ライフサイエンスコーパス: sec

1 .01], 1.9 sec(-1) +/- 0.2 [P < .01], and 1.0 sec(-1) +/- 0.3 [P < .01], respectively, for control fet

2 (-1); P = .01) and total neocortex (HC, 17.0 sec(-1); AD, 17.4 sec(-1); P < .001) and regionally in t

3 oup than in the HC group in the BG (HC, 29.0 sec(-1); AD, 30.2 sec(-1); P = .01) and total neocortex

4 ndard deviation for patients with NASH, 37.0 sec(-1) +/- 16.1; patients with simple steatosis, 61.0 s

5 s follows: none (PDFF, </=5.1%; R2*, </=41.0 sec(-1)), mild (PDFF, >5.1%; R2*, >41 sec(-1)), moderate

6 - 16.1; patients with simple steatosis, 61.0 sec(-1) +/- 17.3; and healthy control subjects, 72.2 sec

7 -planar imaging sequence with a b value of 0 sec/mm(2) yielded high-spatial-resolution T2-weighted MR

8 nd posttreatment mean k(PL), 0.011 and 0.017 sec(-1), respectively, P = .91; baseline and posttreatme

9 ne and treatment mean k(PL), 0.027 and 0.018 sec(-1), respectively, P = .01; baseline and posttreatme

10 MINI identifies the nearest neighbor in 0.05 sec compared to a brute force search time of 0.6 sec.

11 es with SV or AO CHD (DeltaR2* per week, 0.1 sec(-1) +/- 0 [P < .01], 0.2 sec(-1) +/- 0 [P = .01], an

12 algorithms detected apnea within 7.9 +/- 1.1 sec and 5.5 +/- 2.2 sec, respectively, while the estimat

13 07) and temporal (HC, 16.4 sec(-1); AD, 18.1 sec(-1); P < .001) lobes.

14 .001), and delayed time to peak (2.1 of 7.1 sec [29.6%]; P = .005); baseline SvO(2) was lower (-13 o

15 2.5 sec(-1)), high (PDFF: >28.0%; R2*: >70.1 sec(-1)).

16 dose on Day 6, forced expiratory volume in 1 sec (FEV1 ) was measured up to 10 h post-challenge.

17 ssociated with forced expiratory volume in 1 sec (FEV1) decline, with each 10-g increase in lung mass

18 ction in FEV1 (forced expiratory volume in 1 sec) for a 10% increase in CO was 3.33 mL (95% CI: -0.86

19 han ADAMTS-4 (k(cat)/K(m) 1.86 x 10(5) M(-1) sec(-1)), whereas ADAMTS-1 versicanase activity is compa

20 emperature) of Fe-tCDTA (r1 = 2.2 mmol(-1) . sec(-1)) were approximately twofold and fivefold lower,

21 ased T1 relaxivity of oxygen (4.7e-4 mmHg(-1)sec(-1)).

22 ct on WM at either short (2 sec) or long (10 sec) delay intervals.

23 the amino-terminal end of ECL3, and Cys(10)-sec not efficiently labeling any of these residues.

24 range, 99-166; P < .001) and vorticity (108 sec(-1); interquartile range, 84-151; P < .001) found in

25 n than hyperopic children (40 sec arc vs 120 sec arc, P < .001).

26 Results Muscle R2* at 3.0 T (33-125 sec(-1)) depended on the morphology of fat replacement,

27 on in-vitro with an acquisition time of 2.14 sec.

28 d imaging and ADC mapping (b values, 50-1400 sec/mm(2)), MR fingerprinting with steady-state free pre

29 was performed that included DWIBS (b = 1500 sec/mm(2)).

30 Lesions were segmented on b of 1500 sec/mm(2) by two readers in consensus and an additional

31 * per week, 0.1 sec(-1) +/- 0 [P < .01], 0.2 sec(-1) +/- 0 [P = .01], and 0.2 sec(-1) +/- 0 [P = .01]

32 < .01], 0.2 sec(-1) +/- 0 [P = .01], and 0.2 sec(-1) +/- 0 [P = .01], respectively), but not in fetus

33 gantry rotation 0.6 sec., scanning delay 1-2 sec.

34 apnea within 7.9 +/- 1.1 sec and 5.5 +/- 2.2 sec, respectively, while the estimated RR and TV values

35 in the occipital (HC, 19.6 sec(-1); AD, 20.2 sec(-1); P = .007) and temporal (HC, 16.4 sec(-1); AD, 1

36 group in the BG (HC, 29.0 sec(-1); AD, 30.2 sec(-1); P = .01) and total neocortex (HC, 17.0 sec(-1);

37 +/- 17.3; and healthy control subjects, 72.2 sec(-1) +/- 22.0; P = .006 for NASH vs simple steatosis;

38 (~100 nM) to have an elution half-life of ~2 sec, consistent with measured albumin elution, with most

39 63397 had no effect on WM at either short (2 sec) or long (10 sec) delay intervals.

