ライフサイエンスコーパス: selenite

1 , and extensively, throughout the grain than selenite.

2 the selD or the xdh mutant upon addition of selenite.

3 erevisiae, catalyzes high-affinity uptake of selenite.

4 ration of reactive oxygen species induced by selenite.

5 peroxide production and apoptosis induced by selenite.

6 elenate but were low in plants supplied with selenite.

7 ins were up-regulated by chronic exposure to selenite.

8 uced in rats by a single injection of sodium selenite.

9 rticles of elemental selenium (nano-Se) from selenite.

10 hat selenate was taken up 2-fold faster than selenite.

11 vol) fetal bovine serum and 0.1 microM [75Se]selenite.

12 mobile selenate into relatively less mobile selenite.

13 ponic solution supplemented with selenate or selenite.

14 ble Se was comprised of Se oxyanions, mainly selenite.

15 ximately 1 mm from the apex) when exposed to selenite.

16 or the crystallization of templated vanadium selenites.

17 d during four days after soaking with sodium selenite (0, 1, or 2 mg/100 g seeds).

18 al cell bodies were also stained with either selenite (1 hr) or 6-methoxy 8-para-toluene sulfonamide

19 dicates that, in LB medium supplemented with selenite (1 mM), reduction to nano-Se occurs at a rate o

20 ibustion (1 observational study), and sodium selenite (1 RCT, 2 observational studies).

21 Se converted to organic forms was higher for selenite (100%) than for selenate (26%), the absolute co

22 with respect to GPx induction as was sodium selenite (2.2-fold increase at 15 microM).

23 edia containing increasing amounts of sodium selenite (30, 50, and 80 nmol/L).

24 Sodium selenite, a representative of the genotoxic selenium poo

25 We have found that sodium selenite, a selenium compound with chemotherapeutic pote

26 showed that overexpression of Jen1p enables selenite accumulation in yeast compared with a JEN1 knoc

27 indicating the Jen1p transporter facilitates selenite accumulation inside cells.

28 Our results in selenite-adapted cells suggest that selenium may exert i

29 male mice were fed a semipurified diet with selenite added as the source of selenium.

30 sms by which selenium, in the form of sodium selenite, added to serum-free growth medium regulates TR

31 in the medium (NO2- and NO3-) resulted from selenite addition to cell suspensions.

32 vation of intracellular calcium levels after selenite administration resulted in increased levels of

33 The mechanism of selenite adsorption by the new mixed adsorbent composed

34 Herein, the mechanism of selenate and selenite adsorption on NU-1000 is explored to determine

35 esults show that P. putida is able to reduce selenite aerobically, but not selenate, to nano-Se.

36 LNCaP cells treated with selenite also showed p53 translocation to mitochondria,

37 ed in the presence of up to 1.5 mM NaCl, and selenite analysis is even more robust against chloride.

38 G) and glutathione disulfide are formed from selenite and 4 GSH.

39 the Recommended Dietary Allowance of sodium selenite and antiretroviral drugs (ARV) on maternal plas

40 )) through a redox reaction involving sodium selenite and ascorbic acid.

41 otic dissimilatory reduction of selenate and selenite and assimilation of the reduced Se species into

42 vector-only control plants when treated with selenite and exhibited an increased tolerance to Se.

43 In addition, selenite and lactate can inhibit the transport of each o

44 e mechanism of 3D versus 2D A549 cultures to selenite and metabolic reprogramming that can mediate th

45 Se, whereas muskgrass contained Se mainly as selenite and organic Se forms.

46 tRNA(Sec) in vitro in the presence of sodium selenite and purified recombinant E. coli selenophosphat

47 PC3 cells increased cellular sensitivity to selenite and resulted in increased superoxide production

48 imed to produce Se-microparticles (selenate, selenite and Se-organic) using combined methods of micro

49 e NOM with Fe(III) increases the sorption of selenite and selenate by several orders of magnitude.

50 ate differences in selenium speciation, with selenite and selenate co-occurring in most samples.

51 In this study, the metabolization of selenite and selenate fortification at low and high leve

52 th different concentrations of inorganic Se (selenite and selenate) and applied twice to the plants i

53 , were enriched with two selenium compounds (selenite and selenate) to test their suitability as natu

54 lenocysteine and inorganic selenium species (selenite and selenate) were not detected in the dialyzat

55 Besides selenite and selenate, selenosulfate was the most freque

56 Both selenite and selenate, two major inorganic forms of Se,

57 ansported to the grain more efficiently than selenite and selenate.

58 ine, l-selenocystine, methaneseleninic acid, selenite and selenate.

