ライフサイエンスコーパス: sedentary

1 extremely inactive (PAL, <1.40), 26 (34.6%) sedentary (1.40 <= PAL <= 1.69), and 22 (29.3%) active (

2 subjects displaying a higher-than-predicted sedentary 24h-EE lost significantly more fat during the

3 ng, although it was modestly reduced in aged sedentary (-7%) and running (-19%) mice without satellit

4 MTG content in type I fibres (trained: +62%, sedentary: +79%; P < 0.05) but did not affect PLIN prote

5 tasks as expected or unexpectedly performed sedentary activities.

6 passive dispersal of gametes, or sluggish or sedentary adult life habits in the absence of gamete vec

7 Compared with less-sedentary adults (6 sedentary h/d), those who spent 10 s

8 The present analysis examined 20 previously sedentary adults from the HERITAGE Family Study who comp

9 -month randomized controlled trial among 278 sedentary adults with abdominal obesity (75%) or dyslipi

10 rcise on adult neurogenesis and cognition to sedentary aged mice.

11 hat likely drove the Neolithic transition to sedentary agrarian societies in the Fertile Crescent mor

12 stence patterns, from a hunter-gatherer to a sedentary agricultural lifestyle, which ultimately resul

13 for an object location memory task, whereas sedentary and adol ELE mice did not.

14 andy, and sweetened beverages and time spent sedentary and in moderate-to-vigorous physical activity.

15 ins-of migration might promote speciation if sedentary and migratory populations become reproductivel

16 year from which we derived daily minutes in sedentary and moderate-to-vigorous physical activity (MV

17 isrupt skeletal muscle insulin signalling in sedentary and obese individuals.

18 e been associated with obesity phenotypes in sedentary and obese populations, but rarely with skeleta

19 ntify intermediary metabolism fluxes in both sedentary and treadmill-running mice.

20 ining) and from different fathers to compare sedentary- and runner-father progenies.

21 neurons immunoreactive for GABA(Aalpha1) in sedentary animals.

22 the association between objectively measured sedentary behavior (its total volume and accrual in prol

23 earlier sleep onset predicted more next-day sedentary behavior (p < 0.0001), and nights with later s

24 Mean daily steps, hours/day in sedentary behavior (SB) and minutes/week in moderate-to-

25 sical Activity Guidelines recommend reducing sedentary behavior (SB) for cardiovascular health.

26 stimated associations of quartiles of steps, sedentary behavior (SB), and moderate-to-vigorous intens

27 ween sleep and daytime physical activity and sedentary behavior among adolescents from the Fragile Fa

28 ity of either intensity and higher levels of sedentary behavior and also a poor diet quality score (c

29 urement that are providing new insights into sedentary behavior and health.

30 Sedentary behavior and physical inactivity are among the

31 Greater efforts at preventing sedentary behavior and physical inactivity while promoti

32 rt review, we discuss the negative impact of sedentary behavior and physical inactivity, as well as t

33 e evidence that changes in physical activity/sedentary behavior can be maintained long term in indivi

34 Sedentary behavior emerged as an independent predictor o

35 Such evidence has led to new sedentary behavior guidelines and initiatives.

36 In large-scale cohort studies, sedentary behavior has been routinely measured using sel

37 e find that objectively logged reductions in sedentary behavior in the following month are linked to

38 th increasing physical activity and reducing sedentary behavior may attenuate genetic associations wi

39 The degree of observed sedentary behavior post-HTx is surprising, with the majo

40 On days with more sedentary behavior than an individual's average, sleep o

41 tudy, we explored the associations of PA and sedentary behavior with all-cause and cardiovascular dis

42 the association of prolonged, uninterrupted sedentary behavior with glycemic biomarkers in a cohort

43 interaction on obesity, yet the influence of sedentary behavior, distinct from a lack of physical act

44 Parity was associated with sedentary behavior, weight gain, and higher triglyceride

45 howed significant interactions with MVPA and sedentary behavior, with effects on BMI and other adipos

46 om addressing either or both component(s) of sedentary behavior.

47 maintenance efficiency, with daily MVPA and sedentary behavior.

48 tation-related PA, and total sitting time as sedentary behavior.

49 ed by unmeasured factors such as the type of sedentary behavior.

