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1 deactivation of sensory processing regions (secondary somatosensory cortex).
2 l operculum (i.e. the anatomical site of the secondary somatosensory cortex).
3 the bilateral insula, frontal operculum, and secondary somatosensory cortex.
4 s in the parietal operculum, the location of secondary somatosensory cortex.
5 roup 2) showed greater bilateral primary and secondary somatosensory cortex activation with greater d
6 e further demonstrate the specificity of the secondary somatosensory cortex and dorsal posterior insu
7 (P1) was recorded in parallel in the primary/secondary somatosensory cortex and posterior insula (app
8 e major targets are granular insular cortex, secondary somatosensory cortex and several cortical area
9 cupuncture induced greater activation at the secondary somatosensory cortex and stronger functional c
10 ion of ERP amplitude was source localized to secondary somatosensory cortex, and attributed to feedfo
11 ensory aspects of pain processing (thalamus, secondary somatosensory cortex, and posterior insula).
12 of feedback to rat S1: primary motor cortex, secondary somatosensory cortex, and secondary somatosens
13 many regions including primary motor cortex, secondary somatosensory cortex, anterior insula, and the
15 or cingulate cortex (ACC), left insula, left secondary somatosensory cortex, bilateral thalamus, and
16 o feedforward processing between primary and secondary somatosensory cortex by means of dynamic causa
17 in fMRI activity in the premotor cortex and secondary somatosensory cortex contralateral to the affe
19 ort the sensory components of physical pain (secondary somatosensory cortex; dorsal posterior insula)
20 s, arcuate hypothalamic nucleus, primary and secondary somatosensory cortex, ectorhinal cortex, and d
22 primary somatosensory cortex; in frontal and secondary somatosensory cortex, Fos expression was lower
23 the medial insula, medial cingulate cortex, secondary somatosensory cortex, frontal areas, and cereb
25 trate a double dissociation in the patient's secondary somatosensory cortex (increased responses to a
26 g induces robust bilateral activation of the secondary somatosensory cortex, insular cortex, prefront
27 recorded responses of single neurons in the secondary somatosensory cortex of monkeys suggest how th
31 ations that are significantly delayed across secondary somatosensory cortex, premotor, and motor area
33 ing ([Formula: see text]) ICPN compared with secondary somatosensory cortex-projecting ([Formula: see
34 lternative sites of stimulation, such as the secondary somatosensory cortex (rather than primary moto
36 nsely interconnected regions-the primary and secondary somatosensory cortex (S1 and S2)-in mice while
37 om distant sensorimotor areas, including the secondary somatosensory cortex (S2) and primary motor co
38 details of the location and organization of secondary somatosensory cortex (S2) are reported, and ev
40 alateral S1BC, primary motor cortex (M1) and secondary somatosensory cortex (S2) may underlie benefic
41 ions of biocytin into head and limb areas of secondary somatosensory cortex (S2) produced heavy label
42 Here we report that higher-order area 1 and secondary somatosensory cortex (S2) underwent similar sp
43 reas 3b, 3a, 1 and 2, parietal ventral (PV), secondary somatosensory cortex (S2), and primary motor c
44 lar that of deep layer excitatory neurons of secondary somatosensory cortex (S2), contains informatio
45 n area 3b; feedback connections from area 1, secondary somatosensory cortex (S2), parietal ventral ar
50 ect corticocortical pathway was interrupted, secondary somatosensory cortex showed robust activity in
51 ysiological studies suggest that primary and secondary somatosensory cortex (SI and SII, respectively
52 ions, i.e., primary somatosensory cortex SI, secondary somatosensory cortex SII, posterior parietal c
53 ynchronous firing of pairs of neurons in the secondary somatosensory cortex (SII) of three monkeys tr
54 splayed significant increases in ipsilateral secondary somatosensory cortex (SII), although the magni
55 the white matter of the ipsilesional SI and secondary somatosensory cortex (SII), and in the contral
56 bilateral activation was demonstrated in the secondary somatosensory cortex (SII), indicating a bilat
57 mponent, produced little or no activation in secondary somatosensory cortex (SII), whereas light brus
58 tary motor area activity and deactivated the secondary somatosensory cortex, specifically in response
59 mus, the posterior and anterior insulae, the secondary somatosensory cortex, the anterior cingulate c
61 cally inhibiting the thalamus, activation in secondary somatosensory cortex was eliminated, with a su
62 terior cingulate cortex (ACC), amygdala, and secondary somatosensory cortex were all significantly mo
63 eflecting target detection are restricted to secondary somatosensory cortex, whereas activity in insu
64 roelectrodes from the hand region of macaque secondary somatosensory cortex while vibrotactile stimul