40 ') cycle peaking at 1600 mumol photons m(-2) sec(-1) .

41 owed impaired curvature to 0.005 mumol m(-2) sec(-1) unilateral blue light, indicating that regions b

42 an Bland-Altman analysis bias, 0.002 cm(2) . sec(-1) and -1.5 mum, respectively).

43 coefficient was 0.85 +/- 0.04 x 10(-10) m(2)sec(-1) and increased to 1.77 +/- 0.09 x 10(-10) m(2)sec

44 and increased to 1.77 +/- 0.09 x 10(-10) m(2)sec(-1) at 265 K.

45 A mean ADC of 0.0014 m(2)/sec or less (hazard ratio [HR] = 8.3), low ADC signal in

46 f spontaneous abscesses, 0.68 x 10(-3) mm(2)/sec (interquartile range, 0.58-0.79 x 10(-3) mm(2)/sec;

47 toperative abscesses was 1.34 x 10(-3) mm(2)/sec (interquartile range, 1.00-1.62 x 10(-3) mm(2)/sec),

48 usivity (MD) increased by 8.1 x 10(-6) mm(2)/sec (P < .001).

49 MD increased by 10.8 x 10(-6)mm(2)/sec (Pmean < .001) on average in 79% of white matter ske

50 10.8 msec +/- 0.8; ADC, 1.39 x 10(-3) mm(2)/sec +/- 0.02 (reference erector spinae muscle tissue: T1

51 /- 3.6) values and ADCs (1.53 x 10(-3) mm(2)/sec +/- 0.03) compared with those of skeletal muscle.

52 3 msec +/- 1.7; and ADC, 1.40 x 10(-3) mm(2)/sec +/- 0.03).

53 R imaging patterns were 0.360 x 10(-3) mm(2)/sec +/- 0.110, 1.046 x 10(-3) mm(2)/sec +/- 0.232, and 0

54 m(2)/sec +/- 0.232, and 0.770 x 10(-3) mm(2)/sec +/- 0.135, respectively.

55 3) mm(2)/sec +/- 0.110, 1.046 x 10(-3) mm(2)/sec +/- 0.232, and 0.770 x 10(-3) mm(2)/sec +/- 0.135, r

56 0, 169 msec +/- 61, and 1.711 x 10(-3) mm(2)/sec +/- 0.269) (P < .0001 for each) and together produce

57 44, 73 msec +/- 27, and 0.773 x 10(-3) mm(2)/sec +/- 0.331, respectively) were significantly lower th

58 (2)/sec vs [1708.9 +/- 108.1] x 10(-6) mm(2)/sec and [657.3 +/- 129.9] x 10(-6) mm(2)/sec vs [69.2 +/

59 th ADCs between 0.79 and 1.33 x 10(-3) mm(2)/sec and no abscesses with ADCs greater than 1.33 x 10(-3

60 oefficient threshold of 1.275 x 10(-3) mm(2)/sec enabled differentiation between benign and malignant

61 0+/-0.35x10(-3) and 1.34+/-0.36x10(-3) mm(2)/sec in chronic plaques before and after the treatment.

62 (2)/sec, 0.33, 0.45, and 0.27 x 10(-3) mm(2)/sec increases, and 40%, 54%, and 27% increases, respecti

63 e analysis, ADC values of 1.5 x 10(-3) mm(2)/sec or lower were associated with a negative prognosis,

64 ADCs greater than 0.548 x 10(-3) mm(2)/sec showed 100% sensitivity (26 of 26) and 98% specifici

65 eas an ADC greater than 0.597 x 10(-3) mm(2)/sec showed 96% sensitivity (25 of 26) and 100% specifici

66 mpending delivery with a 1921 x 10(-6) mm(2)/sec threshold.

67 coefficients decrease (from 1 x 10(-3)mm(2)/sec to 0.4 x 10(-3)mm(2)/sec), and fiber paths, extracte

68 ) mm(2)/sec, P = .03; D, 0.58 x 10(-3) mm(2)/sec vs 0.42 x 10(-3) mm(2)/sec, P < .001).

69 e increased BMEmax (ADC, 0.67 x 10(-3) mm(2)/sec vs 0.54 x 10(-3) mm(2)/sec, P = .03; D, 0.58 x 10(-3

70 usion coefficient values (875 x 10(-6) mm(2)/sec vs 1111 x 10(-6) mm(2)/sec, respectively; P < .01),

71 rd deviation, [0.89 +/- 0.09] x 10(-3) mm(2)/sec vs [0.9 +/- 0.09] x 10(-3) mm(2)/sec), or fractional

72 delivery ([2406.3 +/- 166.0] x 10(-6) mm(2)/sec vs [1708.9 +/- 108.1] x 10(-6) mm(2)/sec and [657.3

73 (2)/sec and [657.3 +/- 129.9] x 10(-6) mm(2)/sec vs [69.2 +/- 70.2] x 10(-6) mm(2)/sec, respectively)

74 When the ADC value of 1.45x10(-3) mm(2)/sec was used as a threshold value for differentiating Gr

75 sses with ADCs less than 0.79 x 10(-3) mm(2)/sec were found to be spontaneous, while only six (27%) a

76 s with ADCs greater than 1.33 x 10(-3) mm(2)/sec were spontaneous (P < .001).

