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1 ation of ventral mesoderm and formation of a secondary axis.
2 ted into Xenopus embryos, CHL2 RNA induced a secondary axis.
3 on of CHL RNA into Xenopus embryos induced a secondary axis.
4 arly Xenopus embryo, Cngsc induces a partial secondary axis.
5 salize the ventral mesoderm, thus inducing a secondary axis.
6 1 class to induce goosecoid expression and a secondary axis.
7                  Dishevelled (Dsh) induces a secondary axis and can translocate to the membrane when
8  genes patterning the anthozoan aboral pole, secondary axis and endomesoderm support simplification o
9                       Finally, using Xenopus secondary axis and TCF/LEF (T Cell factor/lymphoid enhan
10 ought + heat treatments by 19-57 days, while secondary axis branches were less likely to produce need
11  form side-by-side conjoined twins, with the secondary axis developing as either the left or right si
12 n of xfz8 in ventral cells leads to complete secondary axis formation and can synergize with Xwnt-8 w
13 onical Wnt signaling and blocked Wnt-induced secondary axis formation in a dose-dependent manner, but
14 h reporter activity and beta-catenin-induced secondary axis formation in Xenopus embryos.
15 type Jade-1 in reducing beta-catenin-induced secondary axis formation in Xenopus laevis embryos in vi
16 nopus embryos, Gdf3 misexpression results in secondary axis formation, and induces morphogenetic elon
17 gesting that both 2d and 4d are required for secondary axis formation.
18  into the ventral marginal zone results in a secondary axis formation.
19 R synergistically increases the incidence of secondary axis formation.
20 lantation of the cumulus is able to induce a secondary axis in spiders.
21 servation that NvNoggin1 is able to induce a secondary axis in Xenopus embryos argues that N. vectens
22 ncer factor)-driven transcription and induce secondary axis in Xenopus embryos.
23        CKIepsilon mimicked Wnt in inducing a secondary axis in Xenopus, stabilizing beta-catenin, and
24                                          The secondary axis induced by XOs4 is distinct from that ind
25 ence; also, the presence of Tsg enhances the secondary axis-inducing activity of two products of chor
26 talytic subunit of PP2A (PP2A-C) potentiated secondary axis induction and Siamois reporter gene activ
27          Using epistasis tests and a Xenopus secondary axis induction assay, we show that TLD negates
28 s the response of animal caps to activin and secondary axis induction by smad2.
29 ikewise, Sox9 inhibits beta-catenin-mediated secondary axis induction in Xenopus embryos.
30  hDkk-2, hDkk-3 or Sgy, suppress Wnt-induced secondary axis induction in Xenopus embryos.
31                 By contrast, in an assay for secondary axis induction, extracellularly Wise antagonis
32  initiated twin formation and on whether the secondary axis is on the left or right side of the prima
33 n misexpressed ventrally, derriere induces a secondary axis lacking a head, an effect that is due to
34 , TMEM88 protein inhibits the formation of a secondary axis normally induced by Xdsh.
35 g BMP-4 signaling, leading to induction of a secondary axis on the ventral side of intact embryos and
36         However, endodermal tissue along the secondary axis originated from the host embryo.
37 piralian organizer to this general aspect of secondary axis patterning but highlight the significant
38 e Dpp/BMP2-4 pathway plays a central role in secondary axis patterning in many animals [1-11], but it
39 on may be indicative of their involvement in secondary axis patterning.
40 , the reconstituted embryos failed to form a secondary axis, suggesting that both 2d and 4d are requi
41 ed that neuroectoderm and mesoderm along the secondary axis were derived from the transplanted D quad
42 ntimorph, causing the formation of a partial secondary axis when expressed on the ventral side of the
43 bservations, ALK-4* is capable of inducing a secondary axis when injected into the ventral side of 32
44 enin-deficient micromeres failed to induce a secondary axis when transplanted to the animal pole of u
45 ation gradient, i.e. the potential to form a secondary axis, which is maximal in the head and is grad
46 s grafted into the body column, it induces a secondary axis, while the adjacent Cngsc(-) region has m
47 F hyperactivates Wnt signaling, developing a secondary axis with beta-catenin target gene upregulatio
48 which results in the formation of a complete secondary axis with head and eyes, did not cause the veg