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1 nto the brain by cleaving the chemorepellent semaphorin 3a.
2 n leads to ischemia and angiogenesis through Semaphorin 3A.
3 d myelin-associated glycoprotein, but not to semaphorin 3A.
4 chemia and pathological angiogenesis through Semaphorin 3A.
5 ve guidance molecules, such as ephrin-A2 and semaphorin 3A.
6 towards the retina is guided by netrin-1 and semaphorin 3a.
7 h cone collapse in response to ephrin-A2 and semaphorin 3A.
8                                              Semaphorin 3A, a chemorepellent known to inhibit integri
9                               These included semaphorin 3a, a guidance cue in neural development with
10  required for growth cone steering away from semaphorin-3a, a guidance cue that does not activate ER-
11                                    E7 to E12 semaphorin 3A and 3F and ephrin A2 and A5 expressions di
12                       The guidance molecules Semaphorin 3A and 3F, already known to direct oligodendr
13 lacking the mediators of sensory pathfinding semaphorin 3A and neuropilin-1.
14 teracts cell migration inhibitory signals by Semaphorin 3A and Semaphorin 3F, generating aberrant neu
15 epulsive signal composed at least in part of semaphorin 3A and semaphorin 3F.
16 ce neuropilin-1 (NRP1), a receptor shared by semaphorin 3A and vascular endothelial growth factor (VE
17          Neuropilin-1 (Np-1), a receptor for semaphorin 3A and vascular endothelial growth factor, is
18 ntiates the signaling pathways stimulated by semaphorin 3A and VEGF-A in neuronal and endothelial cel
19                            Astrocyte-derived semaphorins 3a and 6a act to repel OPCs from blood vesse
20 ion and that the molecular cues netrin 1 and semaphorin 3a are likely to be involved.
21                    Similarly, chemorepellent semaphorin-3a binds neuropilin-1 to activate PTEN, which
22 helly et al. show that the axon guidance cue Semaphorin 3A can promote dendrite growth by inhibiting
23 ), cell migration (Ret and EdnrB signalling, semaphorin 3A, cell adhesion molecules, Rho GTPases), an
24                                      NGF- or semaphorin 3A-coupled beads caused a significant increas
25 evealed that decorin-mediated suppression of semaphorin 3A critically depends on erythroblastic leuka
26 nistration of a ligand of plexin-A4, Sema3A (semaphorin 3A), exacerbates the cytokine storm caused by
27                                              Semaphorin 3A expression was observed in deep dorsal and
28 how that decorin has the ability to suppress semaphorin 3A expression within adult rat cerebral corte
29                         We show that loss of Semaphorin 3A function or specific deletion of NRP1 in B
30                           HMGB1 bound at the semaphorin 3A genomic locus, promoted hetrochromatin for
31 nse of dorsal root ganglion (DRG) neurons to Semaphorin 3A gradients can be divided into two steps: g
32 stem scar associated axon growth inhibitors, semaphorin 3A has been shown to be strongly expressed by
33 glycans, decorin has the ability to suppress semaphorin 3A in the injured central nervous system.
34 europilin-1 and suppressed the expression of semaphorin 3a in the podocyte.
35 hosphorylatable active SH3BP4-S246A restores semaphorin-3a induced cell contraction.
36   In the present study, we first showed that semaphorin 3A-induced growth cone collapse in cultured h
37               Raft manipulation also blocked Semaphorin 3A-induced growth cone repulsion, inhibition,
38                  These results indicate that semaphorin 3A initiates growth cone collapse via activat
39 st from beta3GnT2(-/-) OSNs and axons, while semaphorin-3A ligand expression is upregulated.
