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1 -dependent PPS14-specific IgG response after secondary immunization.
2 y CD8+ T cells that are robustly recalled by secondary immunization.
3 mory, but has no effect when injected during secondary immunization.
4 nto effector-memory CD8+ T cells following a secondary immunization.
5 her tissues that was strongly enhanced after secondary immunization.
6 majority of human donors, particularly after secondary immunization.
7 zation but enhanced antibody responses after secondary immunization.
8 e to make alphaGal-specific antibodies after secondary immunization.
9 were analyzed by ELISA following primary or secondary immunization.
10 A response when injected only at the time of secondary immunization.
11 e isotype switching and those resulting from secondary immunization.
12 T lymphocytes isolated following primary or secondary immunization.
13 This population expanded further on secondary immunization.
14 reduced in AP-1 transcription factors, after secondary immunization.
15 interferon (IFN)-gamma levels 1 d following secondary immunization.
16 B7-1 and B7-2 were observed upon primary and secondary immunizations.
17 e response was not observed after primary or secondary immunizations.
18 with MVA-Ag85A IMX313 after both primary and secondary immunizations.
20 Blockade of B7 costimulation at the time of secondary immunization also completely abrogated the est
23 mulation was required only briefly after the secondary immunization compared with after the primary i
24 that was dependent on CD4(+) T cells during secondary immunization, indicating that Pn14 primes for
26 (AFC) in the primary response, shortly after secondary immunization its memory cell progeny produce a
27 dy titers is apparent after both primary and secondary immunization of Aiolos(-)(/)(-) mice with a ra
28 y B cells and PCs failed to expand following secondary immunization of IL-21R.KO mice, and consequent
29 When weak boosting vectors were used for secondary immunization, pre-established CD8+ T cells wer
31 were lower, but they did show boosting upon secondary immunization to the levels achieved in the old
32 rimary kinetics and was highly boosted after secondary immunization, whereas the IgG anti-MCPS respon
33 s strikingly enhanced innate responses after secondary immunization, which was concurrent with enhanc
34 ar and humoral responses were uncoupled upon secondary immunization, which was dramatically affected
35 Foxp3(+)CD4(+) T cells into mice followed by secondary immunization with collagen accelerated the ons
37 accine demonstrated a booster response after secondary immunization with either the MCPS or the conju
38 GBS-III, depletion of CD4(+) T cells during secondary immunization with MenC or another Gram-negativ
41 on of flow-sorted basophils into mice before secondary immunization with PspA significantly protected
42 i-TNF-alpha MAb injected only at the time of secondary immunization with R36A failed to alter the boo
43 primary immunization with RSV was boosted by secondary immunization with RSV or with a chimeric recom
44 regulatory events and Ag presentation after secondary immunization with strong and weak boosting vec
45 92F strain of L. monocytogenes followed by a secondary immunization with wild-type L. monocytogenes r
46 18F strain of L. monocytogenes followed by a secondary immunization with wild-type L. monocytogenes r
47 tibacteriophage antibodies after primary and secondary immunizations with evidence of amplification a
48 educed IgG responses after either primary or secondary immunizations with sheep red blood cells (SRBC