戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ariable effects on the rewarding efficacy of self-stimulation.
2 es into brain areas implicated in electrical self-stimulation.
3 pulsion transition with optogenetic dopamine self-stimulation.
4 paminergic innervation contribute to operant self-stimulation.
5 ain, rewarded by optogenetic dopamine neuron self-stimulation.
6 reward-seeking behaviour measured by optical self-stimulation.
7 ons in anhedonia as measured by intracranial self-stimulation.
8  of other opioids measured with intracranial self-stimulation.
9 ective positive allosteric modulator reduced self-stimulation.
10 ohol reward were examined using intracranial self-stimulation.
11 effect on locomotor activity or intracranial self-stimulation.
12 lso capable of communicating, but must avoid self-stimulation.
13 e internal capsule (IC), which also supports self-stimulation.
14 ossible anatomical substrates supporting MFB self-stimulation.
15 ndle electrodes supported intense electrical self-stimulation.
16 tems in both the CeA and NAc supported laser self-stimulation, amplified incentive motivation for suc
17 avior normalized quickly in the intracranial self-stimulation and 5-choice serial reaction time task
18 ing both a cholinergic pathway that promoted self-stimulation and a dopaminergic pathway that likely
19  itself in classical electrical intracranial self-stimulation and conditioned place preference tests.
20 y was rewarding, as assessed by intracranial self-stimulation and conditioned place preference, where
21                           Using intracranial self-stimulation and fast scan cyclic voltammetry, we fo
22 r GABA neurons produced optical intracranial self-stimulation and place preference.
23 or NMDA receptor antagonists blocked optical self-stimulation and place preference.
24 nd developmentally equivalent cells to avoid self-stimulation and to coordinate their behavior to ach
25 l systems subserving positive reinforcement (self-stimulation) and incentive motivation (relapse).
26 n, shifts sucrose preference, drives optical self-stimulation, and directs sensory discrimination lea
27 generate positive reinforcement, as shown by self-stimulation, and negative reinforcement shown by st
28 hold-lowering effect on lateral hypothalamic self-stimulation, and whether any such effect can be att
29 elements in the anatomical substrate for MFB self-stimulation are discussed.
30 tivation of these projections induced robust self-stimulation behaviors, without activation of the HP
31 iable excitatory synaptic responses, optical self-stimulation behaviour was not observed by activatio
32 whereas CRISPR-Cas9 deletion of Th preserved self-stimulation but abolished place avoidance.
33 n of VTA glutamate neurons not only supports self-stimulation but can also induce avoidance behavior,
34 motivation and reinforcement of intracranial self-stimulation but have not assigned these effects to
35 e disruption of VGLUT2 abolished optogenetic self-stimulation but left real-time place avoidance inta
36 e examined the regulation of thalamostriatal self-stimulation by mGlu(2).
37 ts from the lateral hypothalamus (LH), where self-stimulation can be induced.
38 own that lateral hypothalamic stimulation or self-stimulation can release dopamine in the nucleus acc
39 ng that experiential differences in auditory self-stimulation cannot explain the perceptual change.
40 es project, is activated by vaginal-cervical self-stimulation (CSS) in such women, as visualized by f
41                            Opto-intracranial self-stimulation data showed that abstinent animals exec
42 y neurons proved to be capable of supporting self-stimulation, demonstrating that behavioral reinforc
43 spond, motivation to obtain the stimulation, self-stimulation despite punishment, and cue-induced rei
44                              An intracranial self-stimulation discrete trial procedure that provides
45 used electrical and optogenetic intracranial self-stimulation (eICSS, oICSS) paradigms to study the u
46                   Electrical and optogenetic self-stimulation experiments demonstrate that monkeys an
47 ion among other controls, we show that NKG2D self-stimulation has tumor-promoting capacity.