40 orrespondingly, Ndr2-null mutant mice show a Semaphorin 3A(-/-)-like phenotype of premature dendritic
41                   We further determined that semaphorin 3A-mediated calpain activation and growth con
42              Thus, our results indicate that semaphorin 3A-mediated growth cone collapse is mediated
43                    Here, we demonstrate that semaphorin 3A-mediated growth cone collapse is reduced i
44 ridization and immunostaining confirmed that semaphorin 3A messenger RNA expression and protein level
45  days resulted in a significant reduction of semaphorin 3A messenger RNA expression within injury sit
46 ecorin treatment decreased the expression of semaphorin 3A messenger RNA in cultured rat leptomeninge
47 y into avascular tumor areas is regulated by Semaphorin 3A/Neuropilin-1 signaling; interference with
48  on cultured retinal ganglion cell axons, of semaphorin 3A on dorsal root ganglion sensory axons, and
49                             Stimulation with semaphorin 3A or semaphorin 3C neither disrupts nor enha
50 d in axonal outgrowth and a component of the semaphorin 3A pathway, in switching GTPase signaling whe
51  However, myelin-associated glycoprotein and semaphorin 3A regulate axonal levels of different mRNAs
52 ; in fact, treatment of primary neurons with Semaphorin 3A rescues Ndr2 knock-down-induced dendritic
53                                              Semaphorin 3A (Sema 3A), a member of semaphorin family,
54                                              Semaphorin 3A (Sema 3A), a molecule that inhibits axonal
55                                              Semaphorin 3A (Sema 3A; previously known as semaphorin I
56  linear regression model, rs139438618 at the semaphorin 3A (SEMA3A [OMIM 603961]) locus was significa
57 d in TGF-beta-treated AEECs, which decreased semaphorin 3A (Sema3A) and increased EndMT markers, and
58           Signaling by the axon guidance cue semaphorin 3A (Sema3A) and its receptor complex of neuro
59 ressed genes and we investigated the role of semaphorin 3A (Sema3A) and neuropilin-1 (Nrp-1) in lymph
60  Neuropilin-1 (Npn-1) is a receptor for both semaphorin 3A (Sema3A) and vascular endothelial growth f
61                                              Semaphorin 3A (Sema3A) binds to neuropilin-1 (NP1) and a
62 he growth-promoting NGF or growth-inhibitory semaphorin 3A (Sema3a) compared with control green fluor
63          Secreted semaphorin 3F (Sema3F) and semaphorin 3A (Sema3A) exhibit remarkably distinct effec
64                           Here, we show that semaphorin 3A (Sema3A) expression overcomes the proinvas
65                     We demonstrate here that semaphorin 3A (Sema3A) induces a coordinated rearrangeme
66                                              Semaphorin 3A (Sema3A) is a membrane-associated secreted
67                                              Semaphorin 3A (Sema3A) is a secreted factor known to gui
68 ic genes, and that ventral astrocyte-encoded semaphorin 3a (Sema3a) is required for proper motor neur
69  our laboratory has shown that VEGF-A165 and semaphorin 3A (Sema3A) promote vessel maturation through
70  molecule close homolog of L1 (CHL1) and the semaphorin 3A (Sema3A) receptor, neuropilin 1 (Npn1), im
71 d to produce cell type-specific responses to Semaphorin 3A (Sema3A), a guidance cue that would be pre
72                                              Semaphorin 3A (Sema3A), a known inhibitor of axonal spro
73 h factor (VEGF), an angiogenesis factor, and semaphorin 3A (Sema3A), a mediator of axonal guidance.
74                                              Semaphorin 3A (Sema3A), a secreted guidance cue that pri
75 equired for growth cone collapse elicited by Semaphorin 3A (Sema3A), an axonal guidance cue.
76                                              Semaphorin 3A (Sema3A), in addition to its function as a
77                                We found that semaphorin 3A (SEMA3A), previously shown to act as a sup
78                                              Semaphorin 3A (Sema3A), which lies adjacent to this turn
79 d FGF-2 increased branching by >50%, whereas semaphorin 3A (Sema3A), which repels cortical axons, inh
80          A major component of this signal is semaphorin 3A (Sema3A), which was previously characteriz
81                                              Semaphorin 3A (SEMA3A)-encoded semaphorin is a chemorepe
82 ugh the liberation of repulsive guidance cue semaphorin 3A (Sema3A).
83 etic retinopathy (PDR) had elevated vitreous semaphorin 3A (SEMA3A).
84 protein that binds the secreted guidance cue Semaphorin 3A (Sema3A).
85 te growth cone responses to the guidance cue Semaphorin 3A (Sema3A).
86 p1), which is sensitive to the repellent cue Semaphorin 3A (Sema3A).
87 exins, promotes axon repulsion by binding to semaphorin 3A (SEMA3A).
88  (rs277470) located in a region encoding the semaphorin-3A (SEMA3A) binding domain (meta-analysis p v
89                                              Semaphorin-3A (SEMA3A) functions as a chemorepulsive sig
90                                              Semaphorin-3A (Sema3A) is a major guidance cue in the de
91                                              Semaphorin-3A (Sema3a), a guidance protein secreted by p
92                   Plex 1 alone does not bind semaphorin-3A (Sema3A), but the NP-1/Plex 1 complex has
93 ystal structure of a secreted 65 kDa form of Semaphorin-3A (Sema3A), containing the full semaphorin d
94                    We have demonstrated that semaphorin-3A (Sema3A)-induced growth cone detachment an
95                   Furthermore, expression of semaphorin 3A slightly dorsal and lateral to the turning
96   Exposure of growing neurons to thrombin or semaphorin 3A stimulates a receptor-mediated signaling c
97 copies of A22p, a peptide derived from human semaphorin-3a, that exhibits substantially improved edit
98 toration of nociception in both NGF- and NGF/semaphorin 3A-treated rats.
99                                      NGF and semaphorin 3A treatment also targeted the regeneration o
100  CNCs, cell processes failed to retract with semaphorin 3A treatment.
101 n superficial laminas, adenovirus expressing semaphorin 3A was injected into the ventral spinal cord
102 either of these receptors or the addition of semaphorin 3A (which blocks VEGF-165 binding to Nrp-1) p
103         Moreover, the repulsive guidance cue Semaphorin 3A, which signals through Rac1, destabilizes

 
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