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1 o identify children with active "epilepsy or seizure disorder".
2 ypothyroidism to 3.45; 95% CI, 2.73-4.35 for seizure disorder).
3 f phenytoin, which had been administered for seizure disorder.
4 chiatric symptoms that are concurrent with a seizure disorder.
5 GI1-Ab-E represents a clinically distinctive seizure disorder.
6 reatment for fatigue, or had an uncontrolled seizure disorder.
7 lepsy is a common and frequently intractable seizure disorder.
8 tal delay and an intractable infantile-onset seizure disorder.
9 additional subjects, including one without a seizure disorder.
10 al disability, gastroesophageal reflux and a seizure disorder.
11  pharmacologic and surgical treatment of the seizure disorder.
12 relation has on the management of the actual seizure disorder.
13  pharmacologic and surgical treatment of the seizure disorder.
14  suggest that the deaths were related to the seizure disorder.
15 y could help identify novel therapeutics for seizure disorder.
16 sychoses to supernatural causes, followed by seizure disorder.
17 ndicating functional interactions leading to seizure disorder.
18 ted with a two-fold elevated risk for a late seizure disorder.
19  motor and cognitive impairment severity and seizure disorder.
20 v7/KCNQ channels are linked to a spectrum of seizure disorders.
21 nderstanding of the pathophysiology of human seizure disorders.
22 of therapeutic potential in the treatment of seizure disorders.
23 epileptic drugs have been developed to treat seizure disorders.
24 ion of structural abnormalities that underly seizure disorders.
25 d nausea and vomiting, and certain pediatric seizure disorders.
26 her a surgical treatment could relieve their seizure disorders.
27 r efficacy in children and generalized-onset seizure disorders.
28 ntial for Nav1.1 as a therapeutic target for seizure disorders.
29 tal and epileptic encephalopathies and other seizure disorders.
30 ng the role of GABA(B)Rs in the treatment of seizure disorders.
31 ntal and epileptic encephalopathy associated seizure disorders.
32  (neurocysticercosis) is a frequent cause of seizure disorders.
33 telemetry of epileptic events in humans with seizure disorders.
34    Epileptic spasms are a hallmark of severe seizure disorders.
35 y prescribed to treat anxiety, insomnia, and seizure disorders.
36 se suggest that SCN8A may also contribute to seizure disorders.
37 antiepileptogenic drugs ameliorating TBCID24 seizure disorders.
38 interactions in the genomes of patients with seizure disorders.
39 e a therapeutic benefit for the treatment of seizure disorders.
40 egy for correcting post-traumatic memory and seizure disorders.
41 cularly vulnerable to dysfunction leading to seizure disorders.
42 her therapeutic approach to the treatment of seizure disorders.
43 some spastic quadriplegic cerebral palsy and seizure disorders.
44 e of the pump in human neurodegenerative and seizure disorders.
45  genetic involvement of GABA(A) receptors in seizure disorders.
46  in investigations of the pathophysiology of seizure disorders.
47 pooled CNV data from 10,590 individuals with seizure disorders, 16,109 individuals with clinically va
48 , perinatal complications (4.34, 3.21-5.81), seizure disorders (2.90, 2.24-3.77), and house status (0
49 tal retardation 1.9% versus 1.3% (1.48); and seizure disorders 4.0% versus 1.6% (2.00).
50         Transgenic mice also had a grand mal seizure disorder accompanied by a corresponding dysplasi
51 ific neurodevelopmental delay with co-morbid seizure disorder accounting for 33.3%, 14.8%, 18.5%, 7.4
52                        Epilepsy, a recurrent seizure disorder affecting 1% of the population, can be
53           Current drugs for the treatment of seizure disorders, although effective in many patients,
54 isations was 0.89 (95% CI, 0.86 to 0.93) for seizure disorder and 0.32 (95% CI 0.31 to 0.34) for TGA.
55 articipants, one with a pre-existing complex seizure disorder and another who experienced oral surger
56 nsporter KCC2 generates mice with a profound seizure disorder and confirms the central role of this t
57          Comorbid conditions identified were seizure disorder and obesity (2 cases each).
58 /- mice manifest two key phenotypes: a fatal seizure disorder and retarded growth.
59 iable target for therapeutic intervention in seizure disorders and antiepileptogenesis.
60 could be a novel therapeutic target to treat seizure disorders and epilepsy.SIGNIFICANCE STATEMENT We
61 athogenesis for FIRES and other inflammatory seizure disorders and may provide a valuable biomarker f
62 intervention in obesity, metabolic syndrome, seizure disorders and other neurodegenerative diseases,
63 identified pathway in the pathophysiology of seizure disorders and provide evidence for a more genera
64 activity disorder, autism spectrum disorder, seizure disorder) and neurodegenerative (fragile X-assoc
65 d ventriculoperitoneal shunts, 36% developed seizure disorders, and 20% developed severe ototoxicity.
66                         Correlating with the seizure disorder are enhanced hippocampal levels of neur
67                                        Human seizure disorders are a major health concern due to the
68 vere myoclonic epilepsy of infancy, a severe seizure disorder associated with mutations of the sodium
69  that underlie the M-channel cause the human seizure disorder benign familial neonatal convulsions (B
70                                              Seizure disorders, burn marks, and respiratory problems
71     Temporal lobe epilepsy (TLE) is a common seizure disorder, but the underlying molecular mechanism
72  sodium current, which is a common defect in seizure disorders, cardiac arrhythmias skeletal muscle m
73  focal epilepsy (LFE) is a common and severe seizure disorder caused by epileptogenic lesions, includ
74                  The concept of epilepsy and seizure disorders caused by autoantibodies to specific n
75 eizures recorded over a 6-year period at the Seizure Disorder Center at University of California, Los
76 th multiple neurologic conditions, for which seizure disorders comprise the largest group.