77 tween benign ([1.78 +/- 0.23] x 10(-3) mm(2)/sec) and malignant ([1.03 +/- 0.23] x 10(-3) mm(2)/sec)

78 nd malignant ([1.03 +/- 0.23] x 10(-3) mm(2)/sec) tumors (P = .002).

79 rom 1 x 10(-3)mm(2)/sec to 0.4 x 10(-3)mm(2)/sec), and fiber paths, extracted by tractography, increa

80 ) mm(2)/sec vs [0.9 +/- 0.09] x 10(-3) mm(2)/sec), or fractional anisotropy (0.43 +/- 0.05 vs 0.42 +/

81 nterquartile range, 1.00-1.62 x 10(-3) mm(2)/sec), which was significantly higher than the median ADC

82 nce ranged from -0.04 to 0.02 x 10(-3) mm(2)/sec).

83 ut-offs: 1.21, 1.30, and 1.05 x 10(-3) mm(2)/sec, 0.33, 0.45, and 0.27 x 10(-3) mm(2)/sec increases,

84 hted imaging at 0, 800, 1000, and 1400 mm(2)/sec, and dynamic contrast-enhanced MR imaging, obtained

85 Graves' disease was 2.03+/-0.28x10(-3) mm(2)/sec, and in patients with painless thyroiditis 1.46+/-0.

86 58 x 10(-3) mm(2)/sec vs 0.42 x 10(-3) mm(2)/sec, P < .001).

87 67 x 10(-3) mm(2)/sec vs 0.54 x 10(-3) mm(2)/sec, P = .03; D, 0.58 x 10(-3) mm(2)/sec vs 0.42 x 10(-3

88 (23.7 and 7.7, 8.83, and 1.58 x 10(-3) mm(2)/sec, respectively) and tumor PZ tissue (11.4 and 12.5, 5

89 (20.0 and 8.2, 6.50, and 1.26 x 10(-3) mm(2)/sec, respectively) and tumor TZ tissue (9.8 and 11.2, 4.

90 c +/- 22, and [1.13 +/- 0.19] x 10(-3) mm(2)/sec, respectively) were higher than those in cancers (14

91 c +/- 140 and [0.58 +/- 0.14] x 10(-3) mm(2)/sec, respectively) were lower than those in clinically i

92 c +/- 220 and [0.60 +/- 0.17] x 10(-3) mm(2)/sec, respectively) were lower than those of noncancers (

93 11.4 and 12.5, 5.13, and 1.20 x 10(-3) mm(2)/sec, respectively).

94 (9.8 and 11.2, 4.36, and 1.03 x 10(-3) mm(2)/sec, respectively).

95 mm(2)/sec vs [69.2 +/- 70.2] x 10(-6) mm(2)/sec, respectively).

96 ainless thyroiditis 1.46+/-0.22x10(-3) mm(2)/sec, respectively.

97 c +/- 11, and [0.57 +/- 0.13] x 10(-3) mm(2)/sec, respectively; P < .001 for all).

98 75 x 10(-6) mm(2)/sec vs 1111 x 10(-6) mm(2)/sec, respectively; P < .01), smaller prostate volume (47

99 c +/- 120 and [0.75 +/- 0.17] x 10(-3) mm(2)/sec, respectively; P = .001 for all).

100 c +/- 16, and [0.82 +/- 0.13] x 10(-3) mm(2)/sec, respectively; P = .001 for T1 and ADC and P = .03 f

101 c +/- 120 and [0.81 +/- 0.13] x 10(-3) mm(2)/sec, respectively; P = .006 for T1 and P = .004 for ADC)

102 C less than or equal to 0.905 x 10(-3) mm(2)/sec.

103 remote regions, mean ADC (1.62 x10(-3) mm(2)/sec; 95% CI: 1.59, 1.64) was comparable to that measured

104 gh in the border regions (1.91 x10(-3) mm(2)/sec; 95% CI: 1.89, 1.94; P < .001).

105 n the infarcted regions (2.32 x 10(-3) mm(2)/sec; 95% confidence interval [CI]: 2.28, 2.36) and moder

106 nterquartile range, 0.58-0.79 x 10(-3) mm(2)/sec; P < .001).

107 nificance (mean, 1.42 vs 1.16 x 10(-3) mm(2)/sec; P = .26).

108 ion coefficient [beta] = 1.94 [x10(-3) mm(2)/sec], P = .04).