50 tion and acceleration to capture free-living sedentary behavior.

51 ental evidence on the health consequences of sedentary behavior; second, we describe solutions-focuse

52 Sedentary behaviors and physical inactivity are not only

53 Meal contexts and sedentary behaviors may represent important intervention

54 on (the Hadza of Tanzania) to understand how sedentary behaviors occur in a nonindustrial economic co

55 iseases, body mass index, diabetes, smoking, sedentary behaviors, and alcohol intake.

56 ssages encouraging exercise and discouraging sedentary behaviors.

57 or 7 d, and self-reported meal practices and sedentary behaviors.

58 behaviors in adults; interventions to reduce sedentary behaviors; and the harms of behavioral counsel

59 f-management strategies are needed to reduce sedentary behaviour and enhance motor function.

60 ere depletion of natural stocks, the species sedentary behaviour and possible sampling biases.

61 Obesity and sedentary behaviour are associated with capillary rarefa

62 cioeconomic context of physical activity and sedentary behaviour at an early stage in life.

63 ic support for the hypothesis that increased sedentary behaviour by leisure television watching is a

64 ility (C. cinereum and S. magellanicus), and sedentary behaviour by two terrestrial-foraging species

65 al activity is inversely associated with and sedentary behaviour is positively (and independently) as

66 The lifestyle factors of physical activity, sedentary behaviour, and diet are increasingly being stu

67 beverages and unhealthy snacks, screen-based sedentary behaviour, and physical activity in the interv

68 negative direct effects on smoking, BMI and sedentary behaviour.

69 ng dissociated from the effects of increased sedentary behaviour.

70 gh genetic correlations are observed between sedentary behaviours and neurological traits, including

71 genetic determinants associated with leisure sedentary behaviours and to estimate the potential causa

72 Leisure sedentary behaviours are associated with increased risk

73 omising use of strategies that aim to reduce sedentary behaviours as therapy to improve metabolic fle

74 c flexibility, while physical inactivity and sedentary behaviours trigger a state of metabolic 'infle

75 social isolation, low physical activity, and sedentary behaviours.

76 alth, health behaviours and a range of other sedentary behaviours.

77 yer in the transition between planktonic and sedentary biofilm-associated bacterial lifestyles.

78 60 to 4.30]; P for trend < 0.001) and longer sedentary bout duration (HR, 1.03 [CI, 0.67 to 1.60]; HR

79 High total sedentary time or high sedentary bout duration alone were not associated with d

80 ; however, a significant interaction between sedentary bout duration and total sedentary time was obs

81 oint association of total sedentary time and sedentary bout duration showed that participants in the

82 derate to vigorous physical activity, longer sedentary bout duration was dose-dependently associated

83 Average daily sedentary time and mean sedentary bout duration were the exposures of interest.

84 tics (ie, high total sedentary time and high sedentary bout duration) had the highest levels of homeo

85 entary time; 7.7, 9.6, and 12.4 min/bout for sedentary bout duration) in models that included moderat

86 errupted sedentariness was expressed as mean sedentary bout length.

87 neficial association for replacing prolonged sedentary-bout time with LIPA and MVPA but not for repla

88 nd MVPA but not for replacement with shorter sedentary bouts (per 30 minutes, HR = 1.00; 95% CI: 0.96

89 acing prolonged sedentary bouts with shorter sedentary bouts is associated with reductions in all-cau

90 These findings suggest short sedentary bouts still carry mortality risk and are not a

91 ity (LIPA or MVPA) or 2) replacing prolonged sedentary bouts with shorter sedentary bouts is associat

92 s potentially most hazardous form (prolonged sedentary bouts) to impart health benefit.

93 are not a healthful alternative to prolonged sedentary bouts.

94 Females were bred with chow-fed sedentary C57BL/6 males.

95 10 weeks of exercise training or remained as sedentary, cage-controls.

96 hat participants classified as high for both sedentary characteristics (high sedentary time [>/=12.5

97 participants in the upper quartile for both sedentary characteristics (ie, high total sedentary time

98 esistance (P<0.001 versus low group for both sedentary characteristics) and 2-hour glucose (P=0.002 v

99 r glucose (P=0.002 versus low group for both sedentary characteristics).