77                                              Seizure disorders debilitate more than 65,000,000 people
78  syndromes, traumatic nerve/muscle injuries, seizure disorders, decreased cognitive ability, poor pul
79                                Patients with seizure disorders expressed pain more vocally.
80   Developmental epilepsies are age-dependent seizure disorders for which genetic causes have been inc
81 d speech delay, elevated BMI, short stature, seizure disorders, gait disturbance, and tremors.
82 le spasms, which comprise a severe infantile seizure disorder, have a high morbidity and are difficul
83                                           In seizure disorders, his name is linked to the first descr
84 elopmental delay both in 19.2% (20/104), and seizure disorder in 17.3% (18/104).
85 l lobe epilepsy (TLE), the most common focal seizure disorder in adults, can be instigated in experim
86 Temporal lobe epilepsy is the most prevalent seizure disorder in adults.
87         Febrile seizures are the most common seizure disorder in childhood, associated with a signifi
88 ity, and mutations in these channels cause a seizure disorder in humans.
89 tifying the gene associated with a monogenic seizure disorder in mice, which may ultimately lead to a
90                                    The fatal seizure disorder in Pcmt1-/- mice can be mitigated but n
91 (ID), distinctive behavioral features, and a seizure disorder in two cases.
92 ctivated glia, in the study and treatment of seizure disorders in humans.
93                             The emergence of seizure disorders in old age places an increasing burden
94 ene disorders accounted for a quarter of the seizure disorders in this cohort.
95                                  Epilepsy or seizure disorder is among the least understood chronic m
96 s, but its overall applicability in treating seizure disorders is limited.
97 ion may cause brain abnormalities, including seizure disorders, is unclear.
98 al effects caused by cerebral hemorrhages or seizure disorders, keeps clinicians alert to any improve
99 luding low birth weight, maternal education, seizure disorder, kidney disease duration, and genetical
100 lso been implicated in neurodegenerative and seizure disorders, making RyR2 an attractive therapeutic
101                       Some encephalitides or seizure disorders once thought idiopathic now seem to be
102 ible to unravel whether CVI is caused by the seizure disorder or increased intracranial pressure or b
103  of the tumour leads to focal or generalised seizure disorders or neurological deficits caused by com
104 fter were done to identify children with new seizure disorders or other neurodisabilities.
105  children, with Iraqis more likely to have a seizure disorder (OR: 7.6, 95% CI 3.8-15.0, p < 0.001).
106 nsory perception, behavioural abnormalities, seizure disorders, or a combination of these features.
107  were children with developmental disorders, seizure disorders, or both undergoing GWS between 2014 a
108          Two-thirds of patients had a severe seizure disorder, placing EPG5 within the rapidly expand
109                                              Seizure disorders present an attractive gene therapy tar
110         We aimed to assess the prevalence of seizure disorders, psychoses, and mental retardation in
111 nts (1 day-25 years), 13 children with other seizure disorders receiving B6 supplementation (1 month-
112 nriched in genes associated with cardiac and seizure disorders relative to controls (odds ratio = 9.7
113                                        Human seizure disorders represent a heterogeneous collection o
114 n pathogenesis is suggested by high rates of seizure disorder; research has highlighted the role of s
115 neurologic dysfunction in association with a seizure disorder, resulting in a 1-y period of behaviora
116                     With the hypothesis that seizure disorders share genetic risk factors, we pooled
117  significant loci, 22 of which are novel for seizure disorders, such as deletions at 1p36.33, 1q44, 2
118 get conditions (chronic respiratory failure, seizure disorder, supplemental nutritional dependence, a
119 gical evidence from several animal models of seizure disorder that adenosine possesses endogenous ant
120  double knock-out mice display a progressive seizure disorder that dramatically reduces their median
121 neuronal RNA-binding protein, have a complex seizure disorder that includes both convulsive and non-c
122       Infantile spasms is a severe infantile seizure disorder that is difficult to treat and has a hi
123 s a promising target for further research on seizure disorder therapeutics.
124  been associated with a poor response of the seizure disorder to pharmacotherapy and epilepsy surgery
125                                          New seizure disorder was less common (4.6% vs 6.0%; OR, 0.77
126               To investigate the etiology of seizure disorders, we have used a group of Drosophila mu
127                          Cases with comorbid seizure disorder were excluded from the study.
128 atory syndrome in children, and pre-existing seizure disorder were independently associated with a gr
129 ystem inflammatory syndrome in children, and seizure disorder were independently associated with ICU
130                                              Seizure disorders were noted in 46 (0.3%) children, with
131                         Delayed bone age and seizure disorders were overrepresented in the TTDN1 grou
132                                              Seizure disorders were reported in 25% of patients, incl
133 7.4, and in serum samples from patients with seizure disorders who were treated with phenytoin or car
134 oside biosynthesis, result in an early-onset seizure disorder with profound motor and cognitive decay
135  most Spnb3(-/-) animals develop a myoclonic seizure disorder with significant reductions of EAAT4, E
136 epilepsies are a heterogeneous collection of seizure disorders with a lifetime expectancy risk rate o
137 ts (n=19) who were in good health except for seizure disorder, with stable anticonvulsant drug levels
138 nvestigation of CNVs in epilepsy and related seizure disorders, with potential implications for clini

 
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