109 6 x10(3)mum(2)/sec; right, 1.23 x10(3)mum(2)/sec) demonstrate low initial ADC values, indicating an e

110 7 x10(3)mum(2)/sec; right, 1.15 x10(3)mum(2)/sec), and basal ganglia (left, 1.26 x10(3)mum(2)/sec; ri

111 (2)/sec; right hemisphere, 1.17 x10(3)mum(2)/sec), anterior limb of the internal capsule (left, 1.11

112 x10(3)mum(2)/sec), vermis (1.26 x10(3)mum(2)/sec), thalami (left, 1.17 x10(3)mum(2)/sec; right, 1.15

113 1 x10(3)mum(2)/sec; right, 1.09 x10(3)mum(2)/sec), vermis (1.26 x10(3)mum(2)/sec), thalami (left, 1.1

114 mean ADC: left hemisphere, 1.18 x10(3)mum(2)/sec; right hemisphere, 1.17 x10(3)mum(2)/sec), anterior

115 he internal capsule (left, 1.11 x10(3)mum(2)/sec; right, 1.09 x10(3)mum(2)/sec), vermis (1.26 x10(3)m

116 um(2)/sec), thalami (left, 1.17 x10(3)mum(2)/sec; right, 1.15 x10(3)mum(2)/sec), and basal ganglia (l

117 , and basal ganglia (left, 1.26 x10(3)mum(2)/sec; right, 1.23 x10(3)mum(2)/sec) demonstrate low initi

118 ephosphorylated to various degrees during 20 sec to 10 min postmortem.

119 7 degrees C for 1 min or 42 degrees C for 20 sec) and storage vessels (pellets, 0.25 mL straws or 0.5

120 c diffusion acquisition; b values, 0 and 200 sec/mm(2); six diffusion-encoding directions; five repet

121 g diffusion-weighted MRI (b values of 0-2000 sec/mm(2)).

122 uence (b values 0, 500, 1000, 1500, and 2000 sec/mm(2)) before prostatectomy.

123 d by using maximal b values of 1000 and 2000 sec/mm(2), respectively.

124 se participants: -0.1 ppm +/- 0.04 and 18.21 sec(-1) +/- 3.1; control participants: -0.06 ppm +/- 0.0

125 grae (first MRI: 0.06 ppm +/- 0.06 and 28.26 sec(-1) +/- 9.56; last MRI: 0.07 ppm +/- 0.06 and 25.65

126 ine concentration rose quickly (mean T1/2 27 sec) with a peak amplitude 25% higher in females than ma

127 it was higher in SV CHD (mean DeltaR2*, 1.3 sec(-1) +/- 0.1 [standard error; P < .01], 1.9 sec(-1) +

128 edulla and from 18.3 sec(-1) +/- 1.5 to 16.3 sec(-1) +/- 1.0 in the cortex, indicating increased oxyg

129 sec(-1) +/- 2.5 in the medulla and from 18.3 sec(-1) +/- 1.5 to 16.3 sec(-1) +/- 1.0 in the cortex, i

130 ort, and has a 10% to 90% settling time of 3 sec.

131 ghly diffusion-weighted data (b-value = 3000 sec/mm(2)).

132 +/- 5.16; last MRI: 0 ppm +/- 0.02 and 15.32 sec(-1) +/- 2.49) did not differ between the first and l

133 ol participants: 0.02 ppm +/- 0.02 and 18.38 sec(-1) +/- 2.09) did not differ between groups (suscept

134 ate (first MRI: -0.09 ppm +/- 0.05 and 21.38 sec(-1) +/- 4.72; last MRI: -0.1 ppm +/- 0.05 and 18.75

135 .2 sec(-1); P = .007) and temporal (HC, 16.4 sec(-1); AD, 18.1 sec(-1); P < .001) lobes.

136 total neocortex (HC, 17.0 sec(-1); AD, 17.4 sec(-1); P < .001) and regionally in the occipital (HC,

137 P < .003), from 28.9 sec(-1) +/- 2.3 to 26.4 sec(-1) +/- 2.5 in the medulla and from 18.3 sec(-1) +/-

138 e measured at 85 degrees C is over 3 x 10(4) sec, and no significant degradation is observed in 130 c

139 metropic than in than hyperopic children (40 sec arc vs 120 sec arc, P < .001).

140 Ser9 was reduced by 50% in the brain with 40 sec postmortem, a regular time for tissue processing.

141 /=41.0 sec(-1)), mild (PDFF, >5.1%; R2*, >41 sec(-1)), moderate (PDFF, >14.1%; R2*, >62.5 sec(-1)), h

142 ; 95%CI S: 433; 404-462 sec; O: 412; 382-441 sec; p = 0.378/GLM).

143 se participants: 0.08 ppm +/- 0.06 and 27.46 sec(-1) +/- 5.58; control participants: 0.04 ppm +/- 0.0

144 tween protocols (mean; 95%CI S: 433; 404-462 sec; O: 412; 382-441 sec; p = 0.378/GLM).