100 pathological remodeling and heart failure in sedentary, chronically hypertensive wild-type mice.

101 Sedentary conditions did not significantly affect the nu

102 e (WT, n = 10) littermates under exercise or sedentary conditions were profiled by sequencing rRNA op

103 Here, we tested the hypothesis that sedentary conditions, a major risk factor for CVD, incre

104 a standard environment constituting close to sedentary conditions.

105 y and sympathoinhibitory responses following sedentary conditions.

106 in young competitive athletes compared with sedentary controls, but rarely >40 mm.

107 When compared with sedentary controls, mice that performed a four-week trea

108 atically elevated in the heart compared with sedentary controls.

109 nd FM have systemic hyperalgesia compared to sedentary controls.

110 rved in exercised rodent muscles compared to sedentary controls; however, exercise-induced Mdm2 phosp

111 High fat-fed sedentary dams produced female offspring with impaired g

112 state switching between active (roaming) and sedentary (dwelling) states; however, when provided with

113 teractions between these plant-reprogramming sedentary endoparasites and their infected hosts, focusi

114 Ns belong to a small group of root-infecting sedentary endoparasites that includes cyst and root-knot

115 n roots is less obvious than infestations of sedentary endoparasites; nevertheless, in many instances

116 Sedentary endoparasitic nematodes, root-knot and cyst ne

117 , and sleeping metabolic rate (SMR) and 24-h sedentary energy expenditure (24h-EE) were measured befo

118 behaviour) with respect to wheel running and sedentary female Wistar rats at 8 and 14 weeks of age.

119 ntary (third tertile) versus less time spent sedentary (first tertile) (beta = 0.73 kg/m(2) [SE, 0.10

120 d tetanic and specific force compared to the sedentary group.

121 he all-cause mortality HR comparing the most sedentary (>8 h/day) to the least sedentary (<4 h/day) g

122 adults (6 sedentary h/d), those who spent 10 sedentary h/d had 29% greater risk (HR: 1.29; 95% CI: 1.

123 Compared with less-sedentary adults (6 sedentary h/d), those who spent 10 sedentary h/d had 29%

124 s to have long-term stable distributions and sedentary habits.

125 l (LA) mechanical and electric remodeling in sedentary, healthy middle-aged adults.

126 th of the metabolic heat flux expected for a sedentary individual and can also modulate localized cha

127 We recruited nine healthy sedentary individuals free of any cardiopulmonary diseas

128 The inherent inaccessibility of sweat in sedentary individuals in large volume (>/=10 microL) for

129 lipid storage in muscle between trained and sedentary individuals is unknown.

130 By contrast, in sedentary individuals, lipid infusion only increased the

131 lipid storage in muscle between trained and sedentary individuals.

132 ry artery calcium (CAC) scores compared with sedentary individuals.

133 content in type I fibres of both trained and sedentary individuals.

134 in content was higher in trained compared to sedentary individuals.

135 e of smoking (42.6% to 36.5%; RR = 0.86) and sedentary lifestyle (43.4% to 29.0%; RR = 0.69) declined

136 lic syndrome, smoking, hyperlipidemia, and a sedentary lifestyle are the major modifiable risk factor

137 ts combined with chronic overnutrition and a sedentary lifestyle in Western societies evokes a state

138 A sedentary lifestyle is associated with increased risk fo

139 oking history was predictive of [50] CAD and sedentary lifestyle of [70] CAD.

140 Self-reported sedentary lifestyle was associated with less diary use i

141 moking, poor diet, elevated body mass index, sedentary lifestyle) and three major risk factors (hyper

142 ors including body mass index, healthy diet, sedentary lifestyle, alcohol consumption, smoking, and u

143 Poor nutrition, sedentary lifestyle, and obesity are among the primary r

144 and cardiovascular disease, such as smoking, sedentary lifestyle, and obesity, there are opportunitie

145 A sedentary lifestyle, chronic inflammation and leukocytos

146 This review concludes that a sedentary lifestyle, obesity and ageing impair the vasod

147 Other modifiable risk factors include sedentary lifestyle, smoking, obesity, diabetes mellitus

148 isk factors are obesity, family history, and sedentary lifestyle.

149 d to prevent falls, and reduce smoking and a sedentary lifestyle.