59 increased resistance to the toxic effects of selenite and selenomethionine (SeMet), respectively.

60 3D A549 spheroids in response to anticancer selenite and simultaneously (13)C/(15)N atoms from [(13)

61 Phoma glomerata to effect transformation of selenite and tellurite was investigated including intera

62 pheroids in response to the anticancer agent selenite and that of [(13)C(5),(15)N(2)]-glutamine in 2D

63 ae were also exposed to waterborne dissolved selenite and to dietary selenomethionine as selenized al

64 the form of Se-supplemented (0.4 ppm; sodium selenite) and high Se (1.0 ppm; sodium selenite) diets.

65 <0.01 ppm Se), Se-adequate (0.08 ppm; sodium selenite), and two supraphysiological levels in the form

66 s" and related oxidation products, selenate, selenite, and other species relatable to the quality and

67 We also measured whether oxidized selenium, selenite, and reduced selenium, selenide, would target t

68 nic or organic species of Se (e.g. selenate, selenite, and Se-methionine [Met]) into gaseous Se forms

69 The stoichiometric reductions of selenate, selenite, and selenium dioxide with an iron(II) complex

70 ied with selenate, 38 times higher than from selenite, and six times higher than from SeMet.

71 and MeHgCl) and Se (selenomethionine, sodium selenite, and sodium selenate) compounds.

72 SeMSC accumulation in response to selenate, selenite, and sulfate treatments showed that the BoSMT t

73 re treated with increasing concentrations of selenite, and the effects on DNA-binding activity of NF-

74 The diversity of the selenite anions as an inorganic ligand is demonstrated b

75 ir ability to adsorb and remove selenate and selenite anions from aqueous solutions.

76 ers) occurred at a much higher rate than for selenite (apparently by both passive diffusion and phosp

77 With selenite as the selenium source and the isolated reduced

78 o accumulate selenium by growing with sodium selenite as the selenium source under hydroponic conditi

79 that oxidative stress was induced by sodium selenite at high concentrations in both acute and chroni

80 of loss of cell viability induced either by selenite at pharmacological levels or by growth factor d

81 The observed proton conductivity of the selenite based oxothiometalate species renders them as p

82 differential cytotoxicity observed by sodium selenite between HCT116 and HCT116+Chr.3 cell lines was

83 s was shown by monitoring the consumption of selenite by bacteria incubated in LB broth.

84 ave strong implications for the retention of selenite by corrosion products in nuclear waste reposito

85 Limited studies have shown that selenite can also be immobilized through abiotic precipi

86 It is widely understood that selenite can be biologically reduced to elemental seleni

87 e, but in higher toxic levels (>5-10 microM) selenite can react with essential thiol groups on enzyme

88 he lens nucleus coincident with the onset of selenite cataract.

89 After acute exposure to selenite, cells exhibited mitochondrial injury and cell

90 After chronic exposure to selenite, cells showed growth inhibition caused by cell

91 on pollutants (arsenate, arsenite, selenate, selenite, chromate, and perchlorate) were selected for s

92 - to 3-fold higher from plants supplied with selenite compared with selenate.

93 NOM can sorb Se species, especially selenite, considerably.

94 sprouts that were treated with a high sodium selenite content (2mg/100g seeds).

95 an SG assembly and provide insights into how selenite cytotoxicity may be exploited as an anti-neopla

96 supplemented for 6 wk with 100 microg sodium selenite/day.

97 ministration of high-dose intravenous sodium selenite did not reduce morbidity or mortality.

98 odium selenite) and high Se (1.0 ppm; sodium selenite) diets.

99 increasing selenite levels and, at 7 microM selenite, DNA-binding activity was completely inhibited.

100 th the exception of Salmonella culture using selenite enrichment for which PCR was less sensitive tha

101 Selenite exposed periphyton readily bioconcentrated Se w

102 Selenite exposures resulted in high mortality to embryos

103 icles in the presence of EPS, extracted from selenite fed anaerobic granular sludge, yielded stable c

104 adpoles were exposed to dissolved (75)Se (as selenite) for 7 days and depurated until completion of m

105 as sole electron donor in the reaction with selenite, further conversion of the R-SSeS-R product app

106 of yogurt with Se in the form of free sodium selenite had a low effect on improving the bioaccessibil

107 e resistance, and overexpression resulted in selenite hypersensitivity.

108 much less toxic to the cells than was sodium selenite (IC(50) approximately 17 microM) or the parent

109 The improved understanding of selenite immobilization and the improved model can help

110 at both pathways significantly contribute to selenite immobilization in a microfluidic flow cell havi

111 Jen1p transported selenite in a proton-dependent manner which resembles th

112 S assimilation enzymes were up-regulated by selenite in Col-0 but not Ws-2.

113 ing the cataract formation induced by sodium selenite in male Wistar rat pups.