145 (case participants: 0 ppm +/- 0.03 and 16.49 sec(-1) +/- 3.6; control participants: 0.02 ppm +/- 0.02

146 taR2*, 0.7 sec(-1) +/- 0.2 [P = .01] and 0.5 sec(-1) +/- 0.2 [P = .02], respectively).

147 namic (with an average residence time of 1.5 sec).

148 sec(-1)), moderate (PDFF, >14.1%; R2*, >62.5 sec(-1)), high (PDFF: >28.0%; R2*: >70.1 sec(-1)).

149 eighted images obtained with a b value of 50 sec/mm(2), a T2-weighted gradient-echo (GRE) sequence wa

150 came prominent and reached 5 pM FXIa at >500 sec in the simulation, consistent with anti-FXIa experim

151 lidi (first MRI: 0.13 ppm +/- 0.07 and 27.53 sec(-1) +/- 8.88; last MRI: 0.14 ppm +/- 0.06 and 29.78

152 - 5.9; last MRI: 0.06 ppm +/- 0.02 and 25.55 sec(-1) +/- 4.71), substantia nigrae (first MRI: 0.06 pp

153 icated steps, our fabricated sensor takes 55 sec to deliver sensing response reflecting the meat spoi

154 compared to a brute force search time of 0.6 sec.

155 ess 0.625 mm, pitch 1.3, gantry rotation 0.6 sec., scanning delay 1-2 sec.

156 ion kinase (FAK) activation by 42.6 +/- 12.6 sec.

157 1) and regionally in the occipital (HC, 19.6 sec(-1); AD, 20.2 sec(-1); P = .007) and temporal (HC, 1

158 ng were distinct for each probe, with Cys(6)-sec labeling multiple residues in the carboxyl-terminal

159 of 731 transcripts occurs as early as 20-60 sec following light stress application, and that at leas

160 9 sec(-1) [interquartile range, 59-87] vs 60 sec(-1) [interquartile range, 50-67], respectively; P <

161 g, rats either received extinction trials 60-sec after the last conditioning trial (continuous, no ev

162 ar DTI (20 directions, b values of 0 and 600 sec/mm(2)), regions of interest were placed in the tibia

163 se participants: 0.03 ppm +/- 0.02 and 20.63 sec(-1) +/- 2.44; control participants: 0.02 ppm +/- 0.0

164 lami (first MRI: 0.01 ppm +/- 0.02 and 17.65 sec(-1) +/- 5.16; last MRI: 0 ppm +/- 0.02 and 15.32 sec

165 ol participants: 0.02 ppm +/- 0.02 and 19.65 sec(-1) +/- 3.6), caudate (case participants: -0.1 ppm +

166 9.56; last MRI: 0.07 ppm +/- 0.06 and 25.65 sec(-1) +/- 6.37), globus pallidi (first MRI: 0.13 ppm +

167 ], respectively; P < .001) and vorticity (69 sec(-1) [interquartile range, 59-87] vs 60 sec(-1) [inte

168 etuses with SV or AO CHD (mean DeltaR2*, 0.7 sec(-1) +/- 0.2 [P = .01] and 0.5 sec(-1) +/- 0.2 [P = .

169 rol participants: 0.02 ppm +/- 0.03 and 21.7 sec(-1) +/- 5.26), substantia nigrae (case participants:

170 nal half of ECL2 and throughout ECL3, Cys(7)-sec predominantly labeling only single residues in the c

171 3.51; last MRI: 0.03 ppm +/- 0.02 and 19.73 sec(-1) +/- 3.01), caudate (first MRI: -0.09 ppm +/- 0.0

172 4.72; last MRI: -0.1 ppm +/- 0.05 and 18.75 sec(-1) +/- 2.68), and thalami (first MRI: 0.01 ppm +/-

173 se participants: 0.14 ppm +/- 0.05 and 30.75 sec(-1) +/- 5.14; control participants: 0.08 ppm +/- 0.0

174 mina (first MRI: 0.03 ppm +/- 0.03 and 19.78 sec(-1) +/- 3.51; last MRI: 0.03 ppm +/- 0.02 and 19.73

175 tate (first MRI: 0.06 ppm +/- 0.05 and 25.78 sec(-1) +/- 5.9; last MRI: 0.06 ppm +/- 0.02 and 25.55 s

176 8.88; last MRI: 0.14 ppm +/- 0.06 and 29.78 sec(-1) +/- 6.54), putamina (first MRI: 0.03 ppm +/- 0.0

177 RT SPACE (mean +/- standard deviation, 338.8 sec +/- 69.1).

178 Nonzero minimum b value (100, 600, and 800 sec/mm(2)) did not improve the AUC (0.74; P = .28), and

179 , 10, 20, 30, 50, 80, 100, 200, 400, and 800 sec/mm(2)) was acquired within bone marrow and focal les

180 pression and DW imaging (b = 0, 500, and 800 sec/mm(2)), followed by either sentinel lymph node biops

181 usion-weighted MRI (b = 0, 100, 600, and 800 sec/mm(2)).