150 e change puts them at risk for sequelae of a sedentary lifestyle: weight gain, hypertension, hyperlip

151 e to risk factors such as unhealthy diet and sedentary lifestyle; limited access to risk-reducing beh

152 An aging global population combined with sedentary lifestyles and unhealthy diets has contributed

153 , low socioeconomic position, poor diet, and sedentary lifestyles.

154 and sedentary populations) exceeded those of sedentary lineages.

155 g the most sedentary (>8 h/day) to the least sedentary (<4 h/day) groups was 1.52 (95% confidence int

156 dominantly calcific plaques (72.7%), whereas sedentary males showed predominantly mixed morphology pl

157 ston units or coronary plaques compared with sedentary males with a similar risk profile.

158 (44.3% versus 22.2%; P=0.009) compared with sedentary males, and only male athletes showed a CAC >/=

159 The majority of sedentary marine species are pelago-benthic, meaning the

160 These results suggest that remaining sedentary may reduce neurofunctional readiness for top-d

161 tients spent a smaller proportion of the day sedentary (median, 12% vs. 23%) or lying down (median, 1

162 isms for plaque formation in athletic versus sedentary men.

163 y infusion of serum from exercised mice into sedentary mice and depends on AMPK-mediated signaling in

164 lar dysfunction across the adult lifespan in sedentary mice consuming a non-Western diet, and the und

165 High-fat diet in sedentary mice led to endoplasmic reticulum stress and a

166 n miRNA content, and their administration to sedentary mice reproduces the improvement of glucose tol

167 bates vascular ageing across the lifespan in sedentary mice They also show that lifelong voluntary ae

168 -133a and miR-133b Importantly, treatment of sedentary mice with exosomes isolated from the plasma of

169 and restored youthful wound closure in old, sedentary mice.

170 ons improve arterial stiffness in previously sedentary middle-aged and older men and postmenopausal w

171 Trained (n = 11) and sedentary (n = 10) subjects, matched for age, sex and bo

172 Sedentary nutrient fluxes in the postabsorptive state we

173 biopsies were taken from eight Trained, Lean sedentary, Obese and T2DM subjects.

174 ealth, television-viewing time, and having a sedentary occupation were associated with higher device-

175 wn about the inheritance of these effects to sedentary offspring and the mechanisms involved.

176 Thirty-two sedentary, older adults were randomized to receive eithe

177 1) and GABA(Aalpha2) in the RVLM/RVLM(RE) of sedentary or physically active (10-12 weeks of wheel run

178 e-clip surgery) wild-type and alphaCGRP(-/-) sedentary or voluntary wheel running mice were treated w

179 In these previously sedentary, otherwise healthy middle-aged adults, a 2 yea

180 In these previously sedentary, otherwise healthy, middle-aged adults, 2 year

181 The largest benefits occurred between sedentary patient groups and between those with the high

182 clinics in Rome, 300 physically inactive and sedentary patients with type 2 diabetes were randomized

183 l feasibility studies in extremely unfit and sedentary people.

184 ex and hippocampal formation throughout of a sedentary period of rats who were physically active duri

185 rtical and hippocampal neurons even during a sedentary period of rats.

186 carbohydrates and unsaturated fat, prolonged sedentary periods or limited physical activity have majo

187 Dnmt3a in hypothalamic AgRP neurons causes a sedentary phenotype characterized by reduced voluntary e

188 lux), and 80.03 +/- 2.11% during periods of sedentary physical activity (<320 counts per minute).

189 reduction in health status observed in early sedentary populations without the need to invoke factors

190 ges (i.e., species having both migratory and sedentary populations) exceeded those of sedentary linea

191 species stop migrating and establish founder sedentary populations, a phenomenon documented in birds

192 between 3 types of near visual activities in sedentary posture, namely reading (< 0.5, 0.5-0.9, >=1.0

193 ctivity that is different from chair-sitting sedentary postures used in industrialized populations.

194 olic demands, but was a poor predictor for a sedentary predator that operated well below its energeti

195 pine density was lower in 14- vs. 8-week-old sedentary rats (P = 0.03).