48                                 Intracranial self-stimulation (ICS) is a motivated behavior that resu
49 hat supports this hypothesis is intracranial self-stimulation (ICS), during which animals repeatedly
50                                 Intracranial self-stimulation (ICSS) activates the neural pathways th
51 ts brain reward processes using intracranial self-stimulation (ICSS) and inducible bitransgenic mice
52 aminergic neurons contribute to intracranial self-stimulation (ICSS) and other reward-seeking behavio
53 the CEA and examined effects on intracranial self-stimulation (ICSS) as an index of hedonic state, an
54 ned place preference (CPP), and intracranial self-stimulation (ICSS) assays, we tested the hypothesis
55 atergic neurons produced robust intracranial self-stimulation (ICSS) behavior, which was dose-depende
56                                 Intracranial self-stimulation (ICSS) can be utilized in rodents (rats
57 ate midazolam preference and an intracranial self-stimulation (ICSS) paradigm to evaluate the impact
58 was trained on a discrete trial intracranial self-stimulation (ICSS) procedure interpreted to assess
59                             The intracranial self-stimulation (ICSS) procedure was used to investigat
60  a locomotor activity assay, an intracranial self-stimulation (ICSS) procedure, and a conditioned pla
61 or activation and antagonism on intracranial self-stimulation (ICSS) reward using a discrete-trial cu
62 ty discounting paradigm wherein intracranial self-stimulation (ICSS) serves as the positive reinforce
63                             The intracranial self-stimulation (ICSS) test is sensitive to the functio
64 ts of benzodiazepines using the intracranial self-stimulation (ICSS) test, a procedure with which the
65 pendent) sessions and had daily intracranial self-stimulation (ICSS) thresholds assessed.
66 rebrain bundle, as reflected by intracranial self-stimulation (ICSS) thresholds in rats.
67 the forced swim test (FST), and intracranial self-stimulation (ICSS) thresholds.
68 conditioned stimuli would lower intracranial self-stimulation (ICSS) thresholds.
69                                 Intracranial self-stimulation (ICSS) was used to compare the effects
70                                 Intracranial self-stimulation (ICSS) was used to quantify CSDS-induce
71 bundle (for behavior studies of intracranial self-stimulation (ICSS)) or with cannulae for microdialy
72 eward-seeking behaviors such as intracranial self-stimulation (ICSS), although its precise role remai
73 esynaptic terminals establishes intracranial self-stimulation (ICSS), only Ppp1r1b-stimulated mice ex
74  ventral tegmental area support intracranial self-stimulation (ICSS), yet the cognitive representatio
75 ward sensitivity as measured by intracranial self-stimulation (ICSS).
76 ate with reward learning during intracranial self-stimulation (ICSS).
77 mined the effects on mood using intracranial self-stimulation (ICSS).
78 thalamus and trained to perform intracranial self-stimulation (ICSS).
79  sufficient to support vigorous intracranial self-stimulation (ICSS).
80 n rewarded behaviors, including intracranial self-stimulation (ICSS).
81 neurons produced robust optical intracranial self-stimulation in DAT-Cre mice, supporting an importan
82                                 Intracranial self-stimulation in rats showed that elongating the N-al
83       In addition, we performed intracranial self-stimulation in rats to understand how the chemical
84 activation of these terminals did not induce self-stimulation in the absence of an external reward.
85 ation by itself failed to support behavioral self-stimulation in the absence of any paired external f
86 anges in threshold pulse frequency (pps) for self-stimulation in the ventral tegmental area (VTA).
87 ffect on thresholds for lateral hypothalamic self-stimulation (LHSS) and did not alter the cocaine do
88 r, the effect of ADX on lateral hypothalamic self-stimulation (LHSS) and its facilitation by cocaine
89 of D-amphetamine in the lateral hypothalamic self-stimulation (LHSS) paradigm.
90 erone acetate) decrease lateral hypothalamic self-stimulation (LHSS) reward if rats are denied access
91 curve-shift analysis of lateral hypothalamic self-stimulation (LHSS) to evaluate whether the elevated
92 on of D1 + dentate neurons is sufficient for self-stimulation: mice will press a lever to earn optoge
93 ion of cocaine reinforcing effects, and MPFC self-stimulation (MPFCSS) is mediated by a neural substr
94                                  Optogenetic self-stimulation of acid-sensing TRCs in thirsty animals
95 roposed hypothesis regarding the role in MFB self-stimulation of ascending cholinergic input from the
96           Mice displayed robust intracranial self-stimulation of LH to VTA fibers, an operant behavio
97 o complement the presence of its ligands for self-stimulation of parameters of tumorigenesis.
98         Similar to the PVN(CRF) cell bodies, self-stimulation of PVN(CRF)-VTA projection was dramatic
99 bustly reinforced operant lever pressing for self-stimulation of serotonin neurons, which was exacerb
100  flies could implement behavior that induces self-stimulation of specific neurons in their brains.