182 re obtained with b values of 0, 400, and 800 sec/mm(2).

183 600, 100 and 600, 0 and 800, and 100 and 800 sec/mm(2); AUC, 0.73-0.76) provided results similar to t

184 ating the lesion, images with b value of 800 sec/mm(2), and apparent diffusion coefficient (ADC) maps

185 ol participants: 0.08 ppm +/- 0.07 and 28.82 sec(-1) +/- 6.62), putamina (case participants: 0.03 ppm

186 l participants: -0.06 ppm +/- 0.05 and 18.83 sec(-1) +/- 3.32), and thalami (case participants: 0 ppm

187 se participants: 0.06 ppm +/- 0.04 and 23.87 sec(-1) +/- 4.13; control participants: 0.02 ppm +/- 0.0

188 c(-1) +/- 0.1 [standard error; P < .01], 1.9 sec(-1) +/- 0.2 [P < .01], and 1.0 sec(-1) +/- 0.3 [P <

189 the medulla and cortex (P < .003), from 28.9 sec(-1) +/- 2.3 to 26.4 sec(-1) +/- 2.5 in the medulla a

190 ol participants: 0.04 ppm +/- 0.05 and 24.96 sec(-1) +/- 5.3), globus pallidi (case participants: 0.1

191 Second, a sec-phosphine borane amino ester was saponified and coup

192 hyl and isopropyl groups but remarkably also sec-butyl and t-butyl groups.

193 ination reaction into free [60]fullerene and sec-phosphine borane amino ester compound.

194 nd anteiso positions, terminal isopropyl and sec-butyl, respectively, do not alter CACI-MS/MS diagnos

195 rates were 4.9 au/sec +/- 0.8 versus 4.6 au/sec +/- 0.6, and mean changes in renal flow rate were 2.

196 Mean changes in perfusion rates were 4.9 au/sec +/- 0.8 versus 4.6 au/sec +/- 0.6, and mean changes

197 creted by type II secretory system: PheA (CM-sec), LipA/LesA, VirK, and four families involved in N-g

198 ar velocity (12.8 +/- 1.0 vs 14.9 +/- 3.0 cm/sec and 9.3 +/- 2.0 vs 10.9 +/- 2.0 cm/sec, respectively

199 .0 cm/sec and 9.3 +/- 2.0 vs 10.9 +/- 2.0 cm/sec, respectively), and higher E/E' ratio compared to co

200 Speeds ranged from 1 cm/sec to 4.5 cm/sec.

201 P < .001), hyperemic index (-3.9 of 15.1 cm/sec(2) [-25.8%]; P < .001), and delayed time to peak (2.

202 7 patients (61.2%) did not (mean PSV, 7.1 cm/sec, P < .0001; mean RI, 0.50, P < .0001).

203 pecificity by using a PSV greater than 10 cm/sec and an RI greater than 0.65 as diagnostic criteria.

204 iminatory criteria of PSV greater than 10 cm/sec and RI greater than 0.65 yielded specificity for app

205 iminatory criteria of PSV greater than 10 cm/sec and RI greater than 0.65 yielded specificity for app

206 donor arterial velocity was less than 100 cm/sec (seven of 56 vs four of 126 with velocity >/= 100 cm

207 f 56 vs four of 126 with velocity >/= 100 cm/sec; P = .04).

208 CSF peak-to-peak velocity magnitude (2.2 cm/sec; P = .01).

209 ller (stress, 16.9 cm/sec +/- 4.5 vs 20.5 cm/sec +/- 3.9; P < .0001).

210 r (2) > 0.97), with negligible bias (<0.5 cm/sec) and comparable velocity-to-noise ratios.

211 Speeds ranged from 1 cm/sec to 4.5 cm/sec.

212 with reduced peak velocity (-9.9 of 56.6 cm/sec [-17.5%]; P < .001), hyperemic index (-3.9 of 15.1 c

213 pertension (portal vein velocity, 3.9-5.6 cm/sec of hepatopetal flow), and repeated bleeding of the v

214 ) had proven appendicitis (mean PSV, 19.7 cm/sec; mean RI, 0.69) and 57 patients (61.2%) did not (mea

215 tolic wave was also smaller (stress, 16.9 cm/sec +/- 4.5 vs 20.5 cm/sec +/- 3.9; P < .0001).

216 m Hg, pulmonary vascular resistance >240 dyn-sec/cm(-5) , and pulmonary artery wedge pressure <=15 mm

217 ryl acetamides under TM-free conditions from sec- and tert-arylacetamides and nitroarenes using tert-

218 ody weight (<=510 mg iron; rate <=45 mg iron/sec).

219 n velocity was less than or equal to 4.06 m .sec(-1) at axial SWE (sensitivity, 54.2%; 95% confidence

220 .5, 81.7) and less than or equal to 14.58 m .sec(-1) at sagittal SWE (sensitivity, 58.3%; 95% CI: 36.

