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1 al levels: progressive nonfluent aphasia and semantic dementia.
2 ormed markedly better than the patients with semantic dementia.
3 face and name knowledge in 15 patients with semantic dementia.
4 vanced interpretations of the impairments in semantic dementia.
5 ts observed during the onset and progress of semantic dementia.
6 te semantic primary progressive aphasia from semantic dementia.
7 tices and nucleus accumbens in patients with semantic dementia.
8 tic forms of primary progressive aphasia and semantic dementia.
9 pposed to the 'storage' deficits observed in semantic dementia.
10 ts of picture-naming data from patients with semantic dementia.
11 that is sensitive to semantic impairment in semantic dementia.
12 of a more severe generic semantic deficit in semantic dementia.
13 ith those areas known to be most affected in semantic dementia.
14 visual deficits as compared to patients with semantic dementia.
15 its, but sparing of phonology and fluency in semantic dementia.
16 the widely accepted diagnostic criteria for semantic dementia.
17 ad criteria, excluding only those with clear semantic dementia.
18 ), progressive non-fluent aphasia (PNFA) and semantic dementia.
19 t mortem cases meeting clinical criteria for semantic dementia.
20 h progressive non-fluent aphasia; eight with semantic dementia].
21 on this test of a group of 12 patients with semantic dementia (10 male, mean age: 64.4 years) correl
22 2% bvFTD/motor neuron disease cases), 2 with semantic dementia (5.9% of patients with semantic dement
23 t frontotemporal dementia (71% of cases) and semantic dementia (65% of cases) and in association with
24 ic cases) and heightened responsiveness with semantic dementia (73% of symptomatic cases) and Alzheim
25 er of central conceptual knowledge arises in semantic dementia, a degenerative condition associated w
27 tion, the topography of neuroinflammation in semantic dementia, also known as the semantic variant of
30 expert musicians with clinical diagnoses of semantic dementia and Alzheimer's disease, in comparison
33 l eating behavior in patients with bvFTD and semantic dementia and are likely responsible for the dif
34 heimer's disease, the focal lobar atrophies (semantic dementia and dementia of frontal type) and thre
35 t the performance of three patients-two with semantic dementia and focal temporal lobe atrophy and th
36 teria for behavioural variant FTD (bvFTD) or semantic dementia and had characteristic brain atrophy.
37 asting doubt over the conclusions drawn from semantic dementia and linked basic neuroscience studies.
38 h clinical diagnoses of Alzheimer's disease, semantic dementia and non-fluent primary progressive aph
39 osia is one of the clinical presentations of semantic dementia and not a separate clinical entity.
41 h those observed in a group of patients with semantic dementia and predominant left-sided temporal lo
42 ic prominence, particularly for diagnosis in semantic dementia and prognosis in behavioural syndromes
43 ible for deteriorating semantic knowledge in semantic dementia and separate from 'classic' language a
45 psychological studies of surface dyslexia in semantic dementia and the connectionist triangle model o
46 s an examination of the relationship between semantic dementia and the focal clinical syndrome of pro
47 totemporal dementia with motoneuron disease, semantic dementia and, with one exception, progressive n
48 tients (five with svPPA and two with 'right' semantic dementia) and 12 healthy controls underwent pos
49 ith semantic dementia (5.9% of patients with semantic dementia), and none with progressive nonfluent
50 tients with dementia (19 with bvFTD, 15 with semantic dementia, and 15 with Alzheimer disease) were r
51 f primary progressive aphasia, also known as semantic dementia, and Alzheimer's disease have deficits
52 d in both progressive non-fluent aphasia and semantic dementia, and deficits of semantic processing a
53 clear palsy set), anterior temporal lobes in semantic dementia, and hippocampus and posterior cingula
54 ding of the anatomical changes that occur in semantic dementia, and may further help to explain the d
56 h the volume of right temporal structures in semantic dementia, and with subcallosal gyrus volume in
57 rment of semantic knowledge in patients with semantic dementia appears to influence performance in a
58 ant primary progressive aphasia (also called semantic dementia) are two clinical variants of frontote
59 s suggested by observations on patients with semantic dementia, as well as posterior regions describe
60 a striking literary depiction of collective semantic dementia before the syndrome was recognized in
61 hat differed between patients with bvFTD and semantic dementia but included the cingulate cortices, t