196 Western blot analyses indicated that sedentary rats had lower expression of GABA(Aalpha1) and

197 Sedentary rats had significantly reduced expression of t

198 In aged sedentary rats, compared to young sedentary rats, a 42% increase in IVRT, a 64% decrease i

199 In aged sedentary rats, compared to young sedentary rats, a 42%

200 ins from 8 to 14 weeks of age in running and sedentary rats.

201 alysed by quantitative RT-PCR in age-matched sedentary rats.

202 y GABA(Aalpha2) was lower in the RVLM(RE) of sedentary rats.

203 ecifically, we examined whether total HRV at sedentary rest (measured as the SD of normal-to-normal i

204 teraction between Arabidopsis plants and two sedentary root-parasitic nematode species, the cyst nema

205 gh-sucrose Western diet (WD) in the absence (sedentary, SED) or presence (voluntary wheel running, VW

206 We found that large late hunter-gatherer sedentary settlements in the Levant, c.

207 the transition from hunting and gathering to sedentary societies, laying the foundation for the devel

208 cale, supporting the cookie cutter model for sedentary species, and suggesting that habitat change du

209 species is dramatically larger than for more sedentary species.

210 s an intermediate step between migratory and sedentary states in this family.

211 Because migratory and sedentary strategies involve alternative physiological,

212 ning in preschoolers and diverse adults from sedentary subjects to elite marathoners, totaling 63 sub

213 ither performed regular exercise or remained sedentary their entire lives (average age 62 years).

214 = 0.005), and in those with more time spent sedentary (third tertile) versus less time spent sedenta

215 us physical activity ( P=0.01) and increased sedentary time ( P=0.004).

216 for dominant wrist based on ENMO to classify sedentary time (<50 mg), light PA (50-110 mg), moderate

217 0.5-1.1]; P < .001), and 10.9 vs 11.7 h/d of sedentary time (difference, -0.8 [95% CI, -1.0 to -0.5];

218 multivariable-adjusted models, greater total sedentary time (HR, 1.22 [95% CI, 0.74 to 2.02]; HR, 1.6

219 ociated with less MVPA (p < 0.0001) and less sedentary time (p < 0.0001, p = 0.004) the next day.

220 igh for both sedentary characteristics (high sedentary time [>/=12.5 h/d] and high bout duration [>/=

221 endently associated with weight regain were: sedentary time [2.9% (1.2-4.7), for highest vs lowest qu

222 thropometric outcomes, physical activity and sedentary time among adolescents were objectively measur

223 Dose-response associations of sedentary time and bout duration with CVD were linear (P

224 n device (SitFIT) allowed self-monitoring of sedentary time and daily steps, and a game-based app (Ma

225 ffect suggests that the relationship between sedentary time and DMC may be moderated by unmeasured fa

226 th sedentary characteristics (ie, high total sedentary time and high sedentary bout duration) had the

227 Both the total volume of sedentary time and its accrual in prolonged, uninterrupt

228 e of activity that should be substituted for sedentary time and its potentially most hazardous form (

229 Women jointly classified as having high sedentary time and long bout durations had significantly

230 Both high sedentary time and long mean bout durations were associa

231 The association between sedentary time and LTL was evaluated using multiple line

232 Average daily sedentary time and mean sedentary bout duration were the

233 ildren (49.1% males; 50.9% females) in which sedentary time and physical activity were measured with

234 Primary outcome (objectively measured sedentary time and physical activity) measurements were

235 Evaluation of the joint association of total sedentary time and sedentary bout duration showed that p

236 1.34; CI=1.08-1.65) than women with both low sedentary time and short bout duration.

237 ve associations between objectively measured sedentary time and subcomponents of physical activity wi

238 The association between sedentary time and the risk for CVD is nonlinear with an

239 ew studies have examined whether patterns of sedentary time are associated with higher risk for CVD.

240 lf-reporting to evaluate the total volume of sedentary time as a prognostic risk factor for mortality

241 howed a baseline-adjusted mean difference in sedentary time at 12 months of -1.6 minutes/day (97.5% c

242 arkers of cardiometabolic health, but not in sedentary time at 12 months.

243 ictors of lower physical activity and higher sedentary time from childhood to adulthood.