221 6.3, 95.1) and less than or equal to 5.70 m .sec(-1) at sagittal SWE (sensitivity, 41.7%; 95% CI: 22.

222 n velocity was less than or equal to 4.86 m .sec(-1) at axial SWE (sensitivity, 66.7%; 95% CI: 44.7,

223 MTSWV in volunteers (3.0 m/sec +/- 0.5) was significantly higher than that in patie

224 Mean normal SSP SWV is 3.0 m/sec +/- 0.5.

225 , 1.84 m/sec) and 2.08 m/sec (95% CI: 2.02 m/sec, 2.13 m/sec; P < .001) in cortex, and 1.25 m/sec (95

226 elocity marginally increased (0.19 of 6.05 m/sec [3%]; P = .05).

227 (95% CI: 1.75 m/sec, 1.84 m/sec) and 2.08 m/sec (95% CI: 2.02 m/sec, 2.13 m/sec; P < .001) in cortex

228 Each 1 m/sec increase in PWV, up to 12 m/sec, was associated with

229 Each 1 m/sec increase in PWV, up to 12 m/sec, was associated with mortality, hazard ratio (HR) 1.

230 eiling effect, and PWV was truncated at 12 m/sec.

231 ) and 2.08 m/sec (95% CI: 2.02 m/sec, 2.13 m/sec; P < .001) in cortex, and 1.25 m/sec (95% CI: 1.21 m

232 with an increased mean aortic PWV of 0.19 m/sec (95% CI: 0.03, 0.36) in total and an increased mean

233 .7 m/sec +/- 1.6) than in stable ones (3.2 m/sec +/- 0.9).

234 1) in cortex, and 1.25 m/sec (95% CI: 1.21 m/sec, 1.29 m/sec) and 1.33 (95% CI: 1.27 m/sec, 1.38 m/se

235 2.13 m/sec; P < .001) in cortex, and 1.25 m/sec (95% CI: 1.21 m/sec, 1.29 m/sec) and 1.33 (95% CI: 1

236 m/sec, 1.29 m/sec) and 1.33 (95% CI: 1.27 m/sec, 1.38 m/sec; P = .03) in medulla.

237 on of nerve conduction velocity (0.13-0.28 m/sec), previously unrealized for any human cell-based in

238 , and 1.25 m/sec (95% CI: 1.21 m/sec, 1.29 m/sec) and 1.33 (95% CI: 1.27 m/sec, 1.38 m/sec; P = .03)

239 pulse wave velocity (PWV) (decrease of 1.3 m/sec +/- 0.8).

240 m/sec) and 1.33 (95% CI: 1.27 m/sec, 1.38 m/sec; P = .03) in medulla.

241 l and an increased mean aortic PWV of 0.42 m/sec (95% CI: 0.03, 0.81) in the abdominal segment.

242 ficantly higher than that in patients (2.5 m/sec +/- 0.5; P = .001).

243 Without QM, using Vs of 4.5 m/sec or greater as test positive, SW elastography had les

244 classified benign; lesions with Vs of 4.5 m/sec or greater, malignant.

245 ess than 4.5 m/sec; and 20 (42%), V of 4.5 m/sec or greater.

246 s than 4.5 m/sec; and five (5%), Vs of 4.5 m/sec or greater.

247 Lesions with Vs of less than 4.5 m/sec were classified benign; lesions with Vs of 4.5 m/sec

248 malignant lesions with Vs of less than 4.5 m/sec.

249 lts Total aortic PWV (mean difference, 0.5 m/sec; 95% confidence interval [CI]: 0.3, 0.7) and carotid

250 aphy signal; 20 (42%), Vs of less than 4.5 m/sec; and 20 (42%), V of 4.5 m/sec or greater.

251 aphy signal; 77 (81%), Vs of less than 4.5 m/sec; and five (5%), Vs of 4.5 m/sec or greater.

252 m/sec (95% confidence interval [CI]: 1.51 m/sec, 1.59 m/sec) and 1.69 m/sec (95% CI: 1.64 m/sec, 1.7

253 tex, and medulla, with mean values of 1.55 m/sec (95% confidence interval [CI]: 1.51 m/sec, 1.59 m/se

254 confidence interval [CI]: 1.51 m/sec, 1.59 m/sec) and 1.69 m/sec (95% CI: 1.64 m/sec, 1.74 m/sec; P <

255 , 1.59 m/sec) and 1.69 m/sec (95% CI: 1.64 m/sec, 1.74 m/sec; P < .001), respectively, in parenchyma,

256 val [CI]: 1.51 m/sec, 1.59 m/sec) and 1.69 m/sec (95% CI: 1.64 m/sec, 1.74 m/sec; P < .001), respecti

257 r (P < .001/12) in unstable aneurysms (5.7 m/sec +/- 1.6) than in stable ones (3.2 m/sec +/- 0.9).