64 ecent findings indicate that the syndrome of semantic dementia can inform us about the organisation o
65 a visual decision task in four patients with semantic dementia compared with six age-matched normal c
66 longitudinal bundle, where abnormalities in semantic dementia did not extend caudal to the atrophic/
67 [(18)F]AV-1451, the pathological regions in semantic dementia do not normally contain significant le
69 [(18)F]AV-1451 binding potential, separated semantic dementia from controls with 86% sensitivity and
71 ilateral anterior temporal lobe damage (e.g. semantic dementia), functional neuroimaging and repetiti
72 ative threshold, tensor abnormalities in the semantic dementia group mapped onto the tractographies f
79 performed on a wider cohort of patients with semantic dementia, in which the patients with more exten
82 d the hypothesis that concept degradation in semantic dementia involves a combination of these pan-mo
90 the Alzheimer disease (mean, 710 calories), semantic dementia (mean, 573 calories), and control grou
92 ral variant FTD (n = 26), language variants (semantic dementia, n = 9; and progressive nonfluent apha
93 iant FTD: n = 1337 cases, n = 2754 controls; semantic dementia: n = 308 cases, n = 616 controls; prog
94 havioral variant frontotemporal dementia and semantic dementia, often respond to treatment with selec
95 dial temporal lobe lesions and patients with semantic dementia on nine tests of semantic knowledge an
96 al dementia, progressive non-fluent aphasia, semantic dementia or mixture of these syndromes for muta
97 longitudinal fasciculus were tracked in five semantic dementia patients and eight healthy controls.
99 ', strategies that resonate with attempts by semantic dementia patients to cope with their disease.
101 erall profile masked individual differences: semantic dementia patients with predominant left tempora
102 Three groups of 10 subjects were studied: semantic dementia patients, Alzheimer's disease patients
103 ntic variant primary progressive aphasia and semantic dementia, patients with early right anterior te
104 the tests of new learning, the patients with semantic dementia performed markedly better than the amn
105 r each FTD subtype (behavioural variant FTD, semantic dementia, progressive non-fluent aphasia, and F
106 drome that includes frontotemporal dementia, semantic dementia, progressive nonfluent aphasia and pro
111 wo groups suffering from different diseases: semantic dementia (SD) and herpes simplex virus encephal
113 with frontotemporal dementia (FTD), 19 with semantic dementia (SD) and six with progressive non-flue
117 diverse cortical regions in semantic memory: semantic dementia (SD) is characterized by atrophy of th
118 ed patients with Alzheimer's disease (AD) or semantic dementia (SD) on a visual oddity judgment task
119 ed it to simulate and explain the effects of semantic dementia (SD) on word processing abilities.
121 ral atrophy of the anterior temporal lobe in semantic dementia (SD) produces a gradual degradation of
122 herpes simplex virus encephalitis (HSVE) and semantic dementia (SD) typically affect anterior tempora
126 ccur in both behavioural-variant (bvFTD) and semantic dementia (SD)/semantic-variant primary progress
127 uent aphasia (PNFA) (or a mixed aphasia) and semantic dementia (SD); and to compare the age of onset,
128 onfluent progressive aphasia (NFPA; n = 11), semantic dementia (SD; n = 10), and a third variant term
129 18 progressive nonfluent aphasia [PNFA], 16 semantic dementia [SD]), 22 progressive supranuclear pal
133 rment found across different patient groups (semantic dementia, temporal lobe epilepsy, glioma and st
134 The cortical anatomy of 6 patients with semantic dementia (the temporal lobe variant of frontote
135 convincingly demonstrated that patients with semantic dementia (the temporal variant of frontotempora
141 ite epidemiological and genetic data linking semantic dementia to inflammation, the topography of neu
142 est to evaluate the ability of patients with semantic dementia to use recollection-based memory proce
144 e left language-related cerebral pathways in semantic dementia using diffusion tensor imaging-based t
147 To explore this phenomenon in nine cases of semantic dementia, we designed a set of semantic tests r
148 integrative memory model through the lens of semantic dementia, we propose a number of important exte
150 uced fractional anisotropy for patients with semantic dementia were spatially concordant with each ot
152 frontotemporal dementia alone, one had mixed semantic dementia with frontal features and three had pr
153 Social cognitive changes are also common in semantic dementia, with atrophy centred on the anterior
154 or temporal lobe structures were affected in semantic dementia, with the entorhinal cortex, amygdala,
155 ntext of their implications for the place of semantic dementia within the classification of progressi