114 Selenate was metabolized less than selenite in whole plants, but in grains selenium was pre

115 Glutathione redox status was reduced by selenite in Ws-2 but not in Col-0.

116 lf, there was 30% inorganic Se (selenate and selenite) in addition to 70% MeSeCys.

117 Our previous study showed that sodium selenite induced LNCaP human prostate cancer cell apopto

118 We showed that selenite-induced apoptosis as evidenced by cleavage of p

119 In the current study, we show that selenite-induced apoptosis involves DNA damage.

120 xpressing LNCaP cells were more sensitive to selenite-induced apoptosis than p53-null PC3 cells.

121 to involve DNA topoisomerase II (Top II) as selenite-induced apoptosis was reduced in Top II-deficie

122 Selenite-induced apoptosis was shown to involve DNA topo

123 In the present study, we showed that selenite-induced apoptosis was superoxide mediated and p

124 In the aggregate, these results suggest that selenite-induced apoptosis, which involves ATM/ATR and T

125 e, AnxA5 is protective against cisplatin and selenite-induced apoptotic cell death.

126 In contrast, selenite-induced apoptotic DNA fragmentation was observe

127 N-acetylcysteine amide-only group, a sodium selenite-induced cataract group, and a NACA-treated sodi

128 in the NACA-treated group than in the sodium selenite-induced cataract group.

129 ed cataract group, and a NACA-treated sodium selenite-induced cataract group.

130 Among selenite-induced cataract rats, the Q/siP-c-M-treated ra

131 c sites of MIP N- and C-terminal cleavage in selenite-induced cataractous lenses were identified.

132 Macroarray analysis showed more pronounced selenite-induced increases in mRNA levels of ethylene- o

133 Selenite-induced SGs differ from canonical mammalian SGs

134 Selenite-induced SGs lack several classical SG component

135 Selenite-induced up-regulation of GPx (glutathione perox

136 These results suggest that selenite induces apoptosis by producing superoxide to ac

137 Selenite inhibition was reversed by addition of a dithio

138 V of the spinal cord were stained by sodium selenite injected intrathecally.

139 Furthermore, sodium selenite- injected rat pups had significantly higher lev

140 siderite is essentially a two-step process, selenite ions being immobilized on siderite surface prio

141 Therefore, we postulate selenite is a molecular mimic of monocarboxylates which

142 readily internalized by C. reinhardtii, but selenite is accumulated around ten times more efficientl

143 The reduction of selenate to selenite is catalyzed by a selenate reductase, previousl

144 Inhibition of NF-kappaB activity by selenite is presumed to be the result of adduct formatio

145 Soluble tetravalent selenite is the predominant environmental form and also

146 (DMEM) containing insulin-transferrin-sodium selenite (ITS) supplement (DMEM/ITS) or 10% fetal bovine

147 e DMEM containing insulin-transferrin-sodium selenite (ITS) with 10, 100, and 1000 pg/mL TGF-beta1 or

148 14-(1-pyridinium)-N-octadecanoyl-sphingosine selenite (LCL768) to induce mitophagy and metabolic stre

149 ts was decreased progressively by increasing selenite levels and, at 7 microM selenite, DNA-binding a

150 Se volatilization from selenite may be limited by selenite uptake and by the co

151 , S-nitrosoglutathione, selenodiglutathione, selenite, methylseleninate, and selenocystine.

152 ells with a gradient concentration of sodium selenite, methylseleninic acid and methylselenocysteine

153 n media containing various concentrations of selenite, molybdate, and various purine substrates.

154 nate was 9-fold more toxic to the roots than selenite, most likely due to increased accumulation of o

155 rminated after soaking with different sodium selenite (Na2SeO3) concentrations (0, 1 and 2mg/100g see

156 d fastest uptake rates for both selenate and selenite, of all zirconium-based MOFs studied here.

157 yme-catalyzed reactions that are impacted by selenite or arsenite treatments.

158 r chemopreventive agents and less toxic than selenite or certain naturally occurring selenoamino acid

159 ivities of PH and XDH, increased when either selenite or molybdate was added to the culture medium.

160 on processes for groundwater contaminated by selenite or other contaminants (e.g., uranium(IV)) that

161 With either selenite or selenate as substrates, Se methylation was h

162 atch reactor experiments were performed with selenite or selenate by equilibrating suspensions contai

163 gna unguiculata) exposed to either 20 microM selenite or selenate.

