an integral part of disease pathogen
90      Understanding the factors that regulate sebum production is important in identifying therapeutic
91                                              Sebum production is key in the pathophysiology of acne,
92                                              Sebum production is regulated by the opposing effects of
93 enzonit rile (1), for the topical control of sebum production is reported.
94                     Sebaceous gland size and sebum production may serve as biomarkers for EDAR stimul
95 (13-cis RA)) is the most potent inhibitor of sebum production, a key component in the pathophysiology
96 it resulting from androgen-induced increased sebum production, altered keratinisation, inflammation,
97  strength, sexual function, prostate volume, sebum production, and hematocrit and lipid levels did no
98  strength, sexual function, prostate volume, sebum production, and hematocrit and lipid levels.
99 ons through the regulation of skin immunity, sebum production, and skin barrier function.
100 ma (PPARgamma), a potential cause of reduced sebum production, as well as the potential involvement o
101 s glands is designed to substantially affect sebum production, because over 80% of human sebum compon
102 sebaceous gland differentiation with reduced sebum production, chronic inflammation, and ultimately t
103 linked to multiple factors such as increased sebum production, inflammation, follicular hyperkeratini
104 the contribution of each receptor subtype to sebum production, lipogenesis assays were performed in S
105 ocytes results in enlarged SGs and increased sebum production.
106 crine glands, including a marked decrease in sebum production.
107  may account for the regional differences in sebum production; (iii) to compare the activity of 17bet
108                   TSLP and T cells regulated sebum release and sebum-associated antimicrobial peptide
109 one inflammatory proteins were explored from sebum sampled at the skin surface after oronasal mask ap
110 ts provided blood serum alongside saliva and sebum samples for analysis by liquid chromatography mass
111 he requirements for storage and transport of sebum samples that may be used in clinical assays are le
112                                              Sebum samples were collected from volunteers and stored
113                                   Given that sebum sampling is facile and non-invasive, its potential
114      Moreover, IL-13 was sufficient to cause sebum secretion and adipose loss in vivo and to induce l
115 gated the mechanism by which TSLP stimulates sebum secretion and adipose loss.
116 stimulating lipid loss from the skin through sebum secretion by sebaceous glands, which enhances the
117 on was analyzed by histological analysis and sebum secretion in vivo and by measuring lipogenesis and
118 ied in both Eda mutant and wild-type mice by sebum secretion to levels higher than those observed in
119 ve immunity in skin barrier function through sebum secretion.
120  necessary for TSLP-induced adipose loss and sebum secretion.
121 tmospheric aerosols, cooking oils, and human sebum, unsaturated lipids rapidly decay upon exposure to
122 ereas the gland's main function-excretion of sebum via the holocrine mechanism-can only be evaluated
123 an odor instead originate from precursors in sebum, via reactions that do not involve the skin microb
124 decrease in squalene levels, a biomarker for sebum, was observed.
125 ands (SGs) are holocrine glands that produce sebum, which primarily contains lipids that help to main

 
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