258 ) and 1.69 m/sec (95% CI: 1.64 m/sec, 1.74 m/sec; P < .001), respectively, in parenchyma, 1.80 m/sec

259 y, in parenchyma, 1.80 m/sec (95% CI: 1.75 m/sec, 1.84 m/sec) and 2.08 m/sec (95% CI: 2.02 m/sec, 2.1

260 locities (mean +/- standard deviation, 2.8 m/sec +/- 0.9).

261 An interquartile range increase (3.8 m/sec) tripled the hazard of mortality, HR, 3.21 (95% CI,

262 < .001), respectively, in parenchyma, 1.80 m/sec (95% CI: 1.75 m/sec, 1.84 m/sec) and 2.08 m/sec (95%

263 hyma, 1.80 m/sec (95% CI: 1.75 m/sec, 1.84 m/sec) and 2.08 m/sec (95% CI: 2.02 m/sec, 2.13 m/sec; P <

264 rdware, shielded gradients (45 mT/m; 200 T/m/sec), and advanced imaging methods.

265 her MFR (16.25 +/- 8.02 vs 12.40 +/- 7.05 mL/sec, P = 0.007), and lower IPSS (6.55 +/- 5.86 vs 8.57 +

266 tions involved changes in flow rate (1-12 mL/sec), imaging depth (2.5-7 cm), color flow gain (0%-100%

267 ow (95% limits of agreement: -8.8 and 9.3 mL/sec, respectively) than was Cartesian 4D flow (95% limit

268 trast material and saline injected at 2.5 mL/sec); both protocols had a fixed scan delay of 70 second

269 trast material and saline injected at 2.5 mL/sec; phases 3-4, 40 mL of contrast material and saline i

270 39% +/- 29); maximum urinary flow rate, 6 mL/sec +/- 10 (155% +/- 293); and postvoid residual volume,

271 (95% limits of agreement: -10.6 and 14.6 mL/sec).

272 s 95% CI); ACV was 3.70 mm/sec (2.21-5.18 mm/sec 95% CI); and ADV was 0.88 mm/sec (0.38-1.38 mm/sec 9

273 % CI); and ADV was 0.88 mm/sec (0.38-1.38 mm/sec 95% CI).

274 one, and cortivazol (20.3, 21.3, and 27.5 mm/sec, respectively; P < .003).

275 pliteal LN was at velocities lower than 5 mm/sec.

276 seconds (160-300 ms 95% CI); ACV was 3.70 mm/sec (2.21-5.18 mm/sec 95% CI); and ADV was 0.88 mm/sec (

277 lic acid species formed in vitro was 97.8 mM/sec at 1.5 T and 298 K.

278 .21-5.18 mm/sec 95% CI); and ADV was 0.88 mm/sec (0.38-1.38 mm/sec 95% CI).

279 uously move with a mean velocity of 0.56 mum/sec, suggesting that the movement is directional but not

280 recently found that (11)C-ER176 ((11)C-(R)-N-sec-butyl-4-(2-chlorophenyl)-N-methylquinazoline-2-carbo

281 Spike and burst indices (n/sec/channel) were computed across four 3-second time win

282 reactions including alkenes bearing nitrile, sec-amine, and thioether groups.

283 Consequently, the synthesis of sec-phosphine borane amino acids followed by their use i

284 ntified as spiroborates with two pentacyclic sec-butyl-trihydroxy-methyl-benzo[gh]tetraphen-one ligan

285 isition rate of the pipeline is 3 Gpixel per sec and the net rate is 600 Mpixel per sec with six micr

286 l per sec and the net rate is 600 Mpixel per sec with six microscopes running in parallel.

287 omelanin are redox-active, they can rapidly (sec-min) and repeatedly redox-cycle between oxidized and

288 er's Disease total score (beta = -3.23 score/sec(-1), P = .003) in participants with AD independent o

289 million [ppm]) and R2* (in inverse seconds [sec(-1)]) values of the dentate (case participants: 0.06

290 cing each residue in this motif of secretin (sec), Phe(6), Thr(7), and Leu(10), and cysteines incorpo

291 mice infected with the SL3261/surf or SL3261/sec strain generated large numbers of Th1 CD4(+) ESAT-6(

292 ses when infected with SL3261/surf or SL3261/sec, peak total serum IgG antibody titers were reached m

293 ed more rapidly in mice that received SL3261/sec.

294 ace-bound (SL3261/surf), or secreted (SL3261/sec) antigen.

295 osphination reaction of [60]fullerene by the sec-phosphine borane compounds was performed under PTC t

296 The configuration of the sec-butyl group is found to be (S).

297 Consistent with this, the sec-null mutant displayed reduced responses to GA and br

298 We also recorded the amount of time (sec) before a response was observed.

299 the unreacted enriched DAPS with lithium tri-sec-butylborohydride (commercially distributed as L-Sele

300 roximately 200 um) and real-time (10 volumes/sec) three-dimensional imaging, while further providing