164 es of radioactive selenium 75 in the form of selenite or selenide and measured blood and tissue selen

165 1)) enriched with methylmercury and/or Se as selenite or selenomethionine.

166 lularly and extracellularly, when grown with selenite or tellurite.

167 , (ii) that are highly electrophilic such as selenite, or (iii) that are activated by strain such as

168 amended with the selenium oxyanion selenate, selenite, or selenocyanate, produces volatile organosele

169 B damage to DNA and modulation by vitamin C, selenite, or Trolox, a water-soluble vitamin E analog.

170 eMet and SeMet (but no DMSeP) accumulated in selenite- or SeMet-supplied wild-type plants and in sele

171 um of 8.5 and is also found to function as a selenite oxidase.

172 ics the reaction zone along the margins of a selenite plume undergoing bioremediation in the presence

173 Selenite pretreatment of NB4 cells increases the activit

174 manner and supplementation of 100 nM sodium selenite prevented the detrimental effects of glutamate

175 nium supplementation of 2000 mug/L of sodium selenite prior to cardiopulmonary bypass, 2000 mug/L imm

176 At GSH-to-selenite ratios >4:1, conversion of GSSeSG to a perselen

177 combination of reference spectra showed that selenite reaction with siderite is essentially a two-ste

178 A novel nitrate- and selenite reducing bacterium strain ZYK(T) was isolated f

179 g multiple metalloenzymes, like selenate and selenite reductase.

180 eine thiol groups in reduced rSeBP prevented selenite reduction and selenium binding under comparable

181 regation of the more thermodynamic favorable selenite reduction and the less thermodynamically favora

182 rous oxides, is the main reason for its high selenite removal performance demonstrated by batch and c

183 III) hydrous oxides played a leading role in selenite removal.

184 Conversely, the selenite resistance of Col-0 was reduced in mutants impa

185 Disruption of JEN1 resulted in selenite resistance, and overexpression resulted in sele

186 ndex demonstrated a clear difference between selenite-resistant Col-0 and selenite-sensitive Ws-2.

187 ppb Se) and 380 nM (30 ppb) for selenate and selenite, respectively.

188 use high levels of molybdate, tungstate, and selenite restored growth to wild-type levels, this trans

189 0 micrograms/ml Trolox, and 5 or 12.5 microM selenite resulted in a significant decrease in the numbe

190 feeding mice a diet supplemented with sodium selenite results in an MR-like phenotype, marked by prot

191 rphous elemental selenium precipitate on the selenite-rich side of the mixing zone, while long crysta

192 presence of selenoproteins, suggesting that selenite's neutralization of auranofin is not because of

193 In the form of selenite (Se((IV))), selenium can be incorporated into c

194 When injecting selenite (Se((IV))), the precipitates grew significantly

195 rite and sphalerite, 19% of Se is present as selenite (Se(4+)) in barite, 21% of Se is present as exc

196 olize selenium when grown in the presence of selenite (Se(IV)) and selenate (Se(VI)) was investigated

197 method, the predominant selenium oxyanions, selenite (Se(IV)) and selenate (Se(VI)), can be quantifi

198 es were exposed to dissolved Se (30 ug/L) as selenite (Se(IV)) or selenate (Se(VI)) for 7 days follow

199 or five selenium species; selenate (Se(VI)), Selenite (se(IV)), selenocysteine (SeC), Se-methylseleno

200 dation states, selenate (Se(VI)O(4)(2-)) and selenite (Se(IV)O(3)(2-)) oxyanions can interact with ma

201 ure medium, but only when molybdate (Mo) and selenite (Se) were also added.

202 Sodium selenite (Se)-directed crosslinked hydrogels based on hy

203 reacted Se indicate the formation of a mixed selenite/Se(0) surface phase.

204 in QD exposures, we examined the toxicity of selenite, selenate, and amorphous selenium nanoparticles

205 ntaining anions examined were selenocyanate, selenite, selenate, tellurite, and tellurate.

206 ed and reduced into various inorganic forms (selenite, selenide, or elemental Se) or partially incorp

207 Selenite, seleno-cystine, and seleno-methionine exert th

208 olism of common dietary selenium compounds - selenite, selenomethionine, methylselenocysteine and sel

209 The selenite-sensitive phenotype of Ws-2 was attenuated by t

210 ference between selenite-resistant Col-0 and selenite-sensitive Ws-2.