101                   Mice also acquired operant self-stimulation of thalamostriatal terminals when ChR2
102 nged access to a running wheel on electrical self-stimulation of the lateral hypothalamus (LHSS), a m
103 measured using taste reactivity, and optical self-stimulation of the rostral and caudal shells was al
104 is rewarding, eliciting dopamine release and self-stimulation of these cells.
105 MOR function in DRN-MOR neurons and abnormal self-stimulation of these neurons.
106 or D2R antagonist into the NAc decreased the self-stimulation of these projections.
107 o complement the presence of its ligands for self-stimulation of tumor growth and presumably malignan
108 ewarding as assessed by optical intracranial self-stimulation (oICSS) in DAT-cre mice.
109             However, in optical intracranial self-stimulation (oICSS) maintained by optogenetic stimu
110 uronal activity, and 3) an opto-intracranial self-stimulation paradigm applied to DRN-MOR neurons to
111 we investigated this problem in mice using a self-stimulation paradigm in which specific spontaneous
112                     We used the intracranial self-stimulation paradigm to assess the effects of cocai
113 ard function in rats (using the intracranial self-stimulation procedure) and protein levels of brain-
114                                     Dopamine self-stimulation rapidly and dynamically changes the str
115 threshold) required to maintain intracranial self-stimulation responding in male and female rats, a d
116 ee of negative affect in our model predicted self-stimulation responding.
117  frequency required to maintain half-maximal self-stimulation response rates whereas injecting compar
118 reference scores were highly correlated with self-stimulation responses.
119                        Using an intracranial self-stimulation reward-based foraging task, we investig
120 on of the dMHb in vivo supports intracranial self-stimulation, showing that dMHb activity is intrinsi
121 tal area reward neurons, the distribution of self-stimulation sites in this structure was mapped in 2
122 of these receptors increased thalamostriatal self-stimulation, suggesting that endogenous activation
123 opulation of CB1 receptors modulates optical self-stimulation sustained by activation of PFC afferent
124 neurochemistries along the trajectory of the self-stimulation system has stronger effects on appetiti
125  NAc in the rat brain during an intracranial self-stimulation task in which a cue predicted lever ava
126 sed valence with CRF-containing neuron laser self-stimulation tasks.
127 s motivation as measured in the intracranial self-stimulation test.
128 red before both the 8 and 12 hr post-cocaine self-stimulation tests, reversed the threshold elevation
129 reases in cocaine self-administration and LH self-stimulation that are reversed by dynorphin antagoni
130 ding termination and satiety, locomotion and self-stimulation, the modulation of anxiety-like behavio
131                                  In general, self-stimulation thresholds obtained from lateral sites
132 ated CREB produced increases in intracranial self-stimulation thresholds, a depressive-like sign refl
133 of nicotine-induced lowering of intracranial self-stimulation thresholds.
134 ted plus maze test and elevated intracranial self-stimulation thresholds; both of these effects were
135  "rate-frequency" procedure for intracranial self-stimulation to determine the frequency at which sti
136 properties of nicotine, we used intracranial self-stimulation to measure alterations in the threshold
137 lt Sprague-Dawley rats, we used intracranial self-stimulation to measure effects of the KOR agonist (
138 proximated the human SRT, using intracranial self-stimulation to promote rapid continuous responding
139               The animals would perform both self-stimulation to turn the current on and stimulation-
140 HRBETS also permits optogenetic intracranial self-stimulation under positive or negative operant cond
141 ctly investigates the role of the RRF in MFB self-stimulation using transient lidocaine-induced inact
142 ABA neurons, the degree of responding for IC self-stimulation was proportional to the magnitude of el
143          The threshold for responding for IC self-stimulation was the threshold for electrical coupli
144 ing mice to perform optogenetic intracranial self-stimulation, we examined how self-initiated goal-di
145 red in response to a cue during intracranial self-stimulation were also attenuated by intra-VTA micro
146 lamic areas; of these, roughly 2/3 supported self-stimulation which was widely observed throughout th
147 le rats using social defeat and intracranial self-stimulation, while changes in serotonergic phenotyp
148 ar, by measuring thresholds for intracranial self-stimulation with and without concurrent cocaine adm
149 d GJ blockers increased the threshold for IC self-stimulation without affecting performance.

 
Page Top