244 y) vs. the lowest (<= ~9 hr/day) quartile of sedentary time had higher risk for CVD (HR=1.62; CI=1.21

245 e highest quartile of accelerometer-measured sedentary time had significantly shorter LTL than those

246 ) programme to improve physical activity and sedentary time in male football fans, delivered through

247 Accruing sedentary time in prolonged, uninterrupted bouts may be

248 cise fall far short of replacing most of the sedentary time in the modern lifestyle, because both the

249 s, low levels of physical activity, and high sedentary time increase the risk of cardiovascular disea

250 es have examined whether the manner in which sedentary time is accrued (in short or long bouts) carri

251 ave not examined whether the manner in which sedentary time is accrued (in short or long bouts) carri

252 Evidence that higher sedentary time is associated with higher risk for cardio

253 Excessive sedentary time is ubiquitous in developed nations and is

254 Excessive sedentary time is ubiquitous in Western societies.

255 basis of accelerometer measurements, higher sedentary time may be associated with shorter LTL among

256 High total sedentary time or high sedentary bout duration alone wer

257 This can explain why high amounts of sedentary time produce distinct metabolic and cardiovasc

258 How daily physical activity and sedentary time relate to human judgement and functional

259 ate-to-vigorous physical activity (MVPA) and sedentary time to decision-making competence (DMC) in yo

260 ines should target reducing and interrupting sedentary time to reduce risk for death.

261 Sedentary time was independently associated only with hi

262 Sedentary time was measured by accelerometry between 200

263 Sedentary time was measured using a hip-mounted accelero

264 ured at baseline and at follow-up 7 mo later.Sedentary time was not associated with any of the indivi

265 Accelerometer-measured sedentary time was not associated with LTL in women abov

266 Self-reported sedentary time was not associated with LTL.

267 Sedentary time was objectively measured with a hip-mount

268 on between sedentary bout duration and total sedentary time was observed.

269 Sedentary time was related to lower susceptibility to a

270 Energy intake, HPA, and sedentary time were measured with the use of a 3-d weigh

271 f objectively-measured physical activity and sedentary time with body composition outcomes at 30 year

272 tion with mortality risk for replacing total sedentary time with both LIPA (per 30 minutes, hazard ra

273 Few studies have assessed the association of sedentary time with leukocyte telomere length (LTL).

274 niques to examine whether 1) replacing total sedentary time with light-intensity or moderate to vigor

275 of accelerometer-measured and self-reported sedentary time with LTL in a sample of 1,481 older white

276 ugh safely replacing a large amount of daily sedentary time with physical activity in everyone, regar

277 sessions aimed to improve physical activity, sedentary time, and diet and maintain changes long term.

278 in total or in 10-min bouts or more), daily sedentary time, body-mass index, or fat mass percentage

279 study identified several behaviors (eg, more sedentary time, eating fast food, binge eating, eating c

280 adjusting for WC.Physical activity, but not sedentary time, is prospectively associated with cardiom

281 to vigorous-intensity physical activity, and sedentary time, measured by an accelerometer.

282 ) higher susceptibility to framing with more sedentary time, mediated through lower local and global

283 diet, increased physical activity, decreased sedentary time, or a combination of these among adults w

284 .015, 95%CI: -0.021; -0.009), independent of sedentary time.

285 to mitigate the mortality risks incurred by sedentary time.

286 o predict lower physical activity and higher sedentary time.

287 etabolic risk factors that is independent of sedentary time.

288 ntake, habitual physical activity (HPA), and sedentary time.

289 east moderate intensity rather than reducing sedentary time.

290 ng status, deprivation, body mass index, and sedentary time.

291 2) lower susceptibility to framing with more sedentary time.

292 ets, cereals, animal and dairy products, and sedentary time.

293 ncrease in physical activity and decrease in sedentary time.

294 s willing to pay pound 1,800 per minute less sedentary time/day, and 0.13 probability if society is w

295 nts: 689.7, 746.5, and 799.4 min/d for total sedentary time; 7.7, 9.6, and 12.4 min/bout for sedentar

296 e associations were not independent of total sedentary time; however, a significant interaction betwe

297 repeat expansion shifted yeast cells from a sedentary to a buoyant state, thereby increasing their e

298 support a model in which the transition from sedentary to light activity is associated with an increa

299 pinally projecting C1 and non-C1 neurons and sedentary versus physically active conditions.

300 rous physical activity (MVPA) and time spent sedentary with genetic variants on obesity among 9,645 U