211 reduction-oxidation reaction between aqueous selenite (SeO(3)(2-)) and siderite (FeCO(3(s))) was moni

212 The common selenium oxoanions selenite (SeO3(2-)) and selenate (SeO4(2-)) are toxic at

213 AtSBP1 binds selenium after incubation with selenite (SeO3(2-)) with a ligand to protein molar ratio

214 ubsequent toxicity to consumers of dissolved selenite (SeO3) versus selenate (SeO4) uptake into aquat

215 Simultaneous treatment of selenate with selenite significantly reduced SeMSC production.

216 nsgenic seedlings tolerated Se, particularly selenite, significantly better than the wild type, produ

217 ls were treated with selenocompounds (sodium selenite, sodium selenate, Se-Met, MeSeCys) or SeB [high

218 ated in growth compost irrigated with sodium selenite solution increased by 28- and 43-fold compared

219 administered sodium hydroselenide and sodium selenite solutions (0.05-2.4 mg/kg).

220 capsaicin treatment, which had no effect on selenite stain or MT-III mRNA content in small-diameter

221 When assessed 1 hr after sodium selenite, stain was distributed throughout the neuropil

222 Moreover, we found that selenite supplementation dampens the activity of auranof

223 Selenite supplementation of HIV-infected Malawian women

224 x-ray absorption spectroscopy revealed that selenite-supplied plants accumulated organic Se, most li

225 For both selenate- and selenite-supplied plants, Se accumulation and volatiliza

226 SRM selective inhibitors including selenate, selenite, tellurate, tellurite, nitrate, nitrite, perchl

227 ransverse mixing zone between propionate and selenite that mimics the reaction zone along the margins

228 mpare acute versus chronic effects of sodium selenite, the latter most closely resembling human clini

229 When grown in the presence of selenate or selenite, these bacteria produced both organo-sulfur and

230 ith plants supplied with SeMet, selenate, or selenite; they also accumulated more Se in shoots than i

231 steps in Se volatilization from selenate and selenite, time- and concentration-dependent kinetics of

232 cular mimic of monocarboxylates which allows selenite to be transported by Jen1p.

233 hr) that allows retrograde transport of zinc selenite to cell bodies, only small-diameter neurons and

234 Although the addition of sodium selenite to the growth medium did not affect phage produ

235 xide, which is produced upon the addition of selenite to TSBG.

236 Selenite-treated grains accumulated more selenium.

237 cumulated mainly in the leaves compared with selenite-treated plants where the selenium was retained

238 e peptides inhibited cataract development in selenite-treated rats, which was accompanied by inhibiti

239 Selenite treatment resulted in high levels of superoxide

240 olumbia (Col)-0 and Wassilewskija (Ws)-2, to selenite treatment.

241 aspase-9 activation, and apoptosis following selenite treatment.

242 l p53, Bax, and p21(Waf1) proteins following selenite treatment.

243 r selenium bioavailability is increased with selenite treatment.

244 such as glutathione and cysteine, react with selenite under specific conditions to form selenotrisulf

245 )](54-) (1a), which is templated using eight selenite units and two iron(III) centers.

246 latilization from selenite may be limited by selenite uptake and by the conversion of selenomethionin

247 Selenite uptake by Jen1p had a Km of 0.91 mM, which is c

248 ninic acid, methylselenocysteine, and sodium selenite via reactive oxygen species and facilitates the

249 ryptic soy broth with 1% glucose (TSBG) when selenite was added.

250 Consistent with ATM/ATR involvement, selenite was also shown to stimulate Ser-139 phosphoryla

251 Selenite was bound to the material via inner-sphere comp

252 the generation of reactive oxygen species by selenite was higher in Col-0 than in Ws-2.

253 Sodium selenite was injected intraperitoneally on postpartum da

254 Se in the form of sodium selenite was microencapsulated by spray - drying and add

255 s that selenide reduced damage to the heart; selenite was not effective.

256 we also showed that superoxide production by selenite was p53 dependent.

257 tral evolutions showed that more than 60% of selenite was reduced at the siderite surface after 20 h

258 Selenite was retained at the point of grain entry.

259 Using purified human recombinant Top II, selenite was shown to induce reversible Top II cleavage

260 ation, whereas only approximately 10% of the selenite was translocated.

261 ultured in the presence or absence of Se (as selenite) was used to examine temporal changes in the pr

262 he cooking process the loss of Se, mainly as selenite, was about 7%.

263 All expected forms of selenium except for selenite were determined using LC-MS/MS technique.

264 The effects of selenite were suppressed by pretreatment with a syntheti

265 o foliar Se fertilisers (sodium selenate and selenite) were tested at four rates (0-10-20-40gha(-1))

266 Equilibrating selenate and selenite with a model NOM (Pahokee peat soil), followed

267 e Se-containing substrates selenocystine and selenite with only slightly less activity than the wild-