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1 in the remodeling atrioventricular (AV) and semilunar valves.
2 f Tgf-beta1 in HDAC3-null outflow tracts and semilunar valves.
3 re either atretic or connected distal to the semilunar valves.
6 al crest can result in functionally abnormal semilunar valves and concomitant aortic arch artery abno
7 stal truncus that was patterned into the two semilunar valves and in the proximal conotruncus where m
8 ocol by reporting normative growth trends of semilunar valves and left ventricular dimensions in pigl
9 homeostasis in HDAC3-null outflow tracts and semilunar valves, and pharmacological inhibition of TGF-
10 e is active throughout the developing AV and semilunar valves at E16.5, with more localized expressio
11 icular septal defects and a low incidence of semilunar valve defects, atrioventricular valve defects
13 rta, double outlet right ventricle, aberrant semilunar valve development, bicuspid aortic valve, vent
15 ic Egfr allele waved-2 (Egfrwa2/wa2) exhibit semilunar valve enlargement resulting from over-abundant
19 ptal defects, double outlet right ventricle, semilunar valve hyperplasia and aortic arch artery patte
20 al numbers of labeled cells persisted in the semilunar valves in late fetal, neonatal, and adult hear
21 endocardial cushions (the precursors of the semilunar valves) is required for late gestation valvula
22 es and genes repressed by VEGF between human semilunar valve leaflets from first and second trimester
24 nstream deep cervical lymphatics had typical semilunar valves, long lymphangions and smooth muscle co
25 titative histological assessment of 91 human semilunar valves obtained from fetuses at 14 to 19 and 2
26 rdium of the developing atrioventricular and semilunar valves, the muscular interventricular septum,